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1 ressed relative to myocardial oxygen demand (rate-pressure product).
2 k work rate, maximal oxygen consumption, and rate pressure product.
3 Rest MBF was normalized to rate-pressure product.
4 present after correcting rest flows for the rate-pressure product.
5 d shift in the relationship between MVO2 and rate-pressure product.
6 n increase in heart rate, blood pressure and rate-pressure product.
7 did not demonstrate the expected increase in rate-pressure product.
8 , TA1 and dobutamine similarly increased the rate-pressure product.
9 y (p < 0.02) larger with O3 than with FA for rate-pressure product (1,353 beats/min/mm Hg) and for he
11 line donor vessel absolute flow adjusted for rate pressure product: 177.5 to 139.9 mL/min (difference
12 ats/min, p=0.6) and a comparable increase in rate-pressure product (2.2+/-0.9 vs. 1.9+/-1.2 beats/min
13 /min; P = not significant [ns]) and peak Dob rate.pressure product (20,738 +/- 3,947 vs. 20,047 +/- 3
15 oad after NTG, as indicated by a higher peak rate-pressure product (24 492+/-1054 versus 22 536+/-101
16 and perfusate Ca2+ with similar increases in rate-pressure product (48+/-5%, 42+/-6%, and 51+/-6%, re
18 sed heart rate (79+/-22 to 115+/-28 bpm) and rate-pressure product (9748+/-2862 to 15,157+/-3433 mm H
19 esting MBF and increase in CVR corrected for rate-pressure product after AVR, although these changes
22 atine:ATP ratio that correlated with reduced rate pressure product and increased diastolic pressure.
23 ease in systolic blood pressure, heart rate, rate-pressure product and CO and a fall in PVR were simi
25 dobutamine is in excess of that predicted by rate-pressure product and reflects the unmeasured inotro
27 butamine significantly increased heart rate, rate-pressure product and the first derivative of left v
30 001), blood pressure (P < .005) at rest, and rate-pressure product at peak exercise (P < .003), but e
31 Heart rates, systolic blood pressure and rate-pressure product at peak exercise were not differen
33 e-EN, the ischemic threshold, defined as the rate-pressure product at the onset of 1-mm ST depression
35 by increases in blood pressure, heart rate, rate-pressure product, cardiac index, and stroke work in
36 ardiovascular risk factors, medications, and rate-pressure product change during mental stress, those
37 relatively minor increase in heart rate and rate-pressure product compared with ischemia induced by
38 ed with lower baseline and postmental stress rate-pressure product compared with nifedipine or placeb
39 Over all segments, mean MBF normalized by rate-pressure product ("corrected MBF") was 1.2+/-0.3 mL
41 he isolated heart dose-dependently increased rate pressure product, dP/dt, -dP/dt, and myocardial oxy
42 levations in heart rate, cardiac output, and rate pressure product (estimate of cardiac workload) and
44 ; 95% confidence interval [CI], 0.3 to 0.9), rate pressure product (HR, 0.7; 95% CI, 0.5 to 0.9), lef
45 HR, 2.6; 95% CI, 1.2 to 5.4), lower exercise rate-pressure product (HR, 0.6; 95% CI, 0.5 to 0.8), res
47 Normalization of resting blood flow to the rate pressure product improved the reproducibility signi
49 uring stress, there were equivalent rises in rate pressure product in both groups (DCM, 76+/-15% and
51 og x kg-1 x min-1 IV) caused doubling of the rate-pressure product in both normal and LVR hearts and
56 at a left ventricular pressure of 50 mm Hg, rate-pressure product, left ventricular tension-time ind
57 icant (P < 0.01) and comparable increases in rate-pressure product, measured in beats per minute x mm
58 Ischemic threshold, measured either as the rate-pressure product or the duration of exercise at the
60 mand were seen (tension-time index, P=0.024; rate-pressure product, P=0.046), as well as an increase
61 obutamine in normal segments correlated with rate-pressure product (r=.81, P<.05), the slope of the l
63 mpaired recovery of systolic (24% versus 53% rate pressure product recovery; P<0.05) and diastolic fu
67 eak oxygen uptake ([Formula: see text]O(2)), rate pressure product (RPP), CP and VP compared to those
70 onship between ST-segment depression and the rate-pressure product (RPP) during exercise to determine
73 ic (SBP) and diastolic blood pressure (DBP), rate-pressure product (RPP) oxygen saturation (SpO(2)),
77 damole caused an increase (mean, 21%) in the rate-pressure product secondary to reflex tachycardia re
78 e end of week 12, mean arterial pressure and rate-pressure product significantly decreased in both gr
80 The initial high Ca2+ challenge caused the rate-pressure product to increase by 74 +/- 5% in all he
81 educed mechanical inefficiency (the ratio of rate-pressure product to MVO(2)) and myofilament sensiti
82 tion fraction, myocardial scar and ischemia, rate-pressure product, type of radiotracer or stress age
84 were maintained relatively constant and the rate-pressure product was relatively unchanged, but lact
85 <0.001), while MBF at rest normalized by the rate-pressure product, was similar (1.25 0.36 versus 1.1
87 itude of ST segment depression, and exercise rate-pressure product-were found to be independent predi
88 -diastolic pressure, developed pressure, and rate-pressure product, which was also blocked by DPCPX a