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1 spectively, with values of <0.5% ID/g during receptor blockade).
2 ceptor-dependent (IC(50) shifts 90-fold upon receptor blockade).
3 arized in nonrhythmic slices (following AMPA receptor blockade).
4 n, which was normalized by mineralocorticoid receptor blockade.
5 d hence lead to the potential for incomplete receptor blockade.
6 cells and were completely abrogated by IFN-I receptor blockade.
7 duration and failure rate during cholinergic receptor blockade.
8 refrontal cortex, were prevented by 5-HT2A/C receptor blockade.
9 These effects did not depend on angiotensin receptor blockade.
10 logical reduction of fibrosis by angiotensin receptor blockade.
11 nditions in CGD that can be restored by IL-1 receptor blockade.
12 oduction in the presence or absence of IL-33 receptor blockade.
13 howed dose-dependent sensitivity to dopamine receptor blockade.
14 dysfunction, independent of its angiotensin receptor blockade.
15 -dependent decline in cerebral binding after receptor blockade.
16 , neither group was affected by DLS dopamine receptor blockade.
17 ctance analysis during nicotinic and GABA(A) receptor blockade.
18 e raphe neurons were unaffected by ATP or P2-receptor blockade.
19 an effect attenuated by concomitant 5-HT2A/C receptor blockade.
20 ed in vaccinia virus-treated mice with IL-10 receptor blockade.
21 they were eliminated by systemic muscarinic receptor blockade.
22 -specific component acutely regulated by CB1 receptor blockade.
23 re could be effectively rescued by IFN-alpha receptor blockade.
24 tion, whereas SLEs were blocked by glutamate receptor blockade.
25 ts rely on the same mechanism: dopamine D(2) receptor blockade.
26 ustained attention was abolished by alpha(1)-receptor blockade.
27 opagation becomes stereotyped following GABA receptor blockade.
28 response is sensitive to ET(B) but not ET(A) receptor blockade.
29 was attenuated by D2-like (but not D1-like) receptor blockade.
30 of GABAergic signaling and abolished by AMPA receptor blockade.
31 reased pro-nerve growth factor levels or p75 receptor blockade.
32 lating the solitary tract (ST) under GABA(A) receptor blockade.
33 based on differential extrasynaptic/synaptic receptor blockade.
34 ior and which are reversed by the muscarinic receptor blockade.
35 further antagonism by additional thromboxane receptor blockade.
36 ficacy is mediated by dopamine type 2 (D(2)) receptor blockade.
37 partial gamma-aminobutyric acid(A) (GABA(A)) receptor blockade.
38 from Cx30-deficient mice or with purinergic receptor blockade.
39 etains a bursting behaviour after ionotropic receptor blockade.
40 ut were dramatically reduced after Ang-(1-7) receptor blockade.
41 ptor system counteracts the effects of AT(4) receptor blockade.
42 t not its production) were sensitive to NMDA receptor blockade.
43 induces APOBEC3, inhibited by IFN-alpha/beta receptor blockade.
44 with inhibitors of caspases-1 and -5 or IL-1 receptor blockade.
45 cy calculations when considering therapeutic receptor blockade.
46 or 8 weeks PI and was susceptible to 5-HT(3) receptor blockade.
47 rombosis to a similar extent as prostacyclin receptor blockade.
48 reduced or not affected by chronic glutamate receptor blockade.
49 utamatergic activity after chronic glutamate receptor blockade.
50 potential that normally resulted from IP(3) receptor blockade.
51 iduals, men are more responsive to mu-opioid receptor blockade.
52 the resting membrane potential after GABA(A) receptor blockade.
53 iring is selectively attenuated following D1 receptor blockade.
54 could be targeted with inhibitory checkpoint receptor blockade.
55 t block that could be abrogated by nicotinic receptor blockade.
56 tes to calculate the net rate of reversal of receptor blockade.
57 le ATR levels respond to GABA, but not NMDA, receptor blockade.
58 rability due to more selective dopamine (DA) receptor blockade.
59 c-blocker administration or beta2-adrenergic receptor blockade.
60 ercise ischaemia (PEI) and beta1 -adrenergic receptor blockade.
61 subdiaphragmatic vagotomy or corticosterone receptor blockade.
62 parable in extent to that obtained upon IL-1 receptor blockade.
63 re depletion or inositol 1,4,5-trisphosphate receptor blockade.
64 epression was prevented by MOR- but not GABA-receptor blockade.
65 ce indeed became hyperglycemic after insulin receptor blockade.
66 oid synthesis or highly selective prostanoid receptor blockade.
67 presence of adenosine A1 receptor and GABAB receptor blockade.
68 rome features may benefit from interleukin-1 receptor blockade.
69 the airways, which was reversed after IL-10 receptor blockade.
70 antly reduced by N-methyl-d-aspartate (NMDA) receptor blockade.
71 or AMI, possibly mediated by dopamine type 3 receptor blockades.
72 after 24-hour systemic AT(1)R (Ang II type-1 receptor) blockade.
73 tive beneficial effects over beta-adrenergic receptor blockade, a current pharmacological heart failu
74 sponse to DBP-FITC was not affected by IFN-I receptor blockade, a finding consistent with the known d
75 gamma power is significantly reduced by NMDA receptor blockade, a treatment that paradoxically enhanc
77 tibility of alcohol seeking to aDLS dopamine receptor blockade actually predicted the vulnerability t
80 ognition 4 months after pharmacological NMDA receptor blockade already were affected by disrupted chr
85 BA levels, consistent with the view that NOP receptor blockade and DOP receptor stimulation caused sy
86 d no effect on proliferation, concurrent RON receptor blockade and gemcitabine treatment increased ap
87 nsitivity in the mouse using pharmacological receptor blockade and genetic deletion of the channel.
88 nduced by vascular endothelial growth factor receptor blockade and hypoxia (Sugen/hypoxia) as well as
89 animals, vascular endothelial growth factor receptor blockade and hypoxia caused more severe pulmona
90 an be achieved by PRR stimulation, chemokine-receptor blockade and immune modulation of T cell functi
92 O excitation occurs in the presence of GABAA receptor blockade and its EC50 value is two orders of ma
93 ctivation are sensitive to hippocampal D1/D5 receptor blockade and resistant to adrenoceptor blockade
94 nd that the synergistic effect of inhibitory receptor blockade and stimulation of costimulatory recep
95 from the recent clinical studies using IL-6 receptor blockade, and describe potential mechanisms by
96 baseline femoral neck T-score, interleukin-2 receptor blockade, and proteinuria (HR 2.02, 0.69, 0.4,
98 ety and beneficial effects of angiotensin II receptor blockade (ARB) in patients with structural hear
99 emodynamics and cell biology by neurohumoral receptor blockade are evolving, exploring the role and i
100 eir site of origin, important effects of CB1 receptor blockade are expressed via activation of periph
101 either 5-HT2C receptor activation nor 5-HT2A receptor blockade are sufficient to produce a therapeuti
102 l medical therapies, such as beta-adrenergic receptor blockade, are used to slow pathologic aortic gr
104 lthough we have known that mineralocorticoid receptor blockade attenuates cardiovascular and renal in
105 miting laboratory rodents) and that CeA NMDA receptor blockade attenuates cisplatin-induced anorexia
106 acological data showed that CeA AMPA/kainate receptor blockade attenuates cisplatin-induced pica (a p
107 dies demonstrate for the first time that CB1 receptor blockade attenuates DIO-associated inflammation
111 e sensitive than younger myocytes to thyroid receptor blockade by antagonist, NH3, and to the effects
112 ody weight gain; in contrast, intracore NMDA receptor blockade by AP-5 did not inhibit the energy bal
114 al. now report that combining angiotensin II receptor blockade by losartan with beta-blocker treatmen
116 on of TGF-beta signaling through angiotensin receptor blockade can attenuate CS-induced lung injury i
119 ms and brain regions through which adenosine receptor blockade causes arousal are incompletely unders
120 unusual human subject and in the monkey with receptor blockade decreased exposure to organs with high
121 GABAA and AMPA receptor activation but NMDA receptor blockade decreased oscillations only in the DP
122 ting enzyme inhibition nor mineralocorticoid receptor blockade decreased the primary outcome of posto
124 ient as a consequence of genetic deletion or receptor blockade demonstrated normal recruitment and se
125 se, suppressed replication is rescued by IFN-receptor blockade, demonstrating a role for IFN in restr
127 long-term bioluminescence recordings, GABAA receptor blockade desynchronized the Fbxl3(+/+) but not
129 Based on rates of D-xylose absorption, GLP-1 receptor blockade did not affect gastric emptying of a s
130 ocal ATP application; however, purinergic P2-receptor blockade did not affect their CO2/H(+) responsi
132 induction was TLR2/4 independent because the receptor blockade did not interfere with infection-induc
133 KORs in the LC together with beta-adrenergic receptor blockade did not potentiate KOR-induced reinsta
135 In contrast, colony-stimulating factor 1 receptor blockade diminished C-C chemokine receptor type
138 dy aimed to determine the impact of acute B2 receptor blockade during hemorrhagic shock in angiotensi
141 ogical inhibition of PGE2 synthesis and PGE2 receptor blockade enhanced bacterial killing in Mphis.
142 neurons in nigral slices confirmed that NOP receptor blockade enhanced the DOP receptor-induced effe
144 earchlight decoding approach we show that D2-receptor blockade enhances decoding of reward signals in
145 y changes, we examined the effects of GABA-A receptor blockade, finding that picrotoxin (PTX) recapit
146 We also show that maintained darkness and D1 receptor blockade following maintained illumination and
149 S) that act by both CCR5 internalization and receptor blockade had not been reported until recently (
150 sympathetic drive to the heart through beta-receptor blockade has become a standard component of the
152 tion of neprilysin inhibition to angiotensin receptor blockade has been shown to be even more effecti
154 ntroduction of immunotherapy with checkpoint receptor blockade has changed the treatment of advanced
155 nhibition through neprilysin and angiotensin receptor blockade, has led to groundbreaking findings in
156 , including cancer vaccines and coinhibitory receptor blockade, have demonstrated clinical efficacy i
158 ine-treated mice to elucidate how acute NMDA receptor blockade impacts on the properties of functiona
159 tantly, intra-LA NMDA (N-methyl-d-aspartate)-receptor blockade impaired reward-learning performance a
160 ized controlled trial, long-term aldosterone receptor blockade improved left ventricular diastolic fu
161 ently have we learned that mineralocorticoid receptor blockade improves pancreatic insulin release, i
162 cribe for the first time the effect of G-CSF receptor blockade in a therapeutic model of inflammatory
168 showed that this compound provides 90% NK(1) receptor blockade in rhesus brain at a plasma level of 6
169 l function independent of weight loss or CB1 receptor blockade in the brain, suggesting that peripher
170 BA), neuropeptide Y (NPY), or beta-endorphin receptor blockade in the ipsilateral hypothalamic parave
173 eversal learning was improved after dopamine receptor blockade in the nucleus accumbens; the D1R anta
175 sensus on the relative importance of beta(2) receptor blockade in treating glaucoma may have to be re
176 and by examining of the impact of glutamate receptor blockade in two feeding-related targets of vmPF
178 tor were modest or absent, and selective EP2 receptor blockade increased cytokine release in some ins
180 udies provide novel evidence that VTA amylin receptor blockade increases food intake and attenuates t
181 e mice, while systemic 2-AG depletion or CB1 receptor blockade increases susceptibility in previously
183 by prolonged gamma-aminobutyric acid type A receptor blockade is independent of brain-derived nerve
186 by either PA50 mAb or Fab and suggested that receptor blockade is the mechanism by which PA50 neutral
187 eficiency nor pharmacological glucocorticoid receptor blockade lowered elevated blood glucose levels.
189 Altogether, these results suggest that B2 receptor blockade may be a promising strategy to prevent
191 Behavioral impairment following nicotinic receptor blockade may not be due to the inability of the
193 oma cell interactions through specific Notch receptor blockade may represent a promising treatment st
195 indings raise the possibility that A or A(A) receptors blockade might also confer a disease-modifying
199 hysiologic data, the effects of spinal 5-HT3 receptor blockade on behavioral hypersensitivity and neu
200 degradation offsets the effects of oxytocin receptor blockade on both social place preference and cF
201 n VMH GABA levels and the effects of GABA(A) receptor blockade on counterregulatory responses to a st
202 study was to investigate the effect of 5-HT7 receptor blockade on liver regeneration after partial he
203 s neutropenic, suggesting an effect of G-CSF receptor blockade on neutrophil homing to inflammatory s
204 Here, we examined the effects of muscarinic receptor blockade on rule-related activity in the macaqu
205 quencing to explore the effects of H1 and H2 receptor blockade on the exercise transcriptome in human
206 uences of ventral tegmental area (VTA) CB(1) receptor blockade on the wheel-running performances of w
208 nd the antiproteinuric effect of angiotensin receptor blockade or angiotensin-converting enzyme inhib
211 is similar to published effects of nicotinic receptor blockade or desensitization, and is mediated by
213 release evoked electrically during nicotinic receptor blockade or optogenetically by light activation
214 easurement of thrombus height after specific receptor blockade or use of altered proportions of pepti
217 ombined findings suggest that IFN-alpha/beta receptor blockade, particularly when started at early di
219 y paracrine inhibition, because somatostatin receptor blockade potently stimulated glucagon release w
220 discrete-trial procedures employed here, D1 receptor blockade preferential reduces Pavlovian and ope
227 erpolarizing current injection, but not AMPA receptor blockade, prevents synaptic stimulation from fa
229 signals were mediated by B2 receptors and B2 receptor blockade protected against PAC necrosis evoked
232 inant-negative mutant of AMPKalpha1, and P2Y receptor blockade reduced DOR-stimulated glucose uptake.
238 ast, cardiac afferents, in the absence of TP receptor blockade responded consistently to repeated adm
241 Overall, our results suggest that muscarinic receptor blockade results in a bona fide learning impair
242 mma power is significantly reduced with NMDA receptor blockade, revealing a latent cortical network p
244 ET imaging of 22 patients to assess androgen-receptor blockade showed decreased (18)F-fluoro-5alpha-d
247 to reinvigorate Tex cells through inhibitory receptor blockade, such as alphaPD-1, highlights the the
248 smission also blunted central effects of CB1 receptor blockade, such as fear responses and anxiety-li
250 Furthermore, the differential effects of receptor blockade suggest novel methods of immune respon
252 blockade in the NAc, and not local D(1)-like receptor blockade, suggesting a role for glutamate affer
256 immunoabsorption as well as subsequent IL-6 receptor blockade through tocilizumab, a complete and st
257 urophysiological recordings, and cholinergic receptor blockade to delineate the cholinergic contribut
259 igration assays, and antibody-mediated IL-10 receptor blockade to study plasmacytosis-associated IL-1
262 se results demonstrate that, spurred by AMPA-receptor blockade, up-regulation of betaCaMKII promotes
263 nverting enzyme inhibitors or angiotensin II receptor blockade use (OR, 0.42; [95% CI, 0.22-0.80]; P=
264 -converting enzyme inhibitors/angiotensin II receptor blockade use remained independent predictors fo
266 he impact of intrarenal angiotensin II AT(1) receptor blockade using candesartan and mineralocorticoi
267 kade using candesartan and mineralocorticoid receptor blockade using canrenoic acid potassium salt (C
268 onist exenatide, with or without prior GLP-1 receptor blockade using exendin 9-39, on brain responses
271 his study investigated the efficacy of IL-15 receptor blockade using Mut-IL-15/Fc in an outbred non-h
273 e (NHP) model of schizophrenia based on NMDA-receptor blockade using subanesthetic administration of
274 Delta-18 +/- 2%), an effect prevented by V1a receptor blockade (V2255), supporting local dendritic VP
276 (RR 0.45; 95% CI 0.24-0.83), and angiotensin receptor blockade vs placebo (RR 0.65; 95% CI 0.49-0.85)
277 sin II test showed that complete angiotensin receptor blockade was achieved only in the high-dosage g
280 In this subgroup analysis, interleukin-1 receptor blockade was associated with significant improv
282 isms underlying the hypophagic effect of CB1 receptor blockade, we combined the acute injection of th
283 Using bilateral local intracerebral hormone-receptor blockade, we found that ARs and ERs in the song
285 -converting enzyme inhibitors/angiotensin II receptor blockade, were associated with the presence of
287 Vascular endothelial growth factor (VEGF) receptor blockade, which resulted in EC hyperproliferati
288 therapy with NEP-inhibitors and angiotensin-receptor-blockade, which has been shown being a promisin
291 ission) (study 1) and 2) the effect of GLP-1 receptor blockade with exendin (9-39) on glucose toleran
296 n02 lesioning of vmPFC and acute dopamine D1-receptor blockade with SCH39166 in NAc core or shell) co
299 s largely antagonized by selective serotonin receptor blockade, with little further antagonism by add