ライフサイエンスコーパス: receptor

1 on interface result in monomerization of the receptor.

2 cked substrate with the VirD4-like substrate receptor.

3 d SNP rs3901533 of dectin-1, the beta-glucan receptor.

4 -like protein CDIP1 as a second, alternative receptor.

5 and to increase the binding affinity to the receptor.

6 nd block the interaction with the human ACE2 receptor.

7 ut not that of FcgammaRIIB, an inhibitory co-receptor.

8 a potent orthosteric agonist of the GABA(A) receptor.

9 naling downstream of various innate immunity receptors.

10 nti-CD20 antibodies and lymphoma cancer cell receptors.

11 ng postsynaptic Ca(2+) or blocking nicotinic receptors.

12 or peritumoral cortex tissue expressed P2Y12 receptors.

13 l receptor (TCR), co-receptors, and cytokine receptors.

14 e organization and number of its centromeric receptors.

15 GAP binding and optimal signaling by the two receptors.

16 as a means to pharmacologically modulate the receptors.

17 ng the closely related IsdB and IsdH surface receptors.

18 hanced binding and agonist potency at alpha7 receptors.

19 , as we show for both kainate and GluA2 AMPA receptors.

20 res most probably via inositol trisphosphate receptors.

21 a induced increased fibroblast growth factor receptor 1 (FGFR1) expression in NSCLC cell lines H1975,

22 of its putative protein substrate IFN-gamma receptor 1 (IFNGR1) at the protein level in TNBC.

23 e premetastatic lungs through complement C5a receptor 1 signaling.

24 tive fatty acid receptors, G protein-coupled receptor 120 (GPR120) and cluster of differentiation 36

25 Roundabout guidance receptor 2 (ROBO2) plays an important role during early

26 Transferrin receptor 2 (TFR2) is a transmembrane protein expressed m

27 depletion of transforming growth factor-beta receptor 2 (TGFBR2) in CD4(+) T cells, but not CD8(+) T

28 Tumor necrosis factor receptor 2 (TNFR2) is strongly upregulated on renal tubu

29 ptor (PR), and human epidermal growth factor receptor-2 (HER2) are the three crucial biomarkers for t

30 the pattern recognition receptors, toll-like receptor-2 (TLR2) and -4 (TLR4).

31 l nuclear receptors and/or G-protein-coupled receptors(3,4).

32 cid is a potent ligand for G protein Coupled Receptor 35 (GPR35) and exhibits antinociceptive propert

33 These alarmins bind to the Toll-like receptor 4 and prime the nod-like receptor family pyrin

34 ed amyloid precursor protein (APP) and death receptor-6 (DR6) on MNs as the top predicted ligand-rece

35 r previous study demonstrated that Toll-like receptor 7 (TLR7) is essential in the induction of anti-

36 ependent clustering of P2Y(11)/ P2Y(11)-like receptors, AC5, PKA and Ca(V)1.2 into nanocomplexes at t

37 the envelope to recognise and bind the human receptor ACE2.

38 naptic proteins, primarily the acetylcholine receptor (AChR) and inhibit signaling at the neuromuscul

39 sors, including thermosensitive channels and receptors, act as the initial detectors of temperature c

40 lification (gamma), Binding affinity (K(d)), Receptor activation Efficacy (epsilon), and constitutive

41 ore sustained increase induced by muscarinic receptor activation.

42 Mutations can render the receptor active even in the absence of light.

43 d that during a murine model of sepsis, P2X7 receptor activity is important for maintaining elevated

44 ing in the D. sechellia brain, and find that receptor adaptations are accompanied by increased sensor

45 The receptor adopts two major signaling conformations, one o

46 acokinetics study demonstrated adipoRon (APN receptor agonist) was a blood-brain barrier penetrant.

47 glitazones), glucagon-like peptide-1 (GLP-1) receptor agonists and dipeptidyl peptidase 4 (DPP4) inhi

48 Adhesion G protein-coupled receptors (AGPCRs) are a thirty-three-member subfamily o

49 We identified the aryl hydrocarbon receptor (AHR) pathway as a potent tumour suppressor in

50 of gene expression (SATB1) and the estrogen receptor alpha (ESR1).

51 gulated by peroxisome proliferator-activated receptor alpha (PPARalpha), suggesting that FATP2 activi

52 p involving increased expression of the IL-2 receptor alpha-subunit and activation of the transcripti

53 Estrogen receptor -alpha signaling increased IL-33 release and IL

54 nterleukin 22 (IL-22) signals via both IL-22 receptor alpha1 (IL-22Ralpha1) and the common IL-10R2, b

55 dins (TSPs) and activation of their neuronal receptor, alpha2delta-1.

56 Besides binding to well-established receptors, an extended loop in the C-terminal receptor-b

57 In the presence of target cell receptors, an immunocomplex was formed between anti-CD20

58 2 and 8 K when compared with wild-type A(1)-receptor and A(1)R-Y288A(7.53) (a folding-deficient vari

59 enes such as DCC, which encodes the netrin-1 receptor and has an important role in the development of

60 crease of the cGMP levels generated by these receptors and a change of subcellular cGMP compartmentat

61 increased distinct clusters of acetylcholine receptors and axon terminals exhibited numerous terminal

62 olism and energy balance via several nuclear receptors and/or G-protein-coupled receptors(3,4).

63 protects against ZIKV by serving as a decoy receptor, and plays a protective role in ZIKV-mediated b

64 ors, including the T-cell receptor (TCR), co-receptors, and cytokine receptors.

65 Pre-treatment with SKF-83566, a D1 receptor antagonist, attenuated GBP-induced CPP.

66 ALK5 inhibition and endothelin receptor antagonization inhibited mesenchymal lineage co

67 reconcile the clear contribution of androgen receptor (AR) activity that has been established in prec

68 The androgen receptor (ar) and cytochrome P450 1A genes were associat

69 Loss of androgen receptor (AR) signaling dependence occurs in approximate

70 rates in mouse and human embryos, where EPHA receptors are enriched in pluripotent cells whilst surro

71 LE) peptide ligands in connection with their receptors are important players in cell-to-cell communic

72 Together, these co-receptors are required during embryogenesis to mediate p

73 d glutamate probe that is inert toward these receptors at concentrations that are effective for photo

74 signaling cascade initiated by BDNF and its receptors at the plasma membrane to modulate BDNF-depend

75 the hypothesis that an abnormal ECM-integrin receptor axis contributes to BM megakaryocytosis in JAK2

76 ucleus (VMN), we studied the cholecystokinin receptor B-expressing (CCKBR-expressing) VMN targets of

77 B cell antigen receptor (BCR) signals induce Syk activation followed by

78 that of conventional B cells, through B cell receptor (BCR)-dependent positive selection of fetally d

79 Peroxisome proliferator activated receptor beta/delta (PPARbeta/delta) has pro-angiogenic

80 ed IgG, IgM, and IgA antibodies to the spike receptor binding domain (RBD), S1+S2, nucleocapsid, and

81 st that treatment of COVID-19 with high anti-receptor binding domain IgG titer convalescent plasma is

82 ialysis in July, 2020, using a spike protein receptor binding domain total antibody chemiluminescence

83 ontaining both a stabilized HA and alpha-2,6 receptor binding in tandem pose greater pandemic risk.

84 elements of both the allosteric ligands and receptor binding sites important for these allosteric ac

85 ABM300 was characterized in vitro (receptor binding, beta-arrestin2 recruitment, ERK1/2 pho

86 eceptors, an extended loop in the C-terminal receptor-binding domain (HC) of BoNT/B (HC/B) has been p

87 The receptor-binding domain (RBD) is immunodominant and the

88 antihypertensive activity to the angiotensin receptor blocker (ARB) losartan.

89 ) or by (2) bicuculline (a preferent GABA(A) receptor blocker), suggesting a GABAergic activation ind

90 -converting enzyme inhibitors or angiotensin receptor blockers in patients with COVID-19.

91 is plausible that the ACE inhibitors and ACE receptor blockers may have the potential to prevent and

92 activation, and counterregulation by the co-receptors BTLA, Tim-3, PD-1, TIGIT, and CD73.

93 es the diffusivity of CD19, a stimulatory co-receptor, but not that of FcgammaRIIB, an inhibitory co-

94 The human hepatocyte growth factor receptor (c-MET) signaling pathway is dysregulated in se

95 -encounter, whereas memory B cells expressed receptors capable of neutralizing virus when expressed a

96 g the antitumor activity of chimeric antigen receptor (CAR) T cells.

97 pies, including those using chimeric antigen receptor (CAR)-modified T cells, to solid tumors require

98 unction as third generation Chimeric Antigen Receptors (CAR) T cells demonstrating specific cytolytic

99 reconsolidation requires cannabinoid type 1 receptor (CB1R) signaling based on the fundamental role

100 s expressed elevated levels of the scavenger receptor CD36, accumulated lipids, and used fatty acid o

101 DC intermedilysin, bound to the human immune receptor CD59 in a nanodisc model membrane.

102 effect of eHNP-A1-CD15-LDE225 via scavenger receptor class B type 1 (SR-B1) and CD15 on brain SHH MB

103 ed the biological function of the ACKR2-V41A receptor compared to the wild type allele by measuring i

104 ecruitment to a high-affinity heterotrimeric receptor complex (IL-2Ralpha/IL-2Rbeta/gamma(c)).

105 interaction of a Tat substrate with the Tat receptor complex.

106 The plant Dwarf14-Like (D14L) receptor conditions pre-symbiotic perception of AM fungi

107 e with picrotoxin resistance engineered into receptors containing the delta subunit.

108 a family of cephalopod-specific chemotactile receptors (CRs) to detect poorly soluble natural product

109 pexidartinib, a colony-stimulating factor 1 receptor (CSF1R) inhibitor, to reduce TAM density.

110 found that, like ketamine, HNK reduced NMDA receptor currents in a dose-, pH-, and voltage-dependent

111 e, AP5, and NMDA positively modulate glycine receptor currents.

112 myeloid OC progenitors (OCP) expressing the receptor CX(3)CR1.

113 encephalitis alter the levels of dopamine 1 receptor (D1R) and dopamine 2 receptor (D2R) and cause p

114 of dopamine 1 receptor (D1R) and dopamine 2 receptor (D2R) and cause psychotic-like features in mice

115 supported by decreased available dopamine D2 receptor (D2R) levels and the failure of antipsychotic d

116 ne with Tropomyosin receptor kinase B (TrkB) receptor deletion from D1-expressing cells (D1-Cre-flTrk

117 trongest effect on the minimal perfusion and receptor density for standard and optimized treatments.

118 take, genetically predisposed irregular 5-HT receptor density, or change in sensory bombardment may e

119 Whether this receptor directly regulates the intercalated cell chlori

120 proteins, we found no evidence that a single receptor domain is responsible for the Fls3-sustained re

121 migration of cancer cells nor did it induce receptor downregulation.

122 ed by a molecular modeling study, based on a receptor-driven docking model and including a systematic

123 (RTKs), such as the epidermal growth factor receptor (EGFR), locally increases the abundance of reac

124 Estrogen receptor (ER), progesterone receptor (PR), and human epi

125 Tamoxifen is an antagonist of estrogen receptor (ERalpha), a transcription factor expressed in

126 omoted expression and activation of estrogen receptor (ESR1/ER) and its target genes (PGR, KRT8/CK8,

127 ndicating strong selection of B cell antigen receptors even in the absence of microbiota.

128 to confirm its agonist activity on rat P2X2 receptors expressed in human cells.

129 ated with decreased activation of dopamine 1 receptor-expressing medium spiny neurons (D1R-MSNs).

130 persistently elevated the excitability of D1 receptor-expressing SPNs (D1-SPNs).

131 statin analogs in patients with somatostatin receptor-expressing tumors is often performed using admi

132 and other cell types by analyzing ligand and receptor expression patterns.

133 ignificantly lower frequencies of inhibitory receptor expression, i.e., PD-1 and coexpression of PD-1

134 Toll-like receptor 4 and prime the nod-like receptor family pyrin domain-containing 3 inflammasome i

135 , a member of the adhesion G protein-coupled receptor family, is abundantly expressed in cells of the

136 a transcription factor of the orphan nuclear receptor family.

137 ound to resident cells via its high-affinity receptor, FcepsilonRI.

138 selective binding to the activating Fcgamma receptor FcgammaRIIa results in enhanced ability to prev

139 the neonatal Fc receptor (FcRn) and Fcgamma receptors (FcgammaRs), which confer pleiotropic biologic

140 ypes of receptors, including the neonatal Fc receptor (FcRn) and Fcgamma receptors (FcgammaRs), which

141 he plant defense regulator NPR1 and analyses receptor FLS2.

142 Plasma concentrations of sRAGE (soluble receptor for advanced glycation end products) strongly a

143 a cholesterol-poor ligand that binds to the receptor for cholesterol-rich HDLs, scavenger receptor t

144 KREMEN1 (KRM1) is the entry receptor for the largest receptor-group of hand-foot-and

145 e of its products, T cells bearing alphabeta receptors for antigen.

146 Sialic acids (Sia) are the primary receptors for influenza viruses and are widely displayed

147 successfully identified genes encoding known receptors for previously characterized viruses of Escher

148 proteoglycans (HSPGs) act as alternative co-receptors for RSPO3 using a combination of ligand mutage

149 ors for detection of two putative fatty acid receptors, G protein-coupled receptor 120 (GPR120) and c

150 gnaling in this system involves two distinct receptor-G protein complexes, a conventional ternary com

151 of retinal environment age on their GABA(A) receptor (GABA(A)R) responses, elicited by muscimol.

152 tive allosteric modulators (PAMs) of GABA(A) receptors (GABA(A)Rs) with in vivo anesthetic, anxiolyti

153 increased peroxisome proliferator-activated receptor gamma activity and reduced proliferation.

154 legs, but peroxisome proliferator-activated receptor gamma coactivator 1-alpha and oxidative phospho

155 Glucagon-like peptide 1 receptor (GLP-1R) imaging with radiolabeled exendin has

156 uorescence imaging of glucagonlike peptide 1 receptor (GLP-1R)-positive lesions, using the GLP-1 agon

157 GPR88 is an orphan G protein-coupled receptor (GPCR) considered as a promising therapeutic ta

158 NOS interactors, including G protein-coupled receptor (GPCR) kinase interactor 1 (GIT1), which we fou

159 G protein-coupled receptor (GPCR) kinases (GRKs) play a key role in termin

160 enoceptor (beta(1)AR) is a G-protein-coupled receptor (GPCR) that couples(1) to the heterotrimeric G

161 ar universal expression of G protein-coupled receptors (GPCR) reflects their involvement in most phys

162 G protein-coupled receptors (GPCRs) are a large family of integral membran

163 Multiple G protein-coupled receptors (GPCRs) are targets in the treatment of dement

164 G-protein-coupled receptors (GPCRs) comprise the largest and most pharmaco

165 G protein-coupled receptors (GPCRs) comprise the largest group of membrane

166 -i against seven out of 35 G-protein coupled receptors (GPCRs) within the germline cells also elimina

167 f tyrosine hydroxylase (Th)], glucocorticoid receptor (GR) and plasma corticosterone, as well as brai

168 Glucocorticoid receptor (GR) and STAT3 bind to the same genomic regulat

169 Activation of the glucocorticoid receptor (GR) by the synthetic corticosteroid dexamethas

170 (KRM1) is the entry receptor for the largest receptor-group of hand-foot-and-mouth disease causing vi

171 ovel candidate gene (ILDR1), which encodes a receptor highly expressed in prostate tissue and is rela

172 uced tyrosine kinase activity of the insulin receptor; however, its impact on pancreatic beta-cell fu

173 Here we report that serotonin 2c receptor (Htr2c)-expressing neurons in the lateral parab

174 rotein-coupled receptors like the human Y(1) receptor (hY(1)R) are promising targets in cancer therap

175 Imidazoline I(2) receptors (I(2)-IR), widely distributed in the CNS and a

176 Because endocytosis of TGFbeta receptor II (TbetaRII), in response to TGFbeta stimulati

177 ibe the expression of a functional vitamin D receptor in IDEC.

178 cate classical complement proteins and their receptors in CNS dysfunction, recent data suggest that t

179 PCRs) comprise the largest group of membrane receptors in eukaryotic genomes and collectively they re

180 oach for assessing conformational changes in receptors in living cells at ambient conditions.

181 MALT1 mediates signaling from many immune receptors in mammals, but was not previously implicated

182 ver, difficulty expressing human alpha6beta4 receptors in recombinant systems has precluded drug disc

183 , our results suggest that stimulation of D1 receptors in the dentate gyrus is a potential adjunctive

184 xy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors in vitro after exposure to patients' CSF antib

185 e capacity to interact with diverse types of receptors, including the neonatal Fc receptor (FcRn) and

186 of extracellular signaling through multiple receptors, including the T-cell receptor (TCR), co-recep

187 Binding to Ins(1,4,5)P(3) receptors [Ins(1,4,5)P(3)R] and the ability to release C

188 onents of the necroptotic pathway, including receptor-interacting protein kinase-3 (RIPK3), mixed lin

189 Aryl hydrocarbon receptor-interacting protein-like 1 (AIPL1) is a photore

190 lar patterns (MAMPs) via pattern recognition receptors, internal cellular signaling pathways are indu

191 Recognition of ER proteins by the KDEL receptor is pH dependent, with binding occurring under a

192 We conclude that early blockade of the IL-1 receptor is therapeutic in acute hyperinflammatory respi

193 ectinib is a potent inhibitor of tropomyosin receptor kinase (TRK) A, B, and C, which has been shown

194 We have a mouse line with Tropomyosin receptor kinase B (TrkB) receptor deletion from D1-expre

195 ory role by identifying interleukin 1 type I receptor kinase-1 (IRAK-1) as a Nck1-selective binding p

196 Tropomyosin receptor kinases (TRKs) are promising cancer therapeutic

197 Knockout mice for the kisspeptin receptor, Kiss1r (Kiss1r-/-) and its ligand kisspeptin,

198 evaluated the occupancy of the kappa opioid receptor (KOR) by naltrexone measured with [(11)C]-LY279

199 However, receptor-level IR (e.g., insulin receptor pathogenic var

200 e collected to assess chemokine and integrin receptor levels on monocytes.

201 ine-rich repeat-containing G-protein coupled receptors (LGRs) 4, 5 and 6 (Lebensohn and Rohatgi, 2018

202 of Tnfa and its receptors or major chemokine receptor-ligand subsets persisted in the long term.

203 Toll-like receptor ligands in indoor dust act as environmental adj

204 G-protein-coupled receptors like the human Y(1) receptor (hY(1)R) are prom

205 Receptor-like cytoplasmic kinases (RLCKs) such as ZED1,

206 icient to eradicate the most common cytokine receptor-like factor 2-rearranged (CRLF2-rearranged) Ph-

207 associated kinase (WAK), Catharanthus roseus receptor-like kinase (CrRLK1L) and others involved in ab

208 t is not known whether the plant lysin-motif receptor-like kinase MtLYK10 intervenes in recognition o

209 Receptor-like protein kinases (RLKs) play key roles in r

210 or, myosin XI (a,b), and the putative chitin receptor Lyk5 (a,b,c).

211 ociated truncated O-linked glycans and their receptor, macrophage galactose-type lectin (MGL), on CD1

212 While the inability to utilize the CCR5 receptor maps to a predicted protrusion in the envelope

213 lamine (PE) synergizes with PS to enhance PS receptor-mediated efferocytosis and virus entry.

214 ting cation-independent, mannose 6-phosphate receptor-mediated endocytosis from the cell surface.

215 Withdrawal of GABA(A) receptor-mediated inhibition of the RVLM increases sympa

216 ng us to decouple aggregation from cellular, receptor-mediated internalization.

217 aneous recognition of PE and PS maximizes PS receptor-mediated virus entry and efferocytosis and unde

218 namely, PE synergizes with PS to promote PS receptor-mediated virus infection and clearance of apopt

219 We have recently introduced a quantitative receptor model (SABRE) that includes parameters for Sign

220 old infections (IMIs) after chimeric antigen receptor-modified T-cell (CAR-T-cell) therapy are limite

221 Nuclear receptor modulators, which often feature carboxylic acid

222 h affinity for brain nicotinic acetylcholine receptors (nAChR).

223 nt its inhibition of nicotinic acetylcholine receptors (nAChRs) as a lead for the development of alte

224 vertebrates is based on the assumption that receptor neuron axons exclusively connect to a single gl

225 from patients with anti-N-methyl-d-aspartate receptor (NMDAR) encephalitis alter the levels of dopami

226 activin A regulates phosphorylation of NMDA receptor (NMDAR) subunit GluN2B and that GluN2B-containi

227 is mediated by the NMDA subtype of glutamate receptors (NMDARs).

228 Additionally, BMPR1, a type I BMP receptor normally required for BMP-mediated patterning o

229 D-L1 expression was regulated by the nuclear receptor NR4A1/Sp1 complex bound to the proximal germina

230 The nuclear receptors (NRs), NR2F2 and NR2C2, can bind to (TCAGGG)(n

231 ne activity through the beta-adrenergic-like receptor Octbeta1R drives aversive and appetitive learni

232 s, but whether it represents the main opiate receptor of skin mast cells remains unknown.

233 ion and expands current understanding of the receptors on microglial cells that are engaged by reovir

234 Activation of Tnfa and its receptors or major chemokine receptor-ligand subsets per

235 of the following: (1) L-368899 (an oxytocin receptor [OTR] antagonist) or by (2) bicuculline (a pref

236 r-6 (DR6) on MNs as the top predicted ligand-receptor pair.

237 trogen estetrol and Selective Human Estrogen-Receptor Partial Agonists are being evaluated in endocri

238 However, receptor-level IR (e.g., insulin receptor pathogenic variants, INSR) causes hyperglycemia

239 Here we identify the nuclear receptor peroxisome proliferator-activated receptor (PPA

240 ) and ATP-citrate lyase (ACLY) in a TGF-beta receptor/PI3K/protein kinase B-dependent manner, to regu

241 We have re-examined the roles of the two receptor populations using mice with picrotoxin resistan

242 ression as markers of recurrence in estrogen receptor- positive (ER+) breast cancer tissue.

243 o temperature-sensitive engineered transient receptor potential (TRP) channels.

244 lysosomal calcium efflux channel, transient receptor potential mucolipin 1 (TRPML1).

245 The transient receptor potential vanilloid 1 (TRPV1) channel is activa

246 r receptor peroxisome proliferator-activated receptor (PPAR) delta as key regulator of osteoblast met

247 PGC1alpha [peroxisome proliferator-activated receptor (PPAR) gamma-coactivator 1alpha], PPARalpha, an

248 ipids are endogenous ligands for the nuclear receptor PPARalpha, and we demonstrate that Ppara (-/-)

249 Estrogen receptor (ER), progesterone receptor (PR), and human epidermal growth factor recepto

250 li through the papilla matrix to reach taste receptors, processes that are poorly understood.

251 We compared the requirements for autophagy receptor proteins: OPTN, NBR1, p62, NDP52, and TAX1BP1 i

252 ndividual variability in pattern-recognition receptor (PRR)-induced cytokines in myeloid cells.

253 Peptide receptor radionuclide therapy (PRRT) with (177)Lu-labele

254 Conclusion: Peptide receptor radionuclide therapy can lead to bowel obstruct

255 Before this work, an extracellular auxin receptor - rather than the auxin transporter AUXIN RESIS

256 lentivirus infection alters the frequencies, receptor repertoires, and functions of innate cells in t

257 moke is the first complex odor whose in vivo receptor response pattern has been measured.

258 RIG-I-like receptors (RLR) are cytosolic RNA sensors that signal th

259 re potassium's role in this response and the receptor's effect on intercalated and principal cell fun

260 A number of selective autophagy receptors (SARs) have been characterized that interact b

261 s with HFpEF displayed impaired cardiac beta-receptor sensitivity compared with senior controls.

262 modeling and statistical analyses of T cell receptor sequencing data, we develop a quantitative theo

263 t noncanonical and nontranscription-mediated receptor signalling is more common than hitherto appreci

264 The receptor SpA was attached to BioPE-DOTAP binary lipid bi

265 alanced 5-HT1A/2A activation, either through receptor-specific drug intake, genetically predisposed i

266 It was shown previously that a change in receptor specificity is a hallmark for adaptation to hum

267 on function under redox control include NMDA receptor subunits GluN1 and GluN2A as well as KEAP1 (reg

268 ll known that activation of the kappa opioid receptor system modulates negative affect and dysphoria,

269 pecific innovations in neural, muscular, and receptor systems.

270 Peptides such as gastrin-releasing peptide receptors targeting radiopharmaceuticals are small molec

271 Activation of the T cell receptor (TCR) results in binding of the adapter protein

272 substantial increase in the number of T-cell receptor (TCR) sequences and their cognate antigens.

273 ugh multiple receptors, including the T-cell receptor (TCR), co-receptors, and cytokine receptors.

274 red T cells expressing MCPyV-specific T cell receptors (TCRs) show increased cytokine production, mig

275 sive activation of extrasynaptically located receptors that can impair synaptic plasticity.

276 y subunit-selective agonists and heteromeric receptors that contain binding-impaired subunits, as we

277 lex interaction between the virus and innate receptors that may underlie disease pathogenesis.

278 Besides being regulated by G-protein-coupled receptors, the activity of heterotrimeric G proteins is

279 ective for alpha5-subunit-containing GABA(A) receptors, the derivative SH53d-acid shows superior (>40

280 here that sSORLA binds and activates the EGF receptor to induce downstream signaling through the ERK

281 cell free assay based on the ability of the receptor to prevent alkaline phosphatase from hydrolysin

282 uction of pain depends on coupling of opioid receptors to Galphai/o family members.

283 vo (in animals), using antagonists of opioid receptors to infer endogenous peptide activity, and gene

284 nse to activators of the pattern recognition receptors, toll-like receptor-2 (TLR2) and -4 (TLR4).

285 emory formation, through regulating synaptic receptor trafficking in pyramidal neurons by SorCS2.

286 C-C chemokine receptor type 2 (CCR2) is expressed on monocytes and fac

287 eceptor for cholesterol-rich HDLs, scavenger receptor type B1 (SCARB1).

288 t the first high-resolution structure of the receptor type found in muscle-endplate membrane and in t

289 Stimulation of plasma membrane receptor tyrosine kinases (RTKs), such as the epidermal

290 antibody to urokinase plasminogen activator receptor (uPAR) as a therapeutic strategy to attenuate a

291 to Hebbian plasticity: Ca(2+)-permeable AMPA receptor upregulation, L-type Ca(2+) channel activation,

292 s is followed by discussion of the vitamin D receptor (VDR) that mediates the cellular actions of 1,2

293 unreported post-translational control of LGR receptors via NEDD4/NEDD4L to regulate ISC priming.

294 based on the variable domain of new antigen receptor (VNAR) was used for in vitro selection against

295 sium conductance induced by the somatostatin receptor was also blocked by compound 101 in slices from

296 Ryanodine receptor was protein kinase A-hyperphosphorylated, S-nit

297 here to the surrounding ECM through integrin receptors, we examined the hypothesis that an abnormal E

298 , utilising activated truncated forms of the receptors, we show that ADGRE2/EMR2 and ADGRE5/CD97 are

299 l assays to the evolution of steroid hormone receptors, we show that an ancient hydrophobic interface

300 been identified, together with many "orphan" receptors without known ligands.