ライフサイエンスコーパス: pulse pressure

1 r results were obtained for diastolic BP and pulse pressure.

2 th factor-binding protein 3) were related to pulse pressure.

3 left ventricular dysfunction, and increased pulse pressure.

4 and intraventricular hemorrhage volume, and pulse pressure.

5 cturnal drop in blood pressure (dipping) and pulse pressure.

6 se, body mass index, waist circumference and pulse pressure.

7 fening was assessed from the brachial artery pulse pressure.

8 lus high-density lipoprotein cholesterol and pulse pressure.

9 with forward and reflected wave and carotid pulse pressure.

10 onger predictor of ESRD than diastolic BP or pulse pressure.

11 en in the absence of increases in arteriolar pulse pressure.

12 the relation was strongest for systolic and pulse pressure.

13 ongly related to cardiovascular disease than pulse pressure.

14 onsistent for systolic BP, diastolic BP, and pulse pressure.

15 n long-term average systolic, diastolic, and pulse pressure.

16 analysis with systolic BP or diastolic BP or pulse pressure.

17 P, diastolic BP, mean arterial pressure, and pulse pressure.

18 ng, (5) diet quality, (6) education, and (7) pulse pressure.

19 thesis that OPG is associated with increased pulse pressure.

20 .4-mmHg (95% CI: 0.2, 0.6; p < 0.001) higher pulse pressure.

21 % of the variability in central systolic and pulse pressures.

22 5% confidence interval (CI): 0.25, 1.62] and pulse pressure (0.76 mmHg; 95% CI: 0.22, 1.30).

23 .095 +/- 0.043 SD/SD, P = 0.028) and carotid pulse pressure (-0.114 +/- 0.045 SD/SD, P = 0.013) were

24 CI, -1.8 to 1.6], P=0.9; change in brachial pulse pressure, -0.02 mm Hg [95% CI, -1.6 to 1.6], P=0.9

25 5% CI, -2.3 to 1.2], P=0.5; change in aortic pulse pressure, -0.4 mm Hg [95% CI, -1.9 to 1.0], P=0.6)

26 cPP increased more than peripheral pulse pressure (10.3 and 9.2 mm Hg, respectively; p < 0.

27 14 mm, p = 0.003) dimensions, and decreased pulse pressure (15 +/- 13 vs 29 +/- 22 mm Hg, p = 0.02).

28 re (55 versus 42 mm Hg), a narrowed systemic pulse pressure (43 versus 64 mm Hg), a lower Qp:Qs (0.92

29 /-12.7 vs. 98.2+/-13.0 mmHg, P=0.002), lower pulse pressure (51.6+/-15.1 vs. 61.4+/-15.6 mmHg, P=0.00

30 pressure (135 +/- 1 to 127 +/- 1 mm Hg) and pulse pressure (54 +/- 1 to 48 +/- 1 mm Hg) (both P < 0.

31 5% CI: 0.6, 2.3; p < 0.001), 1.0-mmHg higher pulse pressure (95% CI: 0.4, 1.7; p = 0.001), 0.8-mmHg h

32 otensive participants, elevation of arterial pulse pressure (a surrogate of arterial stiffness) was l

33 Increased pulse pressure, a reflection of aortic stiffness, increa

34 sent recent data unveiling the importance of pulse pressure above that of systolic and diastolic pres

35 sympathetic nerve activity and directly with pulse pressure (all amalgamated R2 > .88, all p < or = .

36 tion of blood pressure (systolic, diastolic, pulse pressure) among UK Biobank participants of Europea

37 dex ([Second/First systolic peak] x 100) and pulse pressure amplification ([Radial/aortic pulse press

38 Central (carotid) to peripheral (brachial) pulse pressure amplification (PPA) was calculated with t

39 mentation index, central pulse pressure, and pulse pressure amplification were not related to cardiov

40 pulse wave velocity, augmentation index, and pulse pressure amplification) are intrinsically limited

41 predictive value of blood pressure (BP) and pulse pressure amplification, a marker of arterial funct

42 Pulse pressure, an index of vascular aging, was associat

43 Increased pulse pressure, an indicator of conduit vessel stiffness

44 n (hazard ratio, 1.41 [CI, 1.18 to 1.69] for pulse pressure and 1.42 [CI, 1.14 to 1.76] for systolic

45 ardiographic images, the association between pulse pressure and AF persisted in models that adjusted

46 X) for CbTX greatly diminished inhibition of pulse pressure and agonist flow responses (with or witho

47 e levels were higher in patients with narrow pulse pressure and clinical warning signs such as severe

48 We assessed changes in pulse pressure and conduit vessel stiffness in a 12-week

49 analyses investigated relationships between pulse pressure and distinct cerebral spinal fluid biomar

50 e compared the accuracy of measured arterial pulse pressure and estimated left ventricular stroke vol

51 o perfuse arterial segments at physiological pulse pressure and flow.

52 he NAV1 lead variant colocalized with higher pulse pressure and higher prevalence of carotid artery s

53 To examine relations between pulse pressure and incident AF.

54 d progressive reductions of R-R interval and pulse pressure and progressive increases of muscle sympa

55 ation because larger (max) and smaller (min) pulse pressure and stroke volume may occur.

56 oad independence and was strongly related to pulse pressure and total arterial stiffness regardless o

57 Greater reductions in pulse pressure and Z(c) in hypertensive subjects treated

58 0.2+/-16.2 versus -3.2+/-16.9 mm Hg, P<0.01) pulse pressures and Z(c) (237+/-83 to 208+/-70 versus 22

59 ender, prior coronary artery disease, higher pulse pressure, and diabetes were significant cardiovasc

60 and 4 BP traits: systolic BP, diastolic BP, pulse pressure, and mean arterial pressure.

61 Independent predictors of PASP were age, pulse pressure, and mitral E/e' (all P< or =0.003).

62 In contrast, augmentation index, central pulse pressure, and pulse pressure amplification were no

63 ht atrial pressure, reduced pulmonary artery pulse pressure, and reduced stroke volume.

64 diastolic BP (DBP), mean arterial BP (MAP), pulse pressure, and renal function during the first year

65 Dynamic changes in systolic pressure, pulse pressure, and stroke volume in patients undergoing

66 Aortic pulse pressure (aoPP) and its components (incident press

67 Peripheral and central pulse pressure, AP, AIx, and aortic and brachial PWV all

68 Diastolic blood pressure and pulse pressure are causally associated with increased ri

69 Reduced arterial compliance and increased pulse pressure are common and major risk factors for car

70 Systolic blood pressure and pulse pressure are substantially higher in older adults.

71 predictive power of systolic, diastolic, and pulse pressure, are described.

72 ere performed, with systolic, diastolic, and pulse pressures as quantitative traits, adjusting for ag

73 Arterial pulse pressure, as well as, left ventricular stroke volu

74 ticipants (13.1%) a median of 12 years after pulse pressure assessment.

75 Increased pulse pressure associated with age-related arterial stif

76 Pulse pressure associations were inverse for abdominal a

77 Higher pulse pressure at any age and higher pulse pressure with

78 Pulse pressure at the time of index PCI is associated wi

79 est for low systolic, high diastolic (narrow pulse pressure) at 2.1% (p = 0.0002).

80 ty, forward pressure wave amplitude, central pulse pressure, augmentation pressure, augmentation inde

81 was to explore whether the brachial/carotid pulse pressure (B/C-PP) ratio selectively predicts the s

82 ith the normal subjects, after adjusting for pulse pressure, baPWV in the BRVO patients was significa

83 hod yielded central systolic, diastolic, and pulse pressure bias and precision errors of -0.6 to 2.6

84 regulatory variants in modifying the traits pulse pressure, blood protein levels, and monocyte count

85 tus, atrial fibrillation, blood pressure and pulse pressure, body mass index, cardiovascular disease,

86 Brachial pulse pressure (bPP), aoPP, and all measures of arterial

87 LBNP decreased pulse pressure, but did not change mean arterial pressur

88 g (95% confidence interval: -1.2, -0.2), and pulse pressure by 1.2 mmHg (95% confidence interval: -1.

89 fice diastolic BP by 8.9/9.5/11.7 mm Hg, and pulse pressure by 13.4/14.2/14.9 mm Hg (n=245/236/90; P

90 itamins and folate, the odds ratios for high pulse pressure by increasing TWA quintiles were 1.00 (re

91 ne) to 5 vascular function measures (central pulse pressure, carotid-femoral pulse-wave velocity, mea

92 ted 3 measures of aortic stiffness: brachial pulse pressure; carotid-femoral pulse wave velocity (CFP

93 ascular disease risk factors, local brachial pulse pressure, CFPWV, and Pf, considered separately, we

94 iables-age, gender, systolic blood pressure, pulse pressure, cholesterol, smoking, ejection fraction,

95 Dynamic changes in arterial pulse pressure closely tracked left ventricular stroke v

96 s (eg, 25% increase in dP/dt(max) and aortic pulse pressure) compared with atrial pacing-LBBB, and th

97 ); similarly, a 1-SD (16 mm Hg) increment in pulse pressure conferred a 55% increased risk for CHF (h

98 stolic blood pressure (SBP and DBP), central pulse pressure (cPP) and flow-mediated dilatation (FMD).

99 examine the progression of central arterial pulse pressure (cPP) in women and the degree to which th

100 gmentation pressure (DeltaP(aug)) to central pulse pressure (cPP), their relation to central arterial

101 Aortic pulse pressure decreased from 76.2 +/- 23.3 mm Hg to 61.

102 After ganglion blockade, systolic and pulse pressure decreased significantly by 13% and 26%, r

103 was associated with lower blood pressure and pulse pressure, decreased systemic vascular resistance,

104 95% CI, 3.3 to 5.4; P<0.0001; central aortic pulse pressure, Delta3.0 mm Hg; 95% CI, 2.1 to 3.9; P<0.

105 otid ligation normalized cerebral arteriolar pulse pressure did not prevent increases in CSA in homoz

106 ulse pressure were associated with childhood pulse pressure (difference per additional average risk a

107 se data reveal that assessment of peripheral pulse pressure does not always reliably predict changes

108 V), which quantifies the changes in arterial pulse pressure during mechanical ventilation, is one of

109 % CI, 0.78-0.96; P = .01), consistent with a pulse pressure effect.

110 Pulse pressure, especially in central arteries, is an in

111 Central pulse pressure, forward pressure wave, reflected pressur

112 blood pressure, mean arterial pressure, and pulse pressure from the International Consortium for Blo

113 m Hg or less (25th percentile) and 23.3% for pulse pressure greater than 61 mm Hg (75th percentile).

114 t for every additional 20-mm Hg increment in pulse pressure >40 mm Hg, there was an OR of 1.49 (CI, 1

115 Odds ratios for high pulse pressure (> or = 55 mmHg) by increasing TWA quinti

116 Pulse pressure had neither a linear nor nonlinear associ

117 n adolescents, cIMT was associated with SBP, pulse pressure, heart rate, BMI, and waist/hip ratio.

118 Thus, wide pulse pressure (high systolic, low diastolic [HSLD]) may

119 Reductions in pump speed led to increases in pulse pressure (high versus low speed: 17 +/- 7 versus 2

120 Pulse pressure, however, was lower in young women and hi

121 was no impact of HR on brachial systolic or pulse pressures; however, there was a highly significant

122 Patients with pulse pressure hypertension >80 mm Hg were 3 times more

123 mic view of the syndrome of systolic or wide pulse pressure hypertension and its hallmark abnormality

124 review the risk of systolic hypertension and pulse pressure hypertension independent of elevated dias

125 The evidence is compelling that wide pulse pressure hypertension is a strong and an independe

126 linkage analyses of log serum creatinine and pulse pressure (i.e., systolic-diastolic BP) provided "s

127 ) was related to systolic blood pressure and pulse pressure; IL (interleukin) 16 was related to diast

128 rachial pressure amplification), and central pulse pressure in 2232 participants (mean age, 63 years;

129 s based on SNPs for aortic root diameter and pulse pressure in adults are associated with the same ou

130 t serum OPG is independently associated with pulse pressure in kidney transplant recipients.

131 c arteries is the primary cause of increased pulse pressure in subjects with degeneration and hyperpl

132 lood pressure, diastolic blood pressure, and pulse pressure in the UK Biobank, we estimate that 9.3%,

133 sociation studies of systolic, diastolic and pulse pressure in up to 776,078 participants from the Mi

134 tral) and 66 (peripheral) of the variance in pulse pressure in younger participants (<50 years) and 9

135 (PAC, ratio of stroke volume over pulmonary pulse pressure), in relation to pulmonary vascular resis

136 ie, augmentation index = (augmented pressure/pulse pressure) increases].

137 rning Score (2.6 vs 3.3; p<0.001) and higher pulse pressure index (0.45 vs 0.41; p<0.001) and tempera

138 Arterial stiffening with increased pulse pressure is a leading risk factor for cardiovascul

139 Pulse pressure is an important risk factor for incident

140 Beside established risk factors, pulse pressure is independently and significantly associ

141 with an SBP >/=120 mm Hg, and thus elevated pulse pressure, low DBP was associated with subclinical

142 ghest quartile of IMT were older age, higher pulse pressure, lower levels of albumin, elevated C-reac

143 >/=90 mm Hg (HR, 1.8; 95% CI, 1.1-2.9), and pulse pressure <50 mm Hg (HR, 1.8; 95% CI, 1.1-2.9), wit

144 measured LVSV, ventriculovascular stiffness (pulse pressure/LVSV(index)), and aortic distensibility a

145 Pulse pressure may also be a valuable risk assessment to

146 Moreover, central aortic pulse pressure may be a determinant of clinical outcomes

147 Increased pulse pressure may help identify hypertensive patients a

148 A wide pulse pressure may serve as a surrogate marker for risk

149 k factors include prehypertension, increased pulse pressure, obstructive sleep apnea, high-level phys

150 in left ventricular stroke volume, and thus pulse pressure, occur in cases of biventricular preload

151 ive 20-year AF incidence rates were 5.6% for pulse pressure of 40 mm Hg or less (25th percentile) and

152 9 mm Hg, diastolic BP of 70 to 79 mm Hg, and pulse pressure of 60 to 69 mm Hg taken as reference.

153 The predictive value of a change in pulse pressure on passive leg raising is inferior to a p

154 The use of changes in pulse pressure on passive leg raising showed a lower dia

155 val analysis examined the effect of baseline pulse pressure on progression to dementia.

156 impact of achieved systolic, diastolic, and pulse pressures on CV outcomes in 1590 adults who had ov

157 t always reliably predict changes in central pulse pressure or arterial stiffness.

158 ard brachial artery blood pressure, brachial pulse pressure, or mean arterial pressure are inadequate

159 rease in systolic blood pressure (P = .005), pulse pressure (P = .02), and mean arterial pressure (P

160 .3 mmHg in CT and TT combined; P = 0.04) and pulse pressure (P = 0.04) at baseline; this association

161 emoral pulse wave velocity (P=0.02), central pulse pressure (P<0.0001), mean arterial pressure (P=0.0

162 lood pressure, diastolic blood pressure, and pulse pressure (p<or=0.01 for all).

163 th mean arterial pressure (P=0.003), central pulse pressure (P=0.001), and forward pressure wave (P=0

164 terial pressure and was inversely related to pulse pressure (P=0.037).

165 p between HR and central aortic systolic and pulse pressures (p < 0.001).

166 (95% CI: 0.02, 0.25; p = 0.021) increase in pulse pressure per month over the course of pregnancy.

167 BP) (p < 0.001) or each 15 mmHg increment in pulse pressure (PP) (p < 0.001).

168 and therefore may not adapt to variations in pulse pressure (PP) amplification (ratio of radial to ce

169 tolic BP (SBP) and diastolic BP (DBP) versus pulse pressure (PP) and mean arterial pressure (MAP) com

170 ow report genome-wide association studies of pulse pressure (PP) and mean arterial pressure (MAP).

171 Ao-PWV, poorer diabetic control, and higher pulse pressure (PP) and systolic BP (SBP) (all P < 0.05)

172 tervals (RR), systolic blood pressure (SBP), pulse pressure (PP) and their coefficients of variation

173 BP (DBP), mean arterial pressure (MAP), and pulse pressure (PP) averaged over multiple years in 46,6

174 on study of mean arterial pressure (MAP) and pulse pressure (PP) in 129 913 individuals in stage 1 an

175 Elevated pulse pressure (PP) is associated with increased cardiov

176 Pulse pressure (PP) provides valuable prognostic informa

177 Pulse pressure (PP) reflects increased large artery stif

178 05, 2010 and 2014; each 1-mm Hg increment in pulse pressure (PP) was associated with 1.6% (95% CI: 0.

179 ssure (SBP), diastolic blood pressure (DBP), pulse pressure (PP), and traditional cardiac risk factor

180 uate the effects of MetS on brachial central pulse pressure (PP), PP amplification, aortic stiffness,

181 Pulse pressure (PP), the difference between systolic and

182 c BP (DBP), mean arterial pressure (MAP) and pulse pressure (PP), we genotyped approximately 50 000 s

183 BP (DBP), mean arterial pressure (MAP), and pulse pressure (PP), we genotyped ~50,000 SNPs in up to

184 signed to facilitate clinical use of central pulse pressure (PP).

185 lic BP (SBP, r(g) = 0.06, P = 0.01), but not pulse pressure (PP, r(g) = -0.01, P = 0.75).

186 er aortic pressure wave pulsatility (central pulse pressure [PP], reflected pressure wave, and reserv

187 Increased pulse pressure predicted increased all-cause mortality,

188 Those with higher baseline pulse pressure progressed to dementia more rapidly (95%

189 Carotid pulse pressure, pulsatility index and carotid-femoral pu

190 pressure (r = 0.079, P = 0.02), and central pulse pressure (r = 0.072, P = 0.035).

191 cluded systolic BP (r = 0.16; p = 0.006) and pulse pressure (r = 0.14; p = 0.02).

192 ) and to a lesser extent with changes in the pulse pressure (r = 0.18) and heart rate (r = 0.09).

193 provements in median stroke volume/pulmonary pulse pressure ratio (2.6 ml/mm Hg [IQR, 1.8-3.5] vs. 1.

194 rimary endpoints and stroke volume/pulmonary pulse pressure ratio, tricuspid annular plane systolic e

195 ssure measurements (systolic, diastolic, and pulse pressure) recorded during pregnancy.

196 und that an analytical method using arterial pulse pressure recording (pressure recording analytical

197 omarker profile exhibited mean (SD) elevated pulse pressure regardless of age (62.0 [15.6] mm Hg for

198 In a stepwise model that evaluated age-pulse pressure relations in the full sample, the late ac

199 er arrival of the reflected wave to elevated pulse pressure remain controversial.

200 .1% of variation in systolic, diastolic, and pulse pressure, respectively, in GERA non-Hispanic white

201 to loss of arterial compliance and increased pulse pressure seen with age, diabetes, and renal insuff

202 ring both SBP and DBP, not just SBP, DBP, or pulse pressure separately.

203 STdep was associated with older age, greater pulse pressure, serum fibrinogen levels and urinary albu

204 ension also were associated with higher mean pulse pressure/stroke volume index (1.24 and 1.15 versus

205 CI, 1.15 to 2.74; P=0.01), and preoperative pulse pressure such that for every additional 20-mm Hg i

206 ith age, yet epidemiological data concerning pulse pressure suggest that large artery stiffening pred

207 index of stroke volume divided by pulmonary pulse pressure (SV/PP) and prospectively gathered data o

208 L6 lead variant and higher eosinophil count, pulse pressure, systolic blood pressure, and carotid art

209 ALT-711 netted a greater decline in pulse pressures than placebo (-5.3 versus -0.6 mm Hg at

210 ized change in central arterial systolic and pulse pressure that is not detected by cuff pressure mea

211 riation in forward wave amplitude paralleled pulse pressure throughout adulthood.

212 Relationships of SBP, DBP, and pulse pressure to cardiovascular disease-related mortali

213 ly adjusted for cardiovascular risk factors (pulse pressure, total/high density lipoprotein cholester

214 ) values at or above the 75th percentile and pulse pressure values below the 75th percentile (P < 0.0

215 ange CCA-IMT, augmentation index, or BP, but pulse pressure variability improved (flavonoid: -0.11 +/

216 Preload responsiveness (pulse pressure variation >13%) was observed in 48% of do

217 luded stroke volume variation (nine trials), pulse pressure variation (one trial), and stroke volume

218 Pulse pressure variation (PPV), which quantifies the cha

219 vena cava diameter (r = 0.42; p < 0.01), and pulse pressure variation (r = 0.87; p < 0.0001) at basel

220 dds ratio were 0.89, 0.88, and 59.86 for the pulse pressure variation and 0.82, 0.86, and 27.34 for t

221 threshold values were 12.5 +/- 1.6% for the pulse pressure variation and 11.6 +/- 1.9% for the strok

222 Measures of arterial pulse pressure variation and left ventricular stroke vol

223 The absolute change in pulse pressure variation and stroke volume variation aft

224 best cutoff values of the absolute change in pulse pressure variation and stroke volume variation aft

225 Esmolol infusion decreased both pulse pressure variation and stroke volume variation as

226 r of breaths sampled may increase calculated pulse pressure variation and stroke volume variation bec

227 Both pulse pressure variation and stroke volume variation dur

228 ta-adrenergic blockade differentially alters pulse pressure variation and stroke volume variation dur

229 ume (tidal volume challenge) are superior to pulse pressure variation and stroke volume variation in

230 e validation is required to define threshold pulse pressure variation and stroke volume variation val

231 is, "tidal volume challenge," the changes in pulse pressure variation and stroke volume variation wil

232 and contractility would independently alter pulse pressure variation and stroke volume variation.

233 idal volume and contractility may also alter pulse pressure variation and stroke volume variation.

234 e variation, stroke volume variation, and/or pulse pressure variation and the change in stroke index/

235 Before fluid administration, we recorded pulse pressure variation and the changes in pulse contou

236 g fluid responsiveness was not different for pulse pressure variation and the passive leg-raising and

237 d-expiratory occlusion test were better than pulse pressure variation at predicting fluid responsiven

238 espiratory system was </= 30 mL/cm H2O, then pulse pressure variation became less accurate for predic

239 Stroke volume variation and pulse pressure variation do not reliably predict fluid r

240 Pulse pressure variation increased progressively with in

241 The changes in pulse pressure variation or stroke volume variation obta

242 However, dobutamine did not alter either pulse pressure variation or stroke volume variation.

243 cteristics curve was significantly lower for pulse pressure variation than for the passive leg-raisin

244 A 10% pulse pressure variation threshold discriminated between

245 riminating patients regarding the ability of pulse pressure variation to predict fluid responsiveness

246 We tested whether the poor ability of pulse pressure variation to predict fluid responsiveness

247 The ability of pulse pressure variation to predict fluid responsiveness

248 Pulse pressure variation was calculated as 100 x (PPmax

249 ex >/= 15% (44% +/- 39%) in 30 "responders." Pulse pressure variation was significantly correlated wi

250 We hypothesized that the magnitude of pulse pressure variation would increase with sampling du

251 ) was observed in 48% of donors (mean +/- sd pulse pressure variation, 19.2% +/- 4.8%).

252 association between stroke volume variation, pulse pressure variation, and/or stroke volume variation

253 The pulse pressure variation, stroke volume variation, and c

254 The pulse pressure variation, stroke volume variation, centr

255 orrelation coefficients between the baseline pulse pressure variation, stroke volume variation, systo

256 er the prediction of fluid responsiveness by pulse pressure variation.

257 riability of inferior vena cava diameter, or pulse pressure variation.

258 characteristic curves was different between pulse pressure variations (0.95; 95% confidence interval

259 (VmaxAo) measured using either approach, and pulse pressure variations (PP) were recorded with the pa

260 onders (10% [8-16] vs. 14% [12-16]), whereas pulse pressure variations were significantly higher in r

261 ial pressure, cardiac index, heart rate, and pulse pressure variations) was observed.

262 d with a pressure recording analytic method, pulse pressure variations, and cardiac output estimated

263 pericardial pressure, and pleural pressure; pulse pressure variations, systolic pressure variations,

264 asuring radial artery augmentation index and pulse pressure velocity.

265 Mean pulse pressure was 60 +/- 21 mm Hg.

266 Among those with more advanced age, higher pulse pressure was also associated with cerebral amyloid

267 Low pulse pressure was associated with increased mortality.

268 ent myocardial infarction or heart failure), pulse pressure was associated with increased risk for AF

269 Pulse pressure was calculated from the arterial pressure

270 1 hypertension, stage 2-3 hypertension), and pulse pressure was calculated.

271 dvancing age, whereas in older participants, pulse pressure was higher and wave reflection was lower

272 Pulse pressure was increased after the removal of all da

273 systole, nondipping was more prevalent, and pulse pressure was increased compared with -CAN.

274 t across adulthood: In younger participants, pulse pressure was lower and wave reflection was higher

275 ave reflection on age-related differences in pulse pressure was minor.

276 and increased systolic BP, diastolic BP, and pulse pressure was observed (eg, adjusted systolic BP me

277 very old participants, a further increase in pulse pressure was observed in those exhibiting both P-t

278 Each 10-mm Hg increase in pulse pressure was related to a smaller aortic root (by

279 rtional-hazards modeling showed that central pulse pressure was significantly associated with a post

280 Furthermore, increased pulse pressure was strongly associated with increased me

281 Arterial pulse pressure was unaffected in both groups.

282 aortic augmentation index from radial artery pulse pressure waveforms.

283 Brachial systolic and pulse pressure were also independently associated with i

284 Weighted and unweighted risk scores for pulse pressure were associated with childhood pulse pres

285 In contrast, wave reflection and pulse pressure were divergent across adulthood: In young

286 Systolic BP, diastolic BP, or pulse pressure were either not associated or inversely c

287 elerated increases in central and peripheral pulse pressure were markedly attenuated when variation i

288 Systolic blood pressure and pulse pressure were significantly reduced by 6 and 3 mm

289 blood pressure, mean arterial pressure, and pulse pressure were weaker predictors of CVD risk in thi

290 mentation index, mean arterial pressure, and pulse pressure) were determined at baseline.

291 index, waist circumference, systolic BP, and pulse pressure, were identified, suggesting that the gen

292 oking, high density lipoprotein cholesterol, pulse pressure, white blood cell count, and fibrinogen.

293 determine whether interventions that reduce pulse pressure will limit the growing incidence of AF.

294 Higher pulse pressure at any age and higher pulse pressure with advancing age is associated predomin

295 the empirically observed chronic changes in pulse pressure with age and the impaired capacity of hyp

296 parate relations of systolic, diastolic, and pulse pressure with risk for heart failure have not been

297 ciation of baseline systolic, diastolic, and pulse pressure with risk for incident CHF was examined i

298 for mean arterial pressure, and 10 SNPs for pulse pressure) with the same outcomes in children (medi

299 55-79 years of age) on clinical measures and pulse pressure x age group interactions were investigate

300 area at diastole)/(lumen area at diastole x pulse pressure)] x 1000, was compared between patients w

301 pulse pressure amplification ([Radial/aortic pulse pressure] x 100) were assessed as predictors of CV