ライフサイエンスコーパス: pupillary

1 sual acuity loss, visual field constriction, pupillary abnormalities, attenuated retinal arteries, lo

2 ing intensity were presented to one eye, and pupillary amplitude and constriction velocity were measu

3 changes in gain, we found that the measured pupillary and behavioral variables were strongly correla

4 This demonstrates that tVNS reliably induces pupillary and EEG markers of arousal beyond the effects

5 measures that indexed motivation level using pupillary and saccadic response to monetary incentives,

6 The pupillary aperture was reduced significantly (42.11+/-20

7 l acuity (BCVA), intraocular pressure (IOP), pupillary aperture, glare, contrast sensitivity, endothe

8 lar lens (IOL) opacification confined to the pupillary area are reported from clinical practice in Lo

9 a showed multiple free-floating cysts in the pupillary area associated with iris neovascularization a

10 Median pupillary area could significantly (p < 0.0001) be reduc

11 Pupillary areas of anesthetized C57BL/6 mice were measur

12 status was measured by retinoscopy along the pupillary axis and at 15 degrees intervals along the hor

13 rrors were measured by retinoscopy along the pupillary axis and at eccentricities of 15 degrees , 30

14 ed by streak retinoscopy performed along the pupillary axis and at eccentricities of 15 degrees, 30 d

15 assessed by retinoscopy performed along the pupillary axis and at eccentricities of 15 degrees, 30 d

16 efractive development was assessed along the pupillary axis by retinoscopy, keratometry, and A-scan u

17 rior vitrectomy and the IOL may be used as a pupillary barrier to prevent loss of lens fragments.

18 In the presence of pupillary block (+PB), AOD500 decreased 25% and 36% for

19 (2) in the presence (+PB) versus absence of pupillary block (-PB) to quantify the effect of dynamic

20 positioning group, and IOP increase (n = 9), pupillary block (n = 1), choroidal effusion (n = 2), CME

21 s: crowded-angle (CR), lens subluxation (LS) pupillary block (PB), and plateau iris syndrome (PL).

22 One case developed postoperative pupillary block and 1 case required endothelial transpla

23 etter efficacy than C3F8 without the risk of pupillary block and thus should be preferred.

24 otruding Soemmering content causing absolute pupillary block became resolved after laser iridotomy an

25 on the first postoperative day to alleviate pupillary block caused by a nonpatent iridotomy.

26 laser peripheral iridotomy to eliminate any pupillary block due to primary angle-closure glaucoma.

27 Pupillary block glaucoma, steroid-induced intraocular pr

28 ions up to 2 years postoperatively comprised pupillary block in 1 eye (successfully reversed by parti

29 compensation, iritis, secondary glaucoma, or pupillary block occurred after surgery in any eye.

30 emmering-capsule-IOL complex caused relative pupillary block similar to a phakic eye and was successf

31 y dynamic pupillary block, but the effect of pupillary block was not as large as that of the dilator

32 Pupillary block was observed in the early postoperative

33 ocation of the dilator muscle and by dynamic pupillary block, but the effect of pupillary block was n

34 atures most consistent with greater baseline pupillary block.

35 perior, inferior, nasal, and temporal to the pupillary center, to create oblique angles of incidence

36 ace impacts the neural processing of others' pupillary changes in early ontogeny.

37 y, infants have been found to mimic observed pupillary changes in others, instantiating a foundationa

38 Among adults, perception of pupillary changes is affected by race.

39 nt, the fundamental process of responding to pupillary changes is impacted by race and interracial in

40 Moreover, when processing other-race pupillary changes, infants recruited the dorsolateral pr

41 sed competing hypotheses about the causes of pupillary changes.

42 case (1.5 vs 1.05), incidence of post-laser pupillary constriction (9.5% vs 1.23%), and anterior cap

43 iris sphincter and ciliary muscle to mediate pupillary constriction and lens accommodation, respectiv

44 ious physiological responses to light, e.g., pupillary constriction and neuroendocrine regulation.

45 in circuits mediating circadian entrainment, pupillary constriction and other non-image-forming visua

46 In sighted individuals, pupillary constriction decreased monotonically for at le

47 at low irradiance levels, and for sustained pupillary constriction during exposure to light in the l

48 y significant difference was observed in the pupillary constriction of the treated eye (P<0.05) compa

49 The melanopsin-mediated, sustained pupillary constriction phase response following cessatio

50 was compared with the photoreceptor-mediated pupillary constriction phase response following cessatio

51 nuation of the melanopsin-mediated sustained pupillary constriction response was significantly associ

52 rations in the melanopsin-mediated sustained pupillary constriction response.

53 hotoreceptors repeatedly, elicited sustained pupillary constriction responses that were more than twi

54 an enhanced pupillary light reflex (PLR)-the pupillary constriction that occurs in response to light

55 ocular motor disorders, such as paradoxical pupillary constriction to darkness, benign tonic upgaze

56 from the observed (or measured) speed of the pupillary constriction to light.

57 n4(-/-) mice, in contrast, could not sustain pupillary constriction under continuous bright illuminat

58 Our data show a clear linear increase in pupillary constriction with increasing log light intensi

59 Cs (ipRGCs) drive circadian-clock resetting, pupillary constriction, and other non-image-forming phot

60 on-image-forming visual responses, including pupillary constriction, circadian photoentrainment and s

61 nses differed from that necessary to trigger pupillary constriction, suggesting that photopotentiatio

62 short isoform (OPN4S) mediates light-induced pupillary constriction, the long isoform (OPN4L) regulat

63 light to drive circadian clock resetting and pupillary constriction.

64 repair visual acuity, postoperative afferent pupillary defect (APD), old age, scleral laceration, and

65 isual acuity (P = .034), a relative afferent pupillary defect (RAPD) (P = .002), or a history of syst

66 sease severity can cause a relative afferent pupillary defect (RAPD).

67 in 8 eyes, iridodialysis in 7 eyes, afferent pupillary defect in 6 eyes, lens dislocation or subluxat

68 retina or choroid, poorer visual acuity, and pupillary defect were associated with visual field defec

69 nsistent with NAION, (3) a relative afferent pupillary defect, (4) observed optic disc swelling, and

70 Fifty participants with relative afferent pupillary defects (RAPDs) confirmed using the swinging f

71 l acuity less than 20/200, relative afferent pupillary defects, optic nerve pallor, and visual field

72 acuity and contrast sensitivity suggest low pupillary dependence for light distribution.

73 so induced a significant decrease in maximum pupillary diameter (0.49+/-0.17 mm, P=.005).

74 he in vivo permeability assay, the change in pupillary diameter at 30 minutes after pilocarpine admin

75 e detected a significant decrease in maximum pupillary diameter by 0.50+/-0.19 mm (P=.011) and in the

76 Changes in pupillary diameter correlated with pilocarpine-induced A

77 We found that the pupillary diameter rapidly varied according to perceived

78 ated sustained pupillary response (mean [SD] pupillary diameter ratios at a point in time, 0.18 [0.1]

79 ataract severity, cataract extraction, small pupillary diameters (<5.5 mm), defocusing, and excessive

80 ast changes could not be responsible for the pupillary differences.

81 Using pupillary dilatation as a measure of central noradrenerg

82 By measuring pupillary dilatation in response to these stimuli-and si

83 rating characteristic curve before and after pupillary dilatation were not found.

84 corneal light reflex axis, is independent of pupillary dilation and phakic status of the eye.

85 Pupillary dilation during cognitive tasks provides a bio

86 thdrawal) dopamine medication, as indexed by pupillary dilation in anticipation of reward.

87 ce of pseudoexfoliation was looked for after pupillary dilation in either or both eyes at 1 or more l

88 Pupillary dilation responses were recorded during a digi

89 Pupillary dilation to increasing levels of monetary rewa

90 an subjects with narrow angles was low after pupillary dilation with tropicamide and oral acetazolami

91 f intersession testing, cataract extraction, pupillary dilation, focal plane, and gain settings on th

92 hermore, death was accompanied by unilateral pupillary dilation, which is indicative of uncal herniat

93 using slit-lamp biomicroscopy after medical pupillary dilation.

94 autonomic functions measured by dynamics of pupillary dilation.SIGNIFICANCE STATEMENT Most of our kn

95 alignment (decentration, tilt, rotation) and pupillary ectopia (4.5 mm iris aperture) were varied upo

96 Pupillary evaluation is a crucial element of physical ex

97 he El Greco fallacy by reviewing some recent pupillary evidence supporting top-down modulation of per

98 A total of 430 patients (8.7%) had a pupillary expansion device used during their cataract su

99 intraoperative vitreous prolapse, and use of pupillary expansion devices.

100 Pupillary fatigue waves became more evident with test du

101 (8/12) showed gradual miosis and periods of pupillary fatigue waves during the recording session.

102 the pupil diameter and the amplitude of any pupillary fatigue waves.

103 The measurement of the pupillary function is an indispensable test in some eye

104 tting, and standing, and eyelid function and pupillary function testing, was completed on 3 young pat

105 tent of rod-, cone-, and melanopsin-mediated pupillary light reflex (PLR) abnormalities in diabetic p

106 ns of rod, cone, and melanopsin to the human pupillary light reflex (PLR) and to determine the optima

107 binocular pupillography was used to measure pupillary light reflex (PLR) in 44 healthy children (23

108 The mammalian pupillary light reflex (PLR) involves a bilateral brain

109 Pupillary light reflex (PLR) is an involuntary response

110 cement of visual function in rd/rd mice: the pupillary light reflex (PLR) returned almost to normal;

111 erwent repeated measurements of quantitative pupillary light reflex (PLR) using the Neurolight-Algisc

112 ng to bright surfaces results in an enhanced pupillary light reflex (PLR)-the pupillary constriction

113 sically photosensitive iris and thus a local pupillary light reflex (PLR).

114 n photoentrainment and severely disrupts the pupillary light reflex (PLR).

115 which include circadian photoentrainment and pupillary light reflex (PLR).

116 including circadian photoentrainment and the pupillary light reflex (PLR).

117 n-image forming visual processes such as the pupillary light reflex and circadian entrainment but als

118 Cs) control non-visual light responses (e.g. pupillary light reflex and circadian entrainment).

119 l centers that mediate behaviors such as the pupillary light reflex and circadian entrainment.

120 ipRGCs dampened the sensitivity of both the pupillary light reflex and circadian photoentrainment, t

121 primarily nonimage visual functions, such as pupillary light reflex and circadian photoentrainment, w

122 n-image-forming visual functions such as the pupillary light reflex and circadian photoentrainment.

123 Intraocular injection of AAQ restores the pupillary light reflex and locomotory light avoidance be

124 The sensitivity of the pupillary light reflex and negative masking (activity su

125 elanopsin and rod-cone photoreceptors to the pupillary light reflex in humans, we compared pupillary

126 ults provide an anatomical substrate for the pupillary light reflex in the cat.

127 In mammals, the pupillary light reflex is mediated by intrinsically phot

128 ediated persistent constriction phase of the pupillary light reflex may represent a surrogate biomark

129 In contrast, sensitivity of the pupillary light reflex was more severely reduced in rd1

130 The pupillary light reflex was reduced in patients with POAG

131 The central pathways subserving the feline pupillary light reflex were examined by defining retinal

132 er with basic neurological examinations (eg, pupillary light reflex) contributed heavily to a linear

133 ritical for competent circadian entrainment, pupillary light reflex, and other non-imaging-forming ph

134 brain to control circadian photoentrainment, pupillary light reflex, and sleep.

135 masking behavior, melatonin suppression, the pupillary light reflex, and sleep/wake cycles.

136 including circadian photoentrainment and the pupillary light reflex, are thought to be mediated by th

137 retinal ganglion cells (ipRGCs) mediate the pupillary light reflex, circadian entrainment, and may c

138 Age, GCS score, injury severity score, pupillary light reflex, CT findings (compressed basal ci

139 onimage-forming visual functions such as the pupillary light reflex, masking behavior, and light-indu

140 esponses to environmental light, such as the pupillary light reflex, seasonal adaptations in physiolo

141 iors like circadian photoentrainment and the pupillary light reflex, the characterization of multiple

142 the rods, cones, and ipRGCs that mediate the pupillary light reflex.

143 n nucleus, indicating its involvement in the pupillary light reflex.

144 o non-image-forming nuclei and an attenuated pupillary light reflex.

145 ation, indicating that these cells serve the pupillary light reflex.

146 kinetic visual field, nystagmus testing, and pupillary light reflex.

147 ympathetic input to the islet grafts via the pupillary light reflex.

148 eration for negative masking but not for the pupillary light reflex.

149 t are different for negative masking and the pupillary light reflex.

150 is sufficient for reaching threshold for the pupillary light reflex.

151 es including circadian photo-entrainment and pupillary light reflex.

152 onse to unilateral ESV, while preserving the pupillary light reflex.

153 synchronization of circadian rhythms and the pupillary light reflex.

154 caque frontal eye fields (FEF) modulates the pupillary light reflex.

155 ite, red, and blue colors; visual field; and pupillary light reflex.

156 rocesses including circadian rhythms and the pupillary light reflex.

157 rcuit that mediates a very basic reflex, the pupillary light reflex.

158 s such as circadian photoentrainment and the pupillary light reflex.

159 al olivary nucleus (PON), which controls the pupillary light reflex; the superior colliculus (SC), wh

160 ses including circadian photoentrainment and pupillary light reflexes and contrast detection for imag

161 the affected dog was functionally blind, and pupillary light reflexes and ERG response amplitudes con

162 lete ophthalmic ocular examination including pupillary light reflexes and laboratory examinations; co

163 Mice lacking rods and cones retain pupillary light reflexes that are mediated by intrinsica

164 The earliest clinical signs (reduced pupillary light reflexes) were seen at 2 to 3 weeks of a

165 logical processes, including light aversion, pupillary light reflexes, and photoentrainment of circad

166 as photic entrainment of circadian rhythms, pupillary light reflexes, etc.

167 s supported by reduced direct and consensual pupillary light reflexes, phenotypic presence of retinal

168 g functions, circadian photoentrainment, and pupillary light reflexes.

169 al dysautonomic features, including impaired pupillary light reflexes.

170 circadian entrainment, sleep induction, the pupillary light response (PLR), and negative masking of

171 ight including circadian entrainment and the pupillary light response (PLR).

172 ted in some cases by objective tests such as pupillary light response and nystagmography.

173 under continuous bright illumination to the pupillary light response and suggest the presence of a p

174 generate mice reduces the sensitivity of the pupillary light response at all wavelengths but does not

175 cells [ipRGCs]) are sufficient to drive the pupillary light response in mice.

176 ment-state distinct from that triggering the pupillary light response itself.

177 ectroretinogram becomes undetectable and the pupillary light response weakens.

178 o subserve circadian photic entrainment, the pupillary light response, and a number of other aspects

179 entrainment of circadian rhythms, and to the pupillary light response.

180 nner and outer retinal photoreception to the pupillary light response.

181 Absent pupillary light responses (FPR 1; 95% CI, 0-7) or absent

182 ecithin-retinol acyl transferase (Lrat) have pupillary light responses (PLR) that are less sensitive

183 Pupillary light responses appear unaffected at BAC level

184 However, photoentrainment and pupillary light responses are preserved.

185 upillary light reflex in humans, we compared pupillary light responses in normally sighted individual

186 a very limited contribution to circadian and pupillary light responses under these conditions.

187 tients had at least a 2 log unit increase in pupillary light responses, and an 8-year-old child had n

188 Nonvisual photoreception (pupillary light responses, circadian entrainment, and in

189 The absence of pupillary light responses, corneal reflexes, and an exte

190 nfidence intervals (CIs) were calculated for pupillary light responses, corneal reflexes, and motor s

191 psin mutant (opn4(-/-)) mice were tested for pupillary light responsiveness by video pupillometry bef

192 rate amplitude (0.5 log) circadian rhythm of pupillary light responsiveness was observed in rd/rd mic

193 -)/(-);rd/rd mice showed significantly lower pupillary light sensitivity than rd/rd mice alone.

194 g of pigmented translucent iris cysts at the pupillary margin of each eye, confirmed with ultrasound

195 In predilation pupillary measurements, we detected a significant decrea

196 ere included, 6 with a unilateral congenital pupillary membrane and 1 with classic persistent fetal v

197 the 6 patients with a unilateral congenital pupillary membrane had 1 or more recurrences after a mem

198 In contrast, histopathology of a recurrent pupillary membrane revealed collagenized fibrovascular t

199 Congenital fibrovascular pupillary membranes in infants are likely a variant of P

200 Histopathologic examination of 2 primary pupillary membranes showed fibrovascular tissue that did

201 The 2 patients without recurrences of the pupillary membranes underwent multiple iris sphincteroto

202 e lens reproliferation into the visual axis, pupillary membranes, and corectopia.

203 The 4 eyes manifested with pupillary membranes, immature anterior chamber angles, l

204 In Parkinson's disease, reduced pupillary modulation by incentives was predictive of apa

205 of control patients indicated that increased pupillary modulation by reward can be predictive of the

206 ine the relationship between complex eye and pupillary movements, collectively referred to as eye met

207 l syndromes) and cardiac (all syndromes) and pupillary (non-fluent variant only) reactivity.

208 ich exhibited severe microphthalmia, reduced pupillary openings, disrupted fiber cell morphology, eve

209 bsent somatosensory-evoked potential, absent pupillary or corneal reflexes, presence of myoclonus, an

210 Thus, the pupillary physiological response reflects the subjective

211 Notably, this atypical pupillary profile was evident despite the fact that both

212 mon neuro-ophthalmologic finding was minimal pupillary reaction to light (25%).

213 Visual acuity (VA), pupillary reaction, and optic disc findings were assesse

214 er age (median 44 vs. 53 yrs), more abnormal pupillary reactions (52% vs. 32%), and more intracranial

215 lasgow Coma Score, hypotension, hypoxia, and pupillary reactions between undocumented immigrants and

216 Coma Scale score, the Injury Severity Score, pupillary reactivity, and presence of midline shift.

217 tomography characteristics, injury severity, pupillary reactivity, mitochondrial haplogroups, and APO

218 -image-forming visual behaviors, such as the pupillary reflex and circadian photoentrainment.

219 nvisual light-sensing functions, such as the pupillary reflex and entrainment of circadian rhythms.

220 sition improvement using lower lid margin-to-pupillary reflex distance was the most common outcome me

221 For the pupillary reflex evaluation, patients were tested in the

222 s that regulate the biological clock and the pupillary reflex in mammals, is homologous to invertebra

223 The pupillary reflex was an important determinant, regardles

224 s also affected saccade velocity and reduced pupillary reflex.

225 d absent corneal reflexes (33.5%) and absent pupillary reflexes (36.2%) at 24 hours, which is earlier

226 w false-positive rates: bilateral absence of pupillary reflexes more than 24 hours after a return of

227 6 months and absent motor response or absent pupillary reflexes or bilateral absent cortical response

228 ing light adaptation, circadian entrainment, pupillary reflexes, and other aspects of non-image-formi

229 ons including circadian photoentrainment and pupillary reflexes.

230 , collapsed PAS-positive lens capsule in the pupillary region.

231 nuation of the melanopsin-mediated sustained pupillary response (mean [SD] pupillary diameter ratios

232 g perception have revealed modulation of the pupillary response according to the brightness of task-r

233 and its activity was associated with larger pupillary response and better performance in the task.

234 atients were OFF dopaminergic drugs, both in pupillary response and saccadic peak velocity response t

235 d ipRGC function caused by glaucoma affected pupillary response and sleep quality.

236 The crucial metric was the growth of the pupillary response and the reduction of this response fo

237 used to characterize the association between pupillary response characteristics and alterations in re

238 ental assessment of various stimulus-induced pupillary response characteristics and was conducted at

239 Association of pupillary response characteristics with alterations in r

240 ERG recordings and tests of the consensual pupillary response confirmed the effectiveness of each d

241 s in eccentric gaze holding and no effect in pupillary response dynamics were observed at levels belo

242 o corneal permeability was quantified as the pupillary response over a 30-minute period to a dose of

243 The pupillary response peaks lagged behind insular activatio

244 ix control subjects we studied the binocular pupillary response to a variety of sharply defined colou

245 pid eye movement latency and the peak of the pupillary response to the blue flash (P = 0.004).

246 atients, like the control subjects, showed a pupillary response to the structured coloured displays,

247 the control subjects, the patients showed no pupillary response when the coloured displays lacked sha

248 Light intensity was a strong predictor of pupillary response, regardless of baseline pupil size.

249 concurrently measured cortical activity and pupillary response, using functional near infrared spect

250 the relationship between light intensity and pupillary response.

251 tive processes can be obtained from the slow pupillary response.

252 crease in arousal levels as reflected by the pupillary response.

253 radiance light, indicating that steady-state pupillary responses are an order of magnitude slower tha

254 with schizophrenia was investigated by using pupillary responses as a biomarker of task effort.

255 pillometer is designed to record and analyze pupillary responses at multiple, controlled stimulus int

256 ing a pupillometer, we recorded and analyzed pupillary responses at varied stimulus patterns (full fi

257 -ramp stimuli, to eccentric gaze holding, to pupillary responses evoked by light flashes.

258 ta/cm(2)/s retinal irradiance) and recording pupillary responses for 50 seconds after light cessation

259 Light-evoked pupillary responses help the eyes of animals perform opt

260 trols, individuals with ASD evinced atypical pupillary responses in the presence versus absence of di

261 sistent with the choices, eye movements, and pupillary responses of subjects who commit to the optima

262 nses; stressed individuals showed attenuated pupillary responses to action, hinting at a noradrenergi

263 explanation for the present findings is that pupillary responses to ambient light reflect the perceiv

264 Pupillary responses to blue light and red light were com

265 isual photoreceptors are required for normal pupillary responses to continuous light exposure at low

266 e photoreceptors and melanopsin in mediating pupillary responses to continuous light.

267 h depression and examined how differences in pupillary responses to emotional stimuli correlate with

268 e infrared camera digitally records afferent pupillary responses to graded light stimuli (-2.9 to 0.1

269 Pupillary responses to high-irradiance blue light associ

270 In glaucomatous eyes, reduced pupillary responses to high-irradiance blue light were a

271 Although dopamine agonist dose did modulate pupillary responses to reward, the pattern of results wa

272 r lactate, lower maximum glucose, and normal pupillary responses were all associated with survival.

273 These pupillary responses were compared with those of 19 age-m

274 In patients with POAG, pupillary responses were evaluated relative to standard

275 Moreover, between-group differences in pupillary responses were observed specifically in respon

276 Effort allocation (pupillary responses) to the task increased as the proces

277 back letter detection test while task-evoked pupillary responses, an established correlate for LC act

278 itudes, goal-directed task effort indexed by pupillary responses, and negative symptoms in schizophre

279 s demonstrated improvement in visual acuity, pupillary responses, color vision, and visual field.

280 cit in action-learning was also reflected in pupillary responses; stressed individuals showed attenua

281 Scotopic vision and pupillary responsiveness have typically been measured us

282 Pupillary responsiveness was expressed as the percent ch

283 Moreover, pupillary reward sensitivity declined with age.

284 nergic medication overnight did not modulate pupillary reward sensitivity in impulse control disorder

285 The pupillary reward sensitivity measure described here prov

286 Beehler pupil dilator, nylon iris hooks, and pupillary rings, including the Perfect Pupil, the Graeth

287 Parameters evaluated include pupillary ruff absence and abnormality, pupil edge pigme

288 Pupillary ruff and associated gonioscopy findings were g

289 Asymmetric pupillary ruff changes were associated with asymmetry in

290 The eye with the most pupillary ruff loss was 25% more likely to have the grea

291 anding of the early origins of responding to pupillary signals in others and further highlights the i

292 aim to establish normative data for scotopic pupillary size and function in the pediatric population

293 i were equiluminant so that constrictions in pupillary size could not be ascribed to changes in light

294 Observed changes in pupillary size provide a range of socially-relevant info

295 nts' brains distinguished between changes in pupillary size.

296 es identified severe cataracts and thickened pupillary sphincter muscle.

297 le miosis was present in only 1 patient, and pupillary supersensitivity to 2.5% phenylephrine was not

298 ffect of supplemental iron and riboflavin on pupillary threshold (PT) and plasma retinol in nightblin

299 central areas of IOL opacification over the pupillary zone, confined to the anterior surface of the

300 om its capsule, which was left behind in the pupillary zone.