ライフサイエンスコーパス: red cell aplasia

1 unlike EPO products, reduce the risk of pure red cell aplasia.

2 e transplant and may be associated with pure red cell aplasia.

3 enia gravis, hypogammaglobulinemia, and pure red cell aplasia.

4 isorders), the most common of which was pure red-cell aplasia.

5 disorder most commonly associated with pure red cell aplasia, (2) the presence of clonal cytogenetic

6 infection may develop that presents as pure red cell aplasia and chronic anemia.

7 one marrow failure syndrome characterized by red cell aplasia and congenital anomalies.

8 s, including transient aplastic crisis, pure red cell aplasia, and hydrops fetalis.

9 sociation between rituximab therapy and pure red cell aplasia, but the diagnostic and therapeutic uti

10 Chronic red cell aplasia can develop in immunocompromised patien

11 tor can correct anemia in patients with pure red-cell aplasia caused by antierythropoietin antibodies

12 d-Blackfan anemia (DBA) is a rare congenital red-cell aplasia characterized by anemia, bone-marrow er

13 graft-vs-host disease-like colitis, and pure red cell aplasia, different from the pattern observed in

14 ore than 80 percent in the incidence of pure red-cell aplasia due to Eprex.

15 B19 causes fifth disease, acute and chronic red cell aplasia, fetal hydrops, arthropathy, and other

16 obtained reports of epoetin-associated pure red-cell aplasia from the Food and Drug Administration a

17 Feline leukemia virus-C (FeLV-C) causes red cell aplasia in cats, likely through its interaction

18 (erythema infectiosum) in children and pure red cell aplasia in immunocompromised patients.

19 ntified 47 adult patients with acquired pure red cell aplasia (median age, 64 years; range, 22 to 84

20 -eight episodes of HPV B19-induced transient red cell aplasia occurred with the following clinical ev

21 n and heme synthesis may be involved in pure red cell aplasia of DBA.

22 teins cause Diamond Blackfan Anemia (DBA), a red cell aplasia often associated with physical abnormal

23 nts with chronic kidney disease who had pure red-cell aplasia or hypoplasia due to antierythropoietin

24 Delayed donor red cell engraftment and pure red cell aplasia (PRCA) are well-recognized complication

25 Acquired pure red cell aplasia (PRCA) can be associated with lymphopro

26 months, respectively, before developing pure red cell aplasia (PRCA) confirmed by bone marrow biopsy.

27 Pure red cell aplasia (PRCA) is a rare complication in patien

28 Pure red cell aplasia (PRCA) is a rare hematological disorder

29 Pure red cell aplasia (PRCA) is a syndrome defined by a normo

30 lobulin (IVIG) therapy in patients with pure red cell aplasia (PRCA) related to human parvovirus B19

31 ped antibodies to epoetin, resulting in pure red cell aplasia (PRCA).

32 ntibodies leading to the development of pure red cell aplasia (PRCA).

33 Pure red-cell aplasia (PRCA) is a rare hematologic disease ch

34 ce of this effect of rituximab therapy: pure red cell aplasia resulting from chronic parvovirus B19 i

35 n in transplant recipients can cause chronic red cell aplasia that generally responds to IVIG therapy

36 Diamond Blackfan anemia (DBA), an inherited red cell aplasia, the bone marrow is characterized by a

37 Pure red-cell aplasia was defined as transfusion-dependent an

38 of administration, the country in which pure red-cell aplasia was identified, and the date on which p

39 the peak incidence of Eprex-associated pure red-cell aplasia was reached in 2001, interventions desi

40 ween 1988 and 1998, antibody-associated pure red-cell aplasia was reported in three patients who had

41 a was identified, and the date on which pure red-cell aplasia was reported.

42 ral history and therapeutic response of pure red cell aplasia we have studied 37 patients.

43 l 2004, 175 cases of epoetin-associated pure red-cell aplasia were reported for Eprex, 11 cases for N