ライフサイエンスコーパス: radiation damage

1 acts form after injury to the eye, including radiation damage.

2 dD, reflecting its role in the DNA repair of radiation damage.

3 for 'aloof' spectroscopy that largely avoids radiation damage.

4 ructures under native conditions and without radiation damage.

5 hotons, better statistics, and lower overall radiation damage.

6 from hypoxia-reoxygenation, and against UVA radiation damage.

7 is accelerated and/or synchronized following radiation damage.

8 ffract x-rays efficiently while withstanding radiation damage.

9 s limited by the thickness of the window and radiation damage.

10 ons imposed upon synchrotron measurements by radiation damage.

11 ace images are obtained without irreversible radiation damage.

12 with micron-sized X-ray beams the effects of radiation damage.

13 photoelectrons are the predominant source of radiation damage.

14 tive stress, and regulation of recovery from radiation damage.

15 ses the recovery of Dr from ultraviolet (UV) radiation damage.

16 tantially alleviates the growth defect after radiation damage.

17 sample or limiting exposure times to prevent radiation damage.

18 could increase the expression and degree of radiation damage.

19 residue is required for effective repair of radiation damage.

20 RecA-dependent process during recovery from radiation damage.

21 e dimensions with resolution limited only by radiation damage.

22 owed that flavopiridol inhibited repair from radiation damage.

23 tization for several phenotypic endpoints of radiation damage.

24 may have related roles in the toleration of radiation damage.

25 unit, crystal variability and sensitivity to radiation damage.

26 d vessels did not show histological signs of radiation damage.

27 represents a novel defense mechanism against radiation damage.

28 S) that may constitute one-third of ionizing radiation damage.

29 d are defective at the G1/S checkpoint after radiation damage.

30 ATM might interact with c-Abl in response to radiation damage.

31 opment of therapeutic targets for mitigating radiation damage.

32 ulate intestinal regeneration after ionizing radiation damage.

33 the high thermal loads, plasma exposure, and radiation damage.

34 ge with great implications for understanding radiation damage.

35 ional structure influence on galactic cosmic radiation damage.

36 through ROS levels in SMG spheres following radiation damage.

37 ts in spaceflight missions to mitigate space radiation damage.

38 ncreasing dose, a dominant factor mitigating radiation damage.

39 s have been shown to recover, or heal, after radiation damage.

40 e as a radiosensitizer to (18)F-FDG-mediated radiation damage.

41 atomic structure of biological molecules is radiation damage.

42 noninvasive tumor imaging and monitoring of radiation damage.

43 lecular structure determination with limited radiation damage.

44 individual biomolecules without significant radiation damage.

45 important regulator of cellular responses to radiation damage.

46 factors, of which one of the most serious is radiation damage.

47 mall sublineages (one to four cells) without radiation damage.

48 zero electron exposure, before the onset of radiation damage.

49 probe protein interactions while minimizing radiation damage.

50 ucceeded by cryo-crystallography to mitigate radiation damage.

51 have similar bone-protective effects against radiation damage.

52 to predict the role of STGBs in annihilating radiation damage.

53 sten films plays a dominant role in reducing radiation damage.

54 e protein in the droplets and the absence of radiation damage.

55 new materials capable of withstanding severe radiation damage.

56 =O vibrational signatures with no observable radiation damage.

57 with X-rays, including the phase problem and radiation damage.

58 on into materials with significantly reduced radiation damage.

59 igh temperatures, corrosive environments and radiation damage.

60 ue that exploits the susceptibility of NC to radiation damage.

61 he only source of energy for the recovery of radiation-damage.

62 hat they are able to tolerate high levels of radiation damage (218.6 Gy).

63 l understanding of the processes controlling radiation damage accumulation is necessary.

64 he development of agents that can ameliorate radiation damage after exposure to radiation has occurre

65 melanin may also protect cells from ionizing radiation damage, against which C. neoformans is extreme

66 There is no evidence of radiation damage, alteration, recrystallisation or defor

67 into the map during refinement and shows how radiation damage alters scattering from negatively charg

68 luding a pigment that can protect cells from radiation damage and a protein that can concentrate DNA

69 cell death is important for both mitigating radiation damage and alleviating the side effects of ant

70 ystal rotation crystallography by mitigating radiation damage and allowing time-resolved studies with

71 Theoretical simulations of radiation damage and charge transport in DNA depend on a

72 TrpRS II is induced after radiation damage and contains an N-terminal extension si

73 c alloy properties that effectively mitigate radiation damage and control a material's response in ex

74 s ([Formula: see text] 10 K) we suppress the radiation damage and enable the acquisition of reliable

75 e prospect of improving our understanding of radiation damage and fostering mechanistic studies of so

76 y, and atomistic simulation to elucidate how radiation damage and interfacial defects interplay to co

77 onucleus (MN) formation has a strong link to radiation damage and is a common bio-dosimeter for acute

78 FEL structure shows little to no evidence of radiation damage and is more complete than a model deter

79 ues of DJ-1, shows an extreme sensitivity to radiation damage and may be subject to other forms of ox

80 nces between Bijvoet mates despite extensive radiation damage and multi-crystal scaling; the pre-scre

81 emtosecond-duration x-ray pulses to minimize radiation damage and obtained a high-resolution room-tem

82 a viable solution, though many are prone to radiation damage and plagued with temperature instabilit

83 ation, understanding the mechanisms by which radiation damage and repair occur is essential.

84 We investigated the effects of radiation damage and sample preparation on the bacteria

85 somatostatin analogs, both by mitigation of radiation damage and the currently observed reduction of

86 rtant underlying biological processes in the radiation damage and tissue repair process.

87 g sufficiently large crystals that withstand radiation damage and yield high-resolution data at synch

88 d as having an unspecified role in repair of radiation damage and, more specifically, DNA double-stra

89 noise ratio and contrast, as well as minimal radiation damage) and subtomogram averaging (three-dimen

90 ring in vitro culture, exhibit resistance to radiation damage, and demonstrate enhanced proliferative

91 the imaging window, high sensitivity toward radiation damage, and low contrast due to a low Z number

92 ss component) effectively anneal the initial radiation damage, and recover the device efficiency, thu

93 Grand Canyon apatites of differing He date, radiation damage, and U-Th zonation yield a self-consist

94 ing that the in vivo macrophage responses to radiation damage are genetically modified processes.

95 To ensure that potential photoreduction and radiation damage are not responsible for the absence of

96 remely low temperatures where the effects of radiation damage are reduced.

97 a dose of 33 MGy, no signs of X-ray-induced radiation damage are visible in this integral membrane p

98 polar condensed media as well as biological radiation damage arising from dissociative electron atta

99 However, detection of radiation damage artefacts has traditionally proved very

100 ce of reliable structural information due to radiation damage artifacts caused by the intense synchro

101 DNA containing oxidative lesions induced by radiation damage at break termini.

102 on (R(g) ) is 5.2-5.4 nm, after allowing for radiation damage at higher concentrations, and that the

103 in certain unstable structures, a prominent radiation damage at room temperature.

104 Specific radiation damage at RT was observed at disulfide bonds b

105 d requiring the study of specific and global radiation damage at RT.

106 silicate band positions indicate accumulated radiation damage at the nanoscale from prolonged space w

107 physicochemical and chemical stages of early radiation damage at the scale of an entire human genome

108 Here, we examine trends in radiation damage behaviour for families of compounds rel

109 diation, most likely by reducing exposure of radiation-damaged breast cells to stimulating effects of

110 nd avoid aggregation, concentration effects, radiation damage, buffer mismatch and other common probl

111 parameters with experimental observations of radiation damage buildup remains elusive.

112 lineated the cellular response of MR-1 to UV radiation damage by analyzing the transcriptional profil

113 n numerous attempts to reduce the effects of radiation damage by cooling the specimen beyond liquid-n

114 Although sample deformation caused by radiation damage can be partly mitigated by cryogenic pr

115 Lattice defects generated by radiation damage can diffuse to grain boundaries (GBs) a

116 us targeting and phosphorylation in ionizing radiation-damaged cells, whereas UV light-induced 53BP1

117 Ionizing radiation damages chromosomal DNA and activates p53-depe

118 -phosphatidylcholines were most sensitive to radiation damage compared to the ester- and ether-linked

119 rinciple of protein's greater sensitivity to radiation damage compared with that of nucleic acid.

120 23)Ra accumulated in bones and induced zonal radiation damage confined to the bone interface, followe

121 e study of biological samples, provided that radiation damage could be prevented.

122 indirect tumor cell death caused by vascular radiation damage could potentially help clinicians inter

123 ronology and U-Th-Pb dating is the effect of radiation damage, created by alpha-recoils from alpha-de

124 However, radiation damage currently limits the application of SAX

125 present the first evidence that LECs process radiation damage differently to blood lymphocytes.

126 Ionizing radiation damages DNA in several ways, including through

127 While ultraviolet (UV) radiation damages DNA, eliciting the DNA damage response

128 is required for expression of an ultraviolet radiation-damaged DNA binding activity and is disrupted

129 f several factors also involved in repair of radiation-damaged DNA, including the DNA-dependent prote

130 , shorter exposure times, and elimination of radiation damage due to capillary effects significantly

131 lar dynamics was employed to investigate the radiation damage due to collision cascades in LiAlO(2) a

132 e fibre layer following occipital lobe/optic radiation damage due to stroke.

133 n particular biological tissues, and reduced radiation damage due to the limited photon energies.

134 -Mn and Mn-Ca distances are less affected by radiation damage due to the their heavy masses, while on

135 anism of FIB milling damage is distinct from radiation damage during cryo-EM imaging.

136 bacterial cell wall membrane did not sustain radiation damage during STEM imaging at low electron dos

137 GT3-Nano for short, to mitigate bone marrow radiation damage during targeted radionuclide therapy.

138 GT3-Nano for short, to mitigate bone marrow radiation damage during targeted radionuclide therapy.

139 Here we use molecular modeling to map the radiation damage during the 10-50 fs to the intensity, t

140 re commonly cryogenically cooled to minimise radiation damage effects from the X-ray beam, but low te

141 Surprisingly, radiation damage effects revealed by x-ray diffraction w

142 e highly durable and especially resistant to radiation damage effects.

143 Ion beams create radiation damage efficiently without material activation

144 that phycoviolobilin is susceptible to X-ray radiation damage, especially as Pg, during single-crysta

145 Understanding and predicting radiation damage evolution in complex materials is cruci

146 mplex oxides is critical to ionic transport, radiation damage evolution, sintering, and aging.

147 Preliminary radiation damage experiments substantiate the prediction

148 rate highlights the potential of accelerated radiation damage experiments.

149 contrast to proteins, there is no spread of radiation damage far from the primary site of ionization

150 ibed as a promising scenario for controlling radiation damage for medical purposes, but it has so far

151 al questions about the detailed mechanism of radiation damage formation remain largely unanswered.

152 of SiC and Si, are analyzed with a model of radiation damage formation which accounts for the fracta

153 The radiation-damage-free FEL structures reveal the full act

154 uction nature of XFEL experiments provides a radiation-damage-free view of the functionally important

155 nsible for eliminating most ultraviolet (UV) radiation damage from DNA, as well as base alterations c

156 dues arise from their greater sensitivity to radiation damage from electron irradiation as determined

157 high-resolution data with minimal effects of radiation damage from sub-10-mum crystals of membrane an

158 Radiation damage from the alpha-particle decay of Pu cre

159 We postulate that this effect is caused by radiation damage from the tracer dose during dosimetry.

160 ional, elementally selective imaging without radiation damage, has had a revolutionary impact in many

161 Efforts to prevent or mitigate radiation damage have included development of antioxidan

162 We propose a novel mechanism of radiation damage healing in metals, which may guide furt

163 Ultraviolet radiation damages human skin and results in an old and w

164 ff of high-resolution information content by radiation damage in a dose-dependent manner.

165 We propose that radiation damage in a few mitochondria is transmitted vi

166 lectrons are the most important component of radiation damage in biological environments because they

167 The formation of stable radiation damage in crystalline solids often proceeds vi

168 The formation of radiation damage in Ge above room temperature is dominat

169 The buildup of radiation damage in ion-irradiated crystals often depend

170 giant surface-to-volume ratio may alleviate radiation damage in irradiated metallic materials as fre

171 ring meiotic recombination, during repair of radiation damage in mature oocytes, and in proliferating

172 -scale simulations of plasticity and primary radiation damage in MoTaVW alloys.

173 direct experimental demonstration of reduced radiation damage in protein crystals with small beams, d

174 Effects of the collision cascade density on radiation damage in SiC remain poorly understood.

175 e, fused Purkinje neurons increase following radiation damage in the developing cerebellum.

176 Structural changes induced by radiation damage in X-ray crystallography hinder the abi

177 p21(-/-) mice had significantly less radiation damage, including 6-fold less scarring, 40% in

178 ng a modified linear quadratic model for the radiation damage, incorporating the effects of hypoxia a

179 rce synchrotron beamlines, but the excessive radiation damage incurred when using capillaries prevent

180 ation was aided by the novel technique of UV radiation damage-induced phasing.

181 nvironment effects can strongly affect local radiation damage-induced structural dynamics.

182 Radiation damage is a major cause of failure in macromol

183 Radiation damage is a major limitation in crystallograph

184 By contrast, below 35 K the radiation damage is frozen in place, permitting the evol

185 These results demonstrate that substantial radiation damage is likely to have occurred during X-ray

186 cal stretcher, no focusing is required, thus radiation damage is minimized and the surface forces are

187 us checkpoint gene products in responding to radiation damage is proposed.

188 undamental property in predicting cumulative radiation damage is the number of atoms permanently disp

189 e microscopy is intrinsically limited by the radiation damage it causes and the degree to which it al

190 The elimination of ultraviolet (UV) radiation-damaged keratinocytes via apoptosis is an impo

191 Radiation damage led to disruption of inter-ring contact

192 Histological analyses demonstrated that radiation damage led to less bone regeneration.

193 Graphene's ability to reduce radiation damage levels to hydrogen bond breakage is dem

194 an experimental strategy that optimizes the radiation damage lifetime of the crystal, or to assign a

195 mode and an exposure beyond the traditional radiation damage limit.

196 n be used to achieve high resolution, beyond radiation damage limits for biological samples.

197 Radiation damage limits the accuracy of macromolecular s

198 The radiation damage may induce conformational changes of th

199 t-enhanced CT had an increased amount of DNA radiation damage (mean increase +/- standard error of th

200 es, yet the resulting ionization and primary radiation damage mechanisms are poorly understood.

201 eletal muscle is particularly vulnerable, as radiation damages muscle precursor cells (MPCs), impairi

202 s that measure close to 5,000 atoms/alpha in radiation-damaged natural zircons.

203 The x-ray exposure at which significant radiation damage occurs has been quantified for frozen c

204 ought that the induced charge and associated radiation damage of atoms in polyatomic molecules can be

205 This methodology minimizes the radiation damage of beam-sensitive materials, such as mi

206 The extranuclear DNA that can result from radiation damage of cells can result in production of th

207 developed which enables the investigation of radiation damage of samples subject to a thermal gradien

208 with X-rays poses fundamental limits due to radiation damage of the highly energetic photons.

209 some mechanistic insight into the effects of radiation damage on DNA, and (2) overcomes specific tech

210 lectron lasers (XFELs) reduce the effects of radiation damage on macromolecular diffraction data and

211 The effects of radiation damage on materials are strongly dependant on

212 gate the impact of water layer thickness and radiation damage on orientation recovery from diffractio

213 and destroy" approach before the effects of radiation damage on the data become significant.

214 due to the thick water layer, the effects of radiation damage on the orientation recovery are relativ

215 the two structures reveal no indications of radiation damage or significant changes within the activ

216 Using an alternate test of DNA repair, i.e., radiation-damaged or undamaged reporter DNA, we introduc

217 cer clonal evolution (potentially induced by radiation damage), or is due to an innately aggressive t

218 Besides fundamental limits imposed by radiation damage, poor detectors and beam-induced sample

219 It influences both the radiation damage process that occurs during illumination

220 Our results suggest that radiation damage produced by ionizing radiation can alte

221 ation of membrane proteins, investigation of radiation damage-prone systems and time-resolved studies

222 proteins can be susceptible to X-ray-induced radiation damage, raising questions about the reliabilit

223 Cryogenic (cryo-) cooling reduces the global radiation damage rate and, therefore, became the method

224 g, sufficient-size crystals while mitigating radiation damage remains a critical bottleneck of serial

225 Radiation damage remains one of the major bottlenecks to

226 accharomyces cerevisiae to tolerate ionizing radiation damage requires many DNA-repair and checkpoint

227 atom structural and chemical analysis of all radiation-damage-resistant atoms present in, and on top

228 sis, sensing, actuation, supercapacitors and radiation-damage-resistant materials.

229 In maize (Zea mays) leaves, UV-B radiation damages ribosomes by crosslinking cytosolic ri

230 the adult murine system, we demonstrate that radiation-damaged salivary glands can be functionally re

231 1 kHz linearly decreases until the extent of radiation damage saturates and the specimen is effective

232 ylation is catalyzed by PP1, we asked if the radiation damage signal to Nek2 was mediated by PP1.

233 prevents autoimmune responses to ultraviolet-radiation damaged skin.

234 uses promotes the homeostasis of ultraviolet radiation-damaged skin.

235 nificantly and consistently downregulated in radiation-damaged skin.

236 Radiation damage studies were also carried out on a surr

237 lacements per atom (DPA) unit in quantifying radiation damage (such as inadequacy in quantifying degr

238 zation, require extremely high resistance to radiation damage, such as resistance to amorphization or

239 ndicated that LiAlO(2) experiences much more radiation damage than LiAl(5)O(8), where the number of L

240 onization of heavy atoms increases the local radiation damage that is seen in the diffraction pattern

241 low symmetry, and that the residue-specific radiation damage that occurs with increasing electron do

242 this beta-emitting scout dose could inflict radiation damage, the extent of which we aimed to quanti

243 Initially considered relatively resistant to radiation damage, the heart has been shown over the past

244 tructural and nanoscale features to mitigate radiation damage, this study demonstrates enhancement of

245 -dihydrothymidine (dHT), formed via ionizing radiation damage to 2'-deoxycytidine and thymidine, resp

246 ribose sugar in the polymer chain restricts radiation damage to a small region and prevents major en

247 omposition, and sufficient stability against radiation damage to allow for multiple images to be obta

248 nging reduction reactions but can also cause radiation damage to biological tissue.

249 se recruitment of BMDCs into the cerebellum, radiation damage to cerebellar cells, or both, increase

250 9 of 9 assessable animals without detectable radiation damage to critical organs, including bone marr

251 -2'-deoxyuridine (dHdU), formed via ionizing radiation damage to cytosine under anoxic conditions and

252 produced in significant amounts by ionizing radiation damage to cytosine under anoxic conditions.

253 f potential synergistic interactions between radiation damage to DNA and oxidative stress-induced dam

254 ex protein signalling systems that recognise radiation damage to DNA and plasma membrane lipids.

255 SC-'236 did not appreciably alter radiation damage to jejunal crypt cells and tissue invol

256 nd therapy as it substantially minimises the radiation damage to non-tumour cells of healthy tissues.

257 vivo half-lives result in poor contrast and radiation damage to normal tissue.

258 Radiation damage to salivary glands results in loss of s

259 ovides a basis for new approaches to prevent radiation damage to the bowel.

260 fy minimally invasive biomarkers of ionizing radiation damage to the CNS that are predictors of late

261 rs in the irradiated group suggests possible radiation damage to the pituitary, with consequent reduc

262 bound water molecules in the early stages of radiation damage to the protein crystal.

263 on, meiosis, and repair of gaps and ionizing radiation damage to the same extent as rad51.

264 ion (IR) can mitigate the chronic effects of radiation damage to the skin.

265 Radiation damage to the whole lung is a serious risk in

266 tudy, we examined whether the enhancement of radiation damage to tumors by TPZ can be predicted from

267 Hf-DBP-QP-SN not only enhances radiation damage to tumors via a unique radiotherapy-rad

268 s) with photosensitizing ligands can enhance radiation damage to tumors via a unique radiotherapy-rad

269 ute to our understanding of the mechanism of radiation damage, to our appreciation of the importance

270 Nanomaterials have been proposed as radiation damage tolerant materials, due to the hypothes

271 Despite signs of volume expansion due to radiation damage, (U,Pu)(Al,Fe)(3)C(3) remains highly X-

272 dose-dependent manner, enabling to quantify radiation damage using a custom Deep Learning algorithm.

273 Radiation damage usually leads to detrimental effects su

274 rials performance, challenging our view that radiation damage usually results in negative effects.

275 cells (BMDCs) into the tumors, restoring the radiation-damaged vasculature by vasculogenesis and ther

276 Susceptibility to radiation damage was determined by irradiating operators

277 based on detecting gas bubbles generated by radiation damage was used to localize internal proteins

278 on and subsequent proton motion can mitigate radiation damage when heavier atoms absorb X-rays.

279 Biological specimens suffer radiation damage when imaged in an electron microscope,

280 may undergo loss of immunoreactivity due to radiation damage when labeled with large amounts of 131I

281 Low-energy electrons do not induce radiation damage, which enables acquiring subnanometer r

282 -ray free electron laser sources to overcome radiation damage, while sample consumption is dramatical

283 explorations of this idea and of outrunning radiation damage with femtosecond pulses led to the deve

284 sed MultiScale Approach to the assessment of radiation damage with ions gives a positive answer to th

285 diffraction information before the onset of radiation damage, yet the majority of structures solved

286 ffusion mechanism whereby iron diffuses into radiation-damaged zircon along the cores of dislocations

287 during low-temperature recrystallization of radiation-damaged zircon in the presence of an aqueous f