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1 ylcholine-dependent memory tasks (recent and reference memory).
2 ion of place-nonreward associations (spatial reference memory).
3 nd locations that remained spatially stable (reference memory).
4 cent memory are more intense than on spatial reference memory.
5 O of Slitrk2 caused abnormalities in spatial reference memory.
6  the model that LTD underlies flexibility of reference memory.
7 t placement, social recognition, and spatial reference memory.
8 s, but this did not appear to impact spatial reference memory.
9 ors compared to wildtype mice but had normal reference memory.
10 n important component of spatial working and reference memory.
11 ory without altering previously consolidated reference memory.
12  route-based learning, spatial learning, and reference memory.
13 ed LTP as well as contextual fear memory and reference memory.
14 re used to assess contextual fear memory and reference memory.
15 TLR3 ligand impaired working memory, but not reference memory.
16 on-spatial working memory and no deficits in reference memory.
17 Morris water maze, AC8 KO mice showed normal reference memory.
18 mination through greater reliance on spatial reference memory.
19 al water maze assessing associative learning/reference memory.
20 quiring only working memory or the update of reference memory.
21 t never contained platforms assessed spatial reference memory.
22  and thus is important for a complex form of reference memory.
23 ion between working/episodic-like memory and reference memory.
24 ampus is needed for both spatial working and reference memories.
25 ivities, goal-directed learning, and spatial reference memories.
26 rats exhibit significant deficits in spatial reference memory acquisition and working memory performa
27 hermore, they had impaired long-term spatial reference memory and a decreased theta frequency in hipp
28  infection with AAV1-I(2CTF) induced spatial reference memory and memory consolidation deficits and a
29 (MWM) to test for time of day differences in reference memory and monitored daily patterns of hippoca
30  Estrous cycle-related variations of spatial reference memory and neurochemistry in intact female mic
31 mice showed a significant decline in spatial reference memory and reversal learning from both young a
32                                     Tests of reference memory and working memory in the water maze fa
33  working memory, non-spatial working memory, reference memory) and exposed to manganese sulfate (15-2
34 Morris water maze assessing spatial learning/reference memory, and a non-spatial water maze assessing
35 ed impairments in contextual memory, spatial reference memory, and nest building.
36 , egocentric learning, allocentric learning, reference memory, and proximal cue learning.
37               Although the effects of NGF on reference memory are well documented, the influence of N
38                    BST and long-term memory (reference memory) are impaired only later (approximately
39 N2 levels experienced a reduction in spatial reference memory as indicated by Y-maze assessment.
40                                      Spatial reference memory, as assessed by performance on the wate
41 owed significantly better performance in the reference memory aspect of the spatial water maze task.
42 ugh no sex differences were found in spatial reference memory at a young age, the mnemonic ability of
43 ired in working memory as well as attenuated reference memory, both functions essential for effective
44 ptor (GluR-A-/- mice) display normal spatial reference memory but impaired spatial working memory (SW
45  nAChR subunit gene, showed improved spatial reference memory compared with APP/PS1 aged-matched litt
46  nAChR subunit gene, showed improved spatial reference memory compared with APP/PS1 aged-matched litt
47 day of testing, when there was a decrease in reference memory compared with their non-restraint contr
48 male C57BL/6 mice learn both the working and reference memory components of a water-escape motivated
49 nutcrackers and jackdaws in both working and reference memory components of the maze.
50 ited compromised learning of the working and reference memory components of the task, and made more w
51                             Both working and reference memory declined with age, but only working mem
52 er maze) and allocentric learning and caused reference memory deficits (Morris water maze), but did n
53 ing Abeta4-42 and completely rescued spatial reference memory deficits after passive immunization.
54                                              Reference memory deficits and declines in performance in
55 /+) causes hippocampus-dependent spatial and reference memory deficits independently of alpha-synucle
56                                       Hence, reference memory demand was uniquely associated with a g
57             Individuals with greater spatial reference memory developed larger home ranges.
58 he number of working memory errors (WME) and reference memory errors (RME) per training day were reco
59 nificantly reduced the number of working and reference memory errors committed over sessions, while N
60 ffer in the commission of working memory and reference memory errors in the radial maze or in the use
61 determining the number of working memory and reference memory errors made in each session, working me
62 ant effects of MLA or DHbetaE were seen with reference memory errors or response latency.
63 by the decrease in the number of working and reference memory errors over sessions.
64 significantly more working memory errors and reference memory errors than all other groups.
65 itted significantly fewer working memory and reference memory errors than females throughout testing.
66 e/wk E(2) group committed significantly more reference memory errors than VEH and Contin E(2) groups,
67 es, supplemented rats made fewer working and reference memory errors; however, the memory enhancing e
68 se results demonstrate that both working and reference memory for temporal information are sensitive
69 eak time indicate a change in the content of reference memory for the remembered time of reinforcemen
70            In addition, the earlier onset of reference memory impairments in females generally coinci
71  6 months of age, indications of working and reference memory impairments were observed in transgenic
72                                              Reference memory improved moderately only in old rats.
73 r conditioning tasks, and normal working and reference memory in a water version of the radial arm ma
74 le mice may overcome age-related deficits in reference memory in an emotional or spatial learning tas
75 mined sex differences in spatial working and reference memory in C57BL/6 mice.
76 y play a critical role in regulating spatial reference memory in female mice.
77 published a method for examining working and reference memory in mice using a spatial version of the
78 o assess spatial and non-spatial working and reference memory in mice.
79 ce of both genders display increased spatial reference memory in the Morris water maze test.
80 mice normally acquired and retrieved spatial reference memory in the Morris water maze, but they were
81 ld not reverse deficits in working memory or reference memory in the radial arm water maze or in visu
82             The medium dose impaired spatial reference memory in the radial-arm maze, whereas all dos
83 d the Barnes circular maze to assess spatial reference memory in transgenic mice overexpressing human
84 rning is assessed across repeated trials and reference memory is determined by preference for the pla
85 f memory compared with vehicle controls in a reference-memory Morris water-maze behavior test that ap
86  but displayed normal olfaction, working and reference memory, motor abilities, anxiety, and repetiti
87                              A major drop in reference memory of the females occurred at the age of 1
88 on 2 spatial memory tasks that tested either reference memory or working memory.
89 In addition, successful watermaze and Y-maze reference memory performance depended on hippocampal fun
90 ent experiment evaluated spatial working and reference memory performance, as well as several physiol
91 le-age male F344 rats, followed by a spatial reference memory probe trial test.
92 controls, despite impairments on the spatial reference memory radial maze task.
93 extent, impaired spatial working memory, but reference memory remained intact.
94 g memory (SWM) tasks, although their spatial reference memory remains normal.
95 ce, Fmr1 KO rats show no deficits in spatial reference memory reversal learning.
96 ze, a complex task that requires working and reference memory simultaneously; at 12 months old, Tg im
97 tial working memory (SWM) but normal spatial reference memory (SRM).
98 ts were impaired relative to controls in the reference memory (successive discrimination) task for ei
99 l fear conditioning but no change in spatial reference memory, suggesting a supportive role for neuro
100                                To assess the reference memory system, and to serve as a control for n
101 ditions: when memory retrieval occurred in a reference memory task after performance had reached asym
102 elationship between performance in a spatial reference memory task and NGF distribution in the aged r
103                               Place-training reference memory task deficits were assessed in the Morr
104                               Place-training reference memory task deficits were observed in rats exp
105 ce on a spatial working memory and a spatial reference memory task in open field.
106 he present study, rats were presented with a reference memory task in which they had to find water th
107 e hippocampus, and their memory on a spatial reference memory task was assessed.
108 yed response) dependent on the PFC, and on a reference memory task with similar motor and motivationa
109 sk when cued by either their internal state (reference memory task) or their previous response (worki
110                                         In a reference memory task, pigeons (Columba livia) were trai
111  performance of Ts65Dn mice on a working and reference memory task.
112 t external, cues impaired performance on the reference memory task.
113 layed trajectories of the next chosen arm in reference memory tasks but the previously visited arm in
114 induced plasticity had no deficit in spatial reference memory tasks, but were impaired in an associat
115 formance was normal in hippocampus-dependent reference memory tasks, including contextual fear condit
116 at of nonstressed rats on both emotional and reference memory tests.
117    For the Morris maze, a measure of spatial reference memory, Tg rats demonstrated significant impai
118 onsumption of caffeine modulates working and reference memory through the antagonism of adenosine A2A
119                                              Reference memory update led to the reward-directed place
120 avoidance task, and both spatial working and reference memory versions of the Morris water maze.
121                                      Spatial reference memory was associated with CA1 transcriptional
122                                      Spatial reference memory was significantly impaired in both J20
123                                      Spatial reference memory was tested in cycling females, ovariect
124 al CA1 principal cells, acquired the spatial reference memory water maze task as well as controls, de
125 s influenced CA1 place cell assemblies while reference memories were partially updated.
126  effects of tau(P301L) expression on spatial reference memory were longitudinally tested using the Mo
127 ocentric strategy and formation of a spatial reference memory were not different between age groups;
128 ippocampal short-term learning and long-term reference memory, which appear to depend on the abundanc
129                                              Reference memory, which was tested in a place discrimina
130 or-delayed win-shift (nonspatial working and reference memory), win-stay (habit learning), and attent
131 idal cell excitability, and impaired spatial reference memory (without influencing learning).

 
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