ライフサイエンスコーパス: pulse wave

1 gestures, acoustic vibrations, and real-time pulse wave.

2 the push and pull regions associated with a pulse wave.

3 Stress/rest PAT ratio (sPAT) of pulse wave amplitude during mental stress/baseline was c

4 Digital pulse wave amplitude was continuously measured using per

5 Hence, we compared finger pulse-wave amplitude (PWA) responses to exercise among 5

6 ection of the heart sounds together with the pulse wave, an attribute not possible with existing phot

7 Comparison of CBP against a pulse wave analysis (PWA) device (Atcor Medical Sphygmoc

8 vascular responses to inhaled salbutamol by pulse wave analysis (PWA) or pulse contour analysis (PCA

9 s), arterial stiffness [pulse wave velocity, pulse wave analysis (PWA)], 24-h ambulatory blood pressu

10 sing radial artery applanation tonometry for pulse wave analysis and modeled in a mixed effects regre

11 automated oscillometric sphygmomanometer and pulse wave analysis every 2 weeks on up to five occasion

12 re derived from brachial pressure and radial pulse wave analysis in 2,073 patients, and 7,146 measure

13 Pulse wave analysis was used to determine augmentation i

14 Pulse wave analysis was used to determine central arteri

15 Radial artery applanation tonometry and pulse wave analysis were used to derive central aortic p

16 Radial artery applanation tonometry and pulse wave analysis were used to derive central aortic p

17 lacement (PAVR) and developments in coronary pulse wave analysis, it is now possible to instantaneous

18 Central hemodynamics was measured by Pulse Wave Analysis, left ventricular mass was assessed

19 sive individuals (age 21-78 yr; 43 male) and pulse wave analysis, wave intensity analysis and wave se

20 Using coronary pulse wave analysis, we calculated the intracoronary dia

21 plethysmography, flow-mediated dilation, and pulse wave analysis.

22 iontophoresis, and arterial stiffness, using pulse wave analysis.

23 Radial artery applanation tonometry and pulse-wave analysis were used to derive central aortic p

24 latory blood pressure monitoring, peripheral pulse-wave analysis, and carotid intima-media thickness.

25 and evaluated central arterial stiffness by pulse-wave analysis.

26 tive self-oscillating gel, in which chemical pulse waves and a stimulus-responsive medium play roles

27 ing) with Doppler assessment (continuous and pulse wave as well as color-flow mapping).

28 Pulsed-wave assessment of PVD flow included S-, D-, and

29 a novel form of mechanical stimulation, or a pulsed wave at the frequency of 1.5 MHz and the duty cyc

30 We studied how the pulsed wave can further increase algal lipid production

31 lastic blood vessels provide capacitance and pulse-wave dampening, which are critically important in

32 Thirteen pulse wave Doppler and 14 tissue Doppler imaging measure

33 We present Z scores for normalized pulse wave Doppler and tissue Doppler imaging in pediatr

34 Z score equations for most left ventricular pulse wave Doppler and tissue Doppler imaging measuremen

35 ETHODS: Ultrasound B-mode, color Doppler and pulse wave Doppler imaging of foot arteries was conducte

36 itant jet-derived pulmonary artery pressure, pulse wave Doppler pulmonary venous flow pattern and two

37 In pediatric echocardiography, pulse wave Doppler, and tissue Doppler imaging velocitie

38 using native tissue harmonics or transmitral pulsed wave Doppler have quantitated PFO functional size

39 The pulsed wave Doppler ratio of peak early transmitral flow

40 luded LV volumes and ejection fraction (EF), pulsed-wave Doppler (PWD)-derived transmitral filling in

41 Pulsed-wave Doppler determination of the pulmonary arter

42 Pulsed-wave Doppler transesophageal echocardiography (TE

43 Standard pulsed-wave Doppler transmitral and pulmonary vein flow

44 ects on diastolic function by load-dependent pulsed-wave Doppler transmitral indices has been variabl

45 myocardial performance indexes quantified by pulsed-wave Doppler ultrasound at day 30, followed by no

46 Pulse wave imaging (PWI) was performed just before infus

47 of spectral analysis of intraocular pressure pulse wave in healthy eyes of a control group (CG), pati

48 h resin casting at electronic microscopy and pulse-wave measurements, respectively.

49 Resin casting and pulse-wave measurements, showed that hrECMs preserves th

50 en to output either continuous wave (CW), or pulsed wave modes (PW).

51 ive, high fidelity, continuous radial artery pulse wave monitoring, which may lead to the use of flex

52 The pulsed waves of ultrasound, generated by a carbon black/

53 Results In normal aortas, the pulse waves propagated at relatively constant velocities

54 was used to measure the local homogeneity of pulse wave propagation within the saccular wall, which i

55 urysms is associated with the homogeneity of pulse wave propagation within the saccular wall.

56 ogram tracings with continuous wave (CW) and pulsed wave (PW) Doppler tracings recorded on the same s

57 ls were degraded by continuous wave (CW) and pulsed wave (PW) ultrasound at 205 kHz using deionized w

58 Here we report the utility of pulsed-wave (PW) Doppler-measured instantaneous flow and

59 lectron microscope results also suggest that pulsed wave stimulation induces shear stress and thus in

60 tty acid composition remains unchanged after pulsed-wave stimulation.

61 ding parameters not included in mTFC such as pulsed-wave tissue Doppler and RV 2-dimensional speckle

62 Long-axis M-mode, pulsed-wave tissue Doppler echograms (lateral, septal, a

63 ipients have biventricular dysfunction using pulsed-wave tissue Doppler imaging early after HT with m

64 ate-diastolic (A') velocities obtained using pulsed-wave tissue Doppler imaging in 380 healthy childr

65 stolic excursion, and systolic and diastolic pulsed-wave tissue Doppler imaging indices were similar

66 Pulsed-wave tissue Doppler imaging studies </=10 days po

67 after HT in children and young adults using pulsed-wave tissue Doppler imaging.

68 Aortic pulse wave transit time from the root of the subclavian

69 Arterial elastic modulus and pulse wave transit time were assessed using ultrahigh fr

70 se system to either active ultrasound (2 MHz pulsed-wave ultrasound for 120 min [sonothrombolysis]; i

71 Pulsed-wave ultrasound increases the exposure of an intr

72 ative blood flow (QBF) measurements that use pulsed-wave US rely on difficult-to-meet conditions.

73 crease in Ep (+155 +/- 193% vs. -5 +/- 28%), pulse wave velocity (+20 +/- 30% vs. -7 +/- 24%), and Ea

74 Carotid-femoral pulse wave velocity (-0.095 +/- 0.043 SD/SD, P = 0.028)

75 ss (-14 +/- 13 g vs. +3 +/- 11 g, p < 0.01), pulse wave velocity (-0.8 +/- 1.0 m/s vs. -0.1 +/- 0.9 m

76 Measurements of aortic-femoral pulse wave velocity (afPWV; n = 446) and large- and smal

77 Aortic pulse wave velocity (Ao-PWV) and albumin creatinine rati

78 Increased aortic pulse wave velocity (aPWV) has been associated with mort

79 f this study was to determine whether aortic pulse wave velocity (aPWV) improves prediction of cardio

80 he basis of having either low or high aortic pulse wave velocity (aPWV), a robust measure of aortic s

81 ns, central augmentation index (AIx), aortic pulse wave velocity (aPWV), blood pressure and heart rat

82 Brachial-ankle pulse wave velocity (baPWV) was measured to determine ar

83 ve hyperemia index (beta = 0.23, p < 0.001), pulse wave velocity (beta = -0.09, p = 0.04), augmentati

84 g flow-mediated vasodilation (FMD), brachial pulse wave velocity (bPWV), circulating angiogenic cells

85 io measure, and a measure of carotid-femoral pulse wave velocity (cf-PWV) and augmentation index (AI)

86 2012-2013) by measuring the carotid-femoral pulse wave velocity (cf-PWV).

87 Carotid-femoral pulse wave velocity (CF-PWV; the gold standard index of

88 ted the relationship between carotid femoral pulse wave velocity (cfPWV) and T-cell activation (defin

89 mean arterial pressure, and carotid-femoral pulse wave velocity (CFPWV) in 1480 participants represe

90 Carotid-femoral pulse wave velocity (CFPWV) is a heritable measure of ao

91 teries (by ultrasonography), carotid-femoral pulse wave velocity (cfPWV), aortic augmentation index,

92 ss: brachial pulse pressure; carotid-femoral pulse wave velocity (CFPWV), which is related directly t

93 ic stiffness as estimated by carotid-femoral pulse wave velocity (cfPWV).

94 ently measured compared with carotid-femoral pulse wave velocity (cfPWV).

95 ar risk factors, both higher carotid-femoral pulse wave velocity (hazard ratio [HR], 1.32; 95% confid

96 A small improvement in the aortic pulse wave velocity (i.e., a decrease of 0.22 m/s; 95% C

97 Pulse wave velocity (index of arterial stiffness) was al

98 peptide were associated with carotid-femoral pulse wave velocity (men: partial correlation, 0.069, P

99 d r = -0.062, P = 0.040), and carotid-radial pulse wave velocity (men: r = -0.090, P = 0.009 and r =

100 ng aortic distensibility and positively with pulse wave velocity (P<0.05).

101 ratio of MPA to aortic size correlated with pulse wave velocity (P=0.0098), strain (P=0.0099), and d

102 Carotid-femoral pulse wave velocity (P=0.02), central pulse pressure (P<

103 Indexed MPA diameter correlated with pulse wave velocity (P=0.04) and with aortic strain (P=0

104 ior diameter (increase of 54.9% +/- 2.5) and pulse wave velocity (PWV) (decrease of 1.3 m/sec +/- 0.8

105 ardiovascular magnetic resonance measures of pulse wave velocity (PWV) and aortic distensibility (AoD

106 of arterial stiffness indices [i.e., aortic pulse wave velocity (PWV) and augmentation (AGI) of caro

107 Aortic blood pressure (BP), pulse wave velocity (PWV) and augmentation index (AIx) w

108 rced vital capacity [FVC]) and a decrease in pulse wave velocity (PWV) and augmentation index up to 2

109 We measured aortic pulse wave velocity (PWV) and brachial PWV to evaluate t

110 Aortic pulse wave velocity (PWV) and carotid augmentation index

111 ein, and arterial stiffness [carotid-femoral pulse wave velocity (PWV) and carotid augmentation index

112 and arterial compliance as assessed by using pulse wave velocity (PWV) and central augmentation index

113 ional stiffness within the aortic arch using pulse wave velocity (PWV) and have found a stronger asso

114 outcomes were changes in carotid to femoral pulse wave velocity (PWV) and plasma 8-isoprostane F2alp

115 Pulse wave velocity (PWV) and the augmentation index (AI

116 ar stiffness was measured by carotid-femoral pulse wave velocity (PWV) and total arterial compliance.

117 We tested this by examining pulse wave velocity (PWV) in brachial arteries of twin s

118 Previous studies have suggested that AIx and pulse wave velocity (PWV) increase linearly with age, ye

119 assessed by magnetic resonance imaging (MRI) pulse wave velocity (PWV) measurements.

120 Higher pulse wave velocity (PWV) reflects increased arterial st

121 Pulse wave velocity (PWV) was assessed three times by fi

122 Pulse wave velocity (PWV) was calculated by the foot-to-

123 Pulse wave velocity (PWV) was calculated using the foot-

124 Pulmonary pulse wave velocity (PWV) was determined by the interval

125 Aortic pulse wave velocity (PWV) was measured in 2007-2009 (Pha

126 Pulse wave velocity (PWV) was measured in the central (c

127 Pulse wave velocity (PWV) was measured invasively (aorti

128 otid artery intima-media thickness (IMT) and pulse wave velocity (PWV) were evaluated in 101 PHIV and

129 ain, incremental elastic modulus (Einc), and pulse wave velocity (PWV) were measured over a TP range

130 ensors can accurately detect and measure the pulse wave velocity (PWV) when skin mounted.

131 at there is a progressive increase in aortic pulse wave velocity (PWV) with age.

132 ular (carotid intima-media thickness (cIMT), pulse wave velocity (PWV)) and cardiac (left ventricular

133 ere 1) arterial stiffness measured by aortic pulse wave velocity (PWV), 2) oxidative stress assessed

134 This study sought to evaluate whether pulse wave velocity (PWV), a noninvasive index of arteri

135 mediated vasodilation (FMD), carotid-femoral pulse wave velocity (PWV), and aortic augmentation index

136 arterial pressure (MAP), augmentation index, pulse wave velocity (PWV), and intima-media thickness.

137 assess hepatic triglyceride content, aortic pulse wave velocity (PWV), and visceral fat.

138 ness of the common carotid artery (CCA-IMT), pulse wave velocity (PWV), augmentation index, blood pre

139 brachial artery blood pressure (BP), aortic pulse wave velocity (PWV), B-mode ultrasonography and wa

140 Disease activity, blood pressure, aortic pulse wave velocity (PWV), brachial artery flow-mediated

141 Progressive increase in pulse wave velocity (PWV), maximal intra-luminal diamete

142 ently, vascular stiffness was assessed using pulse wave velocity (PWV).

143 ng 2007 to 2012, we measured carotid-femoral pulse wave velocity (PWV; SphygmoCor apparatus) 8 weeks

144 Carotid-to-femoral pulse wave velocity (PWVc-f) was assessed at baseline, a

145 cysteine was associated with carotid-femoral pulse wave velocity (r = 0.072, P = 0.036), forward pres

146 ectively measured arterial stiffness (aortic pulse wave velocity [aPWV]) and cardiac biomarkers in 98

147 ved from arterial tonometry (carotid-femoral pulse wave velocity [CFPWV], forward wave amplitude [FWA

148 Measures of arterial stiffness (pulse wave velocity [PWV] and augmentation index correct

149 ple (n = 42), cPP, arterial stiffness (using pulse wave velocity [PWV]) and arterial diameters (using

150 heir relation to central arterial stiffness (pulse wave velocity [PWV]) and arterial diameters, and t

151 s and arterial stiffness (carotid to femoral pulse wave velocity [PWV]) measured at age 17 years.

152 Arterial stiffness (carotid to femoral pulse wave velocity [PWV]) was measured and peripheral b

153 nction (local aortic distensibility and arch pulse wave velocity [PWV]), and LV volumes and mass.

154 r stroke) in relation to arterial stiffness (pulse wave velocity [PWV]), wave reflection (augmentatio

155 ing with iontophoresis), arterial stiffness (pulse wave velocity and analysis), blood pressure, and p

156 male rats characterized for abdominal aortic pulse wave velocity and aortic strain by high-resolution

157 Pulse wave velocity and augmentation index were improved

158 Measures of arterial stiffness (pulse wave velocity and augmentation index) and blood pr

159 aldosterone levels, and arterial stiffness (pulse wave velocity and augmentation index) in 20 adult

160 cardiography, (2) coronary flow reserve, (3) pulse wave velocity and augmentation index, (4) circulat

161 dary outcomes included decreases in arterial pulse wave velocity and carotid artery echodensity and i

162 glyceride content was associated with aortic pulse wave velocity and carotid IMT.

163 condary outcome measures included changes in pulse wave velocity and circulating biomarkers.

164 also led to significant favorable changes in pulse wave velocity and circulating IL-6 levels.

165 In contrast to controls pulse wave velocity and distensibility correlated with a

166 orta and the left ventricle (eg, aortic arch pulse wave velocity and distensibility) as well as the v

167 thelial dysfunction as determined in vivo by pulse wave velocity and ex vivo by atomic force microsco

168 2 aortic stiffness measures, carotid-femoral pulse wave velocity and forward pressure wave amplitude,

169 In vivo MRI revealed that baseline pulse wave velocity and morphology were similar in 6-wee

170 Carotid-femoral pulse wave velocity and radial tonometry-derived central

171 sex-specific genetic determinants for aortic pulse wave velocity and suggest distinct polygenic susce

172 ential relationships observed between aortic pulse wave velocity and telomere length in younger and o

173 tly modifies the relationship between aortic pulse wave velocity and telomere length.

174 r elasticity locally, specifically the local pulse wave velocity and the arterial wall thickness.

175 Local pulse wave velocity and the mean arterial wall thickness

176 t) rats exhibited significantly lower aortic pulse wave velocity and vascular media thickness compare

177 h hypertension and is directly correlated to pulse wave velocity as a measure of vascular stiffness.

178 Carotid femoral pulse wave velocity associated with both urinary albumin

179 We found no differences in pulse wave velocity at 12 months, augmentation index at

180 between-group difference in carotid-femoral pulse wave velocity at 12 months.

181 ApoE(-/-) and WT mice showed that increased pulse wave velocity coincided with the fragmentation of

182 Pulse wave velocity declined 8% with ALT-711 (P<0.05 at

183 ediated dilatation (P < 0.001), while aortic pulse wave velocity decreased (P < 0.001) in all three g

184 artery wall echodensity and carotid-femoral pulse wave velocity demonstrated no significant changes.

185 onometry, blood pressure, and carotid-radial pulse wave velocity did not change.

186 At 96 weeks, pulse wave velocity did not differ significantly between

187 = 0.03), and reduced (i.e. improved) aortic pulse wave velocity from 7.1 +/- 0.3 to 6.1 +/- 0.3 m s(

188 ly decreased aortic root diameters and lower pulse wave velocity in doxycycline-treated Marfan mice s

189 hildren with PAH had significantly increased pulse wave velocity in the ascending aorta (3.4 versus 2

190 aseline independently associated with aortic pulse wave velocity in the complete cohort and progressi

191 OC] and arterial stiffening (aortic pulse wave velocity in young: 3.62 +/- 0.15 m( ) s(-1 )

192 Pulse wave velocity is an independent predictor of the l

193 of 19 %HbO(2) [52.6%]; P < .001); and aortic pulse wave velocity marginally increased (0.19 of 6.05 m

194 ders of magnitude higher), as illustrated by pulse wave velocity measurements, toward hypertension de

195 wave reflection, reflected wave timing, and pulse wave velocity noninvasively in 6417 (age range, 19

196 Mean pulse wave velocity remained stable with both everolimus

197 Carotid to femoral pulse wave velocity showed a significant reduction from

198 Treatment also reduced aortic pulse wave velocity significantly (from 9.09+/-1.77 to 8

199 he weight-loss group, but carotid-to-femoral pulse wave velocity tended to decrease by 0.5 m/s (P = 0

200 systolic blood pressure and carotid-femoral pulse wave velocity to the model, forward pressure wave

201 We newly report that the assessment of local pulse wave velocity via MRI provides early information a

202 was 0.13 (95% CI: 0, 0.26; P = 0.044) lower, pulse wave velocity was 0.29 m/s (95% CI: 0.07, 0.52 m/s

203 phosphate was 1.25 mmol/L (3.87 mg/dl), mean pulse wave velocity was 10.8 m/s, and 81.3% had abdomina

204 Carotid-femoral pulse wave velocity was associated with higher white mat

205 Aortic pulse wave velocity was high-normal (9.2 +/- 2.2 m/s), i

206 Pulse wave velocity was higher in adults after ASO (5.0+

207 Pulse wave velocity was measured at baseline in 449 norm

208 mice, whereas at the age of 18 weeks, local pulse wave velocity was significantly elevated in ApoE(-

209 oral pulse wave velocity, and carotid-radial pulse wave velocity were assessed by tonometry in 1962 p

210 arget-to-background ratios (TBRs) and aortic pulse wave velocity were assessed.

211 ssure, pulsatility index and carotid-femoral pulse wave velocity were each associated with increased

212 n the brachial artery, and carotid to radial pulse wave velocity were measured in all children.

213 ardiac and thoracic aorta calcium scores and pulse wave velocity were measured to evaluate VC progres

214 aorta, and cardiac valve calcium scores and pulse wave velocity were not significantly different amo

215 interval, 2.4-20.7), augmentation index, and pulse wave velocity without changing peripheral blood pr

216 active hyperemia index, aortic hemodynamics, pulse wave velocity) were not differentially altered by

217 measures (distensibility, aortic strain, and pulse wave velocity) were similar across all groups.

218 thickness, peripheral arterial disease, and pulse wave velocity).

219 Mean (+/-SD) carotid-femoral pulse wave velocity, a measure of central aortic stiffne

220 Aortic calcium was reduced by 31%, and pulse wave velocity, an index of stiffness, was decrease

221 elastance (Ea), arterial compliance, aortic pulse wave velocity, and carotid Peterson modulus (Ep).

222 ve, reflected pressure wave, carotid-femoral pulse wave velocity, and carotid-radial pulse wave veloc

223 and fractional shortening), carotid-femoral pulse wave velocity, and central retinal arteriolar and

224 carotid ultrasound (intima-media thickness), pulse wave velocity, and Doppler examination of kidney g

225 , total arterial compliance, carotid-femoral pulse wave velocity, and drug tolerability were assessed

226 mprove aortic wall elasticity as measured by pulse wave velocity, and improve the ultrastructure of e

227 -femoral pulse wave velocity, carotid-radial pulse wave velocity, and venous occlusion plethysmograph

228 Aortic dimensions, distensibility, pulse wave velocity, aortic arch angle, left ventricular

229 aortic stiffness was evaluated by measuring pulse wave velocity, aortic strain, and distensibility.

230 EDD) and aortic stiffening (increased aortic pulse wave velocity, aPWV).

231 e contour analysis, partial rebreathing, and pulse wave velocity, are far less in number and are prim

232 ce or stiffness, elastic modulus, impedance, pulse wave velocity, augmentation index, and pulse press

233 OH)D(3) was not associated with adult aortic pulse wave velocity, blood pressure, fasting glucose, HD

234 Insulin resistance, pulse wave velocity, blood pressure, NO, and overall pla

235 d carotid pressure and flow, carotid-femoral pulse wave velocity, brain magnetic resonance images and

236 ) present repeated measures of aorto-femoral pulse wave velocity, capacitive compliance (C1), and osc

237 elial cells associated with increased aortic pulse wave velocity, carotid intima-media thickness, and

238 rin-mediated dilation (NMD), carotid-femoral pulse wave velocity, carotid-radial pulse wave velocity,

239 RI with gadolinium injection, measurement of pulse wave velocity, extracellular water, 24-hour ambula

240 arterial stiffness were the carotid femoral pulse wave velocity, forward pressure wave amplitude, ce

241 iffness increased markedly with age, eg, for pulse wave velocity, from a few percent in both sexes ag

242 rial stiffness, measured via carotid-femoral pulse wave velocity, has a better predictive value than

243 thickness, echocardiography, measurement of pulse wave velocity, hepatic ultrasonography, retinal fu

244 nces between treatment in carotid-to-femoral pulse wave velocity, high-sensitivity C-reactive protein

245 rtic stiffening, assessed by carotid-femoral pulse wave velocity, is associated with CKD.

246 The values of age, serum phosphate, pulse wave velocity, left ventricular mass (LVM), and LV

247 Patients also underwent assessment of pulse wave velocity, measurement of circulating superoxi

248 ing with iontophoresis), arterial stiffness [pulse wave velocity, pulse wave analysis (PWA)], 24-h am

249 Pulse wave velocity, superoxide, and C-reactive protein

250 nalyzed the primary outcome, carotid-femoral pulse wave velocity, using a linear mixed effects model

251 Aortic stiffness, measured by pulse wave velocity, was approximately 35% greater in El

252 Pulse wave velocity, wave travel times, and lumped press

253 The mean +/- SD carotid-femoral pulse wave velocity, which reflects central aortic stiff

254 ry juice consumption reduced carotid femoral pulse wave velocity-a clinically relevant measure of art

255 nce, pulse contour, partial rebreathing, and pulse wave velocity-based devices have not been studied

256 e severity of arterial stiffness assessed by pulse wave velocity.

257 sessed arterial stiffness by carotid-femoral pulse wave velocity.

258 LV mass, systolic and diastolic function, or pulse wave velocity.

259 ter region of DDB2 gene with carotid-femoral pulse wave velocity.

260 e groups despite considerable differences in pulse wave velocity.

261 Vascular function was assessed by FMD and pulse wave velocity.

262 l arterial stiffness was measured via aortic pulse wave velocity.

263 patients, arterial stiffness as measured by pulse wave velocity.

264 chromosome11, LOD 8.9) in females affecting pulse wave velocity.

265 magnetic resonance) and arterial stiffness (pulse wave velocity/analysis, aortic distensibility) wer

266 8.1 +/- 3.3%), and lower arterial stiffness (pulse wave velocity: mean 6.99 +/- 1.0 m/s vs. 7.05 +/-

267 s, compliance, and distensibility; 2) aortic pulse wave velocity; 3) coronary calcification; and 4) b

268 erformance index (MPI) and aortic stiffness (pulse wave velocity; PWV) were evaluated before and afte

269 ry flow-mediated dilation (FMDBA) and aortic pulse-wave velocity (aPWV) after 4, 8, and 12 weeks.

270 Carotid femoral pulse-wave velocity (cfPWV) measured arterial stiffness

271 line vascular stiffness, indexed by arterial pulse-wave velocity (Doppler) and augmentation index (ca

272 Arterial stiffness was determined by pulse-wave velocity (PWV) of the brachioradial and femor

273 otid artery intima-media thickness (IMT) and pulse-wave velocity (PWV) were evaluated in 101 PHIV and

274 rial distensibility measures, generally from pulse-wave velocity (PWV), are widely used with little k

275 PWV(CR) ) arterial stiffness was measured by pulse-wave velocity (PWV), together with systolic (SBP)

276 ents (62%) were found to present supranormal pulse-wave velocity and 14 patients (38%) presented left

277 elasticity was evaluated by Doppler-derived pulse-wave velocity and left ventricular function by ech

278 surement of AS by applanation tonometry with pulse-wave velocity has been the gold-standard method an

279 terial distensibility, assessed by measuring pulse-wave velocity in vivo.

280 Aortic pulse-wave velocity measured vascular stiffness.

281 there were significant associations between pulse-wave velocity values and left ventricular ejection

282 IMT was 0.71 +/- 0.1 mm, and the mean +/- SD pulse-wave velocity was 5.96 +/- 1.6 meters/second.

283 Pulse-wave velocity was assessed from tonometry and body

284 Pulse-wave velocity was higher in hypertensives (P=0.001

285 Carotid-femoral pulse-wave velocity was significantly (P<0.001) faster a

286 Doppler probes were used to collect pulse-wave velocity waveforms from the right carotid and

287 Left ventricular ejection fraction and pulse-wave velocity were both associated with Hunt and H

288 Left ventricular ejection fraction and pulse-wave velocity were improved between acute aneurysm

289 urine ET-1/creatinine, whereas reduction in pulse-wave velocity, a measure of arterial stiffness, wa

290 ow-mediated dilation of the brachial artery, pulse-wave velocity, and carotid intima-media thickness)

291 ection fraction, B-type natriuretic peptide, pulse-wave velocity, and pulse-wave velocity/left ventri

292 ice a Western diet markedly increased aortic pulse-wave velocity, intima-media thickening, oxidized l

293 res (central pulse pressure, carotid-femoral pulse-wave velocity, mean arterial pressure, forward pre

294 increased carotid intima-media thickness and pulse-wave velocity.

295 Pulse-wave velocity/left ventricular ejection fraction r

296 atriuretic peptide, pulse-wave velocity, and pulse-wave velocity/left ventricular ejection fraction s

297 WCH, MH, sustained hypertension, and aortic pulsed wave velocity by magnetic resonance imaging; urin

298 dependently associated with increased aortic pulsed wave velocity, cystatin C, and urinary albumin-to

299 ntent up to the 6th harmonic of the pressure pulse wave was considered.

300 t of central aortic waveforms analyzed using pulse wave, wave separation, and arterial reservoir mode