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1 e or spinal cord injury), possibly assisting recovery of function.
2 was predominantly expressed in segments with recovery of function.
3 y was seen between segments with and without recovery of function.
4  of necrosis, is independently predictive of recovery of function.
5  months after revascularization to determine recovery of function.
6 6% (91 of 252 patients) demonstrated partial recovery of function.
7 rategies have been associated with increased recovery of function.
8 nhancement occurs in regions that later show recovery of function.
9 ensitivity of 94% and specificity of 48% for recovery of function.
10 trast-enhanced MRI, restoration of flow, and recovery of function.
11 adulthood, indicating very little sparing or recovery of function.
12 tive angiographic parameters did not predict recovery of function.
13 em (PNS) is capable of full regeneration and recovery of function.
14 %) with a lower specificity (43% to 66%) for recovery of function.
15 itivity but a low specificity for predicting recovery of function.
16 VEF (OR 1.831, p < 0.01) but did not predict recovery of function.
17 cclusion that prevents organ reperfusion and recovery of function.
18 of local symptoms and signs of infection and recovery of function.
19 ith either cardiac function or post-ablation recovery of function.
20 sue survives to be a plausible substrate for recovery of function.
21 ns after stroke, which have a causal role in recovery of function.
22 esents a highly efficacious means to promote recovery of function.
23 re of premotor pyramidal neurons and support recovery of function.
24 restoration of action potentials and partial recovery of function.
25  regions to assess effects on impairment and recovery of function.
26 ological neural activity and provide optimal recovery of function.
27 l systems could provide a substrate for some recovery of function.
28 itors before ischemia significantly improved recovery of function.
29 may be crucial for learning, plasticity, and recovery of function.
30 ulation may be a mechanism to facilitate the recovery of function.
31 or the evaluation of interventions promoting recovery of function.
32 sing myocardial viability and predicting the recovery of function.
33  of morphological restoration of tubules and recovery of function.
34  in lumbosacral circuitry contributes to the recovery of function.
35 effect of NCX1015 and its ability to improve recovery of function.
36 myelination of injured CNS axons in vivo and recovery of function.
37 tative response to injury perhaps leading to recovery of function.
38 orarily impaired after saline overload, with recovery of function 1 hour later.
39 d, and used to generate viable explants with recovery of function 14 days postthaw.
40                                              Recovery of function after acute injury to the central n
41 ters to show that axonal fusion enables full recovery of function after axotomy of Caenorhabditis ele
42  and disease and neuroplastic changes during recovery of function after brain lesions.
43 ve values of 79% and 100%, respectively, for recovery of function after CABG in the biopsied segments
44  learning in normal visual experience and in recovery of function after CNS damage.
45 ity also was assessed by dobutamine echo and recovery of function after coronary artery bypass grafti
46 STATEMENT Neuronal plasticity can facilitate recovery of function after cortical injury, but the unde
47  Until recently, the neural bases underlying recovery of function after damage to the cerebral cortex
48  define the mechanisms mediating spontaneous recovery of function after incomplete SCI, we created bi
49 at reflex-conditioning protocols can enhance recovery of function after incomplete spinal cord injuri
50  systemic administration of MSC-EVs enhances recovery of function after injury of the primary motor c
51  in the control of ipsilateral movements and recovery of function after injury.
52  learning, direct multimodal integration and recovery of function after injury.
53 d IPC stimulus, resulted in nearly identical recovery of function after ischemia compared with IPC pe
54 ical trial to test its efficacy in enhancing recovery of function after ischemic stroke.
55 bution 201Tl tomography in the prediction of recovery of function after revascularization and to asse
56 matory reaction have a greater potential for recovery of function after revascularization.
57 nating myocardium, an important predictor of recovery of function after revascularization.
58 d flow are important parameters that predict recovery of function after revascularization.
59 an asynergic region more accurately predicts recovery of function after revascularization.
60 uded vessel and may be useful for predicting recovery of function after revascularization.
61 e to dobutamine can assess the likelihood of recovery of function after revascularization.
62 rating secondary tissue damage and promoting recovery of function after SCI.
63 lucocorticoids, given at high-doses, improve recovery of function after spinal cord injury (SCI) in a
64  Propriospinal neurons play crucial roles in recovery of function after spinal cord injury.
65                                        While recovery of function after spinal root avulsion was rela
66                                              Recovery of function after stroke is now widely consider
67 ired with rehabilitation results in improved recovery of function after stroke or traumatic brain inj
68 ally responsible for rehabilitation-mediated recovery of function after stroke.
69 human studies as an adjuvant tool to promote recovery of function after stroke.
70 acticed movements and is thought to underlie recovery of function after stroke.
71 otension but a more variable time course for recovery of function after the cessation of the infusion
72  that ultimately limit neuronal rewiring and recovery of functions after stroke.
73 an be regenerated; yet, this is critical for recovery of function and a central aim of cell replaceme
74 ce in a deficient state, there is a dramatic recovery of function and a normal histologic appearance
75 =0.6 cm virtually excludes the potential for recovery of function and is a valuable adjunct to DSE in
76  Surviving neural networks are important for recovery of function and non-invasive techniques such as
77 nic temperature of 10 degrees C and complete recovery of function and normal histology.
78 acute inflammatory injury of axons and glia, recovery of function and structural repair, post-inflamm
79 c contributions of individual brain areas to recovery of function and the effect of treatment on cort
80 ng the symptoms of the neglect syndrome, the recovery of function and the use of clinical scans to pr
81 gies for spinal cord injury seek to maximize recovery of function and two strategies that show substa
82  neural tissue in the damaged hemisphere and recovery of function appear to be vulnerable to prolonge
83  down together in stroke and that subsequent recovery of function applies to both domains.
84  integration after spinal cord injury and in recovery of function are broadly significant for basic a
85                          Limited spontaneous recovery of function as a result of brain remodelling af
86 nt regimes influence tissue regeneration and recovery of function as a result of cardiomyocyte prolif
87  recovery in neuronal number correlated with recovery of function as shown by paw withdrawal from a n
88  investigate the pathogenesis of the delayed recovery of function associated with blocking NE functio
89 ild-type FXN levels, we observed significant recovery of function, associated pathology and transcrip
90 also similar if analysis was performed using recovery of function at 3 months as the index of viabili
91                                              Recoveries of function, ATP, and phosphocreatine were hi
92 troke can mediate positive outcomes, such as recovery of function, but can also result in the formati
93 GABAAR in mice with ischemic stroke improved recovery of function by normalizing exaggerated perilesi
94 lowing brain injury demonstrate that partial recovery of function can and does occur.
95 dosis, there was reduced injury and improved recovery of function compared to WT.
96 ) and flow (peak MCIxbeta) better identified recovery of function compared with microvascular density
97 n entirely different set of voxels predicted recovery of function: damage lateral to RFA reduced reco
98 abnormalities on day 1 demonstrated complete recovery of function (ejection fraction in the normal ra
99 rom injury sites and that might prevent full recovery of function, even after successful regeneration
100 ignificant number of patients have transient recovery of function followed by later deterioration, wh
101 g and memory, mood, seizure suppression, and recovery of function following brain damage are mediated
102 herapeutic interventions, seeking to enhance recovery of function following incomplete lesions to the
103                                 The improved recovery of function following ischemia in NCX-KO hearts
104  by reactive lipids in inhibiting repair and recovery of function following ischemic injury, a knock-
105 use inferotemporal acetylcholine facilitates recovery of function following structural damage within
106 ed that nicotinamide (NAM) is beneficial for recovery of function following TBI.
107                                              Recovery of function following traumatic brain injury (T
108               After ischemic stroke, partial recovery of function frequently occurs and may depend on
109                            The half-time for recovery of function from desensitization after a 60-min
110 tangling which axons are responsible for the recovery of function from those that are functionally si
111                          Poststroke loss and recovery of functions have been increasingly correlated
112 ft ventricular contractility had a transient recovery of function; however, ejection fraction deterio
113 ural plasticity may unmask the potential for recovery of function in adulthood.
114 gnition, and a new perspective to understand recovery of function in clinical conditions.
115 imulation of the spinal cord (TESS) promotes recovery of function in humans with spinal cord injury (
116 tramethylpiperidine-N-oxyl) induced complete recovery of function in irradiated TRPM2-/- mice.
117 hese ligands and provides a means to promote recovery of function in multiple preclinical rodent mode
118 of hypoxia but had no effect on postischemic recovery of function in normoxic hearts.
119 we investigated its effects on apoptosis and recovery of function in rats after SCI.
120  novel experimental therapeutic compound for recovery of function in SCI patients.
121 selective removal of unreliable synapses and recovery of function in the persistent fraction.
122 ng mutagenesis of the M4 segment, as well as recovery of function in the polyleucine background, defi
123                    The GC group had a better recovery of function in the year following injury than t
124       This may relate to differences seen in recovery of function in these muscles after injury, such
125 most patients experience spontaneous partial recovery of functions in subacute to chronic phases.
126  P1 and P6 callosotomy groups exhibited full recovery of function; in contrast, the P8 group did not
127  glial borders, larger lesions, and impaired recovery of function, indicating that intrinsic EGFR act
128 axonal sprouting that contributes to partial recovery of function is dependent upon the intrinsic pro
129 rossing the lesion site, suggesting that the recovery of function is due to some other reaction, such
130 psies of dysfunctional segments reveals that recovery of function is inversely proportional to the ex
131                                 However, the recovery of function is markedly different in the two co
132 also demonstrate that substantial sparing or recovery of function is possible when the lesion is made
133 xons, reorganize neural networks and support recovery of function is severely restricted, contributin
134                                        Early recovery of functioning is critical for favorable outcom
135                                              Recovery of function (LVDP) after global ischemia in hea
136                                              Recovery of function may be accelerated by high dose cor
137 aneously even in the absence of reperfusion, recovery of function may be slow and associated with hig
138 ith 12 leucine residues and then constructed recovery-of-function mutants in which specific amino aci
139 er 30% of the myocardial thickness precludes recovery of function.Nagueh et al. [1] evaluated the rel
140                                              Recovery of function occurred in 38% of dysfunctional se
141                                              Recovery of function occurred in 6 (50%) of 12 patients
142                     By contrast, significant recovery of function occurred when proteoliposomes were
143                         Although significant recovery of function occurs, little is currently underst
144 fluorescence in situ hybridization, revealed recovery of function of the dermal-epidermal junction wi
145 hich was dysfunctional, and therefore led to recovery of function on the spontaneous alternation task
146            Similar findings were observed if recovery of function or scintigraphic uptake was analyze
147 higher than dysfunctional myocardium without recovery of function (P<0.001).
148 ute rejection episodes than those with later recovery of function (P=0.003).
149 ally dependant task may be associated with a recovery of function related to the synaptic remodelling
150                                              Recovery of function relies on augmenting neuroplasticit
151 ver, the extent to which transplants improve recovery of function remains unclear and there are contr
152 he role that remyelination plays in clinical recovery of function remains unproven.
153 nd motor function as compared to the partial recovery of function seen in PBS-injected control rats.
154 th suspected hibernation, and it can predict recovery of function similar to T1-201 scintigraphy.
155 e, would have synergistic effects leading to recovery of function that is greater than either alone.
156 ons into host spinal cord circuits underlies recovery of function, the extent to which there is synap
157 n; in contrast, the P8 group did not exhibit recovery of function, thus indicating an abrupt decrease
158 e sensitivity of Peak MCIxbeta >1.5 dB/s for recovery of function was 90% and was similar to Tl(201)
159                                              Recovery of function was defined as improvement in rest
160 no such concentration dependence for time to recovery of function was found when carbachol was used t
161 ve values of dobutamine echocardiography for recovery of function were 100% (95% confidence interval
162 mine is less sensitive but more specific for recovery of function, whereas indexes of 201Tl scintigra
163 at least partially causative to the observed recovery of function, which can also be observed in huma
164 s across the injury site was associated with recovery of function with animals exhibiting plantar foo
165 umatic brain injury (TBI) impedes subsequent recovery of function without enlarging the size of the l

 
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