ライフサイエンスコーパス: pulmonary thromboembolism

1 for the diagnosis of experimentally induced pulmonary thromboembolism.

2 ting to the hemodynamic derangement of acute pulmonary thromboembolism.

3 ected from collagen plus epinephrine-induced pulmonary thromboembolism.

4 f the pulmonary arteries, positive for acute pulmonary thromboembolism.

5 h hyperdense middle cerebral artery sign and pulmonary thromboembolism.

6 s were reported: 2 arterial hyperthension, 1 pulmonary thromboembolism, 1 bleeding, and 2 fatigue.

7 f these 117 resulted from sepsis and 31 from pulmonary thromboembolism, accounted for 51.6%.

8 of deep venous thrombosis of calf veins with pulmonary thromboembolism after tumescent liposuction of

9 Significant rates of pulmonary thromboembolism and of catheter-associated dee

10 The former, typified by chronic pulmonary thromboembolism and primary pulmonary hyperten

11 Five patients (16%) suffered an acute pulmonary thromboembolism and three (9.7%) had a pneumot

12 beta(3)(L746A) mice were resistant to both pulmonary thromboembolism and to ferric chloride-induced

13 Heart failure, systemic and pulmonary thromboembolism, arrhythmia, and sudden cardia

14 rs were also measured in patients with acute pulmonary thromboembolism at baseline conditions and aft

15 samples were drawn from patients with acute pulmonary thromboembolism at baseline conditions and aft

16 However, in contact activation-induced pulmonary thromboembolism by collagen/epinephrine or lon

17 arterial injury and increases mortality from pulmonary thromboembolism following collagen/epinephrine

18 Patients with acute pulmonary thromboembolism had higher plasma hemoglobin c

19 Intracardiac thrombosis and/or pulmonary thromboembolism (ICT/PE) is a rare but devasta

20 rombosis following carotid artery injury and pulmonary thromboembolism in mice without significantly

21 (-/-), but not Fut7(-/-), mice had increased pulmonary thromboembolism-induced mortality and decrease

22 ection of deep venous thrombi and subsequent pulmonary thromboembolism is a serious medical challenge

23 dge, togetherness of hyperdense MCA sign and pulmonary thromboembolism is extremely rare in the liter

24 Pulmonary thromboembolism is the main cause of maternal

25 ry hypertension (CTEPH) develops after acute pulmonary thromboembolism is unknown.

26 f hyperdense middle cerebral artery sign and pulmonary thromboembolism is very rare in the literature

27 Consistent with this, in a pulmonary thromboembolism model, larger vessels were occ

28 ster in PTPN7-null mice than in WT mice in a pulmonary thromboembolism model.

29 iated with a pro-thrombotic state (four with pulmonary thromboembolism), one of whom died; (iv) perip

30 uent to intravascular hemolysis, (2) chronic pulmonary thromboembolism, or (3) upregulated hypoxic re

31 ), whereas autopsy was better at identifying pulmonary thromboembolism (p=0.004).

32 Pulmonary thromboembolism (PEm) is a serious and life th

33 Pulmonary thromboembolism (PTE) is a common disease with

34 t bronchial asthma (BA) enhances the risk of pulmonary thromboembolism (PTE).

35 and were significantly higher in those with pulmonary thromboembolism than those without in coronavi

36 ausing primary HLH regulate pathways linking pulmonary thromboembolism to the presence of SARS-CoV-2

37 ADP-induced platelet pulmonary thromboembolism was decreased in mice previous

38 Pulmonary thromboembolism was detected in one case.