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1 pe generated by ionizing radiation and other radiomimetics.
2 utations confer only weak sensitivity to the radiomimetic agent methyl methane sulfonate (MMS) but co
3 after DNA damage by gamma irradiation or the radiomimetic agent neocarzinostatin.
4 /M phases is inhibited by treatment with the radiomimetic agent, adriamycin.
5  also analyzed the PCNA complex induced by a radiomimetic agent, Bleomycin (BLM), which produces pred
6 cells, and rendered cells sensitive toward a radiomimetic agent, neocarzinostatin.
7 erlying the cellular response to this unique radiomimetic agent.
8 HEJ, and potentiate the cellular activity of radiomimetic agents and IR.
9 ifest increased sensitivity to radiation and radiomimetic agents and show defects in DSB damage repai
10 ntrinsic apoptosis induced by UV mimetic and radiomimetic agents.
11  following exposure to ionizing radiation or radiomimetic agents.
12 phosphorylation at Ser1981, yet unlike other radiomimetics and IR, DNA breaks induced by C-1027 resul
13 hese proteins was required for resistance to radiomimetics, and many were required for resistance to
14 ontrasted cell responses to IR and C-1027, a radiomimetic antibiotic that induces DNA strand breaks.
15 lication stress inducer, hydroxyurea, or the radiomimetic antibiotic, neocarzinostatin.
16  are formed in DNA by ionising radiation and radiomimetic anticancer agents and are thought to be bio
17 que characteristic of ionizing radiation and radiomimetic anticancer drugs is the induction of cluste
18 etylated state and experience treatment with radiomimetic anticancer drugs.
19                           The ability of the radiomimetic anticancer enediyne C-1027 to induce ataxia
20 are formed in cells by ionizing radiation or radiomimetic antitumor drugs.
21 Bs), such as those produced by radiation and radiomimetics, are amongst the most toxic forms of cellu
22                                 Unique among radiomimetics, ataxia-telangiectasia mutated (ATM) is di
23  such as ionizing radiation and conventional radiomimetics because their mechanisms require oxygen to
24 eatments with cobalt-60 gamma irradiation or radiomimetic bleomycin, except after high bleomycin dose
25       Like ionizing radiation (IR) and other radiomimetics, breaks induced by C-1027 efficiently acti
26                                   Like other radiomimetics, C-1027 induced DNA breaks to a lesser ext
27 itivity to DNA-damaging agents, particularly radiomimetic chemicals inducing DNA double-strand breaks
28 e of the transgenic cells to UV light or the radiomimetic chemicals methyl methanesulfonate and mitom
29 uction of DSBs utilize ionizing radiation or radiomimetic chemicals, such as neocarzinostatin (NCS),
30 oma cells to UV-, ribotoxic (anisomycin) and radiomimetic chemicals-induced programmed cell death in
31 ormly in the genome (e.g., by irradiation or radiomimetic chemotherapeutics).
32 ng after exposures to chest radiotherapy and radiomimetic chemotherapies.
33 ms, DNA damage-induced GDR elicited by IR or radiomimetic chemotherapy was monitored in the presence
34  the UV mimetic compound oxaliplatin and the radiomimetic compound doxorubicin promoted apoptosis by
35       After treatment of HCT116 cells with a radiomimetic compound neocarzinostatin, active Chk2 exis
36 cell cycle after treatment with radiation or radiomimetic compounds.
37 nt at bypassing subtle terminal mispairs and radiomimetic damage by direct ligation.
38 ase Chinese hamster ovary CHO cells with the radiomimetic DNA double-strand cleaving agent neocarzino
39 q, we determine numbers of DSBs induced by a radiomimetic drug and replication stress, and reveal two
40 smid DNA containing DNA DSBs produced by the radiomimetic drug bleomycin.
41 mal human cells to gamma-irradiation and the radiomimetic drug neocarzinostatin had no effect on ATM
42 rmore, BGT226 sensitizes cancer cells to the radiomimetic drug neocarzinostatin, suggesting that BGT2
43 in cells treated with gamma-radiation or the radiomimetic drug neocarzinostatin.
44 d with actinomycin D, anisomycin or with the radiomimetic drug neocarzinostatin.
45 rtemis-deficient ES cells are sensitive to a radiomimetic drug, but less sensitive to ionizing radiat
46 ls, TDR1 improved resistance to Bleomycin, a radiomimetic drug.
47 slocations induced by ionizing radiation and radiomimetic drugs are thought to arise by incorrect joi
48 osure of cells to ionizing radiation (IR) or radiomimetic drugs induces Chk2 phosphorylation by ATM,
49  agents, such as ionizing radiation (IR) and radiomimetic drugs is crucial in maintaining genomic int
50 Delta cells are hypersensitive to a range of radiomimetic drugs that share the feature of creating le
51 rgeted DNA and RNA damages from radicals and radiomimetic drugs to design DNA cleaving molecules for
52  recombination, sensitivity to radiation and radiomimetic drugs, and failure to repair a subset of DN
53 are usually caused by ionizing radiation and radiomimetic drugs, but can also occur under normal phys
54 reaks are produced by ionizing radiation and radiomimetic drugs, but it was not known whether they ca
55 t uniquely induced by ionizing radiation and radiomimetic drugs, but their level of production by UVB
56 o DSBs induced by ionizing radiation (IR) or radiomimetic drugs, including bleomycin, in living cells
57 pecially in patients previously treated with radiomimetic drugs, may not be feasible.
58 e that is activated by ionizing radiation or radiomimetic drugs, whereas p95/nbs1 is part of a protei
59 ced in DNA by ionizing radiation and by some radiomimetic drugs.
60 itive to DNA replication inhibitors, but not radiomimetic drugs.
61 (DSBs) caused by ionizing radiation (IR) and radiomimetic drugs.
62 ers treated with ionizing radiation (IR) and radiomimetic drugs.
63 olon carcinoma HCT116 cells by the antitumor radiomimetic enediyne antibiotic C-1027.
64                                          The radiomimetic enediyne C-1027 induces almost exclusively
65 response to DNA strand breaks induced by the radiomimetic enediyne C-1027.
66                For example, the conventional radiomimetic enediyne neocarzinostatin is 4-fold less cy
67 CF7 induced apoptosis, which was enhanced by radiomimetic neocarcinostatin treatment.
68                            Here, we design a radiomimetic photosensitizer, producing DNA radical cati
69 ound maintains the characteristic ability of radiomimetics to cleave DNA in cell-free systems, varyin