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1  host chromosome through a Holliday junction recombination intermediate.
2 embly of a replication complex onto a D-loop recombination intermediate.
3 ions affect the general conformation of this recombination intermediate.
4 st form and then resolve a Holliday junction recombination intermediate.
5 egulator of the pathway of resolution of the recombination intermediate.
6  of chemotheraputics on the structure of the recombination intermediate.
7 al role in promoting replisome assembly on a recombination intermediate.
8 ion, and gain insight into the nature of the recombination intermediate.
9 es branch migration of the Holliday junction recombination intermediate.
10 uired for protection of either type of V(D)J recombination intermediate.
11 , Slx4, and Msh2/Msh3 complex at a 3' tailed recombination intermediate.
12 r, it is dispensable for the accumulation of recombination intermediates.
13 tion for eliminating potentially deleterious recombination intermediates.
14  replication forks and illegitimately formed recombination intermediates.
15 y, crosslink repair and in the resolution of recombination intermediates.
16 nd promotes proper resolution of inversional recombination intermediates.
17 nation process and is necessary to stabilize recombination intermediates.
18 implicated in processing joint molecule (JM) recombination intermediates.
19 or modeling excisive rather than integrative recombination intermediates.
20 that Saw1 targets Rad1/Rad10 to Rad52-coated recombination intermediates.
21 in HR by facilitating the formation of early recombination intermediates.
22 he arrangement of heteroduplex DNA (hDNA) in recombination intermediates.
23  detectable Rad51 foci indicative of meiotic recombination intermediates.
24 ose resembling stalled replication forks and recombination intermediates.
25 ad to models of the structure of the full HJ recombination intermediates.
26 ion, possibly keeping the DNA clear of toxic recombination intermediates.
27 press illegitimate recombination and resolve recombination intermediates.
28 h strand exchange on complex replication and recombination intermediates.
29 cular molecules (many with tails) similar to recombination intermediates.
30 ing with the generation and/or processing of recombination intermediates.
31 eventing transpositional resolution of V(D)J recombination intermediates.
32 g that RRM3 helps prevent formation of toxic recombination intermediates.
33 romotes DNA-PK(cs)-independent resolution of recombination intermediates.
34 strand, allowing DNA synthesis to occur from recombination intermediates.
35 onsistent with localization of ATM to normal recombination intermediates.
36  cycle progression in response to unrepaired recombination intermediates.
37  the repair of mismatches present in meiotic recombination intermediates.
38 ismatch repair of heteroduplex DNA (hDNA) in recombination intermediates.
39 ecific DNA structures found in early meiotic recombination intermediates.
40  consistent with a role for Rad1 in cleaving recombination intermediates.
41 earrangements are formed by misprocessing of recombination intermediates.
42 and DnaT during replication fork assembly at recombination intermediates.
43 e recognition of these mismatches in mitotic recombination intermediates.
44 day junction translocation and resolution of recombination intermediates.
45 otein participates in replication restart at recombination intermediates.
46 t mismatch repair proteins can interact with recombination intermediates.
47 o push invading 3'-ended strands back out of recombination intermediates.
48 recruited to the sites of DSBs such as V(D)J recombination intermediates.
49 esis that FtsK acts on Holliday junction Xer recombination intermediates.
50 uct formation without altering the levels of recombination intermediates.
51 s that resemble D-loop and Holliday junction recombination intermediates.
52 he RAG proteins impairs proper processing of recombination intermediates.
53 eteroduplex DNA, or during the resolution of recombination intermediates.
54 X- and Y-branched structures, reminiscent of recombination intermediates.
55 nition of mismatches present in heteroduplex recombination intermediates.
56 ity, results in a dramatic increase in mtDNA recombination intermediates.
57 G1-3 tail generated during the processing of recombination intermediates.
58 oes not result from excessive degradation of recombination intermediates.
59 ed with RNAs resembling the putative in vivo recombination intermediates.
60 eterologous sequences in three-stranded RecA-recombination intermediates.
61  phosphotyrosine complexes characteristic of recombination intermediates.
62 due to observed changes in the processing of recombination intermediates.
63 single and double HJs contained within large recombination intermediates.
64 ed by population heterogeneity and transient recombination intermediates.
65 , efficiently cleaving replication forks and recombination intermediates.
66 A without any preference for DNA-replication/recombination intermediates.
67 eria and plants, implicated in processing of recombination intermediates.
68 e helicase, Gp41, onto replication forks and recombination intermediates.
69 ing the resolution of replication-associated recombination intermediates.
70 like reannealing denatured DNA and resolving recombination intermediates.
71 o chronic stress can be overcome by reducing recombination intermediates.
72 ructures that arise during the resolution of recombination intermediates.
73 nd its documented activity limiting aberrant recombination intermediates.
74 g stalled replication forks and disassembles recombination intermediates.
75 sing over by binding and stabilizing nascent recombination intermediates.
76 ility by preventing accumulation of aberrant recombination intermediates.
77 loading the replicative helicase, Gp41, onto recombination intermediates.
78 ), which is part of a complex that dissolves recombination intermediates.
79  specific DNA substrates that represent some recombination intermediates.
80 on, inversion) and the accumulation of toxic recombination intermediates.
81 rexpression of RuvAB, suggesting that lethal recombination intermediates accumulate in the absence of
82 ogenous or wild-type transgenic RAG-2, V(D)J recombination intermediates accumulate preferentially in
83  a temperature-sensitive mutant, the knotted recombination intermediates accumulated whether or not g
84 ssover recombination and preventing aberrant recombination intermediate accumulation.
85 eractions of p53, for example, with Rad51 or recombination intermediates, also impacts on HR.
86                        The Holliday junction recombination intermediate, an X-shaped DNA molecule (xD
87 and sliding-clamp formation between a D-loop recombination intermediate and linear duplex DNA.
88 ch supports crossing over(8), binds branched recombination intermediates and associates with MutLgamm
89 veillance system in C. elegans that monitors recombination intermediates and couples their formation
90                                              Recombination intermediates and crossover formation was
91 d on experiments monitoring the formation of recombination intermediates and crossover products, we s
92 ouble-strand break repair model-asymmetry in recombination intermediates and D-loop migration-may be
93 junctions are required for the resolution of recombination intermediates and for the restart of stall
94 Mlh1-Mlh3 endonuclease activity in resolving recombination intermediates and in DNA mismatch repair.
95  MUS81-EME1, however, MUS81-EME2 cuts D-loop recombination intermediates and in so doing disengages t
96 stalled replication forks, the resolution of recombination intermediates and in telomere length maint
97              The Mus81 endonuclease resolves recombination intermediates and mediates cellular respon
98 gamma) complex, which stabilizes CO-destined recombination intermediates and members of the Zip3/RNF2
99 ctions in S phase to both process homologous recombination intermediates and modulate checkpoint acti
100 ng phenotype nor do they accumulate the rDNA recombination intermediates and products that are found
101 ine the effects of truncated RAG proteins on recombination intermediates and products.
102 -linking of Int in trapped Holliday junction recombination intermediates and recombination reactions
103 the Fanconi Anaemia (FA) pathway can resolve recombination intermediates and remodel synthetic replic
104 ivery of DNA structures that mimic mammalian recombination intermediates and should be useful in assa
105 ions would allow PriA to act as initiator on recombination intermediates and stalled replication fork
106 ption of normal mechanisms needed to resolve recombination intermediates and to maintain chromosome s
107 rks, formation of fork-associated homologous recombination intermediates, and hydroxyurea sensitivity
108 sought using ligation-mediated PCR to detect recombination intermediates, and immunohistochemistry wa
109 lease that processes UV-induced DNA lesions, recombination intermediates, and inter-strand DNA crossl
110  elevated telomere loss, unresolved telomere recombination intermediates, and mitotic infidelity.
111 cleases that unhook ICLs, resolve homologous recombination intermediates, and perhaps remove unhooked
112 gene conversion, the molecular structures of recombination intermediates, and the biochemical compete
113 y junctions--crossed DNA structures that are recombination intermediates--and promotes branch migrati
114                                   Persistent recombination intermediates are acted upon by structure-
115        These data indicate that most meiotic recombination intermediates are asymmetrically positione
116 essential for integration; both lysogens and recombination intermediates are detected when gyrase is
117 l-helicase mechanism explains how the looped recombination intermediates are generated and may serve
118                                Despite this, recombination intermediates are not correctly resolved,
119 , here we show that both TCR-gamma and -beta recombination intermediates are readily detectable in no
120 events to ensure that both (a) CO-designated recombination intermediates are reliably resolved as COs
121                                              Recombination intermediates are required to activate the
122                    To ensure that persistent recombination intermediates are resolved prior to cell d
123 data indicate that single-base mismatches in recombination intermediates are substrates for at least
124                          Predicted Ig switch recombination intermediates are substrates for both exon
125 ecific Ercc1/Xpf endonuclease, two potential recombination intermediates are substrates for misproces
126 recombination and replication machinery when recombination intermediates are used as substrates for D
127 cleolytic processing of telomeric homologous recombination intermediates, are suppressed.
128  role for topoisomerase III in disentangling recombination intermediates as an alternative to RuvABC
129 n both mismatch repair and the resolution of recombination intermediates as predicted by genetic stud
130 ver strongly implicates crossover-designated recombination intermediates as the sites of SC initiatio
131 ese checkpoints are blind to replication and recombination intermediates as well as to rearranged chr
132 Mutating Smc6 results in the accumulation of recombination intermediates at centromeres and other uni
133 re reorganization on CSHJ is exerted through recombination intermediates at interstitial chromosomal
134 of the TCR-gamma locus was observed, whereas recombination intermediates at the TCR-beta locus could
135 he factors that coordinate conversion from a recombination intermediate back to a replication fork to
136 pe did not result from accumulation of toxic recombination intermediates, because it was not relieved
137 e necessary for the processing of persistent recombination intermediates before cell division.
138 d recombination and may specifically resolve recombination intermediates before partitioning.
139 s, and cells lacking SLX4 or SAW1 accumulate recombination intermediates blocked at the Rad1/Rad10-de
140                We characterize inter-homolog recombination intermediates bound by RPA in vivo, with p
141 uction of steady-state levels of bimolecular recombination intermediates (BRIs), which block chromoso
142 ersity during NHEJ-dependent repair of V(D)J recombination intermediates, but the roles of the templa
143 dictates the pathway of replication from the recombination intermediate by inhibiting a nonspecific,
144 at the damage arose as a result of repair of recombination intermediates by host cell pathways.
145 suggest that surveillance of T cell receptor recombination intermediates by NBS1 and gamma-H2AX may b
146       Instead, it probably processes meiotic recombination intermediates by nicking double-stranded D
147 emia virus, we show that large quantities of recombination intermediates can be generated, and their
148 ion, coupled with the modest accumulation of recombination intermediates, can suppress defects caused
149         Consistent with a role in processing recombination intermediates, cells depleted of SLX4 are
150 sociated with Sgs1, most notably as an early recombination intermediate chaperone, promoting regulate
151        In wild type, apparent intermolecular recombination intermediates clustered near to but offset
152 nd1 complex from Saccharomyces cerevisiae to recombination intermediates comprising Rad51- and Dmc1-s
153 IIIalpha together effect the resolution of a recombination intermediate containing a double Holliday
154  double-displacement loop (double-D-loop), a recombination intermediate containing two single-strande
155 somerase IIIalpha (TOPO IIIalpha) to resolve recombination intermediates containing double Holliday j
156 th artificial Holliday junctions and meiotic recombination intermediates containing four-way junction
157 ct SPO11-induced double-strand breaks and/or recombination intermediates containing free 3' hydroxyl
158                            The resolution of recombination intermediates containing Holliday junction
159 he Msh proteins act with Sgs1p to unwind DNA recombination intermediates containing mismatches.
160 HU (DrHU), which binds preferentially to DNA recombination intermediates, contains a 47-amino acid ex
161 y release the third strand of the homologous recombination intermediate D-loop structure irrespective
162 vidual replication problems or the resulting recombination intermediates delay the cell cycle.
163 le of robust DNA synthesis at RAD51-mediated recombination intermediates dependent on the processivit
164 pendent shuttling and directed processing of recombination-intermediate [displacement loop (D-loop)]
165 ility complex, we observe persistent meiotic recombination intermediates (DNA joint molecules) resemb
166 sed to extend the displacement loop (D-loop) recombination intermediate, does not influence the lengt
167 ate of Mus81-Mms4 and Sgs1-Top3 is a 3' flap recombination intermediate downstream of replication for
168 during lymphocyte development, joining V(D)J recombination intermediates during antigen receptor gene
169 the processing of DNA ends and resolution of recombination intermediates during double-strand gap rep
170  RuvABC proteins of Escherichia coli process recombination intermediates during genetic recombination
171 ct independently to promote the formation of recombination intermediates during impaired replication.
172 4 is required for the processing of specific recombination intermediates during meiosis.
173 ssover repair, MutLgamma-Exo1 associate with recombination intermediates, followed by direct Cdc5 rec
174 -annealing activity that hastens the rate of recombination intermediate formation.
175                            We show here that recombination intermediates formed between linear duplex
176            Here we address the nature of the recombination intermediates formed by phiC31 integrase w
177 y of the helicase component and ensures that recombination intermediates formed by uvsX/uvsY will eff
178 he two 400-kDa multiprotein Holiday junction recombination intermediates formed during lambda integra
179 cessing of damaged replication forks, or the recombination intermediates formed from damaged forks.
180 esigned substrates representing illegitimate recombination intermediates formed when a displaced DNA
181  that the Sgs1 helicase prevents a subset of recombination intermediates from becoming crossovers, an
182 F/ERCC1 is the active species that processes recombination intermediates generated during the repair
183 x and Esc2 prevent the accumulation of toxic recombination intermediates generated in these processes
184 ity for the Holliday junction (HJ)-a key DNA recombination intermediate-have been purified and charac
185 ith two tetramers of RuvA binding to the DNA recombination intermediate in a co-operative manner.
186 pproximately -4 kcal/mol to stabilizing this recombination intermediate in inverted-repeat sequences.
187 les are consistent with their being an early recombination intermediate in the Red recombination path
188 I stimulates the dissolution of a homologous recombination intermediate in vitro and is essential for
189                   Current mechanisms posit a recombination intermediate in which both 5' ends of doub
190 ishable from the eclipse complex formed as a recombination intermediate in wild-type competent cells.
191   Here we report the first analysis of V(D)J recombination intermediates in a Ku-deficient cell line.
192 es HR by promoting the disassembly of D loop recombination intermediates in a reaction dependent upon
193 elta, and shu1Delta each reduce the level of recombination intermediates in an smc6 mutant when cells
194 itivity to DNA damage or the accumulation of recombination intermediates in cells lacking Sgs1, which
195 ons in the proper assembly and resolution of recombination intermediates in endogenous antigen recept
196              Resolution of Holliday junction recombination intermediates in most Gram-negative bacter
197  the accumulation of potentially deleterious recombination intermediates in mutants of the Smc5/6 com
198                             Here, we compare recombination intermediates in pat1-as2 at 25 degrees C
199 ion and the accumulation of V(beta)-DJ(beta) recombination intermediates in peripheral CD4(+) T cells
200                              The presence of recombination intermediates in plant mitochondria prepar
201                                 The level of recombination intermediates in rho+ mtDNA of Deltamgt1 c
202 s confirm the validity of previous assays of recombination intermediates in S. pombe and provide new
203        mph1 is known to reduce the levels of recombination intermediates in smc6 mutants.
204  over together with evidence for accumulated recombination intermediates in syp-1 mutants indicate th
205 hat CDKG1 is necessary for the processing of recombination intermediates in the canonical ZMM recombi
206  can be restored by increasing the number of recombination intermediates in the double cdkg1-1 fancm-
207 single-strand branch that resembles putative recombination intermediates in the RAD1 RAD10-mediated s
208 We investigated yeast Msh2p association with recombination intermediates in vivo using chromatin immu
209   Repair of single-base mismatches formed in recombination intermediates in vivo was investigated in
210 ructures, we produced test circuits based on recombination intermediates in which 1D translocation ac
211 hat poleta extends DNA synthesis from D loop recombination intermediates in which an invading strand
212 ere we report the first examination of V(D)J recombination intermediates in XRCC4-deficient cells.
213 structure (a model for the Holliday junction recombination intermediate) in which each 'arm' of the 4
214 -6 nt of homology, followed by resolution of recombination intermediates into chromosomal rearrangeme
215 plicated in the biased processing of meiotic recombination intermediates into crossovers by an unknow
216 mm proteins promote the biased resolution of recombination intermediates into crossovers that are dis
217 d ERCC1 form a complex that resolves meiotic recombination intermediates into crossovers.
218 hich the 3'-OH of the invading strand in the recombination intermediate is used as a primer.
219   The efficient and timely resolution of DNA recombination intermediates is essential for bipolar chr
220     The efficient removal of replication and recombination intermediates is essential for the mainten
221 We have examined formation of joint molecule recombination intermediates (JMs) between homologs and b
222 ts show that ICP8 catalyzes the formation of recombination intermediates (joint molecules) between ci
223                                              Recombination intermediates (joint molecules, JMs) accum
224 NA, an in vitro mimic of the in vivo genetic recombination intermediate known as the Holliday junctio
225 r via the formation and biased resolution of recombination intermediates known as double Holliday jun
226 s of minimized and symmetrized surrogates of recombination intermediates lacking the accessory protei
227 nhance checkpoint signaling without reducing recombination intermediate levels.
228 a model in which BLM selectively dissociates recombination intermediates likely to be unfavorable for
229 omologous chromosomes, while crossover-bound recombination intermediates locally stabilize interactio
230 e- and four-strand exchange reactions, using recombination intermediates made by the E. coli RecA pro
231 tion to clear DNA of proteins and to migrate recombination intermediates may be of critical importanc
232 ibution of MutSgamma and RFC-PCNA on meiotic recombination intermediates may drive biased DNA cleavag
233 oth enzymes have profound defects in meiotic recombination intermediate metabolism and crossover (CO)
234 esponse and the formation of RecA*-dependent recombination intermediates necessary for PriA/Pol II-de
235 either differential control of resolution of recombination intermediates or alternative pathways of r
236 ence of DNA damage in the form of unrepaired recombination intermediates or defects in homologous chr
237 ifferential stability of crossover-competent recombination intermediates, or alternatively, the prese
238       We further show that in the absence of recombination intermediates, polycomplexes recapitulate
239 ce of chi recognition is the production of a recombination intermediate possessing a 3'-ssDNA overhan
240  play an essential role in the resolution of recombination intermediates prior to chromosome segregat
241 ecG mutation, which promotes accumulation of recombination intermediates proposed to prime replicatio
242                                        Early recombination intermediates (RAD-51 foci) increase conco
243 vo but does not influence the high levels of recombination intermediates readily detected in the mtDN
244 nt with hemicatenane-related template switch recombination intermediates (Rec-Xs) but not Holliday ju
245 in vitro BLM helicase promotes disruption of recombination intermediates, regression of stalled repli
246 s are reliably resolved as COs and (b) other recombination intermediates reliably mature into noncros
247 tiation and maturation of crossover-specific recombination intermediates requires the cyclin-like CNT
248 1-independent functions that ensure complete recombination intermediate resolution and chromosome seg
249 logous chromosomes and altered processing of recombination intermediates, resulting in increased chia
250 ease Rad1-Rad10 and enzymes known to disrupt recombination intermediates (Sgs1-Top3-Rmi1, Srs2, and M
251                                  Analysis of recombination intermediates showed defects at the cleava
252                              Accumulation of recombination intermediates similarly leads to the demis
253 o END-seq, we identify a SPO11-bound meiotic recombination intermediate (SPO11-RI) present at all hot
254 he recombinase machinery and the presence of recombination intermediates strongly suggest that TCR re
255                                          The recombination intermediate structure between a donor p44
256                 To determine whether the p44 recombination intermediate structure can be generated in
257 LM is well equipped to deal with alternative recombination intermediate structures.
258                  UvsW-catalyzed unwinding of recombination intermediates such as D-loops and static X
259 f PriA-directed replication fork assembly at recombination intermediates such as D-loops.
260 t a model in which Mus81-Mms4 cleaves nicked recombination intermediates such as displacement loops (
261 ng enzymes participate in recombination, and recombination intermediates supply substrates for replic
262 nerate 10-fold higher levels of one class of recombination intermediates, termed signal ends.
263 s that when annealed in vivo approximate the recombination intermediate that Exo should create.
264  These results show that pilin Av produces a recombination intermediate that must be processed by eit
265  repair of a single DSB, suggesting that the recombination intermediates that accumulate cannot be pr
266 the complex is dissolution of toxic X-shaped recombination intermediates that accumulate during repli
267 endent Ddc2 foci, indicating the presence of recombination intermediates that are sensed by checkpoin
268 that these complexes are required to resolve recombination intermediates that arise in response to DN
269 strates fully recapitulate the properties of recombination intermediates that arise within a physiolo
270 to a complementary ectopic DNA site, forming recombination intermediates that can lead to genomic ins
271 COs, likely because of formation of unstable recombination intermediates that cannot be sustained as
272 erase IIIalpha (hTOPO IIIalpha), can process recombination intermediates that contain double Holliday
273         In order to isolate and characterize recombination intermediates that contain Holliday juncti
274 ctor MSH2-MSH3 binds and stabilizes branched recombination intermediates that form during single stra
275              Gene conversion might accompany recombination intermediates that resolve without translo
276 romosome breakage and the formation of toxic recombination intermediates that undermine genomic stabi
277 a transposase capable of inserting the V(D)J recombination intermediate, the signal end DNA fragment,
278                  We identify a novel meiotic recombination intermediate, the single-end invasion (SEI
279  not reflect the accumulation of unprocessed recombination intermediates: the delays in meiotic progr
280 6 complex binds and stabilizes early meiotic recombination intermediates, then coordinates additional
281 straints on which enzymes gain access to the recombination intermediate, thereby controlling the mann
282 male germ cells to promote repair of meiotic recombination intermediates, thereby improving the fidel
283 36 may play an important role in stabilizing recombination intermediates through solvent-mediated pro
284 apitulated a functional equivalent of the HJ recombination intermediate to maintain the structural in
285 ut also on antirecombinases, which dismantle recombination intermediates to allow completion of repai
286 el in which CDK activity links processing of recombination intermediates to cell cycle progression vi
287                             The liability of recombination intermediates to damage is consistent with
288 fluences the choice of proteins that resolve recombination intermediates to form crossovers.
289 system can remove mismatches in heteroduplex recombination intermediates to generate gene conversion
290  that mismatch repair proteins interact with recombination intermediates to prevent recombination, or
291  essentiality of replication fork restart at recombination intermediates under normal growth conditio
292 escuing a proportion of crossover-designated recombination intermediates via a route that is likely A
293                                          The recombination intermediate was recovered in an E. coli s
294   Repair of all 12 single-base mismatches in recombination intermediates was investigated in Chinese
295 studies showed that AtRECQ4A associates with recombination intermediates, we found no evidence that i
296 s regulate recombination by interacting with recombination intermediates, we investigated whether the
297 nical" Rad51 amino acids gives rise to toxic recombination intermediates, which must be actively dism
298       During resolution of Holliday junction recombination intermediates, wild-type Int, but not R169
299                                              Recombination intermediates with a maximum heteroduplex
300 ling the formation and processing of meiotic recombination intermediates with DNA-strand interruption

 
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