ライフサイエンスコーパス: pulse wave velocity

1 thickness, peripheral arterial disease, and pulse wave velocity).

2 e severity of arterial stiffness assessed by pulse wave velocity.

3 sessed arterial stiffness by carotid-femoral pulse wave velocity.

4 LV mass, systolic and diastolic function, or pulse wave velocity.

5 ter region of DDB2 gene with carotid-femoral pulse wave velocity.

6 e groups despite considerable differences in pulse wave velocity.

7 arterial compliance, augmentation index, and pulse wave velocity.

8 hibit increased blood pressure and increased pulse wave velocity.

9 Vascular function was assessed by FMD and pulse wave velocity.

10 l arterial stiffness was measured via aortic pulse wave velocity.

11 kness, while this trend was not observed for pulse wave velocity.

12 -world scenarios for a longitudinal study of pulse wave velocity.

13 patients, arterial stiffness as measured by pulse wave velocity.

14 chromosome11, LOD 8.9) in females affecting pulse wave velocity.

15 increased carotid intima-media thickness and pulse-wave velocity.

16 Carotid-femoral pulse wave velocity (-0.095 +/- 0.043 SD/SD, P = 0.028)

17 ss (-14 +/- 13 g vs. +3 +/- 11 g, p < 0.01), pulse wave velocity (-0.8 +/- 1.0 m/s vs. -0.1 +/- 0.9 m

18 crease in Ep (+155 +/- 193% vs. -5 +/- 28%), pulse wave velocity (+20 +/- 30% vs. -7 +/- 24%), and Ea

19 s, compliance, and distensibility; 2) aortic pulse wave velocity; 3) coronary calcification; and 4) b

20 sional flow magnetic resonance imaging-based pulse wave velocity (4D flow PWV) estimation is a promis

21 Mean (+/-SD) carotid-femoral pulse wave velocity, a measure of central aortic stiffne

22 urine ET-1/creatinine, whereas reduction in pulse-wave velocity, a measure of arterial stiffness, wa

23 ry juice consumption reduced carotid femoral pulse wave velocity-a clinically relevant measure of art

24 assessed by central blood pressure (BP) and pulse wave velocity; adverse cardiac remodeling, capture

25 Measurements of aortic-femoral pulse wave velocity (afPWV; n = 446) and large- and smal

26 tiffness, including increased central BP and pulse wave velocity, along with adverse cardiac remodeli

27 Aortic calcium was reduced by 31%, and pulse wave velocity, an index of stiffness, was decrease

28 magnetic resonance) and arterial stiffness (pulse wave velocity/analysis, aortic distensibility) wer

29 diated dilation, distensibility coefficient, pulse wave velocity and a clustered CVD risk factor scor

30 ing with iontophoresis), arterial stiffness (pulse wave velocity and analysis), blood pressure, and p

31 Pulse wave velocity and aortic augmentation index were m

32 male rats characterized for abdominal aortic pulse wave velocity and aortic strain by high-resolution

33 Pulse wave velocity and augmentation index were improved

34 Measures of arterial stiffness (pulse wave velocity and augmentation index) and blood pr

35 aldosterone levels, and arterial stiffness (pulse wave velocity and augmentation index) in 20 adult

36 cardiography, (2) coronary flow reserve, (3) pulse wave velocity and augmentation index, (4) circulat

37 dary outcomes included decreases in arterial pulse wave velocity and carotid artery echodensity and i

38 glyceride content was associated with aortic pulse wave velocity and carotid IMT.

39 condary outcome measures included changes in pulse wave velocity and circulating biomarkers.

40 also led to significant favorable changes in pulse wave velocity and circulating IL-6 levels.

41 ening as shown by restoration from increased pulse wave velocity and decreased elastin breaks.

42 In contrast to controls pulse wave velocity and distensibility correlated with a

43 orta and the left ventricle (eg, aortic arch pulse wave velocity and distensibility) as well as the v

44 thelial dysfunction as determined in vivo by pulse wave velocity and ex vivo by atomic force microsco

45 2 aortic stiffness measures, carotid-femoral pulse wave velocity and forward pressure wave amplitude,

46 ion working together to significantly reduce pulse wave velocity and improve left ventricular diastol

47 significantly reduced augmentation index and pulse wave velocity and increased compliance immediately

48 controls, with an association between higher pulse wave velocity and more severe molecular and clinic

49 In vivo MRI revealed that baseline pulse wave velocity and morphology were similar in 6-wee

50 ry flow-mediated dilatation, carotid-femoral pulse wave velocity and post-ischaemic brachial artery f

51 Carotid-femoral pulse wave velocity and radial tonometry-derived central

52 sex-specific genetic determinants for aortic pulse wave velocity and suggest distinct polygenic susce

53 ential relationships observed between aortic pulse wave velocity and telomere length in younger and o

54 tly modifies the relationship between aortic pulse wave velocity and telomere length.

55 r elasticity locally, specifically the local pulse wave velocity and the arterial wall thickness.

56 Local pulse wave velocity and the mean arterial wall thickness

57 t) rats exhibited significantly lower aortic pulse wave velocity and vascular media thickness compare

58 ents (62%) were found to present supranormal pulse-wave velocity and 14 patients (38%) presented left

59 elasticity was evaluated by Doppler-derived pulse-wave velocity and left ventricular function by ech

60 Augmentation index, pulse wave velocity, and arterial compliance were identi

61 elastance (Ea), arterial compliance, aortic pulse wave velocity, and carotid Peterson modulus (Ep).

62 ve, reflected pressure wave, carotid-femoral pulse wave velocity, and carotid-radial pulse wave veloc

63 and fractional shortening), carotid-femoral pulse wave velocity, and central retinal arteriolar and

64 carotid ultrasound (intima-media thickness), pulse wave velocity, and Doppler examination of kidney g

65 , total arterial compliance, carotid-femoral pulse wave velocity, and drug tolerability were assessed

66 mprove aortic wall elasticity as measured by pulse wave velocity, and improve the ultrastructure of e

67 -femoral pulse wave velocity, carotid-radial pulse wave velocity, and venous occlusion plethysmograph

68 ow-mediated dilation of the brachial artery, pulse-wave velocity, and carotid intima-media thickness)

69 ection fraction, B-type natriuretic peptide, pulse-wave velocity, and pulse-wave velocity/left ventri

70 Aortic pulse wave velocity (Ao-PWV) and albumin creatinine rati

71 Aortic dimensions, distensibility, pulse wave velocity, aortic arch angle, left ventricular

72 omarkers and measures of arterial stiffness (pulse wave velocity, aortic augmentation index, and aort

73 Aortic pulse wave velocity, aortic distensibility, and other me

74 aortic stiffness was evaluated by measuring pulse wave velocity, aortic strain, and distensibility.

75 Increased aortic pulse wave velocity (aPWV) has been associated with mort

76 f this study was to determine whether aortic pulse wave velocity (aPWV) improves prediction of cardio

77 he basis of having either low or high aortic pulse wave velocity (aPWV), a robust measure of aortic s

78 ns, central augmentation index (AIx), aortic pulse wave velocity (aPWV), blood pressure and heart rat

79 The primary outcome was aortic pulse wave velocity (aPWV).

80 ry flow-mediated dilation (FMDBA) and aortic pulse-wave velocity (aPWV) after 4, 8, and 12 weeks.

81 ectively measured arterial stiffness (aortic pulse wave velocity [aPWV]) and cardiac biomarkers in 98

82 EDD) and aortic stiffening (increased aortic pulse wave velocity, aPWV).

83 e contour analysis, partial rebreathing, and pulse wave velocity, are far less in number and are prim

84 h hypertension and is directly correlated to pulse wave velocity as a measure of vascular stiffness.

85 Carotid femoral pulse wave velocity associated with both urinary albumin

86 We found no differences in pulse wave velocity at 12 months, augmentation index at

87 between-group difference in carotid-femoral pulse wave velocity at 12 months.

88 ce or stiffness, elastic modulus, impedance, pulse wave velocity, augmentation index, and pulse press

89 participants showed that BP, brachial-ankle pulse wave velocity (baPWV) and ankle brachial index (AB

90 Brachial-ankle pulse wave velocity (baPWV) was measured to determine ar

91 nce, pulse contour, partial rebreathing, and pulse wave velocity-based devices have not been studied

92 ve hyperemia index (beta = 0.23, p < 0.001), pulse wave velocity (beta = -0.09, p = 0.04), augmentati

93 uctions in both native T1 mapping and aortic pulse wave velocity between groups favoring the interven

94 y scan, such as tonometry of carotid femoral pulse wave velocity, bioelectrical impedance analysis, a

95 hial artery flow-mediated dilatation, aortic pulse wave velocity, blood pressure and circulating lipi

96 OH)D(3) was not associated with adult aortic pulse wave velocity, blood pressure, fasting glucose, HD

97 Insulin resistance, pulse wave velocity, blood pressure, NO, and overall pla

98 g flow-mediated vasodilation (FMD), brachial pulse wave velocity (bPWV), circulating angiogenic cells

99 d carotid pressure and flow, carotid-femoral pulse wave velocity, brain magnetic resonance images and

100 WCH, MH, sustained hypertension, and aortic pulsed wave velocity by magnetic resonance imaging; urin

101 ) present repeated measures of aorto-femoral pulse wave velocity, capacitive compliance (C1), and osc

102 diac cycle length, carotid to femoral artery pulse wave velocity, carotid artery pulse waves (by appl

103 bclinical CVD were assessed: carotid-femoral pulse wave velocity, carotid intima media thickness, and

104 elial cells associated with increased aortic pulse wave velocity, carotid intima-media thickness, and

105 rin-mediated dilation (NMD), carotid-femoral pulse wave velocity, carotid-radial pulse wave velocity,

106 ss and pressure pulsatility (carotid-femoral pulse wave velocity, central pulse pressure [CPP], and f

107 io measure, and a measure of carotid-femoral pulse wave velocity (cf-PWV) and augmentation index (AI)

108 Carotid-femoral pulse wave velocity (CF-PWV) and brachial and central pu

109 , along with blood pressure, carotid-femoral pulse wave velocity (cf-PWV), lipids/lipoproteins, and g

110 2012-2013) by measuring the carotid-femoral pulse wave velocity (cf-PWV).

111 Carotid-femoral pulse wave velocity (CF-PWV; the gold standard index of

112 ted the relationship between carotid femoral pulse wave velocity (cfPWV) and T-cell activation (defin

113 three metro areas underwent carotid-femoral pulse wave velocity (cfPWV) assessment between 2012 and

114 mean arterial pressure, and carotid-femoral pulse wave velocity (CFPWV) in 1480 participants represe

115 Carotid-femoral pulse wave velocity (CFPWV) is a heritable measure of ao

116 Carotid-femoral pulse wave velocity (cfPWV) is regarded as the gold stan

117 teries (by ultrasonography), carotid-femoral pulse wave velocity (cfPWV), aortic augmentation index,

118 ss: brachial pulse pressure; carotid-femoral pulse wave velocity (CFPWV), which is related directly t

119 ently measured compared with carotid-femoral pulse wave velocity (cfPWV).

120 ic stiffness as estimated by carotid-femoral pulse wave velocity (cfPWV).

121 Carotid femoral pulse-wave velocity (cfPWV) measured arterial stiffness

122 ved from arterial tonometry (carotid-femoral pulse wave velocity [CFPWV], forward wave amplitude [FWA

123 ApoE(-/-) and WT mice showed that increased pulse wave velocity coincided with the fragmentation of

124 dependently associated with increased aortic pulsed wave velocity, cystatin C, and urinary albumin-to

125 Pulse wave velocity declined 8% with ALT-711 (P<0.05 at

126 ediated dilatation (P < 0.001), while aortic pulse wave velocity decreased (P < 0.001) in all three g

127 artery wall echodensity and carotid-femoral pulse wave velocity demonstrated no significant changes.

128 onometry, blood pressure, and carotid-radial pulse wave velocity did not change.

129 At 96 weeks, pulse wave velocity did not differ significantly between

130 line vascular stiffness, indexed by arterial pulse-wave velocity (Doppler) and augmentation index (ca

131 diographic quantification of early diastolic pulse-wave velocity (E) to mitral annular velocity (e')

132 to evaluate the prognostic role of estimated pulse wave velocity (ePWV), a marker of arterial stiffne

133 RI with gadolinium injection, measurement of pulse wave velocity, extracellular water, 24-hour ambula

134 Arterial stiffness, as measured by pulse wave velocity, for the early non-invasive screenin

135 arterial stiffness were the carotid femoral pulse wave velocity, forward pressure wave amplitude, ce

136 = 0.03), and reduced (i.e. improved) aortic pulse wave velocity from 7.1 +/- 0.3 to 6.1 +/- 0.3 m s(

137 esult in a significant improvement in aortic pulse wave velocity from baseline.

138 iffness increased markedly with age, eg, for pulse wave velocity, from a few percent in both sexes ag

139 re change in blood pressure, cardiac output, pulse wave velocity, glomerular filtration rate, natriur

140 surement of AS by applanation tonometry with pulse-wave velocity has been the gold-standard method an

141 rial stiffness, measured via carotid-femoral pulse wave velocity, has a better predictive value than

142 ar risk factors, both higher carotid-femoral pulse wave velocity (hazard ratio [HR], 1.32; 95% confid

143 thickness, echocardiography, measurement of pulse wave velocity, hepatic ultrasonography, retinal fu

144 nces between treatment in carotid-to-femoral pulse wave velocity, high-sensitivity C-reactive protein

145 A small improvement in the aortic pulse wave velocity (i.e., a decrease of 0.22 m/s; 95% C

146 ly decreased aortic root diameters and lower pulse wave velocity in doxycycline-treated Marfan mice s

147 hildren with PAH had significantly increased pulse wave velocity in the ascending aorta (3.4 versus 2

148 aseline independently associated with aortic pulse wave velocity in the complete cohort and progressi

149 OC] and arterial stiffening (aortic pulse wave velocity in young: 3.62 +/- 0.15 m( ) s(-1 )

150 terial distensibility, assessed by measuring pulse-wave velocity in vivo.

151 score); arterial stiffness (carotid-femoral pulse wave velocity); incident hypertension, diabetes, c

152 In vivo aortic stiffness (pulse wave velocity) increased progressively with age in

153 Pulse wave velocity (index of arterial stiffness) was al

154 ice a Western diet markedly increased aortic pulse-wave velocity, intima-media thickening, oxidized l

155 Pulse wave velocity is an independent predictor of the l

156 rtic stiffening, assessed by carotid-femoral pulse wave velocity, is associated with CKD.

157 The values of age, serum phosphate, pulse wave velocity, left ventricular mass (LVM), and LV

158 Pulse-wave velocity/left ventricular ejection fraction r

159 atriuretic peptide, pulse-wave velocity, and pulse-wave velocity/left ventricular ejection fraction s

160 of 19 %HbO(2) [52.6%]; P < .001); and aortic pulse wave velocity marginally increased (0.19 of 6.05 m

161 res (central pulse pressure, carotid-femoral pulse-wave velocity, mean arterial pressure, forward pre

162 8.1 +/- 3.3%), and lower arterial stiffness (pulse wave velocity: mean 6.99 +/- 1.0 m/s vs. 7.05 +/-

163 Aortic pulse-wave velocity measured vascular stiffness.

164 Patients also underwent assessment of pulse wave velocity, measurement of circulating superoxi

165 ders of magnitude higher), as illustrated by pulse wave velocity measurements, toward hypertension de

166 peptide were associated with carotid-femoral pulse wave velocity (men: partial correlation, 0.069, P

167 d r = -0.062, P = 0.040), and carotid-radial pulse wave velocity (men: r = -0.090, P = 0.009 and r =

168 wave reflection, reflected wave timing, and pulse wave velocity noninvasively in 6417 (age range, 19

169 mediated dilation (FMD) and change in aortic pulse wave velocity over 12 months.

170 ng aortic distensibility and positively with pulse wave velocity (P<0.05).

171 ratio of MPA to aortic size correlated with pulse wave velocity (P=0.0098), strain (P=0.0099), and d

172 =0.008) and strain (P=0.004) and aortic arch pulse wave velocity (P=0.01) with the aerobic exercise t

173 Carotid-femoral pulse wave velocity (P=0.02), central pulse pressure (P<

174 Indexed MPA diameter correlated with pulse wave velocity (P=0.04) and with aortic strain (P=0

175 stensibility (P=0.016) and an 8% decrease in pulse wave velocity (P=0.058).

176 There were no between-group differences in pulse wave velocity (P=0.958) or left ventricular mass (

177 ing with iontophoresis), arterial stiffness [pulse wave velocity, pulse wave analysis (PWA)], 24-h am

178 Single-point pulse wave velocities (PWV; Bramwell-Hill) and axial sti

179 ior diameter (increase of 54.9% +/- 2.5) and pulse wave velocity (PWV) (decrease of 1.3 m/sec +/- 0.8

180 ardiovascular magnetic resonance measures of pulse wave velocity (PWV) and aortic distensibility (AoD

181 of arterial stiffness indices [i.e., aortic pulse wave velocity (PWV) and augmentation (AGI) of caro

182 Aortic blood pressure (BP), pulse wave velocity (PWV) and augmentation index (AIx) w

183 rced vital capacity [FVC]) and a decrease in pulse wave velocity (PWV) and augmentation index up to 2

184 We measured aortic pulse wave velocity (PWV) and brachial PWV to evaluate t

185 Aortic pulse wave velocity (PWV) and carotid augmentation index

186 ein, and arterial stiffness [carotid-femoral pulse wave velocity (PWV) and carotid augmentation index

187 and arterial compliance as assessed by using pulse wave velocity (PWV) and central augmentation index

188 ional stiffness within the aortic arch using pulse wave velocity (PWV) and have found a stronger asso

189 outcomes were changes in carotid to femoral pulse wave velocity (PWV) and plasma 8-isoprostane F2alp

190 Pulse wave velocity (PWV) and the augmentation index (AI

191 ar stiffness was measured by carotid-femoral pulse wave velocity (PWV) and total arterial compliance.

192 or the crossover study was the difference in pulse wave velocity (PWV) between treatment with placebo

193 Arterial pulse wave velocity (PWV) correlates with the level of s

194 ysis of regional stiffness, as calculated by pulse wave velocity (PWV) for large-, medium- and small-

195 al time (PAT), Pulse transit time (PTT), and Pulse Wave Velocity (PWV) have all been used as metrics

196 We tested this by examining pulse wave velocity (PWV) in brachial arteries of twin s

197 ng SB would decrease blood pressure (BP) and pulse wave velocity (PWV) in sedentary adults.

198 Previous studies have suggested that AIx and pulse wave velocity (PWV) increase linearly with age, ye

199 Pulse wave velocity (PWV) independently predicts cardiov

200 Increased arterial stiffness measured by pulse wave velocity (PWV) is an important parameter in t

201 assessed by magnetic resonance imaging (MRI) pulse wave velocity (PWV) measurements.

202 Higher pulse wave velocity (PWV) reflects increased arterial st

203 ness using PA relative area change (RAC) and pulse wave velocity (PWV) to identify early signs for PA

204 Pulse wave velocity (PWV) was assessed three times by fi

205 Pulse wave velocity (PWV) was calculated by the foot-to-

206 Pulse wave velocity (PWV) was calculated using the foot-

207 Aortic pulse wave velocity (PWV) was calculated.

208 Pulmonary pulse wave velocity (PWV) was determined by the interval

209 Aortic pulse wave velocity (PWV) was measured in 2007-2009 (Pha

210 Pulse wave velocity (PWV) was measured in the central (c

211 Pulse wave velocity (PWV) was measured invasively (aorti

212 ediated dilatation (FMD), blood pressure and pulse wave velocity (PWV) were assessed as secondary out

213 otid artery intima-media thickness (IMT) and pulse wave velocity (PWV) were evaluated at baseline and

214 otid artery intima-media thickness (IMT) and pulse wave velocity (PWV) were evaluated in 101 PHIV and

215 ain, incremental elastic modulus (Einc), and pulse wave velocity (PWV) were measured over a TP range

216 ure (cSBP), central pulse pressure (cPP) and pulse wave velocity (PWV) were measured with the Sphygmo

217 -mediated dilation (FMD) and carotid-femoral pulse wave velocity (PWV) were measured.

218 ensors can accurately detect and measure the pulse wave velocity (PWV) when skin mounted.

219 at there is a progressive increase in aortic pulse wave velocity (PWV) with age.

220 ular (carotid intima-media thickness (cIMT), pulse wave velocity (PWV)) and cardiac (left ventricular

221 ion (FMD) and flow-mediated slowing (FMS) of pulse wave velocity (PWV), 10-and 60-min after a high-in

222 ere 1) arterial stiffness measured by aortic pulse wave velocity (PWV), 2) oxidative stress assessed

223 Pulse wave velocity (PWV), a measure of vascular stiffne

224 This study sought to evaluate whether pulse wave velocity (PWV), a noninvasive index of arteri

225 The main outcome was carotid-femoral pulse wave velocity (PWV), an established biomarker of l

226 mediated vasodilation (FMD), carotid-femoral pulse wave velocity (PWV), and aortic augmentation index

227 arterial pressure (MAP), augmentation index, pulse wave velocity (PWV), and intima-media thickness.

228 measures, including augmentation index (AI), pulse wave velocity (PWV), and the recently proposed har

229 assess hepatic triglyceride content, aortic pulse wave velocity (PWV), and visceral fat.

230 ness of the common carotid artery (CCA-IMT), pulse wave velocity (PWV), augmentation index, blood pre

231 brachial artery blood pressure (BP), aortic pulse wave velocity (PWV), B-mode ultrasonography and wa

232 Disease activity, blood pressure, aortic pulse wave velocity (PWV), brachial artery flow-mediated

233 nal studies addressing the impact of NSPT on pulse wave velocity (PWV), carotid intima-media thicknes

234 indicators of arterial stiffness, including pulse wave velocity (PWV), central augmentation index (C

235 Exhaled carbon monoxide (CO), pulse wave velocity (PWV), malondialdehyde (MDA) and thr

236 Progressive increase in pulse wave velocity (PWV), maximal intra-luminal diamete

237 derived functionally, e.g. by measurement of pulse wave velocity (PWV), or morphologically, e.g. by a

238 , carotid intima-media thickness (CIMT), and pulse wave velocity (PWV), respectively.

239 miR-92a level was positively correlated with pulse wave velocity (PWV), systolic blood pressure (SBP)

240 on carotid artery intima media thickness and pulse wave velocity (PWV), were evaluated at baseline an

241 ently, vascular stiffness was assessed using pulse wave velocity (PWV).

242 ccompanied by vascular fibrosis and elevated pulse wave velocity (PWV).

243 cted by increased flow velocities, mainly by pulse wave velocity (PWV).

244 exercise on arterial stiffness, measured by pulse wave velocity (PWV).

245 vascular damage is reflected by increases in pulse wave velocity (PWV; indicating arteriosclerosis),

246 ng 2007 to 2012, we measured carotid-femoral pulse wave velocity (PWV; SphygmoCor apparatus) 8 weeks

247 Arterial stiffness was determined by pulse-wave velocity (PWV) of the brachioradial and femor

248 otid artery intima-media thickness (IMT) and pulse-wave velocity (PWV) were evaluated in 101 PHIV and

249 rial distensibility measures, generally from pulse-wave velocity (PWV), are widely used with little k

250 PWV(CR) ) arterial stiffness was measured by pulse-wave velocity (PWV), together with systolic (SBP)

251 Measures of arterial stiffness (pulse wave velocity [PWV] and augmentation index correct

252 ple (n = 42), cPP, arterial stiffness (using pulse wave velocity [PWV]) and arterial diameters (using

253 heir relation to central arterial stiffness (pulse wave velocity [PWV]) and arterial diameters, and t

254 s and arterial stiffness (carotid to femoral pulse wave velocity [PWV]) measured at age 17 years.

255 Arterial stiffness (carotid to femoral pulse wave velocity [PWV]) was measured and peripheral b

256 nction (local aortic distensibility and arch pulse wave velocity [PWV]), and LV volumes and mass.

257 r stroke) in relation to arterial stiffness (pulse wave velocity [PWV]), wave reflection (augmentatio

258 erformance index (MPI) and aortic stiffness (pulse wave velocity; PWV) were evaluated before and afte

259 Carotid-to-femoral pulse wave velocity (PWVc-f) was assessed at baseline, a

260 cysteine was associated with carotid-femoral pulse wave velocity (r = 0.072, P = 0.036), forward pres

261 Mean pulse wave velocity remained stable with both everolimus

262 Carotid to femoral pulse wave velocity showed a significant reduction from

263 Treatment also reduced aortic pulse wave velocity significantly (from 9.09+/-1.77 to 8

264 Pulse wave velocity, superoxide, and C-reactive protein

265 he weight-loss group, but carotid-to-femoral pulse wave velocity tended to decrease by 0.5 m/s (P = 0

266 d dilation to evaluate endothelial function, pulse wave velocity to assess arterial stiffness, and le

267 systolic blood pressure and carotid-femoral pulse wave velocity to the model, forward pressure wave

268 nalyzed the primary outcome, carotid-femoral pulse wave velocity, using a linear mixed effects model

269 there were significant associations between pulse-wave velocity values and left ventricular ejection

270 We newly report that the assessment of local pulse wave velocity via MRI provides early information a

271 was 0.13 (95% CI: 0, 0.26; P = 0.044) lower, pulse wave velocity was 0.29 m/s (95% CI: 0.07, 0.52 m/s

272 phosphate was 1.25 mmol/L (3.87 mg/dl), mean pulse wave velocity was 10.8 m/s, and 81.3% had abdomina

273 Carotid-femoral pulse wave velocity was associated with higher white mat

274 Aortic pulse wave velocity was high-normal (9.2 +/- 2.2 m/s), i

275 Pulse wave velocity was higher in adults after ASO (5.0+

276 Pulse wave velocity was measured at baseline in 449 norm

277 Pulse wave velocity was measured using cardiac magnetic

278 mice, whereas at the age of 18 weeks, local pulse wave velocity was significantly elevated in ApoE(-

279 Functionally, pulse wave velocity was significantly higher in patients

280 IMT was 0.71 +/- 0.1 mm, and the mean +/- SD pulse-wave velocity was 5.96 +/- 1.6 meters/second.

281 Pulse-wave velocity was acutely significantly reduced at

282 Pulse-wave velocity was assessed from tonometry and body

283 Pulse-wave velocity was higher in hypertensives (P=0.001

284 Carotid-femoral pulse-wave velocity was significantly (P<0.001) faster a

285 Aortic stiffness, measured by pulse wave velocity, was approximately 35% greater in El

286 Pulse wave velocity, wave travel times, and lumped press

287 Doppler probes were used to collect pulse-wave velocity waveforms from the right carotid and

288 oral pulse wave velocity, and carotid-radial pulse wave velocity were assessed by tonometry in 1962 p

289 arget-to-background ratios (TBRs) and aortic pulse wave velocity were assessed.

290 ssure, pulsatility index and carotid-femoral pulse wave velocity were each associated with increased

291 n the brachial artery, and carotid to radial pulse wave velocity were measured in all children.

292 ardiac and thoracic aorta calcium scores and pulse wave velocity were measured to evaluate VC progres

293 ac markers such as NT-proBNP, Troponin T and pulse wave velocity were monitored.

294 aorta, and cardiac valve calcium scores and pulse wave velocity were not significantly different amo

295 Left ventricular ejection fraction and pulse-wave velocity were both associated with Hunt and H

296 Left ventricular ejection fraction and pulse-wave velocity were improved between acute aneurysm

297 active hyperemia index, aortic hemodynamics, pulse wave velocity) were not differentially altered by

298 measures (distensibility, aortic strain, and pulse wave velocity) were similar across all groups.

299 The mean +/- SD carotid-femoral pulse wave velocity, which reflects central aortic stiff

300 interval, 2.4-20.7), augmentation index, and pulse wave velocity without changing peripheral blood pr