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1 a and was blocked by the NF-kappaB inhibitor pyrrolidinedithiocarbamate.
2 rophages, and these effects are prevented by pyrrolidinedithiocarbamate.
3 teine, dimethyl- and tetramethylthiourea, or pyrrolidinedithiocarbamate abrogated the increase in mRN
4  Herein, we demonstrate that the antioxidant pyrrolidinedithiocarbamate activates cAMP-dependent prot
5      CMV-UV-stimulated IL-6 was inhibited by pyrrolidinedithiocarbamate (an inhibitor of the transcri
6 stimulation of ICAM-1 could be suppressed by pyrrolidinedithiocarbamate, an inhibitor of NF-kappaB ac
7 as signaling agents in HPV, the antioxidants pyrrolidinedithiocarbamate and ebselen and the Cu,Zn sup
8                             The antioxidants pyrrolidinedithiocarbamate and N-acetyl-l-cysteine atten
9                              The antioxidant pyrrolidinedithiocarbamate and the proteasome inhibitor
10 ated HASMCs was inhibited by the antioxidant pyrrolidinedithiocarbamate and the serine protease inhib
11        Herein, we show that the antioxidants pyrrolidinedithiocarbamate and vitamin E induce apoptosi
12                              Similar to NAC, pyrrolidinedithiocarbamate, another antioxidant, was als
13  chemical inhibitors such as lactacystin and pyrrolidinedithiocarbamate as well as with an I kappa B
14 um-sulfur bonds in [Cm(pydtc)(4)](-) (pydtc, pyrrolidinedithiocarbamate) at high pressures (up to 11
15          The NF-kappa B inhibitors MG132 and pyrrolidinedithiocarbamate efficiently inhibited the ind
16 ulation of NF-kappa B by the immunomodulator pyrrolidinedithiocarbamate enhanced the cytopathogenicit
17 NF-kappaB inhibitors Bay11-7082 and ammonium pyrrolidinedithiocarbamate inhibited an early postentry
18  inhibited by the commonly used antioxidants pyrrolidinedithiocarbamate (PDTC) and butylated hydroxya
19                  Although the ROS scavengers pyrrolidinedithiocarbamate (PDTC) and N-acetylcysteine (
20                                              Pyrrolidinedithiocarbamate (PDTC) and N-acetylcysteine (
21    We report here that the known antioxidant pyrrolidinedithiocarbamate (PDTC) leads to time and dose
22 kappaB (IkappaB)-alpha, or with low doses of pyrrolidinedithiocarbamate (PDTC) selectively inhibited
23         Preincubation of cells with 10(-7) M pyrrolidinedithiocarbamate (PDTC), an inhibitor of nucle
24                             The antioxidants pyrrolidinedithiocarbamate (PDTC), N-acetylcysteine, 6-h
25                      The NFkappaB inhibitors pyrrolidinedithiocarbamate (PDTC; 100 microM), caffeic a
26      We show that inhibition of NF-kappaB by pyrrolidinedithiocarbamate prevents poly(I-C) + IFN-gamm
27  by the antioxidants N-acetyl-L-cysteine and pyrrolidinedithiocarbamate, suggesting the involvement o