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1 ressed relative to myocardial oxygen demand (rate-pressure product).
2 k work rate, maximal oxygen consumption, and rate pressure product.
3                   Rest MBF was normalized to rate-pressure product.
4  present after correcting rest flows for the rate-pressure product.
5 d shift in the relationship between MVO2 and rate-pressure product.
6 n increase in heart rate, blood pressure and rate-pressure product.
7 did not demonstrate the expected increase in rate-pressure product.
8 , TA1 and dobutamine similarly increased the rate-pressure product.
9 y (p < 0.02) larger with O3 than with FA for rate-pressure product (1,353 beats/min/mm Hg) and for he
10  flow (3.3+/-2.5 x rest) exceeded normalized rate-pressure product (1.9+/-0.8 x rest; P<.05).
11 line donor vessel absolute flow adjusted for rate pressure product: 177.5 to 139.9 mL/min (difference
12 ats/min, p=0.6) and a comparable increase in rate-pressure product (2.2+/-0.9 vs. 1.9+/-1.2 beats/min
13 /min; P = not significant [ns]) and peak Dob rate.pressure product (20,738 +/- 3,947 vs. 20,047 +/- 3
14      On exercise, oxygen demand was highest (rate-pressure product: 22,157 +/- 5,497 beats/min/mm Hg
15 oad after NTG, as indicated by a higher peak rate-pressure product (24 492+/-1054 versus 22 536+/-101
16 and perfusate Ca2+ with similar increases in rate-pressure product (48+/-5%, 42+/-6%, and 51+/-6%, re
17                                  The resting rate pressure product (6628 +/- 504 versus 5225 +/- 807)
18 sed heart rate (79+/-22 to 115+/-28 bpm) and rate-pressure product (9748+/-2862 to 15,157+/-3433 mm H
19 esting MBF and increase in CVR corrected for rate-pressure product after AVR, although these changes
20                                 However, the rate pressure product, an index of myocardial work, tend
21           Rest blood flows normalized to the rate-pressure product, an index of cardiac work, were si
22 atine:ATP ratio that correlated with reduced rate pressure product and increased diastolic pressure.
23 ease in systolic blood pressure, heart rate, rate-pressure product and CO and a fall in PVR were simi
24                In controls, during ischemia, rate-pressure product and oxygen consumption decreased b
25 dobutamine is in excess of that predicted by rate-pressure product and reflects the unmeasured inotro
26 spite similar values for peak heart rate and rate-pressure product and similar severity of CAD.
27 butamine significantly increased heart rate, rate-pressure product and the first derivative of left v
28                                              Rate-pressure product and wall motion score index, simil
29       Changes in heart rate, blood pressure, rate-pressure product, and cardiac output were greater w
30 001), blood pressure (P < .005) at rest, and rate-pressure product at peak exercise (P < .003), but e
31     Heart rates, systolic blood pressure and rate-pressure product at peak exercise were not differen
32                                     The mean rate-pressure product at peak pacing was 22,313+/-5,357
33 e-EN, the ischemic threshold, defined as the rate-pressure product at the onset of 1-mm ST depression
34                                              Rate pressure product both at baseline and at peak adeno
35  by increases in blood pressure, heart rate, rate-pressure product, cardiac index, and stroke work in
36 ardiovascular risk factors, medications, and rate-pressure product change during mental stress, those
37  relatively minor increase in heart rate and rate-pressure product compared with ischemia induced by
38 ed with lower baseline and postmental stress rate-pressure product compared with nifedipine or placeb
39    Over all segments, mean MBF normalized by rate-pressure product ("corrected MBF") was 1.2+/-0.3 mL
40                    Similarly, heart rate and rate pressure product declined from the baseline dipyrid
41 he isolated heart dose-dependently increased rate pressure product, dP/dt, -dP/dt, and myocardial oxy
42 levations in heart rate, cardiac output, and rate pressure product (estimate of cardiac workload) and
43                                          The rate-pressure product for the group as a whole increased
44 ; 95% confidence interval [CI], 0.3 to 0.9), rate pressure product (HR, 0.7; 95% CI, 0.5 to 0.9), lef
45 HR, 2.6; 95% CI, 1.2 to 5.4), lower exercise rate-pressure product (HR, 0.6; 95% CI, 0.5 to 0.8), res
46 c complications, organ dysfunction, or heart rate pressure product identified.
47   Normalization of resting blood flow to the rate pressure product improved the reproducibility signi
48                                 In addition, rate-pressure product improved suggesting decreased card
49 uring stress, there were equivalent rises in rate pressure product in both groups (DCM, 76+/-15% and
50 assessed by measuring developed pressure and rate pressure product in Langendorff mode.
51 og x kg-1 x min-1 IV) caused doubling of the rate-pressure product in both normal and LVR hearts and
52                                          The rate-pressure product in postischemic hearts was 46% of
53                               In CHF hearts, rate-pressure product increased only 40% in response to
54 ated (p < 0.001) with a 2.2-fold increase in rate-pressure product induced by dobutamine.
55                 They also correlated with RV rate-pressure product (K(mono), R(2) = 0.41, P = 0.0005;
56  at a left ventricular pressure of 50 mm Hg, rate-pressure product, left ventricular tension-time ind
57 icant (P < 0.01) and comparable increases in rate-pressure product, measured in beats per minute x mm
58   Ischemic threshold, measured either as the rate-pressure product or the duration of exercise at the
59                 Endothelial NO decreased the rate-pressure product (P<0.05), upregulated cytochrome c
60 mand were seen (tension-time index, P=0.024; rate-pressure product, P=0.046), as well as an increase
61 obutamine in normal segments correlated with rate-pressure product (r=.81, P<.05), the slope of the l
62                                              Rate-pressure product reactivity was calculated as the m
63 mpaired recovery of systolic (24% versus 53% rate pressure product recovery; P<0.05) and diastolic fu
64 did not alter blood pressure, heart rate, or rate-pressure product relative to placebo.
65          Heart rate, mean arterial pressure, rate-pressure product, respiratory rate, and catecholami
66         HYP showed a 13% to 26% reduction in rate pressure product (RPP) and impaired dP/dt versus SH
67 eak oxygen uptake ([Formula: see text]O(2)), rate pressure product (RPP), CP and VP compared to those
68                                     Baseline rate-pressure product (RPP) (6559+/-1590 versus 7144+/-1
69             Exercise BP, heart rate (HR) and rate-pressure product (RPP) at maximal and submaximal wo
70 onship between ST-segment depression and the rate-pressure product (RPP) during exercise to determine
71            Cold induced similar increases in rate-pressure product (RPP) in smokers and nonsmokers.
72  E) were repeated at 2.5 and 5 minutes for a rate-pressure product (RPP) less than 20% baseline.
73 ic (SBP) and diastolic blood pressure (DBP), rate-pressure product (RPP) oxygen saturation (SpO(2)),
74                                          The rate-pressure product (RPP) was calculated as the mean s
75                    Measurements were made of rate-pressure product (RPP=LV developed pressure x heart
76               MBF was also normalized to the rate.pressure product (RPP).
77 damole caused an increase (mean, 21%) in the rate-pressure product secondary to reflex tachycardia re
78 e end of week 12, mean arterial pressure and rate-pressure product significantly decreased in both gr
79                              The increase in rate-pressure product tended to be greater in patients w
80   The initial high Ca2+ challenge caused the rate-pressure product to increase by 74 +/- 5% in all he
81 educed mechanical inefficiency (the ratio of rate-pressure product to MVO(2)) and myofilament sensiti
82 tion fraction, myocardial scar and ischemia, rate-pressure product, type of radiotracer or stress age
83                                       The RV rate-pressure product was also determined by means of ri
84  were maintained relatively constant and the rate-pressure product was relatively unchanged, but lact
85 <0.001), while MBF at rest normalized by the rate-pressure product, was similar (1.25 0.36 versus 1.1
86      Left ventricular developed pressure and rate pressure product were significantly depressed in th
87 itude of ST segment depression, and exercise rate-pressure product-were found to be independent predi
88 -diastolic pressure, developed pressure, and rate-pressure product, which was also blocked by DPCPX a