ライフサイエンスコーパス: redox

1 e (MIOX) is associated with altered cellular redox.

2 These results validate the utility of redox-activated metal complexes as hypoxia-selective H(2

3 general scope for detecting a broad range of redox active and nonredox active species simultaneously,

4 es that facilitate voltammetric detection of redox active neurochemicals in a multianalyte solution.

5 raised much attention as organocatalysts and redox active polymers.

6 macrocyclic polyamide cage that incorporates redox-active 1,4-dithiin units.

7 n of O(2) into H(2)O(2), based on the use of redox-active carbenium species.

8 ate single-electron devices wherein a single redox-active cluster is connected to two macroscopic ele

9 ile electrodeposition process for creating a redox-active crystalline layer (denoted as RZx) on pyrol

10 t for bacteria and an essential cofactor for redox-active cuproenzymes.

11 luate the change in (64)Cu-ATSM signal after redox-active drug treatment.

12 standing how strong trans effect ligands and redox-active ligands work together to enable rapid elect

13 Eumelanin is also a promising redox-active material for energy conversion and storage,

14 While redox-active materials based on conducting and semicondu

15 ial system to explore the rational design of redox-active molecules in nonaqueous systems.

16 redox sensing, we found that antioxidant and redox-active molecules were capable of reducing the expr

17 sed molecules represent a class of promising redox-active organics for potential grid-scale energy st

18 It has been shown recently that many non-redox-active proteins are good electronic conductors, th

19 ioelectronics research has mainly focused on redox-active proteins because of their role in biologica

20 chemically mediated carbon capture utilizing redox-active sorbents such as quinones is emerging as a

21 tron transfer kinetics and thermodynamics of redox-active species encapsulated into supramolecular as

22 two maquette systems containing one pH- and redox-active tyrosine (alpha(3)Y and peptide A), and two

23 motion of mixed-valence interactions between redox-active, pai-conjugated scaffolds is of interest wh

24 With the reversible redox activity of azo group in pai-conjugated structures

25 for aqueous RFBs by utilizing the reversible redox activity of azo groups.

26 with stimuli-controlled electronic behavior, redox activity, and modularity could be used as a bluepr

27 taxel-methylene blue conjugate (PTX-MB) with redox activity.

28 or as an electron reservoir with reversible redox activity.

29 derlying features and relationship for anion redox and cationic redox to occur reversibly.

30 derstanding of melanin optical, paramagnetic redox, and conductivity properties, including photocondu

31 Moreover, increased staining of the redox-associated markers 8-OHdG, GADD45 and GP-1 were al

32 phyrins have been anticipated to exhibit the redox-associated variation of Huckel aromaticity as a me

33 ether, our results demonstrate a beta2AR-ROS redox axis, which if disturbed, interferes with proper r

34 lic/fluxomic redirection leading to restored redox balance imparted by palmitate helps explain mainta

35 oxaloacetate transaminase 1 (GOT1)-dependent redox balance in PCa, which could be exploited for novel

36 Palm may help improve function via enhanced redox balance in T2DM hearts during peaks of hyperglycae

37 Furthermore, treatments that maintain redox balance promote T cell self-renewal and enhance an

38 Abeta accumulation affects mitochondrial redox balance, increasing oxidative stress status, which

39 w application of an emerging electrochemical redox based treatment modality.

40 nter and ligand, [Fe(tpyPY2Me)](2+) exhibits redox behavior at potentials 640 mV more positive than t

41 The first evaluation of redox behavior of buckyball- or tetracyanoquinodimethane

42 tammetric and pulse techniques confirmed the redox behaviour of the new compound with concentration a

43 atifying to witness the enormous progress in redox biology brought about by the science community in

44 , and that the rate of electron flux through redox buffering circuits is directly linked to changes i

45 is reduced to H(2)O by electrons drawn from redox buffering circuits supplied by NADPH, and that the

46 The relevant redox buffering reactions, however, remain poorly constr

47 ancer Genome Atlas, we predict that elevated redox capacity is a general feature of melanomas, not pr

48 coupling, borylation, and hydrogenation in a redox catalytic regime involving S(4)(* -)/S(4)(2-) and

49 d-light-mediated dual transition-metal/photo-redox-catalyzed C-H arylation and intermolecular atom-tr

50 way to explore transient states in multiple redox-center containing proteins (homo and hetero metal

51 three-dimensional networks of closely spaced redox centers that appear to be nearly ideal for multist

52 aracterize elusive intermediates in multiple redox-centre containing proteins.

53 Cell cycle-dependent redox changes can mediate transient covalent modificatio

54 The redox changes were associated with increased NF-kappaB (

55 Reduction/oxidation (redox) changes in the glutathione pool (GSH), glutaredox

56 ndicate which compounds are energy minima in redox chemical space across a set of pH values and elect

57 the context of ligand-based and metal-based redox chemistry in the enzymatic oxygen atom transfer re

58 )Fe(2)(mu-H)](-1) by pulse EPR revealed that redox chemistry induces significant changes in Fe-C cova

59 Selenium (Se) redox chemistry is a determining factor for its environm

60 deepen our understanding of proton-dependent redox chemistry of transition metal oxide surfaces.

61 p understanding of these phenomena and their redox chemistry remains incomplete.

62 con electrodes originate from silica-silicon redox chemistry.

63 and its reduced forms NAD(P)/NAD(P)H are all redox cofactors with key roles in energy metabolism and

64 to glucose and thereby yields the necessary redox cofactors.

65 rategically chosen as the vdW heterojunction redox component.

66 )Mn(0.75)]O(2), present in almost all oxygen-redox compounds, is lost on charging, driven in part by

67 To better understand their fate when soil redox conditions change, that is, from flooding to drain

68 Structures determined under various pH/redox conditions illuminate the role played by previousl

69 re observed, revealing new information about redox conditions within the treatment zone.

70 rtidal communities are adapted to changes in redox conditions.

71 oundwater, reflecting greater sensitivity to redox conditions.

72 ndwater systems under changing or stratified redox conditions.

73 Here, we quantified how oxidation-reduction (redox) conditions impact the fate of a Green fluorescent

74 We then construct an eCRISPR based redox conduit in both E. coli and Salmonella enterica.

75 d hence for the release and oxidation of its redox content during the collision onto UMEs.

76 en associated with a highly abnormal loss of redox control in TNBC cells.

77 ecules regulating interneuron function under redox control include NMDA receptor subunits GluN1 and G

78 materials as well as in the construction of redox-controlled supramolecular assemblies.

79 llowing the injection, despite the lack of a redox counter-reaction or conventional electrolyte, pers

80 ctrodes for the oxidation of an outer-sphere redox couple (ferrocene methanol) and two inner-sphere r

81 ein A (SpA) in the presence of electroactive redox couple ferri/ferro cyanide (K(3)/K(4)[Fe(CN)(6)]).

82 and environments for tuning the Ni(II)/(III) redox couple such as strongly donating thiolates in Ni s

83 studied NTO reduction by the hematite-Fe(2+) redox couple to assess the importance of this process fo

84 ffect dye regeneration (with I(-)/I(3)(-) as redox couple) and hole transport in NiO-based p-DSCs.

85 NADH and NAD(+) are a ubiquitous cellular redox couple.

86 ysts on Cu-modified CeO(2) supports based on redox-coupled atomic layer deposition.

87 le (ferrocene methanol) and two inner-sphere redox couples (potassium ferrocyanide and dopamine).

88 pies (XAS), which together indicate that all redox couples are ligand-localized.

89 First, the major redox couples in the mitochondrial matrix (NAD, NADP, th

90 ss common due to less-accessible metal-based redox couples.

91 ng S(4)(* -)/S(4)(2-) and S(3)(* -)/S(3)(2-) redox couples.

92 t PYO:eDNA interactions support an efficient redox cycle with rapid EET that is faster than the rate

93 g(0) as a crucial step in the atmospheric Hg redox cycle.

94 tic activity in ascorbate oxidation based on redox cycling between Cu(I) and Cu(II) , as well as thei

95 plained by major U speciation changes due to redox cycling in the wetland.

96 eployment of HsmA to capture heme and reduce redox damage caused by inflammatory mediators of protect

97 yocytes, or adult LV myocytes isolated from "redox dead" (Cys17Ser) PKARIalpha knock-in mice and thei

98 ammatory cytokine signatures associated with redox dependent induction of ASK1 and activation of stre

99 off) 19,000-fold, thus creating a reversible redox-dependent switch with 70-fold faster dissociation

100 ensity functional theory (DFT) calculations, redox disproportionation forms [Cu(III)](C=CAr)(R) speci

101 The electrochemical detection of synthetic redox DMPC (1,2-dimyristoyl-sn-glycero-3-phosphocholine)

102 ity, the electrochemical detection of single redox DMPC liposome collisions at polarized UMEs was inv

103 etect current spikes corresponding to single redox DMPC liposome collisions with K(3)Fe(CN)(6)/K(4)Fe

104 d and validate the mechanism of bias-induced redox-driven color switching.

105 biochemical level, analyzing the subcellular redox dynamics of NAD in living plant tissues has been c

106 Converging evidence implicates redox dysregulation as a pathological mechanism driving

107 oadditions, leads to concise, divergent, and redox-economic total syntheses of several polycyclic mem

108 This redox-economical approach can be used to append nearly a

109 The respiratory complex I transduces redox energy into an electrochemical proton gradient in

110 in mitochondria involving complex I converts redox energy into chemical energy and likely evolved fro

111 e known to be tightly linked to the cellular redox/energy status of the cell, the regulatory mechanis

112 1-mediated H(2)O(2) production regulates the redox environment and fine tunes Rho activity across the

113 in of SrrB sense and respond to the cellular redox environment to regulate S. aureus survival and pat

114 these findings, we propose that both pH and redox environments regulate cargo binding to a hydrophob

115 for the study of electron transfer in other redox enzymes and paves the way to explore transient sta

116 s such as fibroblast growth factor 2 (FGF2), redox enzymes such as thioredoxin, and proteins most exp

117 omplement proteins, carbonic anhydrases, and redox enzymes that ostensibly contribute to the therapeu

118 anism to convert absorbed photon energy into redox equivalents for enzyme catalysis.

119 Aqueous redox flow batteries (RFBs) are promising alternatives f

120 lly deployed as a catholyte in a non-aqueous redox flow cell with butyl viologen as the anolyte to yi

121 der conditions commonly found within aquifer redox fluctuating and transition zones where both arseni

122 c manipulations that increased mitochondrial redox generation promoted proline biosynthesis, while re

123 cytosolic, the molecular basis of the thiol redox homeostasis in the single mitochondrion of these p

124 arbon balance, phytohormone biosynthesis and redox homeostasis.

125 ogenesis, iron-sulfur cluster formation, and redox homeostasis.

126 samples from sources to sinks or across the redox/hydrological/trophic interfaces.

127 neous current switching (driven by molecular redox) in memristors based on Ru-complexes of azo-aromat

128 her 5f (n) configurations, rendering it both redox-inactive and resistant to forming chemical bonds t

129 olutions (ITIES) is ideally suited to detect redox-inactive ions by their ion transfer.

130 utions (ITIES) are often performed to detect redox-inactive species; unfortunately, due to the inhere

131 ral [Zn(tpyPY2Me)](2+) analog containing the redox-inactive Zn(II) ion.

132 ic responses at 0.3 V, in the absence of any redox indicators, allowed a single E. coli cell detectio

133 emonstrated an improvement in sleep pattern, redox, inflammatory profiles, and biomarkers of endothel

134 acids for intramolecular electron-transfer, redox-innocent Lewis acids separate these two functions

135 r myocytes expressing wild-type SERCA2b or a redox-insensitive mutant in which C674 is replaced by se

136 stal structure, spectroscopic properties and redox interconversions of a set of Ni(II) complexes comp

137 However, the sluggish S redox kinetics, especially under high S loading and lean

138 med quantitative in situ hepatic glutathione redox mapping in zebrafish larvae carrying targeted muta

139 a new SECM method to in situ investigate the redox mechanism of cardiomyocytes at a single-cell level

140 acer experiments support a Mars van Krevelen redox mechanism where CH(4) is activated by reaction wit

141 dy advances our fundamental understanding of redox mechanisms in disordered rocksalt oxyfluorides, hi

142 Photo-redox mediated ring-opening metathesis polymerization (p

143 Redox-mediated HIF-1alpha inactivation also decreased th

144 The reversibility of the redox-mediated initiation and propagation steps enable s

145 emerging ROMP technique that uses an organic redox mediator and a vinyl ether initiator, in contrast

146 Cathodic reduction at the cobaltocenium redox mediator substantially weakens the homolytic nitro

147 They differ from PEMFCs in their use of redox mediators dissolved in liquid electrolyte to condu

148 e that the combined loss-of-function of both redox metabolism-related systems configure a perfect sto

149 liver cancer cells altered the abundance of redox metabolites, including an increase in glutathione.

150 Redox modification of the conserved Cys243 inhibits the

151 ive to other cell cycle phases suggests that redox modifications could play prominent roles in regula

152 Redox modulators have been developed as an attractive ap

153 on was related to both the glutathione (GSH) redox molecule and the enzyme peroxidase (PO), which wer

154 microsensors were insensitive to interfering redox molecules and surpassed similar microelectrodes co

155 Attributed to the redox neutral and strong acid/base-free reaction conditi

156 acid (HOSA) has been explored as an in situ redox-neutral directing group for the formation of N-PAH

157 Herein, we introduce a microfluidic redox-neutral electrochemistry (muRN-eChem) platform tha

158 The facilitation of redox-neutral reactions by electrochemical injection of

159 may offer a general blueprint for affecting redox-neutral S(N)1 substitutions under mild conditions.

160 of TEMPO(.) catalysis for the development of redox-neutral transformations are rare.

161 n a homogeneous setting through the use of a redox non-innocent terpyridine-based pentapyridine ligan

162 The redox noninnocence of the TAML scaffold in cobalt-TAML (

163 nd can transfer electrons to a physiological redox partner.

164 The position of the redox peak was found to shift -60 +/- 2 mV pH(-1) at 25

165 ce, we report for the first time nonturnover redox peaks that are very likely intrinsic to the redox

166 s found to offer the optimal balance of high redox potential (E(1/2) = +1.19 V vs Fc(+/0)) and charge

167 Redox potential (Eh) measurements are widely used as ind

168 e biosynthesis, while reducing mitochondrial redox potential and/or ATP synthesis impaired proline bi

169 er will be formed outside templates when the redox potential exceeds the potential windows of nonaque

170 e transport model capable of reproducing the redox potential in space and time together with the site

171 try show that the cage anodically shifts the redox potential of the encapsulated quinone by a signifi

172 catalysis smoothly proceeds at the 1((II/I)) redox potential.

173 D tumor model, governed by the Cu(+2)/Cu(+1) redox potential.

174 e porphyrin (AlPor, AlPorF(3), or AlPorF(5)) redox potentials are tuned by the substitution of phenyl

175 were conducted with two paddy soils covering redox potentials from E(H) -260 to +200 mV.

176 ectrochemical studies that gave insight into redox potentials of the four mitochondrial complexes and

177 zed structures with reduced optical gaps and redox potentials.

178 as compared to previous work with a ferroin redox probe confined in a thin solution layer.

179 esence of ferrocenemethanol as an additional redox probe in the aqueous solution (at room temperature

180 The nanoMIP tagged with a redox probe, combines both recognition and reporting cap

181 6)/K(4)Fe(CN)(6) as the encapsulated aqueous redox probe.

182 Although results show that outer-sphere redox probes are unproductive for particle characterizat

183 echanism of conversion of the boronate-based redox probes to oxidant-specific products.

184 ifies that the linker type and the number of redox probes within the cage are useful handles to fine-

185 ed two ratiometric electrochemical molecular redox probes, Formaldehyde oxidative latent probe (FOLP)

186 s discussed, as well as the intrinsic oxygen redox process at high potentials, which is 75 % reversib

187 oride, Li(2)MnO(2)F, we show that the oxygen redox process in such materials involves the formation o

188 e effects of each molecular interface on the redox process of surface-adsorbed protein species.

189 des functioning via coupled cationic-anionic redox processes as a potential way of achieving higher c

190 ogen and/or the premonolayer palladium oxide redox processes at Pd.

191 The ability to harness cellular redox processes for abiotic synthesis might allow the pr

192 e describe the fundamental photochemical and redox processes that can enable its realization.

193 nds that undergo 1e(-)/1H(+) and 2e(-)/2H(+) redox processes.

194 ionally rely on chromogenic, fluorogenic, or redox properties for analyte detection that, in many ins

195 The redox properties were studied by cyclic voltammetry, whe

196 . distribution, circulation time, excretion, redox properties) is also mentioned.

197 lo-cations that have distinct electronic and redox properties.

198 peaks that are very likely intrinsic to the redox protein(s) performing the HET.

199 mHAMs preferentially react with redox proteins including disulfide oxidoreductase enzyme

200 w specific TPEF markers, namely, the optical redox ratio and mitochondrial fractal dimension, correla

201 omics analysis revealed decreasing levels of redox ratios in synchrony with the cycles.

202 e tumor microenvironment, namely, acidic pH, redox reactants, and hypoxia, are exploited.

203 the kinetics and electron flux of the cyclic redox reaction are key to the layer-to-tunnel structure

204 PhC(NtBu)(2)], has been synthesized from the redox reaction of the dipotassium bis(silylenyl)-nido-ca

205 dating the catalytic mechanism of this multi-redox reaction on metal-oxide photoanodes remains a sign

206 ation, structural evolution, electrochemical redox, reaction mechanism, kinetics, and degradation.

207 time, a mechanistic study for catecholamine redox reactions at LSGE as the electron transfer-chemica

208 phologies of manganese oxides, which undergo redox reactions coupled to sodium ion uptake and release

209 de iodine(V) complexes are unreactive toward redox reactions due to the fact that in such cases two e

210 ution can potentially promote and homogenize redox reactions in battery materials.

211 dislocations, are reported to influence the redox reactions in battery particles through single-part

212 the potential of utilizing reversible oxygen redox reactions in conventional layered oxides for high-

213 indicating that dislocations may facilitate redox reactions in layered oxides during initial chargin

214 imensional interplay among coupled catalytic redox reactions occurring in an EBFC-based sensor and pr

215 l electrochemical method to mimic the cyclic redox reactions occurring over long geological time scal

216 s)(18)] into the CV treated polymer promotes redox reactions through generation of alternative electr

217 bifurcation uses free energy from exergonic redox reactions to power endergonic reactions.

218 These findings provide evidence that redox reactions within beta-oxidation and the electron t

219 new insights for natural biotic and abiotic redox reactions, and explained the dominance of todoroki

220 nine dinucleotide (NAD(+)) is a coenzyme for redox reactions, making it central to energy metabolism.

221 ionship between crystallographic defects and redox reactions.

222 ocks, solvents, metal catalysts, energy, and redox reagents.

223 l mechanisms that specify cysteines that are redox regulated by EGF stimulation, we performed time-re

224 enabled unbiased interaction mapping for the redox regulated sensor protein, KEAP1, for the first tim

225 olism, collagen biosynthesis and remodeling, redox regulation and immunomodulatory proteins typify a

226 We place special emphasis on redox regulation and inflammation, with a focus on IL-1b

227 de insights into the origin and evolution of redox regulation in the protein kinase superfamily and m

228 ar dynamics simulations indicated widespread redox regulation of cryptic cysteine residues that are s

229 PR/Cas9 screen against NRF2 target and other redox regulatory genes in both 2D- and 3D-culture system

230 pper on a gold substrate followed by in situ redox replace reaction in a Pd salt solution.

231 pportunity to covalently link multi-electron redox responsive POM cores with virtually any (bio)organ

232 ated MMP9 (matrix metalloprotease 9) via its redox-responsive regulatory sites, causing a cascade of

233 nnic acid (TA), a polyphenolic compound with redox scavenging and anti-inflammatory effects, and poly

234 onnection between this signaling pathway and redox sensing, we found that antioxidant and redox-activ

235 ic determinants of 2-OG levels, we uncover a redox sensitive mitochondrial lipoylation pathway, depen

236 effect may become critical in volatile-rich, redox sensitive rocks such as carbonate-rich lithologies

237 substrate-binding domain of FBXL5 contains a redox-sensitive [2Fe-2S] cluster that, upon oxidation, p

238 ures of the Aurora A kinase domain delineate redox-sensitive cysteine residues that, upon covalent mo

239 ed the co-occurrence of four health-relevant redox-sensitive elements (U, As, V, and Cr) in 1494 grou

240 nd protein interaction "social network" of a redox-sensitive protein in cells with high temporal reso

241 Empirically, sustained expression of the redox-sensitive S70pBcl2 prevents oxidative stress-induc

242 phosphorylation at serine-70 functions as a redox sensor to prevent drug-induced oxidative stress-me

243 Here, we propose that redox shifts can also arise from small perturbations tha

244 in yields from coupling reactions with added redox shuttles (generally >80%) and those without (gener

245 veral diseases are associated with disturbed redox signaling and altered metabolism of sulfur-contain

246 n and implicate mitochondrial energetics and redox signaling as therapeutic targets for cardiac aging

247 ts positive side in physiology and health in redox signaling, "oxidative eustress," whereas at higher

248 establish several paradigms of physiological redox signaling.

249 es oxidative stress and Nox isoform-specific redox signalling important in vascular dysfunction assoc

250 veal that Na(+) controls OXPHOS function and redox signalling through an unexpected interaction with

251 ysiological levels and their central role in redox signalling via different post-translational modifi

252 veral other reactive species are involved in redox signalling, for instance nitric oxide, hydrogen su

253 troactive drugs on the basis of the target's redox signature.

254 The AgNC provides a new additional redox site, capturing externally supplied electrons with

255 y chemical transformations require metal and redox sites in proximity at interfaces; however, in trad

256 cleaning experiments and detecting benchmark redox species in solution.

257 ed by three subsequent sections bridging the redox spectrum from pure anionic, to a mix of cationic a

258 stitution of tungsten with tellurium induces redox stability, directing the functionality of the pero

259 amined for cell cytotoxicity, proliferation, redox state and migration using mouse embryonic fibrobla

260 ing key influence on Earth's climate and the redox state of (sub)surface Earth.

261 These data suggest that the redox state of adipocytes controls the development of ur

262 rturbations that alter oxygen consumption or redox state support a model in which surfactin-mediated

263 en different oligomeric states, depending on redox state, pH, posttranslational modifications, and ot

264 certain specific aspects, like the endosomal redox state, remain less characterized.

265 for GSSG, only weakly affected the cytosolic redox state.

266 e coordination geometry upon a change in the redox state.

267 olic profiles, controlled by their different redox states and expression of Activating Transcription

268 us to obtain real-time live cell imaging of redox states in TfR1-mediated endocytosis, attesting a r

269 tine structures of the Fe(III) and Fe(IV) =O redox states of a B-type DyP.

270 Obtaining structures of intact redox states of metal centers derived from zero dose X-r

271 Individual redox states of the reductase CntB and the catalytic com

272 er reorganization energy and more accessible redox states than the monomeric analogues.

273 with substrates and antagonists in different redox states.

274 2)GPI exists in two almost equally populated redox states: oxidized, in which all the disulfide bonds

275 redoxin (TXN), a major regulator of cellular redox status and, in addition, identified PRDM5 as a nov

276 a multiforme (GBM) cells would have abnormal redox status due to bio-thiols, like glutathione (GSH),

277 hat the production of 16:1t is linked to the redox status of the chloroplast through PRXQ associated

278 of glutathione, a key regulator of cellular redox status, are reduced in the medial prefrontal corte

279 resolving a cascade of fast and co-occurring redox steps in enzymatic systems such as ferritin.

280 Presented herein is the design of a redox stimuli activatable metal-free photosensitizer (aP

281 In this context, photonic and redox stimuli represent highly appealing modes of activa

282 vating the p47(phox) subunit that results in redox stress and accelerated fibrosis in the aged.

283 profiles in cells experiencing metabolic or redox stress confirmed that KEAP1 sheds many basal inter

284 Redox studies indicate that normal fused sapphyrins are

285 l alpha9 helix (C175) of BFL-1 operates as a redox switch to control the accessibility of the anti-ap

286 Here we show how harnessing the redox-switchable chelating and donating properties of an

287 A large number of Cys peptides (412) were redox switched, representing central pathways of mitocho

288 We identify a global operation of thiol redox switches that is required for optimal usage of ene

289 t is first explored to establish features of redox system in the CSD subjects compared to a healthy p

290 a detrimental imbalance in the mitochondrial redox system, which will lead to neuron death.

291 , exclusively SNc neurons showed an oxidized redox-system, i.e., a low reduced/oxidized glutathione (

292 s method provides a means by which bacterial redox systems can be exploited to generate "unnatural" p

293 d physiological studies have shown that both redox systems, NTRC and FDX-FTR-TRXs, participate in fin

294 In addition to describing the redox thermodynamics behind HET, we suggest that the cor

295 nd relationship for anion redox and cationic redox to occur reversibly.

296 Numerous redox transformations that are essential to life are cat

297 Redox-triggerable polymer self-immolation promotes drug

298 We propose that redox vulnerabilities could be exploited for therapeutic

299 The isoindigo 4-7 shows multi-redox waves with a low electrochemical band gap, which s

300 omenon is attributed to the nature of oxygen redox which is very likely mostly associated with Mn.