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1 proximately 0.1 %) of the pigment content in red cones.
2 n--about 10% of total opsin--in dark-adapted red cones.
3  required for the maintenance or function of red cones.
4 st sensitivity, 5.5-fold higher than that of red cones.
5 possess ultraviolet and green cones, but not red cones.
6 ole in repressing UV cone genes in zebrafish red cones.
7 e principal member originated from ancestral red cones, although the origin of the accessory member i
8 hromophore did not affect the ability of the red cone and blue cone/green rod pigments to activate tr
9                                          The red cone and blue cone/green rod pigments were unstable
10 t important in the activation pathway of the red cone and blue cone/green rod pigments.
11 SPR-mediated knockout of THRB causes loss of red cones and DC-P, but not DC-A, and the appearance of
12  purpose greyscale system based on ancestral red cones and rods to mediate visual body stabilization
13                       In samd7(-/-) retinas, red cones are transformed into hybrid red/ultraviolet (U
14 We propose that DC-P arose from an ancestral red cone, as revealed by expression of the red cone cell
15 tina, loss of thrb resulted in an absence of red cones at both larval and adult stages without disrup
16 E65 was selectively expressed in human green/red cones but absent from blue cones and mediated ester
17 f photoresponsiveness of bleached salamander red cones but not of bleached salamander red rods.
18 ased opsin expression in individual rods and red cones, but decreased opsin expression in individual
19 tinal is reversible in darkness in amphibian red cones, but essentially irreversible in red rods.
20 l red cone, as revealed by expression of the red cone cell fate determinants thyroid hormone receptor
21             Finally, we demonstrate that the red-cone-enriched tetratricopeptide repeat protein 39B (
22 s an approximately 2-fold desensitization in red cones, equivalent to that produced by a steady light
23                        Implicit times of the red cone ERGs were slightly faster for the Neuroline ski
24 normally bias their connectivity in favor of red cones fail to precisely recapture this synaptic part
25 y TH receptors and a requirement for thrb in red cone fate determination.
26              Bipolar cells were dominated by red-cone inputs, often alongside equal sign inputs from
27 lectrode recordings from the hypothalamus of red cone knockin mice (Opn1mwR).
28 us laevis genes, Prph2 (also called RDS) and red cone opsin (RCO) using a polymerase chain reaction-b
29 sis suggests that separate enhancers control red cone opsin expression in DC-P and DC-A, consistent w
30 sion resulted in a decrease in rhodopsin and red cone opsin expression levels in Xenopus retinas.
31 y that Rx is co-expressed with rhodopsin and red cone opsin in maturing photoreceptors and demonstrat
32                                 When rod and red cone opsin probes were combined, the number of label
33 n of rAAV5-hCNGB3 with a long version of the red cone opsin promoter in younger animals led to a stab
34 nt therapy with different forms of the human red cone opsin promoter led to the restoration of cone f
35 monstrate that Rx binds to the rhodopsin and red cone opsin promoters in vivo.
36 r of photoreceptors expressing rod opsin and red cone opsin, and decreased the number of photorecepto
37 ess this deficit using mice expressing human red cone opsin, in which rod-, cone-, and melanopsin-dep
38 eceptor-specific genes such as rhodopsin and red cone opsin.
39        We also show that the degeneration of red cone photoreceptors in the mutants occurs independen
40 rotenoid biosynthesis (feather follicles and red cone photoreceptors), and genetic evidence implicate
41  of long-wavelength-sensitive opsin (lws) in red cone photoreceptors, while in retinal pigment epithe
42 stability of the chromophore within the deep red cone pigment binding sites.
43                           Interestingly, the red cone pigment containing the retinal analogue remaine
44 f rhodopsin, the green cone pigment, and the red cone pigment in H2O (D2O) are found at 1656 (1623),
45 s question by expressing human or salamander red cone pigment in Xenopus rods, and human rod pigment
46                                     However, red cone pigment isomerizes spontaneously 10,000 times m
47 ow overcome this problem by expressing human red cone pigment transgenically in mouse rods in order t
48                             However, for the red cone pigment, the 9-methyl group of retinal appears
49 corresponding control cultures regarding the red cone pigment, which was expressed in all cases, and
50 nel catfish orthologues of rhodopsin and the red cone pigment-the full complement of retinal opsins i
51 Raman spectra of recombinant human green and red cone pigments have been obtained to examine the mole
52 f seven cone pigments indicate that the deep red cone pigments select 6- s- trans chromophore conform
53 igate theoretically the hypothesis that deep red cone pigments select a 6- s- trans conformation of t
54 pes, there exist subpopulations of green and red cones (previously shown to be located in the ventral
55 flash cone, light-adapted 3.0 Hz flicker and red cone responses were analysed, as well as their respe
56 flash cone, light-adapted 3.0 Hz flicker and red cone responses were up to four times larger when rec
57 d opsin is the only known molecule unique to red cones, these data suggest that a novel gene is requi
58 e of transgenic cone pigment to native human red cones, we obtained a dark rate of approximately 10 f
59 tina, retinal progenitors destined to become red cones were transfated into ultraviolet (UV) cones an