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1 There was no evidence of either recurrent or radiation-induced tumor.
2 the Gadd45a gene are susceptible to ionizing radiation-induced tumors.
3 cer in humans and to a high rate or ionizing radiation-induced tumors and mortality in mice.
4 carcinogen, the number of genetic changes in radiation-induced tumors, and the pathways responsible f
5                                 By contrast, radiation-induced tumors are associated with paracentric
6                                        These radiation-induced tumors are distinctive in their high h
7                       Interestingly, diverse radiation-induced tumors arising in wild-type and Nf1(+/
8  found on chromosome 9 near the Atm locus in radiation-induced tumors, but in 50% of the cases the de
9 found near the p63 locus on chromosome 16 in radiation-induced tumors, but these frequently included
10               An inflammatory response after radiation-induced tumor cell death recruits a variety of
11    These data indicate that Gleevec enhances radiation-induced tumor cell killing and suggest that th
12 ld increase tumor oxygenation and potentiate radiation-induced tumor cell killing.
13 could more actively target to the ligands of radiation-induced tumor cells, which could accumulate mo
14 p110delta is a therapeutic target to enhance radiation-induced tumor control.
15 the untreated group) and histologic signs of radiation-induced tumor damage.
16  tumor cells and immune cells that occurs in radiation-induced tumor equilibrium and suggest a potent
17  whether host immune responses contribute to radiation-induced tumor equilibrium in animal models.
18 ors often involve whole chromosomes, whereas radiation-induced tumors exhibit a high frequency of loc
19                              The majority of radiation-induced tumors exhibit one of three 'signature
20                             More than 30% of radiation-induced tumors from both p53+/- and p53-/- mic
21                             More than 50% of radiation-induced tumors from p53+/- and p53-/- mice sho
22  tumor stroma sensitivity as determinants of radiation-induced tumor growth delay.
23           Antiangiogenic therapy can enhance radiation-induced tumor growth inhibition.
24                                  Unlike some radiation-induced tumors, no activating point mutations
25 ment with dimethylbenzanthracene or ionizing radiation induced tumors of various histologic types tha
26 d breaks; the rearrangements common in these radiation-induced tumors reflect their etiology.
27                                        Among radiation-induced tumors, ret/PTC3 was found in 58%, ret
28                                        Among radiation-induced tumors, solid variant of papillary car
29 ted here whether Ang2 blocking could enhance radiation-induced tumor vascular damage.