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2 for both the NLST (Hazard Ratio (HR) = 2.20, pv = 0.01) and the SPORE cohorts (HR = 2.15 and pv = 0.0
6 = 0.01) and the SPORE cohorts (HR = 2.15 and pv = 0.044), respectively, after adjusting for key clini
7 Xanthomonas oryzae pathovar (pv.) oryzae and pv. oryzicola produce numerous transcription activator-l
8 eudomonas syringae pathovar (pv.) tomato and pv. maculicola were made by insertion of the luxCDABE op
9 gens Xanthomonas oryzae pv. oryzae (Xoo) and pv. oryzicola (Xoc) encode numerous secreted transcripti
11 odis pv. citrumelo to those of X. axonopodis pv. citri and X. campestris pv. vesicatoria provides val
20 identified unique effectors in X. axonopodis pv. citrumelo that may be related to the different host
22 he complete genome sequence of X. axonopodis pv. citrumelo to those of X. axonopodis pv. citri and X.
23 pAI, and hrpW were absent from X. axonopodis pv. citrumelo while present in X. axonopodis pv. citri.
24 anker (CBC) caused by Xanthomonas axonopodis pv. citri (Xac) was first documented in India and Java i
25 The protein Clp from Xanthomonas axonopodis pv. citri regulates pathogenesis and is a member of the
27 ght (CBB), incited by Xanthomonas axonopodis pv. manihotis (Xam), is the most important bacterial dis
29 Mb genome sequence of Xanthomonas axonopodis pv. punicae strain LMG 859, the causal agent of bacteria
30 v library were conjugated into X. campestris pv. campestris (Xcc) and exconjugants were scored for an
32 tomato bacterial spot pathogen X. campestris pv. vesicatoria 85-10, with a completely different host
33 of X. axonopodis pv. citri and X. campestris pv. vesicatoria provides valuable insights into the mech
34 to B. cinerea infection and to X. campestris pv. vesicatoria, correlated with cuticle permeability an
37 a Abra43 (Abra43) and Xanthomonas campestris pv. campestris 8004 (Xcc8004), on the structure and func
39 family encoded by the Xanthomonas campestris pv. campestris str. ATCC 33913 genome (GI:21233491).
41 lt disease, caused by Xanthomonas campestris pv. musacearum (Xcm), is a major threat to banana produc
42 e-genome sequences of Xanthomonas campestris pv. raphani strain 756C and X. oryzae pv. oryzicola stra
43 confers resistance to Xanthomonas campestris pv. vesicatoria (Xcv) pathogenic strains which contain t
46 ecreted effector from Xanthomonas campestris pv. vesicatoria, is a desumoylating enzyme with strict s
50 pic mechanism of thermal transport in MgSiO3 pv, and provide reference data for understanding heat co
52 esults support the hypothesis that X. oryzae pv. oryzae commandeers the regulation of otherwise devel
53 ive resistance to PthXo2-dependent X. oryzae pv. oryzae due to promoter variations of OsSWEET13 in ja
54 esults corroborate a model whereby X. oryzae pv. oryzae enhances the release of sucrose from host cel
55 These results suggest that the X. oryzae pv. oryzae PhoPQ TCS functions in virulence and in the p
58 ain and found it to be impaired in X. oryzae pv. oryzae virulence and no longer able to activate the
62 estris pv. raphani strain 756C and X. oryzae pv. oryzicola strain BLS256, pathogens that infect the m
63 he strains in the African clade of X. oryzae pv. oryzicola, representing the first dominant resistanc
64 m and mesophyll pathogens Xanthomonas oryzae pv. oryzae (Xoo) and pv. oryzicola (Xoc) encode numerous
65 hway of the rice pathogen Xanthomonas oryzae pv. oryzae (Xoo) in a semi-isolated environment represen
66 The biotrophic pathogen Xanthomonas oryzae pv. oryzae (Xoo) produces a sulfated peptide named RaxX,
67 ce to a broad spectrum of Xanthomonas oryzae pv. oryzae (Xoo) races that cause bacterial blight disea
68 bacterial blight pathogen Xanthomonas oryzae pv. oryzae (Xoo) that deliver the TAL effector AvrXa27.
69 livered by the pathogenic Xanthomonas oryzae pv. oryzae (Xoo), in revealing the new function of two p
71 ceptor confer immunity to Xanthomonas oryzae pv. oryzae (Xoo), the causal agent of bacterial leaf bli
72 n by pathogenic bacterium Xanthomonas oryzae pv. oryzae (Xoo), which causes a vascular disease in ric
84 xtraordinary diversity of Xanthomonas oryzae pv. oryzae genotypes and races that have been isolated f
85 ing disease by strains of Xanthomonas oryzae pv. oryzae is dependent on major transcription activatio
87 levated upon infection by Xanthomonas oryzae pv. oryzae strain PXO99(A) and depends on the type III e
90 21, confers resistance to Xanthomonas oryzae pv. oryzae strains producing the type one system-secrete
91 ative bacterial pathogen, Xanthomonas oryzae pv. oryzae upon recognition of a small protein, Ax21, th
94 tibility to M. oryzae and Xanthomonas oryzae pv. oryzae, hemibiotrophic pathogens, but enhanced resis
96 the gamma-proteobacterium Xanthomonas oryzae pv. oryzae, which utilizes a group of type III TAL (tran
100 Gram-negative bacterium, Xanthomonas oryzae pv. oryzae; NH1, the rice ortholog of NPR1, a key regula
102 e rice bacterial pathogen Xanthomonas oryzae pv. oryzicola (Xoc) has been demonstrated to contain a b
105 native TAL effector from Xanthomonas oryzae pv. oryzicola drives expression of a target with an EBE
106 cognizes a rice pathogen, Xanthomonas oryzae pv. oryzicola, which causes bacterial streak disease.
108 rice pathogens Xanthomonas oryzae pathovar (pv.) oryzae and pv. oryzicola produce numerous transcrip
109 ana pathogens Pseudomonas syringae pathovar (pv.) tomato and pv. maculicola were made by insertion of
110 mal conductivity kappa of MgSiO3 perovskite (pv) by ab initio lattice dynamics calculations combined
111 their control proteins HrpV and HrpG from Ps pv. tomato DC3000 in vitro establish that HrpRS forms a
113 ene production and increases growth of P. s. pv. tomato and severity of speck disease on susceptible
114 st response to the large collection of P. s. pv. tomato strains that express different combinations o
115 the host Pto kinase, which recognizes P. s. pv. tomato strains that express the effector AvrPto.
118 n both the olive tree pathogen P. savastanoi pv. savastanoi and the human opportunistic pathogen Pseu
119 Deletion of the dgcP gene in P. savastanoi pv. savastanoi NCPPB 3335 and P. aeruginosa PAK reduced
120 aused by the bacteria Pseudomonas savastanoi pv. savastanoi (Psv) on the epiphytic and endophytic fun
121 e olive tree pathogen Pseudomonas savastanoi pv. savastanoi is dependent on the integrity of its GGDE
123 Both Sanger sequencing of 50 P. syringae pv. atrofaciens mutant clones for each genotype and popu
126 phaseolicola, and pPMA4326A from P. syringae pv. maculicola encoded a type IVA T4SS (VirB-VirD4 conju
127 owth of the universally virulent P. syringae pv. maculicola ES4326 among more than 100 Arabidopsis ec
130 P. syringae pv. syringae 61 and P. syringae pv. phaseolicola 1448A restored HR elicitation and patho
131 report on the genome sequence of P. syringae pv. phaseolicola isolate 1448A, which encodes 5,353 open
133 ngae pv. syringae, pPh1448B from P. syringae pv. phaseolicola, and pPMA4326A from P. syringae pv. mac
136 P. syringae pathovars, hrpP from P. syringae pv. syringae 61 and P. syringae pv. phaseolicola 1448A r
137 g a plasmid-encoded T3SS and the P. syringae pv. syringae 61 effector gene hopA1 increased in planta
140 Here we characterize a gene in P. syringae pv. syringae B728a and P. syringae pv. tomato DC3000, te
141 e supporting the hypothesis that P. syringae pv. syringae B728a produces both of these siderophores.
142 contributions of 4,296 genes in P. syringae pv. syringae B728a were determined by genome-wide fitnes
145 welve PFPs along with pPSR1 from P. syringae pv. syringae, pPh1448B from P. syringae pv. phaseolicola
147 ana did not confer resistance to P. syringae pv. tabaci (Pta) expressing avrPto or avrPtoB, but recog
149 fect was suppressed by wild-type P. syringae pv. tabaci and P. fluorescens heterologously expressing
150 cens, a TTSS-deficient mutant of P. syringae pv. tabaci, or flg22 (a flagellin-derived peptide elicit
152 AD(P) leaking into the ECC after P. syringae pv. tobacco DC3000/avrRpt2 infection is sufficient for i
153 y related to the tomato pathogen P. syringae pv. tomato (Pto), including strains isolated from snowpa
154 ficantly when co-inoculated with P. syringae pv. tomato but not when co-inoculated with a type III se
156 not prevent the HR activated by P. syringae pv. tomato DC3000 + avrB, avrRpm1, avrRps4 or avrRpt2, b
157 and related T3SS substrates for P. syringae pv. tomato DC3000 and three other sequenced strains.
159 d to test the ability of several P. syringae pv. tomato DC3000 effectors for their ability to suppres
160 ated with the Hrp T3SS system in P. syringae pv. tomato DC3000 have predicted lytic transglycosylase
163 variance of basal resistance to P. syringae pv. tomato DC3000 in the Col-0 x Fl-1 F(2) population.
165 the loss of swarming motility of P. syringae pv. tomato DC3000 on medium containing a low percentage
167 repertoire of the model pathogen P. syringae pv. tomato DC3000 were deleted to produce polymutant DC3
168 ogenetically divergent pathovar, P. syringae pv. tomato DC3000, revealed a strong degree of conservat
169 syringae pv. syringae B728a and P. syringae pv. tomato DC3000, termed phcA, that has homology to a f
172 1-1, hopS1, and hopS2 operons in P. syringae pv. tomato DC3000; these operons encode three homologous
174 a virulence protein by promoting P. syringae pv. tomato growth and enhancing symptoms associated with
178 ine family transporter (BCCT) in P. syringae pv. tomato strain DC3000 that mediates the transport of
179 In this study, we found that P. syringae pv. tomato strain DC3000 was distinct from most bacteria
180 hat were susceptible to virulent P. syringae pv. tomato strain DC3000, but resistant to DC3000 expres
181 efined 29 type III proteins from P. syringae pv. tomato, and 19 from P. syringae pv. phaseolicola rac
183 The introduction of Pseudomonas syringae pv. actinidiae (Psa) severely damaged the New Zealand ki
186 the bacterial pathogen Pseudomonas syringae pv. maculicola (Pma) ES4326 and activation of SA synthes
187 nced resistance to both Pseudomonas syringae pv. maculicola (Psm) ES4326 and Hyaloperonospora parasit
190 s locally infected with Pseudomonas syringae pv. maculicola Whole transcriptome shotgun sequencing an
192 real bacterial pathogen Pseudomonas syringae pv. oryzae, transgenic EFR wheat lines had reduced lesio
193 from the bean pathogen Pseudomonas syringae pv. phaseolicola (Pph) is driven by exposure to the stre
195 with the bean pathogen Pseudomonas syringae pv. phaseolicola illustrate how exposure to resistance m
196 opsis is a non-host for Pseudomonas syringae pv. phaseolicola NPS3121 (Pph), a bacterial pathogen of
197 rming enzyme (EFE) from Pseudomonas syringae pv. phaseolicola PK2 is a member of the mononuclear nonh
198 with the plant pathogen Pseudomonas syringae pv. phaseolicola where isolates that have lost the genom
201 n effector protein from Pseudomonas syringae pv. pisi, triggers RPS4-dependent immunity in resistant
202 ete hrp/hrc region from Pseudomonas syringae pv. syringae 61 into the genome of the soil bacterium Ps
203 ated as a phytotoxin by Pseudomonas syringae pv. syringae B301D contains a 4-Cl-L-Thr-9 moiety where
204 ete genomic sequence of Pseudomonas syringae pv. syringae B728a (Pss B728a) has been determined and i
209 transcript profiles of Pseudomonas syringae pv. syringae B728a support a model in which leaf surface
210 surfactant produced by Pseudomonas syringae pv. syringae B728a that was not detected by traditional
213 in and syringopeptin by Pseudomonas syringae pv. syringae is controlled by the regulatory genes salA
216 lowing inoculation with Pseudomonas syringae pv. tabaci carrying avrPto, aconitase-silenced N. bentha
219 mplement recognition of Pseudomonas syringae pv. tomato (Pst) bacteria expressing either avrPto or av
220 (COR) toxin produced by Pseudomonas syringae pv. tomato (Pst) DC3000 functions to overcome plant immu
221 ility of Arabidopsis to Pseudomonas syringae pv. tomato (Pst) DC3000 independently of the phyB/PIF th
222 the bacterial pathogen Pseudomonas syringae pv. tomato (Pst) DC3000, including increased disease res
223 is hyper-susceptible to Pseudomonas syringae pv. tomato (Pst) DC3000, while Arabidopsis lines overexp
227 lent bacterial pathogen Pseudomonas syringae pv. tomato (Pst) DC3000/avrRpt2, and renders plants susc
229 the bacterial pathogen Pseudomonas syringae pv. tomato (Pst) in the cultivated tomato, Lycopersicon
230 retion system (TTSS) of Pseudomonas syringae pv. tomato (Pst) injects into the plant cell effector pr
231 The bacterial pathogen Pseudomonas syringae pv. tomato (Pst) strain DC3000 infects tomato and Arabid
232 the bacterial pathogen Pseudomonas syringae pv. tomato (Pst), contains two MAMPs, flg22 and flgII-28
241 acterial plant pathogen Pseudomonas syringae pv. tomato DC3000 (DC3000) causes disease in Arabidopsis
242 The plant pathogen Pseudomonas syringae pv. tomato DC3000 (DC3000) is found in a wide variety of
244 sigma factor encoded by Pseudomonas syringae pv. tomato DC3000 (DC3000), a plant pathogen that is lik
245 on after infection with Pseudomonas syringae pv. tomato DC3000 (Pst DC3000), demonstrating that the t
246 st avirulent strains of Pseudomonas syringae pv. tomato DC3000 (Pst) carrying AvrRpt2, AvrB, or AvrPp
248 g22 or inoculation with Pseudomonas syringae pv. tomato DC3000 (PstDC3000) hrcC mutant, which is defi
251 the biotrophic bacteria Pseudomonas syringae pv. tomato DC3000 and for susceptibility to the necrotro
253 stance to the bacterium Pseudomonas syringae pv. tomato DC3000 and the fungal pathogen Botrytis ciner
254 ll as non-host pathogen Pseudomonas syringae pv. tomato DC3000 and the general elicitor cryptogein-in
255 the virulent bacterium Pseudomonas syringae pv. tomato DC3000 and the necrotrophic fungus Botrytis c
256 to "mine" the genome of Pseudomonas syringae pv. tomato DC3000 for the metabolic potential to biosynt
258 fection with pathogenic Pseudomonas syringae pv. tomato DC3000 lacking hopQ1-1 [PtoDC3000(DeltahQ)] w
259 The bacterial pathogen Pseudomonas syringae pv. tomato DC3000 must detoxify plant-produced hydrogen
260 odel plant pathosystem, Pseudomonas syringae pv. tomato DC3000 on tomato and Arabidopsis thaliana hos
261 ut not to the bacterium Pseudomonas syringae pv. tomato DC3000 or to the oomycete Hyaloperonospora ar
262 signalling, for example Pseudomonas syringae pv. tomato DC3000 produces coronatine (COR), a jasmonic
264 how the plant pathogen Pseudomonas syringae pv. tomato DC3000 responds to iron limitation and have f
265 The bacterial pathogen Pseudomonas syringae pv. tomato DC3000 suppresses the two-tiered plant innate
267 nse, the plant pathogen Pseudomonas syringae pv. tomato DC3000 uses the virulence factor coronatine t
270 ne were challenged with Pseudomonas syringae pv. tomato DC3000, which is a bacterial pathogen of bras
275 esistance to strains of Pseudomonas syringae pv. tomato expressing AvrRpt2 and Ralstonia pseudosolana
276 in tomato to strains of Pseudomonas syringae pv. tomato expressing the (a)virulence proteins AvrPto o
277 eraction of tomato with Pseudomonas syringae pv. tomato is an established model system for understand
278 ersicum), resistance to Pseudomonas syringae pv. tomato is elicited by the interaction of the host Pt
279 tor protein AvrPto from Pseudomonas syringae pv. tomato is secreted into plant cells where it promote
280 nomical relevant tomato-Pseudomonas syringae pv. tomato pathosystem is widely used to explore and und
283 ity to the hemibiotroph Pseudomonas syringae pv. tomato strain DC3000 (Pto DC3000) was attenuated in
284 cted a mutant screen of Pseudomonas syringae pv. tomato strain DC3000 to identify genes that contribu
285 se has been cloned from Pseudomonas syringae pv. tomato strain DC3000, Pseudomonas putida KT2440, and
286 genic bacteria, such as Pseudomonas syringae pv. tomato strain DC3000, the causative agent of tomato
289 ses upon recognition of Pseudomonas syringae pv. tomato strains expressing the AvrPto or AvrPtoB prot
291 d by the plant pathogen Pseudomonas syringae pv. tomato, has a carboxy-terminal domain that is an E3
292 enhanced resistance to Pseudomonas syringae pv. tomato, which is consistent with a previous study sh
298 t the bacterial pathogen Pseudomonas syringe pv. tomato (Pst) DC3000 in the salicylic acid (SA)- and
299 t the wheat pathogen Xanthomonas translucens pv. undulosa elevates expression of the host gene encodi
300 resistance to the pathogen Xanthomonasoryzae pv. oryzae (Xoo), suggesting the presence of a related d