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1 f the PR8 NS gene for that of a Vero-adapted reassortant virus.
2 he time of onset of virus shedding in triple-reassortant viruses.
3 salts was also mapped to L3/lambda1 by using reassortant viruses.
4 e wholly Eurasian lineage H5N8 and two novel reassortant viruses.
5  resulting in new genetic constellations and reassortant viruses.
6 raminidase activity and virion morphology of reassortant viruses.
7 tions also strongly affected the genotype of reassortant viruses.
8 the viability and replicative ability of the reassortant viruses.
9 be incorporated into progeny virions to give reassortant viruses.
10 s segments to diminish their ability to form reassortant viruses.
11  also contributed to the pathogenesis of the reassortant viruses.
12 igs with reverse genetics-created HA plus NA reassortant viruses.
13 cs to generate A/Anhui/1/13 (H7N9) and three reassortant viruses (2:6 H7N9) which contained the hemag
14 influenza virus (A/Memphis/31/98, H3N2) in a reassortant virus A/NWS/33(HA)-A/Mem/31/98(NA) (H1N2) an
15 ray analysis with a high infectivity "triple reassortant" virus ((A/Swine/MN/593/99 (H3N2)) that spre
16                        Infection with the NP reassortant virus also resulted in the upregulation of g
17 of these amino acid changes into an H7N9 PR8 reassortant virus also significantly improved viral tite
18                                     Exemplar reassortant viruses also replicated to similar titers in
19                              While both 1918 reassortant viruses also were highly pathogenic, the H5N
20 ween the capacity of T1L x T3A and T1L x T3D reassortant viruses and field isolate strains to inhibit
21 surface protein mu1 by genetic analyses with reassortant viruses and recoated cores.
22                         A genetic study with reassortant viruses and subsequent biochemical analyses
23 ole for the RNP complex in the generation of reassortant viruses, and a functional interaction of PB2
24 athogenicity avian H7N3 virus and that these reassortant viruses are stable and not attenuated in an
25 th pandemic potential, including avian-human reassortant viruses as tested here, may emerge.
26        We found that an H9N2 avian influenza reassortant virus bearing a human-origin H1N1/2009 PA ge
27 and A(H7) viruses leads to the generation of reassortant viruses bearing A(H9N2) internal genes with
28                From studies with single-gene reassortant viruses bearing one gene segment from the AA
29  the circulating swIAVs, generating many new reassortant viruses bearing pdm/09-derived genes globall
30 th the 1918 and 1918HA/K173 viruses, whereas reassortant viruses bearing the 1918 viral RNA polymeras
31 es, we generated all the 254 combinations of reassortant viruses between A/chicken/South Kalimantan/U
32 m for the generation of single and quadruple reassortant viruses between A/Teal/HK/W312/97 (H6N1) and
33      To evaluate the pathogenic potential of reassortant viruses between currently cocirculating avia
34  the context of single-, double-, and triple-reassortant viruses between PR8 and SPN04; PR8/SPN04 M,
35 re, we examined the biological properties of reassortant viruses between the 1918 virus and a contemp
36                                              Reassortant viruses between the two BatIVs were also suc
37  ability to enhance the pathogenicity of the reassortant viruses but could do so by cooperating with
38 ed the functionality of the H7N3:PH1N1 7 + 1 reassortant viruses by measuring the inflammatory respon
39                We created otherwise isogenic reassortant viruses by site-directed mutagenesis that co
40 w that following adaptation in the ferret, a reassortant virus carrying the surface proteins of an av
41                        Finally, we show that reassortant viruses carrying gene segments of CIV and hI
42                                        Using reassortant viruses carrying representative HAs, we anal
43                 Previously, we showed that a reassortant virus composed of wild-type avian H9N2 surfa
44 f infection with HPAI (the most virulent), a reassortant virus containing 1918 hemagglutinin and neur
45                               In contrast, a reassortant virus containing an oseltamivir-resistant se
46 ere isolated from infected animals: a double-reassortant virus containing genes similar to those of h
47 ose of human and swine viruses, and a triple-reassortant virus containing genes similar to those of h
48 logenetic analyses show that IND1982/01 is a reassortant virus containing genome segments derived fro
49                      Here, we evaluated if a reassortant virus containing the hemagglutinin and neura
50                                          The reassortant virus containing the original wt A/Anhui/1/2
51  We also used reverse genetics to generate a reassortant virus containing the PB1 gene of the A/PR/8/
52                  In this study, we generated reassortant viruses containing human seasonal HA and neu
53 s of IAV have resulted in the circulation of reassortant viruses containing human-origin genes in pig
54 the spread of virus to the MLN (P = 0.02) in reassortant viruses containing segment 7 from the spread
55 whether transmissible H5 subtype human-avian reassortant viruses could be generated in vivo.
56  humans, raising the concern that H9N2:pH1N1 reassortant viruses could emerge.
57                                 Studies with reassortant viruses demonstrated that expression of the
58                   In assays using a panel of reassortant viruses derived from these strains, the diff
59 man lung epithelial A549 cells (however, the reassortant virus did not replicate more efficiently tha
60                                            A reassortant virus (Dk78/Eng62N2) did not propagate in du
61     We also observed the generation of novel reassortant viruses during chicken transmission which in
62 rograms to monitor the emergence of human H5 reassortant viruses, especially those containing a PB2 s
63                                            A reassortant virus expressing only the HA and neuraminida
64                                            A reassortant virus expressing the 1918 viral RNA polymera
65                                           In reassortant viruses, factory costaining for cUb mapped p
66 ial clinical implications of the avian-swine reassortant virus for pathogenicity in pigs or other spe
67       Given the relatively high incidence of reassortant viruses from tissues of the ferret upper air
68                             AC resistance of reassortant viruses generated from a cross of wt type 1
69 reased infectivity, using laboratory-derived reassortant viruses generated from sialic acid binding r
70  outbreak, and studying the pathogenicity of reassortant viruses generated with the index wild bird v
71                   Two antigenically distinct reassortant viruses (H3N2) were isolated from infected a
72             The spread of H5N8 and the novel reassortant viruses, H5N2 and H5N1 (H5Nx), in domestic p
73                                         This reassortant virus had a higher titer and total protein y
74                                  Some of the reassortant viruses had a greater replication in chicken
75                            Nineteen of these reassortant viruses had high-growth phenotypes in vitro,
76         Here, we show that the seven main H5 reassortant viruses had various combinations of gene seg
77 SIVs) into U.S. swine in 1998, H1N2 and H1N1 reassortant viruses have emerged from reassortment betwe
78 e HA of human A/Udorn/307/72 (H3N2) permit a reassortant virus [human Udorn HA, with all other genes
79        The inclusion phenotypes of T1L x T3D reassortant viruses identified the viral M1 genome segme
80           A genetic analysis using T1L x T3D reassortant viruses implicated the L3 and M1 gene segmen
81 etics of the wholly human virus and a triple-reassortant virus in 7-week-old pigs that were infected
82 y, growth kinetics, and pathogenicity of the reassortant virus in mice.
83 e of Ngari virus, emphasizing a role of this reassortant virus in severe human illness throughout Eas
84                                    Growth of reassortant viruses in ammonium chloride-treated cells s
85 o exchange genetic sequences and produce new reassortant viruses in swine.
86 ts the need for increased attention to novel reassortant viruses in swine.IMPORTANCEThe emergence of
87                                    Growth of reassortant viruses in the presence of E64 segregated ex
88                                    Growth of reassortant viruses in the presence of E64 segregated wi
89                     The neuraminidase of the reassortant viruses is most closely related to that of B
90                                  Analysis of reassortant viruses isolated from crosses of an MA mutan
91                                      We used reassortant viruses isolated from crosses of wild-type (
92 the presence of E64 were identified by using reassortant viruses isolated from independent crosses of
93 PI viruses in the presence of E64, we tested reassortant viruses isolated from independent crosses of
94 s, and propose that the 1918 virus was not a reassortant virus (like those of the 1957 and 1968 pande
95           However, some gene segments in two reassortant viruses likely originated from domestic anse
96 tween this wholly human virus and the triple-reassortant viruses may affect their replication efficie
97 gglutinin (HA) binding and NA specificity in reassortant viruses may be related to the emergence of n
98  the A(H1N1)pdm09 influenza virus, a complex reassortant virus of swine origin, highlighted the impor
99 d species, with interspecies transmission of reassortant viruses peaking during the winter.
100           Approximately 34% of the recovered reassortant viruses possessed the H5 hemagglutinin (HA)
101 are a 2004 human H5N1 Vietnam isolate with 2 reassortant viruses possessing the 1918 hemagglutinin (H
102                         The transmissible H5 reassortant virus preferentially recognized human-type r
103                                          The reassortant viruses replicated relatively poorly in embr
104                                              Reassortant viruses represented by A/Chicken/Hong Kong/G
105                        Analysis of T1L x T3D reassortant viruses revealed that the mu1-encoding M2 ge
106 in vivo evaluation of the H7N3:PH1N1 (7 + 1) reassortant viruses revealed that the PB2, NA, or M segm
107                     Analysis of T1L x T3D-RV reassortant viruses revealed that the viral S4 gene segm
108                                              Reassortant virus S7, which was unable to reach the live
109                                          The reassortant viruses shed by contact chickens also showed
110    Plaque purification and genotyping of the reassortant viruses shed via the oropharynx of contact c
111                   An experiment with genetic reassortant viruses showed that in newborn mice the M se
112 ly higher proportion compared to most of the reassortant viruses, showed significantly increased repl
113                      In 2009, a swine-origin reassortant virus spilled over to humans, causing a glob
114 nd that prime-boost vaccinated pigs had less reassortant viruses than nonvaccinated pigs, likely due
115                        We demonstrate that a reassortant virus that expresses the M2 gene-encoded mu1
116  birds, increases the risk of emergence of a reassortant virus that persists in wild bird populations
117                Here, we show that three H3N2 reassortant viruses that contain 3 (NP, M, and NS) or 5
118        Influenza co-infections can result in reassortant viruses that contain gene segments from mult
119 EA virus and a pdm/09 virus yielded 55 novel reassortant viruses that could be categorized into 17 ge
120 st this possibility, we generated a panel of reassortant viruses that expressed the NA genes of human
121  increases the risk of generating H5 subtype reassortant viruses that may be shed from upper airway s
122                             We identified 54 reassortant viruses that were grouped in 17 single and 1
123  growth in cured cells, we used T1L X PI 3-1 reassortant viruses to genetically map the capacity of t
124  a series of human 1918-avian H1N1 influenza reassortant viruses to identify the genetic determinants
125              We used T1L x T3A and T1L x T3D reassortant viruses to identify viral genes associated w
126  PI 2A1 sigma1 protein, we used T1L X PI 2A1 reassortant viruses to identify viral genes associated w
127          The capacity for different reovirus reassortant viruses to induce acute myocarditis in mice
128 e capacities of both T1L x T3A and T1L x T3D reassortant viruses to inhibit cellular DNA synthesis an
129 nsfection-based inoculation system to select reassortant viruses under in vivo selective pressure.
130 -adapted (ca) influenza A/Kawasaki/86 (H1N1) reassortant virus vaccine given with or apart from child
131       In addition, infection with the triple-reassortant virus was associated with moderate to severe
132   Replication capacity of the individual 1:7 reassortant viruses was assessed in mouse lungs and huma
133 that the internal gene complex of the triple-reassortant viruses was associated with three recent phy
134 tify factors that restrict the generation of reassortant viruses, we cotransfected human embryonic ki
135   To investigate the virulence of pH1N1-H5N1 reassortant viruses, we created pH1N1 (A/California/04/2
136                           By using T1L x T3D reassortant viruses, we found that differences in the ca
137                              Using T1L x T3D reassortant viruses, we found that strain-specific diffe
138                                        Using reassortant viruses, we showed that differences in the p
139                                          Two reassortant viruses were assessed in ferrets and showed
140 Recently, three genetically distinct A(H5N1) reassortant viruses were detected in live poultry market
141 umber of clade 1.1.2 and 2.3.2.1c interclade reassortant viruses were detected with different combina
142  H3N2 virus backgrounds and found that these reassortant viruses were fully competent in mammalian ho
143                   We show here that multiple reassortant viruses were generated during natural coinfe
144                                         Four reassortant viruses were generated, with three of them s
145                                These chicken reassortant viruses were highly similar to the human iso
146 lowing the TRIG cassette emergence, numerous reassortant viruses were isolated in nature, suggesting
147                                      Most of reassortant viruses were more pathogenic and contagious
148 n (HA), we found that 51 of the 127 possible reassortant viruses were viable and able to be rescued.
149 B2, and NP genes impacted replication of the reassortant viruses while also affecting the expression
150                       In this model, an H3N2 reassortant virus with avian virus internal protein gene
151 rissa virus) suggested that it was a genetic reassortant virus with S and L segments derived from Bun
152                                          The reassortant virus with the HA and NA from the chicken vi
153 ic H1N1 (pH1N1) influenza A virus (IAV) is a reassortant virus with two polymerase components, PA and
154 ic H1N1 (pH1N1) influenza A virus (IAV) is a reassortant virus with two polymerase components, PA and
155  H5N8 HPAI viruses in mallards, we generated reassortant viruses with 2014 and 2016 viruses.
156               Together, these data show that reassortant viruses with a novel genetic composition gen
157                                              Reassortant viruses with avian polymerases and human PA
158 es in pigs, which could give rise to progeny reassortant viruses with enhanced virulence and transmis
159 art to two intrasubtypes, cocirculating H3N2 reassortant viruses with fitness advantages over the par
160 n but inefficient transmission, whereas H5N1 reassortant viruses with four or six human virus interna
161 ted in an increase in the virus yield of the reassortant viruses without a significant loss of NA act

 
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