ライフサイエンスコーパス: rabies virus

1 ho would otherwise succumb to infection with rabies virus.

2 d to identify potent small molecules against rabies virus.

3 Mononegavirales, including measles virus and rabies virus.

4 cell-type-specific promoters, and a modified rabies virus.

5 iral infection involving an agent other than rabies virus.

6 ed each area's monosynaptic inputs using the rabies virus.

7 ear, and hence avoid an epizootic fadeout of rabies virus.

8 of Cre-dependent helper virus and a modified rabies virus.

9 etrograde spread of a genetically modifiable rabies virus.

10 lary ventrolateral column were infected with rabies virus.

11 als were given an intramuscular challenge of rabies virus.

12 e exposed, through aerosol, to 3 variants of rabies virus.

13 the behavioural changes in hosts infected by rabies virus.

14 city, is an important restriction factor for rabies virus.

15 dangerous zoonotic pathogens, like Ebola or rabies virus.

16 , such as HSV, Zika virus, dengue virus, and rabies virus.

17 with high homology (89 to 93%) to clade I of rabies viruses.

18 rences between vaccine strains and wild-type rabies viruses.

19 re less susceptible to lethal infection with rabies viruses.

20 ealed using Cre-conditional pseudorabies and rabies viruses.

21 4 experimental cross-species inoculations of rabies virus, a widespread zoonosis which in nature exhi

22 nd to be an important restriction factor for rabies virus, acting directly or indirectly against vira

23 mples; 2) showing how cross species jumps of rabies virus among bat populations can be readily identi

24 against heterologous CDV strains.IMPORTANCE Rabies virus and canine distemper virus (CDV) cause high

25 own that a Th1-biased seroconversion to both rabies virus and MARV glycoproteins (GPs) is beneficial

26 hysiological recording system, combined with rabies virus and optogenetic cell-type identification, t

27 All bats survived exposure to aerosolized rabies virus and produced rabies neutralizing antibody.

28 cles of two recombinant rhabdoviral vectors, rabies virus and vesicular stomatitis virus (VSV), expre

29 including Ebola, Marburg, Nipah, Hendra, and rabies viruses and severe acute respiratory syndrome-cor

30 s using homologous (inactivated Pitman Moore rabies virus) and heterologous (inactivated vesicular st

31 members of NNS RNA viruses, such as measles, rabies virus, and Ebola virus.

32 , lymphocytic choriomeningitis virus (LCMV), rabies virus, and Lassa virus.

33 s, including Ebola virus, Lassa virus, LCMV, rabies virus, and Marburg virus, which was substituted f

34 accinated subjects developed protective anti-rabies virus antibody titers.

35 Rabies virus antigen was detected in archived autopsy br

36 s, rabies virus neutralizing antibodies, and rabies virus antigens were conducted on available specim

37 n gene do not suffice to render a pathogenic rabies virus apathogenic and are not a viable and safe a

38 By using orthogonal tract tracing and rabies virus approaches in transgenic SVZ-lineage-tracin

39 continued the investigation into aerosolized rabies virus as a potential route of transmission.

40 We therefore applied a genetically modified rabies virus as a retrograde tracer to fill the dendrite

41 We used a G-deleted rabies virus as a retrograde tracer to label CG neurons

42 Using rabies virus as a retrograde transneuronal tracer in mic

43 Here we use rabies virus as a retrograde transynaptic tracer to show

44 Finally, using rabies-virus-assisted monosynaptic tracing, we show that

45 Over the last decade, rabies viruses associated with the silver-haired bat (SH

46 nlethal infections, is necessary to maintain rabies virus at levels consistent with field observation

47 e the mouse IFN-gamma gene into a pathogenic rabies virus backbone, SPBN, to produce the recombinant

48 These rabies virus-based approaches permit the linking of conn

49 We used rabies virus-based circuit-mapping tools to reveal the i

50 LORAB1, a recombinant, bivalent, inactivated rabies virus-based EBOV vaccine, in rhesus and cynomolgu

51 use primary visual cortex (V1) with modified rabies virus-based input mapping, we have determined the

52 Here we use a recombinant rabies virus-based method to label a specific type of lo

53 Rabies virus-based monosynaptic tracing has been used to

54 Rabies virus-based retrograde tracing has developed into

55 sults demonstrate an important limitation of rabies virus-based retrograde tracing of sensory neurons

56 Using a monosynaptic rabies virus-based tracing technique, we studied the evo

57 Using a modified monosynaptic rabies virus-based transsynaptic tracing strategy, we sy

58 Using rabies virus-based transsynaptic tracing, we find that a

59 t rabies virus, illustrating that a bivalent rabies virus-based vaccine against CDV induces protectiv

60 Rabies virus-based vectors have been proven to be effici

61 Using a monosynaptic rabies viruses-based transneuronal tracing method combin

62 Rabies-virus-based input mapping indicated that LC-NE ne

63 ht help to protect wild dog populations from rabies virus, but not from canine distemper virus.

64 hrlichia canis, Neospora caninum and perhaps rabies virus, but not with exposure to canine distemper

65 eins of the Ebola, vesicular stomatitis, and rabies viruses by Nedd4 family HECT domain E3 ligases is

66 Here we show that deletion-mutant rabies virus can be specifically targeted to cells local

67 In this study, we demonstrate that rabies virus can infect sensory neurons in the somatosen

68 Recombinant rabies viruses can encode genes of interest for labeling

69 r stomatitis virus (VSV), like its relative, rabies virus, can cause neuropathy in mice if it enters

70 eported that a viral protein (G-protein from rabies virus) capable of interfering with protein-protei

71 hereas all animals that received recombinant rabies viruses carrying only the CDV attachment protein

72 ts immunized twice with a mix of recombinant rabies viruses carrying the CDV fusion and attachment gl

73 RNA viruses, a group that includes Ebola and rabies viruses, catalyze RNA-dependent RNA polymerizatio

74 such as vesicular stomatitis virus (VSV) and rabies virus, catalyzes the transfer of 5'-phospho-RNA (

75 The rabies virus causes a fatal encephalitis and can be tran

76 nstrate that the clearance of the attenuated rabies virus CVS-F3 from the CNS is an exception; increa

77 ) but not during clearance of the attenuated rabies virus CVS-F3 from the CNS tissues.

78 Here we combine rabies-virus-dependent retrograde mono-trans-synaptic la

79 s backbone, SPBN, to produce the recombinant rabies virus designated SPBNgamma.

80 DFA) detection remains the gold standard for rabies virus diagnostics.

81 transfection of neurons with deletion-mutant rabies virus encoding channelrhodopsin-2, and used this

82 We have constructed a deletion-mutant rabies virus encoding EGFP and find it to be an excellen

83 and the membrane proteins thought to mediate rabies virus endocytosis (neural cell adhesion molecule,

84 r spinal cord, which are the known sites for rabies virus entry into the CNS, and enhancements in bra

85 copy, we demonstrated that pseudotyping with rabies virus envelope glycoprotein (RV-G) enabled the ax

86 tion of pseudotyped and genetically modified rabies virus evidence was found for direct synaptic inpu

87 Rabies virus exhibits a small genome that encodes a limi

88 These new rabies viruses express useful neuroscience tools, includ

89 retrogradely labeled following injections of rabies virus expressing enhanced green fluorescent prote

90 Monosynaptically restricted rabies virus facilitates the anatomical investigation of

91 Rabies virus found worldwide and prevalent throughout th

92 e used retrograde transneuronal transport of rabies virus from single muscles of rhesus monkeys to id

93 ng the transneuronal retrograde transport of rabies virus from the diaphragm with the immunohistochem

94 by the retrograde transneuronal transport of rabies virus from the diaphragm, were immunopositive for

95 e used retrograde transneuronal transport of rabies virus from the rat kidney to identify the areas o

96 rabies viral vectors, recovery of G-deleted rabies viruses from cDNA, amplification of the viruses,

97 Incorporation of IFN-gamma into the rabies virus genome highly attenuated the virus.

98 ding to the first 19 nucleotides (nt) of the rabies virus genome, we demonstrate that L alone initiat

99 Using sequences encoding 78% of the rabies virus genome, we explored the extent, repeatabili

100 f a prophylactic mRNA-based vaccine encoding rabies virus glycoprotein (CV7201).

101 Seroconversion against rabies virus glycoprotein (G) and SHIV(89.6P) Env was de

102 (VSV) to encode a fluorophore and either the rabies virus glycoprotein (RABV-G) or its own glycoprote

103 re we show that a short peptide derived from rabies virus glycoprotein (RVG) enables the transvascula

104 To this end, a SAM vaccine encoding the rabies virus glycoprotein (RVG) was used.

105 etylcholine receptor (AchR)-binding peptide [rabies virus glycoprotein (RVG)-9R] effectively suppress

106 ibution studies, nanoparticles modified with rabies virus glycoprotein (RVG29) were loaded with small

107 lly in an anterograde direction, whereas the rabies virus glycoprotein gave a specifically retrograde

108 The neurotoxin-like region of the rabies virus glycoprotein inhibited acetylcholine respon

109 Furthermore, as this p137 RNA is fused to a rabies virus glycoprotein peptide that facilitates deliv

110 inding of this study is that a region in the rabies virus glycoprotein, with homologies to snake toxi

111 te at a molecular level its interaction with rabies virus glycoprotein.

112 vitro, as did full length ectodomain of the rabies virus glycoprotein.

113 Naturally occurring herpes and rabies viruses have been used for transneuronal circuit

114 Although modified rabies viruses have emerged as a powerful tool for traci

115 ew, most notably the alpha herpesviruses and rabies virus, have evolved to enter the NS efficiently a

116 ic transgenic complementation of recombinant rabies virus holds great promise for obtaining cellular-

117 results can explain observed constraints on rabies virus host shifts, describe a previously unrecogn

118 respective roles of ecology and evolution in Rabies virus host shifts.

119 rus infection were attributed to aerosolized rabies virus; however, little work has been done to addr

120 complete labeling of neurons with a modified rabies virus identified these neurons as pyramidal cells

121 animals developed protective titers against rabies virus, illustrating that a bivalent rabies virus-

122 haracterized, was demonstrated to neutralize rabies virus in a fluorescent antibody virus neutralizat

123 Using pseudotyped rabies virus in a transgenic Gpr151-Cre mouse line, mono

124 abies virus (RABV) has been used as a street rabies virus in laboratory investigations.

125 stence mechanisms of vampire-bat-transmitted rabies virus in Latin America, we use data from a spatia

126 ere validated by efficacy against infectious rabies virus in mammalian cell culture.

127 We used transneuronal transport of rabies virus in monkeys and rats to identify regions of

128 erized vaccine-induced cellular responses to rabies virus in naive adult volunteers.

129 a set of gene sequences from an epidemic of rabies virus in North American raccoons.

130 performed retrograde injections of modified rabies virus in the visual cortex and pulvinar of the Lo

131 sceptible to infection with EnvA-pseudotyped rabies virus in tumor virus A receptor transgenic mice,

132 fter restimulation of PBMCs with inactivated rabies virus in vaccinated, but not in unvaccinated, ind

133 in the raphe nuclei were also infected with rabies virus, indicating that midline cells utilizing mu

134 Rabies virus induces drastic behaviour modifications in

135 The rabies viruses infecting the donor and the deceased kidn

136 essing cells, allowing us to control initial rabies virus infection and subsequent monosynaptic retro

137 rs across the BBB is critical to surviving a rabies virus infection and that HPA axis activity may in

138 es involved in innate immune response during rabies virus infection and that the M protein of wild is

139 Using rabies virus infection as an example, we discuss the adv

140 se neurotropic viruses in neuronal cells and rabies virus infection in mouse brains.

141 Between 1956 and 1977, 4 human cases of rabies virus infection were attributed to aerosolized ra

142 IV5-G protected mice as late as 6 days after rabies virus infection.

143 sing vaccine for prevention and treatment of rabies virus infection.

144 tein (G) of rabies (PIV5-G) as a therapy for rabies virus infection: we have found that PIV5-G protec

145 ABV is associated with the majority of human rabies virus infections in the United States and Canada.

146 We report here on experiments that use rabies virus injections into V4 to retrogradely label mo

147 We injected rabies virus into lobules VB-VIIIB of the vermis and use

148 his relay directly, we injected transynaptic rabies virus into several extrastriate visual areas.

149 Glycoprotein-deleted (DeltaG) rabies virus is a powerful tool for studies of neural ci

150 n and that the M protein of wild isolates of rabies virus is a viral immune-modulatory factor playing

151 Rabies virus is an important zoonotic pathogen.

152 Based on rabies virus, it is genetically targetable, allows high-

153 rapid production of murine IFN-gamma by the rabies virus itself would induce a more robust antiviral

154 Vampire bat rabies virus lineages associated with antigenic variant

155 iciently long incubation periods, allows for rabies virus maintenance.

156 e order Mononegavirales, which also includes rabies virus, measles virus, and respiratory syncytial v

157 virus (VSV) and related rhabdoviruses (e.g., rabies virus) mediate both cell attachment and membrane

158 Using rabies virus -mediated monosynaptic retrograde tracing t

159 In this study, we developed a modified rabies virus-mediated monosynaptic retrograde tracing me

160 Glycoprotein-deleted rabies virus-mediated monosynaptic tracing has become a

161 Here, we use G-deleted rabies virus-mediated monosynaptic tracing to identify i

162 rategy combining retroviral birthdating with rabies virus-mediated putative retrograde trans-synaptic

163 Here we combine rabies virus-mediated retrograde tracing with retroviral

164 The virus is now termed dog rabies virus-Mexico (DRV-Mexico).

165 Using rabies virus monosynaptic tracing, we mapped cocaine-ind

166 ary analyses indicate multiple barriers that Rabies virus must overcome through adaptation.

167 Rabies viruses, negative-strand RNA viruses, infect neur

168 luorescent-focus inhibition test (RFFIT) for rabies virus neutralising antibody measurement.

169 etermine the lowest dose of CV7201 to elicit rabies virus neutralising titres equal to or greater tha

170 organ recipients remained asymptomatic, with rabies virus neutralizing antibodies detected in their s

171 wo arms assessed as percentage of those with rabies virus neutralizing antibodies titers >= 0.5 IU/mL

172 ct rabies virus-specific binding antibodies, rabies virus neutralizing antibodies, and rabies virus a

173 ce immunized with LBNSE-CXCL13 produced more rabies virus-neutralizing antibodies (VNAs) and develope

174 This insight into the architecture of the rabies virus nucleocapsid highlights the surprising stru

175 V is a negative strand RNA virus, similar to rabies virus or Ebola virus, that has a unique mechanism

176 s of a 30-yr data set covering a large-scale rabies virus outbreak among North American raccoons reve

177 Here we show that inactivated rabies virus particles containing the MERS-CoV S1 protei

178 ndidates based on recombinant vaccine strain rabies virus particles, which concurrently display the p

179 jor unanswered research questions related to rabies virus pathogenesis, especially those connecting t

180 Despite its ability to infect all mammals, Rabies virus persists in numerous species-specific cycle

181 outs of the LC8 recognition motif within the rabies virus phosphoprotein (RavP) result in completely

182 Understanding the interactions between rabies virus (RABV) and individual host cell proteins is

183 oviral vaccine vector platforms, recombinant rabies virus (RABV) and recombinant vesicular stomatitis

184 both survival from infection with attenuated rabies virus (RABV) and reduction of neurological sequel

185 rly chemokines, is an important mechanism of rabies virus (RABV) attenuation.

186 Both canine distemper virus (CDV) and rabies virus (RABV) cause lethal disease in wild and dom

187 Rabies virus (RABV) causes a severe and fatal neurologic

188 Rabies virus (RABV) causes fatal encephalitis in more th

189 Rabies virus (RABV) causes rabies disease resulting in >

190 la virus (EBOV) vaccine based on inactivated rabies virus (RABV) containing EBOV glycoprotein (GP) in

191 Our studies demonstrate that wild-type (wt) rabies virus (RABV) does not activate DCs.

192 letion variant of the SAD-B19 vaccine strain rabies virus (RABV) has been the reagent of choice in mo

193 A canine rabies virus (RABV) has been used as a street rabies vir

194 al spread rate, persistence and incidence of rabies virus (RABV) in raccoons (Procyon lotor).

195 Untreated rabies virus (RABV) infection leads to death.

196 sible for vaccine-induced protection against rabies virus (RABV) infections in postexposure settings.

197 Rabies virus (RABV) maintenance in bats is not well unde

198 Rabies virus (RABV) P gene mRNA encodes five in-frame st

199 asome recognizes RNA viruses and its role in rabies virus (RABV) pathogenicity and immunogenicity rem

200 MIP-1alpha in mouse brain further decreased rabies virus (RABV) pathogenicity.

201 Rabies virus (RabV) phospho (P) protein, which is largel

202 The rabies virus (RABV) phosphoprotein P is a multifunctiona

203 Rabies virus (RABV) postexposure prophylaxis (PEP) requi

204 dress this need, we developed an inactivated rabies virus (RABV) that contains the MERS-CoV spike (S)

205 In this study, a recombinant rabies virus (RABV) that expressed murine interleukin-7

206 l infections, we investigated the ability of rabies virus (RABV) to activate DCs.

207 -deficient, and (iii) chemically inactivated rabies virus (RABV) vaccines expressing Zaire Ebola viru

208 the live but not UV-inactivated recombinant rabies virus (RABV) variant TriGAS in mice induces the r

209 ptive chimpanzees to test oral delivery of a rabies virus (RABV) vectored vaccine against Ebola virus

210 Its pathogen, rabies virus (RABV), can utilize its viral proteins, suc

211 ic and sometimes deadly viruses that include rabies virus (RABV), human respiratory syncytial virus (

212 Rabies, caused by rabies virus (RABV), is a fatal encephalitis in humans a

213 Rabies, caused by rabies virus (RABV), is an ancient zoonosis and still a

214 such as vesicular stomatitis virus (VSV) and rabies virus (RABV), possess an unconventional mRNA capp

215 Replication-deficient rabies virus (RABV)-based vaccines induce rapid and pote

216 proteins in the brains of mice infected with rabies virus (RABV).

217 cing the protective immune responses against rabies virus (RABV).

218 loRab1 PBV vaccines and challenged them with rabies virus (RABV).

219 ted GL261 tumor-bearing mice with attenuated rabies virus (RABV).

220 r the RNA-dependent RNA polymerase (RdRP) of rabies virus (RABV).

221 Replication-deficient rabies viruses (RABV) are promising rabies postexposure

222 Attenuated rabies viruses (RABV) are unique tools to study CNS immu

223 ent study, it was found that the recombinant rabies viruses rB2c-K1685A and rB2c-K1829A, carrying mut

224 eurological disease caused by infection with rabies virus, registers throughout recorded history.

225 distinctive antigenic patterns were found in rabies viruses related to rabies in vampire bats in Mexi

226 Recombinant rabies viruses rendered replication-deficient by the del

227 amma directly in the infected tissue reduces rabies virus replication and spread, limiting its pathog

228 tional significance of the components of the rabies virus replication machinery is incomplete.

229 In Latin America, vampire-bat-transmitted rabies virus represents a key example of how such uncert

230 ecific infection of MEC-LII with recombinant rabies virus results in unambiguous labeling of monosyna

231 Retrograde circuit mapping with modified rabies viruses revealed that the On-DSGCs project to the

232 Retrograde monosynaptic tracing with rabies virus reveals that this results in synaptic conta

233 inserted into the genome of the recombinant rabies virus (rRABV) strain LBNSE, and the effect of the

234 To overcome these problems, we used rabies virus (RV) as a retrograde transneuronal tracer.

235 ermeability in mice, whether associated with rabies virus (RV) clearance or CNS autoimmunity, is unal

236 Rabies virus (RV) infection is a fatal nervous system di

237 We injected rabies virus (RV) into selected regions of the cerebella

238 While the glycoprotein (G) of rabies virus (RV) is known to play a predominant role in

239 tudies indicate that the interaction between rabies virus (RV) phosphoprotein and the dynein light ch

240 A major goal in rabies virus (RV) research is to develop a single-dose p

241 Previous comparisons of different rabies virus (RV) strains suggested an inverse relations

242 plementary targeting system for monosynaptic rabies virus (RV) tracing that identifies direct inputs

243 The nonpathogenic phenotype of the live rabies virus (RV) vaccine SPBNGAN is determined by an Ar

244 Although current postexposure prophylaxis rabies virus (RV) vaccines are effective, approximately

245 after a single immunization, live-attenuated rabies virus (RV) vaccines could be particularly useful

246 Rabies virus (RV), a member of the Rhabdoviridae family,

247 Viral vectors based on influenza virus, rabies virus (RV), and vaccinia virus (VV) were used to

248 ng with EnvB pseudotyped lentivirus (LV) and rabies virus (RV), to selectively coinfect ErbB4-express

249 Recombinant rabies virus (RV)-based vectors have demonstrated their

250 Postexposure treatment (PET) of wild-type rabies virus (RV)-infected mice with a live-attenuated t

251 tically from hindlimb extensor muscles using rabies virus (RV).

252 , West Nile virus [WNV], Sindbis virus [SV], rabies virus [RV], and influenza A virus [IAV]) remains

253 Using a data set of hundreds of rabies viruses sampled from 23 North American bat specie

254 Dengue virus, tick-borne encephalitis virus, rabies virus, severe acute respiratory syndrome coronavi

255 vidual viral lineages revealed that although rabies viruses shared consistent three-stage processes o

256 Silver-haired bat rabies virus (SHBRV) infection induces a strong virus-sp

257 Surprisingly, experiments using monosynaptic rabies virus showed that proopiomelanocortin (POMC) and

258 ion, we developed a self-inactivating DeltaG-rabies virus (SiR) that transcriptionally disappears fro

259 Laboratory tests to detect rabies virus-specific binding antibodies, rabies virus n

260 erstand the molecular machinery required for rabies virus spread in the nervous system.

261 an infectious clone of the highly pathogenic rabies virus strain CVS-N2c and replaced its cognate gly

262 mice infected with an attenuated laboratory rabies virus strain.

263 The matrix (M) protein of wild isolates of rabies virus such as Tha (M-Tha) was previously shown to

264 s captured in Trinidad in the 1950s during a rabies virus surveillance program.

265 on in these circuits, we used a monosynaptic rabies virus system to generate brain-wide maps of neuro

266 We used a retrograde trans-synaptic rabies virus system to generate brain-wide maps of the i

267 We used the monosynaptic rabies virus system, in conjunction with mice expressing

268 omplish this, we used a genetically modified rabies virus that acts as a retrograde tracer and fills

269 Using a modified rabies virus that expresses green fluorescent protein, w

270 synaptic infection from the spinal cord with rabies viruses that carry glycoproteins in their envelop

271 Here we generated recombinant inactivated rabies viruses that carry one of the CDV glycoproteins o

272 Using paired cytopathic and noncytopathic rabies viruses that differ by only two amino acids, we i

273 We used a monosynaptic rabies virus to define the circuit's functional connecti

274 We also show that engineering a lethal rabies virus to express IFN-gamma directly in the infect

275 Here, we used transneuronal transport of rabies virus to identify the areas of the primate cerebr

276 e used retrograde transneuronal transport of rabies virus to identify the cortical areas that most di

277 a recently developed tracing system based on rabies virus to overcome all three constraints.

278 ice, recombinant adeno-associated virus, and rabies virus to produce sparse but binary labeling of se

279 nables trans-synaptic spreading of G-deleted rabies viruses to directly connected, presynaptic neuron

280 Using monosynaptically-restricted rabies virus tracing of OPC afferents, we identified ext

281 n Tau(VLW) DGCs, and monosynaptic retrograde rabies virus tracing showed that these cells are disconn

282 Using rabies virus tracing strategies Weissbourd et al. (2014)

283 Combining rabies-virus tracing, optical clearing (CLARITY), and wh

284 We employed cell-type-specific monosynaptic rabies virus tracings to characterize afferent connectio

285 Unlike the 2 previous clusters of rabies virus transmission through solid organ transplant

286 icular stomatitis virus (VSV), a relative of rabies virus transmitted by insect bites, that SCS macro

287 Using a modified rabies virus transsynaptic tracing strategy, we labeled

288 y recipient were consistent with the raccoon rabies virus variant and were more than 99.9% identical

289 Genetic analysis revealed a canine rabies virus variant found only in the patient's home co

290 pulation lives in a country where the canine rabies virus variant is endemic and dog bites are common

291 differences in rabies due to canine and bat rabies virus variants have been noted, but no detailed s

292 differences in the route of viral spread of rabies virus variants in the nervous system, although ce

293 erlying geographic expansions of vampire bat rabies virus (VBRV) in Peru.

294 We have developed an inactivated rabies virus-vectored MARV vaccine (FILORAB3) to protect

295 the safety and immunogenicity of attenuated rabies virus vectors expressing simian-human immunodefic

296 that multiple neurotropic viruses, including rabies virus, vesicular stomatitis virus, Semliki Forest

297 er VLPs, as well as vesicular stomatitis and rabies viruses (VSV and RABV, respectively).

298 Using a genetically modified rabies virus, we demonstrated that molecular layer perfo

299 immunopositive neurons that were infected by rabies virus were significantly less prevalent in the po

300 Rabies viruses whose glycoprotein (G) gene is deleted fr