ライフサイエンスコーパス: receptor subunit

1 (LIF), signal via the common GP130 cytokine receptor subunit.

2 d in the low pH treatment, including a GABAA receptor subunit.

3 ion of membrane expression of the GluN1 NMDA receptor subunit.

4 utation in Gabrg2, a gene encoding a GABA(A) receptor subunit.

5 mologous segments from the rat GluK2 kainate receptor subunit.

6 832407) in GRIK1, encoding the kainate GluK1 receptor subunit.

7 xpression of the GluK5 high-affinity kainate receptor subunit.

8 d in the ion channel pore of the GluN2A NMDA receptor subunit.

9 ately 5 mum, along with GluA1 and GluA2 AMPA receptor subunits.

10 unbiased capture of five alpha or beta GABAA receptor subunits.

11 tor expression and a decrease in GluN2B NMDA receptor subunits.

12 MDA receptors (NMDARs) in PSD via GluN2-type receptor subunits.

13 te, located at the interface of two adjacent receptor subunits.

14 ocated at the interface between two adjacent receptor subunits.

15 e complementary genetically modified GABA(A) receptor subunits.

16 terodimerization of the IL-2Rbeta and gammac receptor subunits.

17 eto2 at these structurally discrete sites on receptor subunits.

18 ls and to other neurons that coexpress these receptor subunits.

19 as exemplified by deficiency of IL-10 or its receptor subunits.

20 tenin-1-mediated dendritic transport of NMDA receptor subunits.

21 eptor (AMPAR) and GluN1 N-methyl-D-aspartate receptor subunits.

22 by reducing proteasomal degradation of GluA1 receptor subunits.

23 and through AMPA and kainate-like glutamate receptor subunits.

24 expression of AMPAR and N-methyl-D-aspartate receptor subunits.

25 UR2 and GLUR3) and GABAA (GABAA1 and GABAA2) receptor subunits.

26 nriched for specific nicotinic acetylcholine receptor subunits.

27 naling were identified, including four GABAA receptor subunits.

28 enzymes with variable substrate-adaptor and -receptor subunits.

29 raction between heterodimeric hIL-12 and its receptor subunits.

30 vity is dependent on the IL-17RA and IL-17RB receptor subunits.

31 ns showed relatively low expression of GABAA receptor subunits.

32 We studied GABAB receptor subunit 1 (GABAB1) splicing in alcoholic postmo

33 thyl-4-isoxazolepropionic acid receptor AMPA receptor subunit 1 (GluA1) through phosphorylation at Se

34 mpetition for IFN-alpha2A binding to the IFN receptor subunit 1 (IFNAR1) and do not involve inhibitin

35 protein causes the degradation of type I IFN receptor subunit 1 (IFNAR1).

36 interferon receptor (IFN-alpha and IFN-beta receptor subunit 1 [IFNAR1]), T cells, and B cells in sp

37 h its receptor IFNAR1 (interferon alpha/beta receptor subunit 1) is vital for host-protective anti-vi

38 ibodies against IFN-gamma and IFN-alpha/beta receptor subunit 1, and their cellular sources were iden

39 ss of PHF6 and the interferon alpha and beta receptor subunit 1.

40 receptor delta and gamma-aminobutyric acid B receptor subunit 1; their differential methylation was a

41 ecific to the gamma-aminobutyric acid type B receptor subunit 1a (GABA(B)R1a).

42 osphorylation of N-methyl-D aspartate (NMDA) receptor subunit 2 (GluN2) that is critical for neuropla

43 ve inhibiting IFN-alpha2A binding to the IFN receptor subunit 2 (IFNAR2).

44 using the N-terminal 150 residues of kainate receptor subunit 2 (KA2) to the recently discovered high

45 ith IL-4 or IL-13 for (1) expression of IL-4 receptor subunits, (2) neutrophil extracellular trap (NE

46 cted down-regulation of N-methyl-D-aspartate receptor subunits 2A and 2B (GluN2A and GluN2B) in the m

47 my (T286A) or binding to NMDA-type glutamate receptor subunit 2B (GluN2B; formerly NR2B; I205K).

48 tic proteins, including N-methyl-d-aspartate receptor subunit 2B (NR2B) and PSD-95.

49 uccessive impairment of N-methyl-d-aspartate receptor subunit 2B (NR2B), postsynaptic density-95 (PSD

50 s the mechanism of KIF17 binding to the NMDA receptor subunit 2B (NR2B).

51 Since the discovery of the glutamate NMDA receptor subunit 3A (GluN3A), the functional role of thi

52 nversely, abrogating the expression of IL-17 receptor subunit a (IL-17Ra) in the neurons of the S1DZ

53 variant in the gene encoding the GluN2A NMDA receptor subunit: a N615K missense variant in the M2 por

54 nd on synaptic mobilization of the glutamate receptor subunit A1 (GluA1) mediated by GR-PO(4) Togethe

55 In addition, we find that all three receptor subunits accumulate rapidly within a subpopulat

56 ull mice developing MM, the Galphai2-coupled receptor subunit activated MEK/ERK and PI3K, resulting i

57 nnels, CaM kinase kinase, and the GluA2 AMPA receptor subunit, akin to a homeostatic process.

58 s did plasma concentrations of interleukin-6 receptor subunit alpha (also based on a single variant;

59 trol and especially identify the IL-27/IL-27 receptor subunit alpha (IL-27RA) axis as a predictor of

60 )-mediated pathway regulating interleukin-27 receptor subunit alpha (IL-27Ra) expression on hematopoi

61 explained by their higher expression of IL-2 receptor subunit alpha (IL-2Ralpha) and gamma chain and

62 eased levels of messenger RNAs encoding IL15 receptor subunit alpha (IL15RA) and IL21 compared with t

63 ndritic cells that overexpress interleukin-3 receptor subunit alpha (IL3RA or CD123).

64 apolipoprotein E and nicotinic acetylcholine receptor subunit alpha 5 genes are associated with lifes

65 re, we showed that macrophages express IL-22 receptor subunit alpha-1 (IL-22Ra1) during pneumococcal

66 c4, Ampd3, Trim63 (MuRF1), and acetylcholine receptor subunit alpha1 (Chrna1).

67 Mice with disruption of the interleukin 11 receptor subunit alpha1 gene (Il11ra1(-/-)) mice and Il1

68 rincipal neurons were found to express GABAA receptor subunits alpha1 , alpha3 , beta2/3 , gamma2 , a

69 However, GABAA receptor subunits alpha1 and gamma2 were upregulated, an

70 Here, the expression of GABAA receptor subunits alpha1, alpha2, alpha3, alpha5, beta2,

71 investigated the distribution of 10 GABA(A) receptor subunits (alpha1, alpha2, alpha3, alpha4, alpha

72 the memory deficits and had reduced GABA(A) receptor subunit alpha2 (GABRA2) expression in lateral h

73 utative trafficking sequences in two GABA(A) receptor subunits: alpha4 and delta.

74 e lacking platelets or the platelet integrin receptor subunit alphaIIb established that the survival

75 ion of Neto1 and 2 with pore-forming kainate receptor subunits also increases the duration of bursts

76 Sharing of receptor subunits among different cytokine receptor comp

77 s a paradoxical enhancement in membrane AMPA receptor subunits, AMPA responsiveness, and the motivati

78 n-6 (IL-6) and IL-27 signal through a shared receptor subunit and employ the same downstream STAT tra

79 ed cell-surface expression of the GluR2 AMPA receptor subunit and increased intracellular Ca(2+) sign

80 The postsynaptic gamma2-GABA(A)-receptor subunit and the presynaptic vesicular inhibitor

81 nregulation and internalization of glutamate receptor subunits and alterations of the dendritic spine

82 the spider Cupiennius salei have 15 Cys-loop receptor subunits and an acetylcholine-binding protein (

83 to investigate changes in synaptic glutamate receptor subunits and functional consequences.

84 Transcripts of NMDA receptor subunits and GABAergic interneuron markers, as

85 dition to expected components including GABA receptor subunits and gephyrin, several novel proteins w

86 lexity in the mutual regulation of the TRC40 receptor subunits and raise the question as to the role

87 ession analysis revealed upregulation of IL6 receptor subunits and signal transducers CD126 and GP130

88 ng the levels of N-methyl-D-aspartate (NMDA) receptor subunits and the HSA21 proteins amyloid beta (A

89 We observed that NMDA receptor subunits and their transcripts are increased in

90 ociated with delayed expression of glutamate receptor subunits and transporters.

91 ts of a progressive loss of GABA(A) alpha(1)-receptor subunits and up-regulation of alpha(2/3)-recept

92 electively inhibited GluK1 and GluK2 kainate receptor subunits, and also GluK1/GluK5 and GluK2/GluK5

93 Our results reveal a novel GABA(A) receptor subunit- and input-specific form of inhibitory

94 eto1 and Neto2, which impact KAR gating in a receptor subunit- and Neto isoform-specific manner.

95 Receptor subunits are arranged in a 1-2-1-2 fashion, dem

96 We find that receptor subunits are arranged nonstochastically, with t

97 variants of several nicotinic acetylcholine receptor subunits are associated with nicotine dependenc

98 1a(+/-) ), and in which the alpha(2) GABA(A) receptor subunits are expressed at higher levels relativ

99 in the absence of filamin, type-A glutamate receptor subunits are lacking at the postsynapse, while

100 A disadvantage is that the different receptor subunits are not identified.

101 GABA alpha1 receptor subunits are significantly down-regulated in la

102 We found that the minor receptor subunit, ASGR2, is not required for effective i

103 ncrease in phosphorylation of the AMPA GluA1 receptor subunit at serine 831 (S831), a CaMKII site, al

104 ons and the expression of the alpha6 GABA(A) receptor subunit at the mossy fiber-granule cell synapse

105 enhanced ability to increase glutamate AMPA receptor subunits at the cell surface of wild type neuro

106 3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor subunits at the putative time of memory restabi

107 ine in the transcript encoding the glutamate receptor subunit B, glutamate receptor ionotropic AMPA 2

108 ene encoding the GluN2A N-methyl-d-aspartate receptor subunit being most often affected.

109 f patients and treatment with IL-1 and IL-12 receptor subunit beta 1 (Rb1) antibodies may also be use

110 he gamma-aminobutyric acid type A (GABA(A) ) receptor subunit beta2.

111 RG3, encoding gamma-aminobutyric acid type A receptor subunits (beta3, alpha5, gamma3).

112 - and GluA2-containing AMPARs, regardless of receptor subunit binding specificity, through increased

113 nteracts with the bone morphogenetic protein receptor subunits BMPR1a and BMPR1b.

114 depended on the ectopic expression of GM-CSF receptor subunits by tumors.

115 Receptor subunits can exist in surface-bound or soluble

116 ich incomplete agonist occupancy at the four receptor subunits can provide activation without inducin

117 way mediated via the nicotinic acetylcholine receptor subunit CHRNA2.

118 .SIGNIFICANCE STATEMENT The alpha5 nicotinic receptor subunit (Chrna5) is important for attention, bu

119 and function of the nicotinic acetylcholine receptor subunit cluster of alpha3, alpha5, and beta4.

120 AT3(glo)/CMKII mice revealed changes in NMDA receptor subunit composition and altered NMDA-dependent

121 utamatergic transmission and changes in AMPA receptor subunit composition at 72 h postsurgery.

122 lates with a change in glutamate and glycine receptor subunit composition quantified via mRNA levels.

123 agonist is heavily dependent upon the GABAA receptor subunit composition underpinning tonic inhibiti

124 iderable alignment of synaptic AMPA and NMDA receptor subunit composition within specific subtypes of

125 ive zones and altered postsynaptic glutamate receptor subunit composition, coinciding with a reductio

126 Met) mice, suggesting a modification of NMDA receptor subunit composition.

127 very likely resulting from changes in GABAA receptor subunit composition.

128 icity, demonstrating its reliance on GABA(A) receptor subunit composition.

129 he cell type studied, their respective GABAA receptor subunit compositions, and critically, on the am

130 for glycinergic and glutamatergic ionotropic receptor subunits, confirming a switch from Glyalpha2 to

131 ayer 5; we found increased numbers of alpha1 receptor subunit-containing GABAergic synapses detected

132 istinct interneuron types have similar GABAA receptor subunit content.

133 ata indicate that the GluN2B and GluN2D NMDA receptor subunits contribute to synaptic activity in the

134 ceptor to both Glu and IVM, and improved the receptor subunits' cooperativity.

135 ) blockade or genetic deletion of the GB(1a) receptor subunit disrupts homeostatic regulation of syna

136 The addition of the beta1a dihydropyridine receptor subunit enhanced FRET to the II-III loop, thus

137 e-protective EPO receptor, comprising an EPO receptor subunit (EPOR) and the common beta-chain (CD131

138 pliced, flip and flop variants of GluA1 AMPA receptor subunit exhibit no functional difference in hom

139 Differences in GABAA receptor subunit expression between the human and rodent

140 Furthermore, reducing nicotinic ACh receptor subunit expression in MBONs compromises odor-ev

141 rotein 130 (gp130) is the signal transducing receptor subunit for cytokines of the interleukin-6 (IL-

142 pentameric GABAA receptors require multiple receptor subunits for their synaptic localization in bas

143 including the expression switch of the NMDA receptor subunits from 2B to 2A, which is dependent on b

144 inhibition is marked by a switch in glycine receptor subunits from neonatal to adult form around the

145 al staining intensity for all positive GABAA receptor subunits from the dorsolateral pole to ventrome

146 We examined GABA(A) receptor subunits GABA(Aalpha1) and GABA(Aalpha2) in the

147 ositive for a gamma-aminobutyric acid (GABA) receptor subunit (GABAA Ralpha1 ), and that a synaptic r

148 Further, expression of an important GABAA receptor subunit, GABAAR alpha1, was significantly atten

149 of CHOP prevented heterodimerization of the receptor subunits GABAB1 and GABAB2 and subsequent forwa

150 short splicing variants in addition to GABAB receptor subunit GABAB1a, the longest known major transc

151 xploratory behavior) and whole-brain GABA(A) receptor subunits (gabra1, gabra2, gabrd, & gabrg2) in r

152 To address this, we created 2 NOD- scid IL-2 receptor subunit gamma ( IL2rg) (null) (NSG) strains tha

153 r nucleases (TALENs) to target interleukin-2 receptor subunit gamma (IL2RG) in pronuclear stage marmo

154 in-interacting domain of GABA type A (GABAA) receptor subunit gamma2 (TAT-GABAgamma2) and muscimol, a

155 s variant rs16969968 in the alpha5 nicotinic receptor subunit gene (CHRNA5) is the strongest genetic

156 ediate early genes Fos and FosB and the NMDA receptor subunit gene Grin2a in only Fos-positive neuron

157 ants, and those variants, as in GluA2-4 AMPA receptor subunits, generally show different properties.

158 0 epilepsy-related genes including all GABAA receptor subunit genes (GABRs), and we identified six de

159 Mutations in GABAA receptor subunit genes are frequently associated with ep

160 sense variants in the integrin alphaIIbbeta3 receptor subunit genes ITGA2B and ITGB3 identified by wh

161 ating synaptic transmission, including GABAA receptor subunit genes.

162 the "photoswitch-ready" version of a GABA(A) receptor subunit genomically replaces its wild-type coun

163 in the common gamma (gammac) chain cytokine receptor subunit give rise to severe combined immunodefi

164 -hydroxy-5-methyl-4-isoxazolepropionic acid) receptor subunit GluA1 C terminus (Ser818, Ser831, Thr84

165 r phosphorylation of the AMPA-type glutamate receptor subunit GluA1 on Ser-845 by PKA and for synapti

166 ceptors regulate the phosphorylation of AMPA receptor subunit GluA1 via a signaling pathway linking c

167 ated region of Gria1, which encodes the AMPA receptor subunit GluA1, to pull down miRNAs binding to i

168 hydroxy-5-methyl-4-isoxazole propionic acid) receptor subunit GluA1.

169 sities of synapse-sized clusters of the AMPA receptor subunit GluA1.

170 droxy-5-methyl-4-isoxazole propionate (AMPA) receptor subunits GluA1 and GluA2 in prefrontal neurons.

171 lted in increased surface expression of AMPA receptor subunits GluA1 and GluA2.

172 -fold), GluK1b (5-fold), as well as the AMPA receptor subunit GluA1i (5-fold).

173 2 may be mediated by the AMPA-type glutamate receptor subunit GluA2, which can become associated with

174 . (2017) reveal a critical role for the AMPA receptor subunit GluA3 in cerebellar synaptic plasticity

175 EMENT We report novel roles of the glutamate receptor subunit GluA3 in synaptic transmission in synap

176 The AMPA-receptor subunit GluA4 is expressed transiently in CA1 p

177 on PV interneurons are dependent on the AMPA receptor subunit GluA4, which is regulated by presynapti

178 mpal neurons, we discovered that the kainate receptor subunit GluK2 and the auxiliary subunit Neto2 s

179 ex and the transmembrane M3 helix of kainate receptor subunit GluK2.

180 aled decreased expression levels of the NMDA receptor subunit GluN1 and the postsynaptic density prot

181 on function under redox control include NMDA receptor subunits GluN1 and GluN2A as well as KEAP1 (reg

182 PFC that include ERK/MAP kinase and the NMDA receptor subunits, GluN1 and GluN2B.

183 ressing recombinant NMDA family of glutamate receptor subunits, GluN1/GluN2A, we found that NPA has a

184 , we found that one of these positions, NMDA receptor subunit, GluN2A(F636), can strongly regulate et

185 inase II (CaMKII) to the NMDA-type glutamate receptor subunit GluN2B is an important control mechanis

186 sphatase (STEP) are known to target the NMDA receptor subunit GluN2B on tyrosine 1472, which is a cri

187 3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor subunit GluR1, GABABR1, and GABABR2 levels, whe

188 3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor subunit GluR4.

189 ontrols the synaptic levels of the glutamate receptor subunits GluRIIA, GluRIIC, and GluRIID.

190 he disease state, including AMPA and kainate receptor subunits, glutamate transporters EAAT1 and EAAT

191 al treatment with antisense directed to IL-6 receptor subunit gp130 (gp130), but not to tumor necrosi

192 r1), Bdnf and its receptor (Trkb), glutamate receptor subunits (Gria1, Gria3, Grm1), and epigenetic e

193 substitution (p.A636T) occurs in a glutamate receptor subunit, GRIA1.

194 the homologous but distinct mouse glutamate receptor subunit Grid2 is associated with Lurcher ataxia

195 t-term habituation in mice lacking the NMDAR receptor subunit Grin2a (which also shows association to

196 with NRF1, leading to suppression of a NMDA receptor subunit Grin2A.

197 tonin receptors (Htr1a, Htr2a) and glutamate receptor subunit Grin2b, were modified in the limbic sys

198 munication Variations in genes encoding NMDA receptor subunits have been found in a range of neurodev

199 y number variation studies [3-7], fewer GABA receptor subunits have been observed in the post-mortem

200 ngle gene coding for a high-affinity kainate receptor subunit (i.e., grik4) in a limited area of the

201 IFN-alpha/beta or IFN-I) signals through two receptor subunits, IFNAR1 and IFNAR2, to orchestrate ste

202 cropatterning of the type I interferon (IFN) receptor subunit IFNAR2 fused to the HaloTag was achieve

203 riptional silencing of the unique IFN-lambda receptor subunit (IFNLR1) in a cell-type-specific manner

204 in muscles specifically increased glutamate receptor subunit IIA (GluRIIA) level and the frequency o

205 ional assays have identified residues of the receptor subunit IL-1Rrp2 needed for cytokine recognitio

206 in M receptor beta and the cytokine-specific receptor subunit IL-31Ralpha, of which there are several

207 tibodies against IL-13 or the IL-4 and IL-13 receptor subunit IL-4Ralpha, as well as an antibody agai

208 Upregulation of IL-13, its receptor subunits IL-13Ralpha1 and IL-13Ralpha2, and sha

209 okine family comprising IL-17A to IL-17F and receptor subunits IL-17RA to IL-17RE represents a geneti

210 dy against IL13, prevents its binding to the receptor subunits IL13RA1 and IL13RA2.

211 Our results showed that Il25 and its receptor subunit, Il17rb, were upregulated during a prim

212 oxed CNTFRalpha mice depletes this essential receptor subunit in a large subset of motor neurons (and

213 nclude that GluK1 is the predominant kainate receptor subunit in cb1 and cb3 Off bipolar cells.

214 fragments that bridge together two different receptor subunits in a cytokine-receptor complex, precis

215 ment or specific deletion of beta2 nicotinic receptor subunits in dopamine neurons mitigates aberrant

216 To identify the role of individual receptor subunits in ethanol-induced behaviors, we devel

217 flippase-dependent expression of GFP-tagged receptor subunits in single neurons and 'FlpTag', a vers

218 pecific role of GluN2B, GluN2A, and dopamine receptor subunits in the actions of NMDAR PAM vs. antago

219 tor subunits and up-regulation of alpha(2/3)-receptor subunits in the dentate nucleus, and correspond

220 xtensive hydrophobic interfaces between AMPA receptor subunits in the ion channel.

221 The distribution of GABA(A) receptor subunits in the rhesus monkey was highly hetero

222 xpressed with Agam/Orco (odorant receptor co-receptor subunit) in Xenopus oocytes and assayed by two-

223 in schizophrenia of CHRNA7, the gene for the receptor subunit, in the neurobiology of deficits in att

224 iple factors including assembly of different receptor subunits, interaction with auxiliary subunits,

225 structural differences in binding subsites, receptor subunit interfaces, or transmembrane regions.

226 g to propofol-selective binding within GABAA receptor subunit interfaces, with stable hydrogen bonds

227 ased hippocampal expression of the GABAB(1b) receptor subunit is associated with a depression-like ph

228 ken together, the data suggest that GABAB(1) receptor subunit isoforms differentially regulate the de

229 Fibrinogen receptor subunits Itga2b (alphaIIb) and Itgb3 (beta3) mR

230 , we trained wild-type and alpha-9 nicotinic receptor subunit knock-out (KO) mice, which lack choline

231 Using multiple cytokine and cytokine receptor subunit knockout mice, we demonstrate that stem

232 GRIA4 encodes an AMPA receptor subunit known as GluR4, which is found on excit

233 en the interleukin-2 cytokine and one of its receptor subunits, leading to a 15-fold enhancement in i

234 A multitude of 18 iGluR receptor subunits, many of which are diversified by spli

235 style and that expression of select GABA(A) receptor subunits may be one of the underlying mechanism

236 4, alpha3, and beta2 nicotinic acetylcholine receptor subunit mRNA levels in the nucleus accumbens an

237 NrCAM formed a complex in brain with Sema3F receptor subunits Neuropilin-2 (Npn-2) and PlexinA3 (Ple

238 antibodies (AB) against N-methyl-D-aspartate receptor subunit NR1 (NMDAR1) are highly seroprevalent i

239 an upregulation of the N-methyl-D-aspartate receptor subunits NR1 and NR2A.

240 eroprevalence (~10%) of N-methyl-D-aspartate-receptor subunit-NR1 (NMDAR1) autoantibodies (AB) in hea

241 While clonal depletion of the NMDA receptor subunit NR2 results in their rapid elimination

242 Furthermore, the cAMP effector PKA, the NMDA receptor subunits NR2A and -B, as well as PSD95, were te

243 toxicity, as the N-methyl-d-aspartate (NMDA) receptor subunit NR2B was up-regulated in the cultures.

244 re we show that the F-box protein FBXL2 (the receptor subunit of one of 69 human SCF (SKP1, CUL1, F-b

245 -binding CXXHX16H motif on the sulphonylurea receptor subunit of the channel, and mutagenesis togethe

246 130 (gp130) is the common signal-transducing receptor subunit of the interleukin-6 (IL-6) family, whi

247 enes is GRIK4, a gene coding for a glutamate receptor subunit of the kainate type.

248 the USP47-associated betaTrCP, the substrate-receptor subunit of the SCF(betaTrCP) ubiquitin ligase,

249 thickness and an upregulation of GluA3 AMPA receptor subunits on bushy cells.

250 ce implicates gamma-aminobutyric acid type A receptor subunits on chromosome 15q12 as candidate genes

251 membrane protein translocase but not the two receptor subunits, one of which is essential for protein

252 ng that is mimicked by activating lesions in receptor subunits or downstream mediators as well as abe

253 e cytokine subunit p19 and the soluble alpha receptor subunit p40, binds to a receptor complex consis

254 fly antennal tissue and identified IR8a as a receptor subunit physically associated with IR64a by mas

255 rs (USERs) that allow activation of a single receptor subunit population sensitized to extremely low

256 r targeted-ABPP and observed a lack of GABAA receptor subunit protection.

257 ably, we only observed epsilon acetylcholine receptor subunit protein upregulation and activity in 3D

258 The discovery of interleukin (IL)-6 and its receptor subunits provided a foundation to understand th

259 ion increases the surface expression of AMPA receptor subunits, providing insight to the mechanism by

260 pha1 , alpha3 , beta2/3 , gamma2 , and GABAB receptor subunits R1 and R2.

261 p(-/-) neurons as the alpha1 to alpha2 GABAA receptor subunit ratio was increased.

262 compression, the pattern of AMPA and GABAA/B receptor subunits remain significantly altered in a laye

263 mploy small diffusible molecules and involve receptor subunits resembling highly conserved G-protein

264 Based upon (3)H incorporation into receptor subunits resolved by SDS-PAGE, there was etomid

265 uitinated in cells, especially the ubiquitin receptor subunit, Rpn13.

266 tations in CSF2RA or CSF2RB, encoding GM-CSF receptor subunits); secondary PAP results from various u

267 reduction in the expression of distinct NMDA receptor subunits selectively in identified mesolimbic D

268 tion demonstrates striking interfacial GABAA receptor subunit selectivity in the native milieu, sugge

269 ction of differential N-glycan processing or receptor subunit selectivity.

270 and spatiotemporal correlation of individual receptor subunits showed ligand-induced dimerization and

271 also known as GluRA or GluR1) AMPA glutamate receptor subunit, shows genome-wide association to schiz

272 Here we investigate the input and GABA(A) receptor subunit specificity of inhibitory synaptic plas

273 ng proteins (including the high-affinity IgE receptor subunits, spleen tyrosine kinase, and phospholi

274 pregulated the expression of IL-33 and IL-25 receptor subunits (ST2 and IL-17RA).

275 h had altered relative expression of GABA(A) receptor subunits, suggesting that some other still unkn

276 eased taste bud expression of mRNA for sweet receptor subunits T1R (Taste receptor type) 2 and 3, as

277 population has defined three regions in each receptor subunit that are under selective pressure, whic

278 (BLA) revealed an increase in the GluA1 AMPA receptor subunit that correlated with SEFL.

279 This targeting of a receptor subunit that is common to all members of an oth

280 th mice deficient in GluK1 and GluK2 kainate receptor subunits to assess the role of GluK1 kainate re

281 ab) therapy targeting cell surface-expressed receptor subunits to inhibit T cell proliferation has on

282 h age-dependent suppression of amygdala AMPA receptor subunit trafficking, (2) maternal presence supp

283 amygdala (BLA), and plasticity-related AMPA receptor subunit trafficking.

284 age both the BLA and plasticity-related AMPA receptor subunit trafficking.

285 3, dynorphin-B, and phosphorylated glutamate receptor subunit, type 1 expression.

286 ide important insight into the role of GABAA receptor subunit under- or overexpression in disease sta

287 expression and phosphorylation of glutamate receptor subunits underlie the alterations in LTP and th

288 amma molecule (gammac) is a shared signaling receptor subunit used by six gammac-cytokines.

289 This requires bridging receptor subunits via covalent attachment of 4,4'-bis(ma

290 rformed, and subcellular expression of GABAA receptor subunits was analyzed semiquantitatively.

291 3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor subunits was impaired in the GluD1 knockout mic

292 stably expressing different combinations of receptor subunits, was developed.

293 gene expression levels of glutamate and GABA receptor subunits were compared between sedentary and ph

294 and signaling in cells in which both hIL-12 receptor subunits were functionally deleted.

295 gene expressions of AMPA and NMDA glutamate receptor subunits were markedly increased after cisplati

296 uning and switch in the N-methyl-D-aspartate receptor subunit, which are relevant to autism and other

297 ng advantage of various agonists binding the receptor subunits with different affinities and rate con

298 ising number of mutations found in glutamate receptor subunits, with the GRIN2A gene encoding the Glu

299 beta2/3 , and gamma2 ) and GABAB (R1 and R2) receptor subunits within the normal human STN.

300 We then show that GluN1 and GluA1 receptor subunits within these neuronal subpopulations m