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1 e was sequenced from 471 pharyngitis and 127 pyogenic and blood isolates recovered from 598 patients
2 controls competence in a parallel way in all pyogenic and bovis streptococci.
3        Alleles shared among the pharyngitis, pyogenic, and blood samples were identified in throat is
4  circuit linked to the sigX genes of mutans, pyogenic, and bovis streptococci that uses a novel small
5 ellulitis (OR = 1.75; CI, 1.1-2.8; P = .02), pyogenic arthritis (OR = 4.2; CI, 1.8-9.6; P = .001), di
6  < 0.001), bacterial meningitis (P < 0.001), pyogenic arthritis (P < 0.001), internal organ abscess (
7 mmon features of human PAPA syndrome such as pyogenic arthritis and skin inflammation were not recapi
8 sis of the synovium in patients with chronic pyogenic arthritis identified dramatic neovascularizatio
9 al onset multisystemic inflammatory disease; pyogenic arthritis pyoderma gangrenosum and acne; Muckle
10 9 disease outcomes (gout, gouty arthropathy, pyogenic arthritis, essential hypertension, coronary ath
11 ts with an autosomal dominant disease called pyogenic arthritis, pyoderma gangrenosum, and acne (PAPA
12 utations were shown to cause the syndrome of pyogenic arthritis, pyoderma gangrenosum, and acne (PAPA
13                                              Pyogenic Arthritis, Pyoderma Gangrenosum, and Acne Syndr
14 and tapetoretinal degeneration (MRST); and a pyogenic arthritis, pyoderma gangrenosum, and acne syndr
15 mic manifestations, skin cellulitis/abscess, pyogenic arthritis, tuberculosis, longer hospital stays,
16 s arise following parenchymal infection with pyogenic bacteria and are typified by inflammation and e
17 long thought to be selectively vulnerable to pyogenic bacteria, but also have a high risk of hypoxemi
18 rm in response to a parenchymal infection by pyogenic bacteria, with Staphylococcus aureus representi
19  intra-macrophagic infections, interleukin-6 pyogenic bacterial diseases and interleukin-17A/F mucocu
20  mutations in the TIR-MyD88 pathway underlie pyogenic bacterial diseases in childhood.
21 ad to severe immunodeficiency in response to pyogenic bacterial infection during childhood, little is
22 ogens is essential for successful control of pyogenic bacterial infection.
23 cause peripheral neutropenia underlying mild pyogenic bacterial infections.
24 mia (SAB) is uniquely characterized by focal pyogenic complications that might not be apparent clinic
25                                    A case of pyogenic flexor tenosynovitis associated with C. cellula
26 lar, the mutant mice exhibited a substantial pyogenic form of the granulomatous response compared wit
27 al dystopia (3 cases), trichiasis (2 cases), pyogenic granuloma (2 cases), eyelid margin nodule (1 ca
28 pared for: 1) I-RL (n = 14), 2) peri-implant pyogenic granuloma (I-PG) (n = 5), 3) peri-implant perip
29  = 9), 4) T-RL (n = 44), 5) tooth-associated pyogenic granuloma (T-PG) (n = 21), and 6) tooth-associa
30 his is the first case in the literature of a pyogenic granuloma associated with a routine guided tiss
31 t dislocation in the Evicel group, 1 case of pyogenic granuloma in the Tisseel group, and no complica
32             Groesser et al. demonstrate that pyogenic granuloma is a RAS pathway-driven tumor.
33                                            A pyogenic granuloma is an exuberant growth of granulation
34 ried for case reports that included the term pyogenic granuloma or lobular capillary hemangioma.
35         This case is interesting because the pyogenic granuloma was found associated with guided tiss
36 ncluded cyst formation, infection, chemosis, pyogenic granuloma, and corneal abrasion.
37 on average and included tearing, discomfort, pyogenic granuloma, and dacryocystitis.
38      Reviews of conjunctival myxoma, corneal pyogenic granuloma, and limbal juvenile xanthogranuloma
39 aluation of the tissue was consistent with a pyogenic granuloma.
40                                    Cutaneous pyogenic granulomas (PGs) are commonly encountered, beni
41                                              Pyogenic granulomas generally are soft, painless, and de
42 s gingival lesions that clinically resembled pyogenic granulomas or periodontal abscesses, but with a
43 ntrol children, suggests that ocular surface pyogenic granulomas respond to topical timolol treatment
44 cutive children with acquired ocular surface pyogenic granulomas treated at Boston Children's Hospita
45                                              Pyogenic granulomas, acquired vascular lesions, form on
46  angiogenic response such as in psoriasis or pyogenic granulomas, the proliferating ECs markedly over
47 s group (348 episodes; 40%), followed by the Pyogenic group (215; 25%).
48 is, and species of the bovis, salivarius and pyogenic groups of streptococci, the pheromone XIP is se
49 itis/vertebral body osteomyelitis simulating pyogenic infection occur, sometimes with relative disc s
50 have a protective role in murine immunity to pyogenic infection.
51         Most M1 GAS subclones recovered from pyogenic infections and blood were abundantly represente
52 ean of 9.8 months before their recovery from pyogenic infections and blood, which indicates that sele
53 rder of phagocyte function, characterized by pyogenic infections and granuloma formation caused by de
54 se (CGD), an immunodeficiency with recurrent pyogenic infections and granulomatous inflammation, resu
55 ciated with increased susceptibility to both pyogenic infections and to inflammatory disorders.
56 itment, mediated by beta2 integrins, combats pyogenic infections but also plays a key role in ischemi
57                                          Non-pyogenic infections of the spine are most frequently cau
58          Most patients die in childhood from pyogenic infections or an unusual lymphoma-like conditio
59 erican male presenting with severe recurrent pyogenic infections was found to have C2 deficiency (C2D
60 s a rare disorder characterized by recurrent pyogenic infections, defective neutrophil chemotaxis and
61 oning from early observations of superficial pyogenic infections.
62           However, the relation of JPDD with pyogenic liver abscess (PLA) has not been reported.
63 ata suggesting that patients who experienced pyogenic liver abscess (PLA) have higher CRC incidence r
64        Although endophthalmitis secondary to pyogenic liver abscess (PLA) is becoming a globally emer
65 train, NTUH-K2044, from a community-acquired pyogenic liver abscess (PLA) patient.
66                                            A pyogenic liver abscess (PLA) represents a pus-filled cav
67  the most common infectious source of EKE is pyogenic liver abscess (PLA).
68                                     Although pyogenic liver abscess (PPLA) fatalities are decreasing
69 ng the viral hepatitides) include amebic and pyogenic liver abscess and cholangitis.
70 siella pneumoniae causing community-acquired pyogenic liver abscess complicated with metastatic menin
71      An emerging population of patients with pyogenic liver abscess includes those with complications
72         In contrast to amebic liver abscess, pyogenic liver abscess is associated with greater morbid
73      The overall prognosis for patients with pyogenic liver abscess is improving, although poor outco
74                                              Pyogenic liver abscess is most often cryptogenic in orig
75                                              Pyogenic liver abscess was predominantly managed by surg
76                                              Pyogenic liver abscess, although commonly occurring in p
77 ed by the same symptoms and signs as classic pyogenic liver abscess, but further distinguished by the
78  that of monomicrobial Klebsiella pneumoniae pyogenic liver abscess, characterized by the same sympto
79 ncomitant human immunodeficiency virus/AIDS, pyogenic liver abscess, infectious endocarditis, cirrhos
80 phasized that the prognosis of patients with pyogenic liver abscess, who do not have underlying comor
81 leading to a new generation of patients with pyogenic liver abscess.
82             As of 1999, the vast majority of pyogenic liver abscesses should be approached therapeuti
83                              Between NTM and pyogenic lymphadenitis, the model achieved an AUC of 90%
84 s nitric oxide (.NO) in host defense against pyogenic microorganisms is unclear, and the actual inter
85 his article describes the pathophysiology of pyogenic MSK infections, including the route of contamin
86 fly review standards of treatment of various pyogenic MSK infections.
87    The type II ComRS system is shared by the pyogenic, mutans, and bovis streptococci, including the
88 ry episodes lead to accumulation of sterile, pyogenic, neutrophil-rich material within the affected j
89               The model classifying nodes as pyogenic, NTM, reactive, or proliferative lymphadenopath
90         Liver abscess is typically either of pyogenic or amebic origin.
91 e group's charter and its first guideline on pyogenic osteomyelitis.
92 roject, to construct clinical guidelines for pyogenic osteomyelitis.
93                Brain abscesses result from a pyogenic parenchymal infection commonly initiated by Gra
94  Common comorbidities with tuberculosis were pyogenic pneumonia in 26 patients (33%) and anaemia in 1
95 ison of MR images of patients diagnosed with pyogenic spondylodiscitis and tuberculous spondylodiscit
96 erise the evolving epidemiological trends of pyogenic spondylodiscitis in Germany, and concurrently e
97                   Characteristic features of pyogenic spondylodiscitis include: involvement of the lu
98 scitis is the commonest spine infection, and pyogenic spondylodiscitis is the most common subtype.
99 ish between tuberculous spondylodiscitis and pyogenic spondylodiscitis on MR images.
100 rentiate between features of tuberculous and pyogenic spondylodiscitis on MR images.
101                                              Pyogenic spondylodiscitis presents significant diagnosti
102 nts with confirmed spondylodiscitis (18 with pyogenic spondylodiscitis, and 16 with tuberculous spond
103  rates, and length of stay, in patients with pyogenic spondylodiscitis.
104 servative versus early surgical treatment of pyogenic spondylodiscitis.
105  is the most common infectious agent causing pyogenic spondylodiscitis.
106 d autoinflammatory syndrome characterized by pyogenic sterile arthritis and less frequently accompani
107  disease, most notably in the PAPA syndrome: pyogenic sterile arthritis, pyoderma gangrenosum, and ac
108                                              Pyogenic sterile arthritis, pyoderma gangrenosum, and ac
109 bulinemia D with periodic fever syndrome and pyogenic sterile arthritis, pyoderma gangrenosum, and ac
110                               PAPA syndrome (pyogenic sterile arthritis, pyoderma gangrenosum, and ac
111 r-X (fasX) were identified in four different pyogenic streptococcal species.
112 te proteins involved in haem uptake found in pyogenic streptococci and Clostridium novyi.
113  required for genetic transformation, yet no pyogenic streptococci are known to develop competence.
114 he proportion of impetigo lesions containing pyogenic streptococci declined following MDA.
115  between arms) but the proportion containing pyogenic streptococci fell significantly (63% vs 23%, P
116                                Patients with pyogenic vertebral osteomyelitis may have an increase in

 
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