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1 le KT dimensions and improve marginal tissue recession.
2 linical treatments in patients with gingival recession.
3 pratarsal location after levator aponeurosis recession.
4 ot adult mass and experience subsequent mass recession.
5 ns following initial bilateral medial rectus recession.
6 e 64 untreated sites experienced increase in recession.
7 in consumption volume triggered by economic recession.
8 nfluence the degree of mass overshooting and recession.
9 (73%) treated with lateral rectus muscle re-recession.
10 26 with unilateral lateral rectus muscle re-recession.
11 es has been in decline since the most recent recession.
12 r AHP was corrected by performing a bimedial recession.
13 n, radiographic bone fill (RBF), and mucosal recession.
14 blems before and during the current economic recession.
15 used significant crayfish mortality and gill recession.
16 DM-adjacent untreated sites without baseline recession.
17 mental health effects of the global economic recession.
18 hniques at reducing CDH in cases of gingival recession.
19 est was significantly greater for teeth with recession.
20 ed, and the ADM-adjacent sites with baseline recession.
21 No patients underwent medial rectus recession.
22 econstruction, without needing medial rectus recession.
23 sustained calving losses and more rapid ice recession.
24 ew and existing recipients of SNAP after the recession.
25 e effect against the negative impact of this recession.
26 flap (CAF) procedures in localized gingival recessions.
27 rioration in quality of care during economic recessions.
28 e treatment of Miller Class I or II gingival recessions.
29 ortality among older workers declines during recessions.
30 ia following bilateral lateral rectus muscle recessions.
31 zes the financial system and causes economic recessions.
32 topic increase during and immediately after recessions.
35 ection or resection combined with antagonist recession (12 with esotropia and 19 with exotropia; mean
36 cation or plication combined with antagonist recession (13 with esotropia and 9 with exotropia; mean
39 of both elevators (bilateral superior rectus recession 5-8 mm, bilateral inferior oblique recession o
40 cession-resection (bilateral superior rectus recession 6-9 mm; bilateral inferior rectus resection 5-
41 has increased since the onset of the global recession, a trend that may have long-term health and so
42 dal behavior rise with the continuing global recession, additional suicide prevention efforts and lon
44 elected from 11 patients with Miller Class I recessions affecting canines or premolars in the maxilla
48 l patients underwent bilateral medial rectus recession and a lateral rectus resection in the nondomin
50 d to assess the association between economic recession and adult mortality in Brazil and to ascertain
56 eories were unable to foresee the last Great Recession and could neither predict its prolonged durati
60 mals of extreme elevation respond to glacial recession and past harvest, we combined our fieldwork wi
61 r fluid (GCF) samples from sites of gingival recession and saliva; and 2) clinical outcomes of corona
62 association between the presence of gingival recession and the condition of radiographic buccal bone,
64 ncisors and molars and with visible [>=2 mm] recession) and to calculate propensity scores (PSs); 2)
65 es were measured at every visit whereas IPD, recession, and bleeding on probing were assessed at 8, 1
68 clinician-reported intercostal or subcostal recession, and clinician-reported wheeze on auscultation
69 o improve patient comfort, to prevent future recession, and in conjunction with restorative, orthodon
70 acing record-breaking unemployment, economic recession, and reduced wages, we can anticipate an incre
71 017 classification of phenotype and gingival recession, and to stress why it should be fully integrat
72 clinical attachment level (CAL); 3) gingival recession; and 4) percentage of sites with bleeding on p
73 previous orthodontic treatment; 4) gingival recession; and 5) band of keratinized gingiva for each o
74 h increased volatility and deep, short-lived recessions; and alternation of high- and low-output stat
75 riods of higher unemployment rates, that is, recessions, are associated with a moderate but significa
76 an recession depth (0.21 versus 0.39 mm) and recession area (0.31 versus 0.53 mm(2)) after 6 months w
77 ls, case series, and case reports evaluating recession areas that were treated by means of RC procedu
78 ent untreated sites which had presented with recession at baseline but were not treated showed a sign
81 During the 12-month study period the mean recession at sites with PreGR >/=2 mm decreased signific
82 ators, included CAL, probing depth, gingival recession, bleeding on probing (BOP), visible plaque, su
83 the post-surgery increase in buccal gingival recession (bREC), effect of a connective tissue graft (C
84 ed to snow drought and earlier soil moisture recession, but summer precipitation changes remain highl
85 evaluate the treatment of localized gingival recessions by using gingival unit grafts (palatal tissue
87 erent clinical attachment level and gingival recession changes by the end of the maintenance period.
88 nd narrow gingiva tend to have more gingival recession compared with those with thick and wide gingiv
89 MW, or CAL of Miller Class I and II gingival recessions compared with the other treatment modalities.
92 verage recession coverage (ARC) and complete recession coverage (CRC) were evaluated 3 and 6 months a
93 ue grafting for soft tissue augmentation and recession coverage at teeth and dental implant sites.
94 ession defect treatment, to provide adequate recession coverage, the periodontal profession continues
95 er-decadal variability in growth cycles (the recession cycle), highlighting periods of large slow gro
97 CAF) have been deemed the gold standard for recession defect treatment, to provide adequate recessio
99 s presenting with twenty-one Millers Class I recession defects (isolated or adjacent multiple) were s
100 st or control treatments for Miller Class II recession defects appear to lead to stable, clinically e
101 rformed on single tooth Miller Class I or II recession defects at either premolar or anterior sites i
106 aterial) influence root coverage outcomes in recession defects treated with a coronally advanced flap
107 y, 15 patients with 38 Miller Class I and II recession defects underwent CTG or GTR according to rand
108 d controlled trial, Miller Class II gingival recession defects were treated with either a connective
110 ngle-tooth recession defects, multiple-tooth recession defects, and additional focused questions on r
111 l comparison of matched-pair, within-patient recession defects, collagen matrix (CMX) + CAF therapy w
112 iew, which covered treatment of single-tooth recession defects, multiple-tooth recession defects, and
113 ss the treatment outcomes for multiple-tooth recession defects, oral sites other than maxillary canin
114 tion: For Miller Class I and II single-tooth recession defects, SCTG procedures provide the best outc
116 possible for single-tooth and multiple-tooth recession defects, with SCTG procedures providing the be
122 eeding score (FMBS), probing depth (PD), and recession depth (RD) were recorded at baseline and 1, 3,
124 mary outcomes were the changes in slopes for recession depth (REC), keratinized tissue width (KTW), a
125 cedures can provide significant reduction in recession depth and CAL gain for Miller Class I and II r
127 There was no difference in the midfacial recession depth and recession width at the test and cont
128 on probing depth, clinical attachment level, recession depth and width, amount of keratinized tissue,
129 The primary outcome parameter, change in recession depth at 5 years, demonstrated statistically s
130 ugh there were no significant changes in the recession depth between 3 and 240 +/- 12 months in both
131 lagen matrix led to the best improvements of recession depth, clinical attachment level (CAL) gain, a
132 procedures provide significant reduction in recession depth, especially for Miller Class I and II re
136 dies) showed significantly increased odds of recession development long term, regarding either number
139 Considering looming fatality and economic recession, effective policy making based on ongoing COVI
141 the development and progression of gingival recession, especially when restorative margins may inter
144 es (control group), with or without gingival recession (GR) and with attached gingiva, were left untr
145 evaluate the 2-year term results of gingival recession (GR) associated with non-carious cervical lesi
146 of attached gingiva associated with gingival recession (GR) at baseline were treated with FGGs in a p
147 tcomes after treatment of localized gingival recession (GR) by a coronally advanced flap (CAF) combin
148 s presenting with 21 Miller Class I gingival recession (GR) defects (isolated or adjacent multiple) w
149 g-term outcomes of untreated buccal gingival recession (GR) defects and the associated reported esthe
150 roaches for the treatment of single gingival recession (GR) defects are documented in the literature.
151 he treatment of localized maxillary gingival recession (GR) defects, 1 and 5 years after surgical pro
157 tached gingiva (AG) associated with gingival recession (GR) treated with FGG; and 2) contralateral si
158 clinical attachment level (CAL) and gingival recession (GR) were evaluated at 3 and 6 months and bone
159 ), clinical attachment level (CAL), gingival recession (GR), and radiographic bone level (BL) were co
160 nt level (CAL), probing depth (PD), gingival recession (GR), bleeding on probing (BOP), plaque index
161 en toothbrushing and development of gingival recession (GR), but relevant GR data for the multidirect
165 ], clinical attachment level [CAL], gingival recession [GR]) and radiographic (defect Bone level [(DB
166 linical attachment level [CAL], and gingival recession [GR]) and radiographic (defect bone level [DBL
167 CTG + CAF resulted in greater reductions in recession, greater percentage of root coverage, and incr
170 ea (AERSA) as a prognostic test for gingival recessions (GRs) and to compare the predictive value of
173 ing to root coverage (3,539 treated gingival recessions [GRs]), and 10 for non-root coverage procedur
176 th (PD), clinical attachment level (CAL) and recession height (GRH), recession width (GRW) and KGW me
178 depth (PD), clinical attachment level (CAL), recession height (RH), width of keratinized gingiva (WKG
180 Data on Class I, II, III, and IV gingival recessions, histologic attachment achieved after treatme
182 , keratinized tissue width (KTW), horizontal recession (HR), mucogingival junction localization, and
183 nts to factors capable of amplifying glacier recession in addition to climatic change along the Himal
184 ), determinate growth patterns, such as mass recession in birds (weight loss prior to fledging, prece
186 trends in Spain before and during the Great Recession in different socioeconomic groups, quantifying
188 probing, clinical attachment level, gingival recession, interleukin-1beta, tumor necrosis factor (TNF
191 dv), 89 underwent UMRadv with lateral rectus recession (LRc), and 34 underwent bilateral medial rectu
197 D = -0.48, 95% CI = -0.70 to -0.27), mucosal recession (MR) (WMD = -0.60 mm, 95% CI = -0.85 to -0.36
198 ere included, with data on midfacial mucosal recession (MR) of immediately placed implants following
199 CI = 0.12 to 0.59 mm, P = 0.003) for mucosal recession (MR); a WMD of 0.13 mm (95% CI = -0.11 to 0.36
200 uccal (bREC) and interdental (iREC) gingival recession observed at 6 months after treatment of period
202 < 0.0001), and minimal increase in gingival recession of 0.23 +/- 0.62 mm (P = 0.168) were observed.
203 SNAP) expanded significantly after the Great Recession of 2008-2009, but no studies have characterize
208 of the total glacier population (~10%), the recession of lake-terminating glaciers accounted for up
209 ides a principled rationale for the apparent recession of the ascending pathway in motor cortex.
212 at these differences corresponded to general recession of the midface and superior displacement of th
213 wever, NIPSA resulted in significantly lower recession of the tip of the interdental papilla compared
214 tissue graft (CTG) for treatment of multiple recessions of Miller Classes I and II over a short perio
216 ur understanding of the effects of the Great Recession on health and add to growing literature sugges
218 ates during and after the 1980 and 1981-1982 recessions, on rates of subsequent adolescent substance
219 es showed a tendency to increase in existing recession or develop new recession during the 18- to 35-
221 atch with prey such that the extent of range recession or local adaptation may appear as a geographic
224 ed statistically significant improvements in recession over baseline, although intergroup comparisons
226 many advantages, carries a risk of gingival recession, papilla loss, collapse of ridge contour, and
227 nt decreases were recorded in both groups of recession parameters compared with baseline measurements
229 solitarious phase of each subspecies during recession periods to understand whether both subspecies
230 ch from a harmless solitarious stage, during recession periods, to swarms of gregarious individuals t
231 xamination including probing depth, gingival recession, plaque index, and bleeding on probing was per
232 D, clinical attachment level (CAL), gingival recession, plaque index, GI, and bleeding on probing (BO
233 d that as unemployment rates increase during recessions, population mortality actually declines.
234 ntries, our analysis suggests that, although recessions pose risks to health, the interaction of fisc
235 plaque score), bleeding on probing, gingival recession, probing depth (PD), and vertical (VAL) and ho
237 to describe the spatial-temporal spread and recession process of floodwaters in urban road networks.
238 ediment supply and rapid hydrograph rise and recession produced the conditions for these exceptional
239 tarious distributions and forecast potential recession range shifts under two extreme climate change
240 orthern clade could contract its solitarious recession range, while the southern clade is likely to e
242 of multiple Class III-IV Miller periodontal recession (REC) defects on mandibular anterior teeth.
243 D), clinical attachment loss (CAL), gingival recession (REC), and bleeding on probing (BoP) being mea
244 ation of this new classification of gingival recessions, recent articles still report data based on p
246 nd of T3, 83% of the 64 treated sites showed recession reduction (RecRed), whereas 48% of the 64 untr
249 abismus who underwent surgical correction by recession, resection, advancement, or a combination of b
253 uated the relative effectiveness of combined recession-resection of vertical rectus muscles versus su
254 From 1 to 9 years, the ADM-treated isolated recessions showed a relapse from 77% to 62% mRC (P <0.05
255 017 classification of phenotype and gingival recession successfully incorporated the most relevant pr
256 s related to the 2016-2017 Nigerian economic recession that influenced care-seeking and hospital func
258 During the period encompassing the Great Recession, the magnitude of these associations doubled i
259 tions is unclear and the ability of gingival recession to predict underlying buccal bone deficiencies
261 advocate the true clinical effect of PRF on recession treatment with CAF + CTG, and additional trial
262 centered outcomes in patients with bilateral recession type 1 multiple gingival recessions after 6 mo
266 h at least one Miller Class I or II gingival recession underwent a surgical root coverage procedure.
271 rs of plaque index, gingival index, vertical recession (VR), probing depth, clinical attachment level
273 e WMD of CAL gain was 0.10 mm and the WMD of recession was -0.18 mm; again, no significant difference
278 ooting adult mass followed by extensive mass recession was most prevalent at our freshwater colony, b
280 mine if such trends continue during economic recessions, we analyzed mortality rates in Spain before
281 ry anterior teeth with pre-existing gingival recession were more likely to have thin (<1 mm) buccal b
282 st one site of Miller Class I or II gingival recession were treated by a coronally advanced flap with
283 o the test group (TG), and the contralateral recessions were assigned to the control group (CG).
287 ith lack of attached gingiva associated with recessions, were treated with marginal or submarginal fr
288 nsisting of short illness, temperature, age, recession, wheeze, asthma, and vomiting (mnemonic STARWA
289 lated to the presence or absence of gingival recession, while patient sex, age, and the apico-coronal
290 ment level (CAL) and recession height (GRH), recession width (GRW) and KGW measurements were recorded
291 apilla bleeding index, recession depth (RD), recession width (RW), and root surface area were evaluat
292 s presented reduction of recession depth and recession width and gain of keratinized tissue thickness
293 ference in the midfacial recession depth and recession width at the test and control sites at baselin
295 Post-restoration, clinical (probing depth, recession, width of keratinized mucosa, bleeding on prob
297 emissions were largely a result of economic recession with changes in fuel mix (for example, substit
298 rtical rectus muscles versus superior rectus recession with inferior oblique weakening for patients w
299 ital, iris depigmentation and thinning, iris recession with retinal necrosis and hypotony, a filterin
300 onstrates the possibility of treating buccal recessions with gingival unit grafts as an alternative t