ライフサイエンスコーパス: redox system

1 ell as at 25 degrees C (with the glutathione redox system).

2 to behave as an attractive organoboron multi-redox system.

3 t, to a depleted mitochondrial thioredoxin-2 redox system.

4 he robustness of the subordinate glutathione redox system.

5 ications despite their relatively well-known redox system.

6 provides only static information on a given redox system.

7 that are formed by the poxvirus cytoplasmic redox system.

8 isulfide bonded via the poxvirus cytoplasmic redox system.

9 y factors that varied with the monolayer and redox system.

10 to the function of a large number of E. coli redox systems.

11 efense and functions importantly in cellular redox systems.

12 olarity to control PCET reactions in organic redox systems.

13 aning in the context of the endogenous thiol redox systems.

14 and the redox state of the stromal Cys-based redox systems.

15 for designing scalable, photosensitizer-free redox systems.

16 herto hidden dynamics of endogenous cellular redox systems.

17 n the reductive states of NAD(H) and NADP(H) redox systems.

18 pid electron transfer in a number of dynamic redox systems.

19 olypeptide chain is exactly the same in both redox systems.

20 versible voltammetric response for all these redox systems.

21 and IrCl(6)2-/3-) and nonaqueous (ferrocene) redox systems.

22 d active voltammetric response for all three redox systems.

23 se (TGR), that exhibits specificity for both redox systems.

24 For the Fe redox system, a limit of detection of 10(-8) M was calcu

25 as examined as a representative two-electron redox system; a Nernst slope of -30.8 mV was obtained.

26 luence of ionizable residues on a particular redox system, an important step toward understanding the

27 o histones caused loss of homeostasis of the redox system and in [Ca(2+)]i, as well as defects in mit

28 fragilis lacks the glutathione/glutaredoxin redox system and possesses an extensive number of putati

29 also contain a fully functional glutathione redox system and the low-molecular-weight thiol glutathi

30 establish the relationship between these two redox systems and its impact on plant development.

31 ethod to characterize the EEI in solid-state redox systems and reactive electrochemistry at precisely

32 Both flavodoxin and ferredoxin redox systems are able to support this enzymatic activit

33 It is proposed that the Fd-FTR-Trx and NTRC redox systems are linked by the redox balance of 2-Cys P

34 t three different temperatures (with the DTT redox system) as well as at 25 degrees C (with the gluta

35 obtain near 100% CO selectivity in a cyclic redox system at 750-935 degrees C, which is a significan

36 nthesis of a new class of such a three-state redox system based on the intermolecular reaction of a l

37 sfer in several characterized extracytosolic redox systems but the breadth of functions of this modif

38 ts rapid electron exchange with outer-sphere redox systems, but not with dopamine, which requires ads

39 s method provides a means by which bacterial redox systems can be exploited to generate "unnatural" p

40 arked accumulation of intracellular cystine, redox system collapse and rapid cell death, which can be

41 other disulfide molecules, NADPH depletion, redox system collapse, and rapid cell death (likely disu

42 can be activated in vitro by a two-component redox system comprised of soluble cytochrome b5 and P450

43 -state analysis of a M. tuberculosis class I redox system comprising flavoprotein reductase A (FprA),

44 Within the apicoplast, the ferredoxin redox system consists of plant-type ferredoxin-NADP(+) r

45 pH and the predominance of the two-electron redox system (DPDE)Rh(I)/(DPDE)Rh(III) at both low and h

46 uch a gradient allows the differentiation of redox systems due to their intrinsic difference in therm

47 Two different thiol redox systems exist in plant chloroplasts, the ferredoxi

48 rements are performed in the presence of the redox system ferri-/ferrocyanide and show an increase in

49 ating the importance of a full physiological redox system for activation/inactivation.

50 Thionine was used as redox system for electrochemical probe.

51 nd relies on the CoA/CoA disulfide reductase redox system for protection from oxidative damage.

52 riments show that dopamine is a well-adapted redox system for SECM in feedback mode and in unbiased c

53 ctase (TrxB)/Trx system is the major or sole redox system for thiol/disulfide cellular homeostasis in

54 of 2-Cys PRXs with NTRC and the FDX-FTR-TRXs redox systems for fine tuning chloroplast performance in

55 solic redox couples, such as the NADH/NAD(+) redox system, for the regulation of Ca(2+) homeostasis h

56 n 1(2+), thereby establishing an organoboron redox system fully characterized in all three redox stat

57 a bifunctional active surface containing two redox systems has been obtained.

58 , exclusively SNc neurons showed an oxidized redox-system, i.e., a low reduced/oxidized glutathione (

59 sion, we demonstrate that a polyph-dependent redox system in blood cells is necessary to maintain the

60 on the potential role of the plasma membrane redox system in caloric restriction-induced pathways res

61 erent complications arising from isolating a redox system in multiple oxidation states without drasti

62 ates a fine-tuned and balanced intracellular redox system in plant defence responses to biotic and ab

63 er significance for the CoASH/CoAS-disulfide redox system in prokaryotic thiol/disulfide homeostasis.

64 t is first explored to establish features of redox system in the CSD subjects compared to a healthy p

65 the major, if not the sole, thiol/disulfide redox system in this anaerobe required for survival and

66 his failure, including the complexity of the redox system in vivo.

67 (GSH) systems are considered to be two major redox systems in animal cells.

68 nent in the thioredoxin system, one of major redox systems in mammals that links NADPH and thiol-depe

69 In this review, we discuss the two redox systems in plants and systematically compare them

70 Specifically, the use of ambipolar redox systems in proton-coupled electron transfer (PCET)

71 hemical responses for aqueous and nonaqueous redox systems in the mid- and far-IR.

72 Redox systems including extracellular cysteine/cystine (

73 rapid electron-transfer kinetics for aqueous redox systems, including Fe(CN)(6)(-3/-4) and Ru(NH(3))(

74 idence that light- and NADPH-dependent thiol redox systems interact at the level of Trx f1 and NTRC t

75 Of the six rainwater redox systems investigated, only manganese speciation ap

76 y, cytoskeletal proteins, protein synthesis, redox system, ion channels and those with unknown functi

77 This important redox system is believed to be driven by an enzyme or en

78 We show that the G6PD-NADPH redox system is important for HK2 stability and metaboli

79 The interaction between the two thiol redox systems is indispensable to sustain photosynthesis

80 c molecular chaperone in the thiol/disulfide redox system, is unknown.

81 teractions between the PPF surface and these redox systems lead to their slow electron-transfer kinet

82 -Hush-Sutin methodology for strongly coupled redox systems lying in the (Robin-Day) Class II category

83 removed by eight endogenous antioxidant and redox systems, many components of which are expressed un

84 Thus, the NTRC, 2-Cys Prxs, and Fd-FTR-Trxs redox systems may act concertedly, but the nature of the

85 rejection and that manipulation of cellular redox systems may provide a means to protect xenogeneic

86 an explanation for how NADPH required by the redox system might be generated in Plasmodium spp.

87 d physiological studies have shown that both redox systems, NTRC and FDX-FTR-TRXs, participate in fin

88 lation of cell growth and stimulation of the redox system of plasma membranes are correlated.

89 Here we explore the effects of cellular redox systems on the aggregation of hSOD1 proteins.

90 s with agents that modulate the antioxidants/redox system or boost endogenous antioxidants would be a

91 Plasma membrane-associated redox systems play important roles in regulation of cell

92 Thiol-based redox systems play pivotal roles in the success and surv

93 dative damage, but the involvement of the PM redox system (PMRS) in these processes is unknown.

94 on an HT-BDD electrode the [Fe(CN)6](3-/4-) redox system presents a reversible voltammetric behavior

95 Moreover, the combined deficiency of both redox systems provokes aberrant chloroplast ultrastructu

96 wth depended on the last enzyme IspH and its redox system ptFd and ptFNR.

97 hange rate constant implies that this CuII/I redox system represents a true geometric "entatic state.

98 These parasites possess a unique thiol redox system required for DNA synthesis and defense agai

99 Treatment with inhibitors of these redox systems resulted in an increase of hSOD1 aggregate

100 Dysregulation in cellular redox systems results in accumulation of reactive oxygen

101 lectrochemical response, with five different redox systems serving as probes (Fe(CN)(6)(3)(-)(/4)(-),

102 iline film using Fe(2+)/Fe(3+) in HCl as the redox system shows five distinct linear segments (bands)

103 ansfer rates similar to those on GC, but for redox systems such as Fe3+/2+, ascorbic acid, dopamine,

104 ion, chloroplasts contain an NADPH-dependent redox system, termed NTRC, which allows the use of NADPH

105 ytes contain an unidentified plasma membrane redox system that can reduce extracellular monodehydroas

106 GSH) is a critical component of the cellular redox system that combats oxidative stress.

107 nd thioredoxin (Trx) define a major cellular redox system that maintains cysteine residues in numerou

108 n oxidoreductase central to the unique thiol-redox system that operates in trypanosomatid protozoa, h

109 Organic redox systems that can undergo oxidative and reductive (

110 ve shown that in addition to these cytosolic redox systems the parasite also has an important mitocho

111 For all five redox systems, the forward reaction peak current varied

112 e proposed method was validated with a model redox system then further evaluated by determining ascor

113 Here, we exploit bacterial redox systems to induce a copper-mediated radical polyme

114 as implied by the inability of other related redox systems to substitute for Rv3230c.

115 decade, that a rapid development of various redox systems took place, thus triggering a renaissance

116 these pathways and their crosstalk with the redox system under shear stress is lacking.

117 ve detection for the study of solution-phase redox systems under strict diffusion control.

118 d PrnB was inactive when coupled with common redox systems under various conditions.

119 itroreductases (Nrs) play important roles in redox system via NADPH or NADH as a reductant.

120 n the stability and reversibility of the new redox system, we could demonstrate by charge-discharge e

121 lectrodes was evaluated using nine different redox systems; well-defined and stable diffusion-limited

122 es including proteasome system and mycothiol redox system were also identified as conditionally essen

123 ate constants, k degrees (app), for all five redox systems were determined from deltaE(p)-nu experime

124 eneous electron-transfer kinetics of various redox systems were evaluated.

125 th ultramicroelectrodes (UMEs) even for fast redox systems, where different sizes of UMEs are used to

126 forded 1, making a fully reversible 1 <--> 3 redox system, whereas two-electron oxidation with AuCl p

127 e phylum Apicomplexa, harbors the ferredoxin redox system which supplies electrons to enzymes of vari

128 a detrimental imbalance in the mitochondrial redox system, which will lead to neuron death.

129 ion could be indicative of the presence of a redox system, which would regulate the activity of this

130 orted here feature a reversible two-electron redox system with regular or inverted redox potentials f

131 New redox systems with three oxidation states are highly sou

132 rapid rates of electron-transfer for soluble redox systems without conventional pretreatment, long-te