ライフサイエンスコーパス: pulmonary regurgitation

1 2.4 0.6 m/s, P>0.1; only 2 patients had mild pulmonary regurgitation).

2 ment, and all but 1 patient had mild or less pulmonary regurgitation.

3 ventricular to pulmonary artery stenosis or pulmonary regurgitation.

4 stent (BMS), although this treatment causes pulmonary regurgitation.

5 etralogy of Fallot) underwent PVR for severe pulmonary regurgitation.

6 er methods cannot reliably be used to assess pulmonary regurgitation.

7 n the management of conduit obstructions and pulmonary regurgitation.

8 te relief of the obstruction and significant pulmonary regurgitation.

9 he pulmonary valve in 6 young pigs to induce pulmonary regurgitation.

10 No patient had more than mild pulmonary regurgitation.

11 the late deleterious consequences of chronic pulmonary regurgitation.

12 However, BMS resulted in free pulmonary regurgitation (21.3+/-10.7% versus 41.4+/-7.5%

13 n repaired tetralogy of Fallot (TOF) reduces pulmonary regurgitation and decreases right ventricular

14 y valve placement is an emerging therapy for pulmonary regurgitation and right ventricular outflow tr

15 Pulmonary regurgitation and RV volumes were assessed by

16 cy and childhood leads to reintervention for pulmonary regurgitation and stenosis in later life.

17 ables (including tricuspid regurgitation and pulmonary regurgitation) and invasive central venous pre

18 childhood had more than or equal to moderate pulmonary regurgitation, and fulfilled defined criteria

19 nt in MRI-defined ventricular parameters and pulmonary regurgitation, and results in subjective and o

20 peration, reintervention, moderate or severe pulmonary regurgitation, and/or mean RVOT gradient >40 m

21 ventricular systolic function and/or severe pulmonary regurgitation are at increased risk for advers

22 Nonsurvivors were more likely to have pulmonary regurgitation at any gestational age (61% vers

23 t of sustained ventricular tachycardia, with pulmonary regurgitation being the predominant haemodynam

24 er repair because it is associated with less pulmonary regurgitation, better exercise tolerance, and

25 Recent evidence suggests that pulmonary regurgitation causes significant morbidity, pr

26 ng of the RV to PVR in patients with chronic pulmonary regurgitation did not result in a measurable e

27 1% with data); no patient had more than mild pulmonary regurgitation early after implantation or duri

28 icuspid regurgitation (TR) and end-diastolic pulmonary regurgitation (EDPR) gradients in outpatients

29 Both HRV and BRS decreased with pulmonary regurgitation, elevated right ventricular end

30 n the past, residual defects such as chronic pulmonary regurgitation following repair of tetralogy of

31 ity, left ventricular ejection fraction, and pulmonary regurgitation fraction; 0.81 [0.72-0.89]; P=0.

32 ent of the tricuspid regurgitation gradient, pulmonary regurgitation gradient, pulmonary artery strok

33 inal synchrony improved significantly in the pulmonary regurgitation group (maximum wall delay P=0.03

34 A monocusp valve can prevent pulmonary regurgitation; however, valve failure resultin

35 MRI demonstrates the progression of pulmonary regurgitation in growing swine.

36 Fetuses with pulmonary regurgitation, indicating circular shunt physi

37 mm Hg on echocardiography without important pulmonary regurgitation (less than mild or regurgitant f

38 tients (n=31) and subgroups with predominant pulmonary regurgitation (n=13) or stenosis (n=18).

39 er surgical RV revalvulation for significant pulmonary regurgitation (n=21).

40 95% confidence interval, 1.1-1.5; P<0.001), pulmonary regurgitation (odds ratio, 2.9; 95% confidence

41 g factors in the assessment of the impact of pulmonary regurgitation on RV size and function.

42 can treat this condition without consequent pulmonary regurgitation or cardiopulmonary bypass.

43 ients, but 58 (8.5%) had moderate or greater pulmonary regurgitation or maximum Doppler gradients >40

44 ventricular systolic function and/or severe pulmonary regurgitation (OR, 10.3) in a previously propo

45 ventricular ejection fraction and/or severe pulmonary regurgitation (OR, 9.0) and smoking history (O

46 ndocarditis (n=3, 2 with stenosis and 1 with pulmonary regurgitation), or right ventricular dysfuncti

47 he determinants of improvement after PVR for pulmonary regurgitation over a wide range of patient age

48 e whole cohort (P<0.001) and both subgroups (pulmonary regurgitation P=0.01; pulmonary stenosis P=0.0

49 RV) volume overload in patients with chronic pulmonary regurgitation, persistent RV dysfunction and s

50 ent (33+/-24.6 to 19.5+/-15.3, P<0.001), and pulmonary regurgitation (PR) (grade 2 of greater before,

51 t ventricular (RV) function in patients with pulmonary regurgitation (PR) after tetralogy repair rema

52 paired tetralogy of Fallot are monitored for pulmonary regurgitation (PR) and right ventricular (RV)

53 Repair of TOF often results in chronic pulmonary regurgitation (PR) and right ventricular (RV)

54 to examine the prevalence and predictors of pulmonary regurgitation (PR) following balloon dilation

55 diac flow measurement method for quantifying pulmonary regurgitation (PR) in an in vitro and a chroni

56 Pulmonary regurgitation (PR) is an important determinant

57 145 with mitral regurgitation (MR), 101 with pulmonary regurgitation (PR), 71 with multivalve disease

58 to quantify biventricular size and function, pulmonary regurgitation (PR), and myocardial viability.

59 ventricular (RV) outflow tract stenosis and pulmonary regurgitation (PR), percutaneous pulmonary val

60 digital color Doppler method for quantifying pulmonary regurgitation (PR), using an animal model of c

61 ith repaired tetralogy of Fallot and chronic pulmonary regurgitation (PR).

62 , and 94% of patients had moderate or severe pulmonary regurgitation (PR).

63 tricular outflow tract (RVOT) obstruction or pulmonary regurgitation (PR).

64 aired right ventricular outflow tract (RVOT) pulmonary regurgitation (PR).

65 mic right ventricle, pulmonary hypertension, pulmonary regurgitation, pulmonary atrioventricular valv

66 These abnormalities are associated with pulmonary regurgitation, right ventricular enlargement,

67 similar TOF group, late PVR for symptomatic pulmonary regurgitation/RV dilation did not reduce the i

68 increased for the whole cohort (P=0.02) and pulmonary regurgitation subgroup (P=0.05); circumferenti

69 No correlation was found between pulmonary regurgitation velocities and either mean pulmo

70 Pulmonary regurgitation was </=2+ in 97% of patients.

71 Severe pulmonary regurgitation was found in 64.5% of ES3 and 10

72 Pulmonary regurgitation was graded as mild (RF<20%), mod

73 Before implantation, pulmonary regurgitation was moderate or severe in 92 pat

74 Pulmonary regurgitation was recorded in 10 patients (25%

75 Pulmonary regurgitation was the main underlying haemodyn

76 At least moderate neoaortic and pulmonary regurgitation were present in 3.4% and 6.6%, r

77 aying surgical replacement, but it creates a pulmonary regurgitation when crossing the valve.

78 with repaired tetralogy of Fallot often have pulmonary regurgitation, which is frequently overlooked

79 ular patch (TAP) exposes patients to chronic pulmonary regurgitation, while valve-sparing (VS) proced

80 Patients with pulmonary regurgitation with or without stenosis after r

81 position in patients with moderate to severe pulmonary regurgitation with or without stenosis.