ライフサイエンスコーパス: reflux

1 aspirations in children with extraesophageal reflux.

2 lfones or beta-hydroxysulfones in toluene at reflux.

3 s an adaptive response to gastro-oesophageal reflux.

4 ly milder conditions, such as vesicoureteral reflux.

5 cough due to reflux, LPR, and asthma due to reflux.

6 ealth, home oxygen use, and gastroesophageal reflux.

7 and the non-prescription medicines for acid reflux.

8 to facilitate diagnosis of pathological bile reflux.

9 ients required conversion to RYGB for severe reflux.

10 on 100 consecutive patients with pathologic reflux.

11 diarrhea, weight loss, and gastroesophageal reflux.

12 d to have esophageal remnants in addition to reflux.

13 with THF.BH(3) have to be carried out under reflux.

14 ating better function), and gastroesophageal reflux.

15 ive sleep apnea (+1 point), gastroesophageal reflux (+1 point), and depression (+1 point) was predict

16 plication for dysphagia - 5; redo Nissen for reflux - 1; paraesophageal hernia -1).

17 allergic rhinitis (62.4%), gastroesophageal reflux (42.1%), sinusitis (37.9%), nasal polyposis (30.2

18 terior fundoplication (Nissen conversion for reflux - 6), and 7 underwent revision following Nissen f

19 time (2-16 h), with a moderate temperature (reflux: 65 degrees C), and low concentration (0.014 M) o

20 scopically, 20/23 (87%) had macroscopic bile reflux (74% yellowish bile lakes, 13% greenish bile lake

21 In reactions conducted in refluxing acetic acid, however, the 3-(chloromethyl)coum

22 ated with 4-nitrobenzenesulfenyl chloride in refluxing acetonitrile, N-propargylic beta-enaminones pr

23 he belt was impaired esophageal clearance of refluxed acid (median values of 23.0 seconds without bel

24 23 had acidic reflux, whereas 9 had combined reflux [acidic + weakly acidic reflux (WAR)].

25 hageal acid exposure than impedance-measured reflux activity in patients with symptomatic gastro-oeso

26 Heating a toluene solution of 3a or 3b at reflux afforded the rearranged species ArSn(3-tricyclo[2

27 n of impaired clearance was that of rapid re-reflux after peristaltic clearance.

28 Esophagitis, whether caused by acid reflux, allergic responses, graft-versus-host disease, d

29 Secondary valves prevent reflux and allow for the generation of propulsive pressu

30 ageal pressure gradient was the mechanism of reflux and appeared to relate to the non-compliant proxi

31 is associated with chronic gastroesophageal reflux and esophageal cancer.

32 ted and unconjugated BAs existed in non-bile reflux and healthy juice.

33 lly diagnosed as gastritis with/without bile reflux and healthy subjects for BA profiles measurements

34 stomy in reducing macro and microscopic bile reflux and impact on dyspepsia related quality of life i

35 in fluxes through plasmodesmata enable auxin reflux and increase total root-tip auxin.

36 Barrett's esophagus (BE) is associated with reflux and is implicated the development of esophageal a

37 improvements in symptoms of gastroesophageal reflux and rhinitis, bronchial reversibility, and exhale

38 Delayed gastric emptying and bile reflux are common concerns in long-term survivors after

39 addition, extraesophageal manifestations of reflux are most effectively diagnosed with a stepwise ap

40 iencing gastroesophageal and extraesophageal reflux are often prescribed proton pump inhibitors (PPIs

41 , digital ulceration, and gastro-oesophageal reflux, are now treatable.

42 No longitudinal studies of reflux-associated erosive tooth wear and of reflux chara

43 Gastroesophageal acid reflux at each of the pH sensors extending 5.5 cm proxim

44 Interventions to treat reflux attempt to restore the integrity of the EGJ.

45 exposure of esophageal cells to acidic bile reflux (BA/A).

46 t eroGFP (and other ER luminal proteins) to "reflux" back to the reducing environment of the cytosol

47 s exposure arises via compromise of the anti-reflux barrier and reduced ability of the esophagus to c

48 symptoms also occur in the context of normal reflux burden, when there is either poor epithelial resi

49 es suggest that inhibition of ROS induced by reflux can be a useful strategy for preventing DNA damag

50 Nissen fundoplication for gastroesophageal reflux can be followed by troublesome side effects.

51 also present megaureters and vesicoureteral reflux, caused by failure of ureters to separate from Wo

52 reflux-associated erosive tooth wear and of reflux characteristics have been reported to date.

53 Postoperative serum HMGB1 and reflux cholangitis indicated recurrence and unfavorable

54 Though there was macroscopic bile reflux, clinical symptoms and microscopic changes were m

55 Interestingly, under reflux conditions, the microwave-heated (MWH) reaction d

56 arbituric/N,N-dimethylbarbituric acids under reflux conditions.

57 ctive study of patients with pathologic acid reflux confirmed by esophageal pH testing undergoing MSA

58 success, but with trade-offs between better reflux control vs. more side-effects after Nissen fundop

59 ociated with hemorrhage than cortical venous reflux (CVR) in patients with lateral sinus dural arteri

60 The outcome was recurrence of reflux, defined as use of antireflux medication (proton

61 The presence of gastroesophageal reflux disease (26%), esophageal stricture (39%), or bot

62 0%), sinusitis (47.8%), and gastroesophageal reflux disease (46.3%).

63 e (NERD) compared with patients with erosive reflux disease (ERD) or Barrett's esophagus (BE).

64 hageal disorders, including gastroesophageal reflux disease (GERD) and eosinophilic esophagitis (EoE)

65 sophageal manifestations of gastroesophageal reflux disease (GERD) and to compare the most recent tec

66 Management of gastroesophageal reflux disease (GERD) commonly starts with an empiric tr

67 variable rates of recurrent gastroesophageal reflux disease (GERD) have been reported.

68 The prevalence of gastroesophageal reflux disease (GERD) in Africa is not known but is beli

69 w data on the prevalence of gastroesophageal reflux disease (GERD) in the United States.

70 Gastroesophageal reflux disease (GERD) increases EAC risk, but whether an

71 Gastroesophageal reflux disease (GERD) is a common comorbidity among pati

72 Endoscopic management of gastroesophageal reflux disease (GERD) is being employed increasingly.

73 Gastroesophageal reflux disease (GERD) is caused by gastric acid entering

74 The pathogenesis of gastroesophageal reflux disease (GERD) is complex and involves changes in

75 Gastroesophageal reflux disease (GERD) is defined by recurrent and troubl

76 Gastroesophageal reflux disease (GERD) is prevalent worldwide, particular

77 Gastro-esophageal reflux disease (GERD) is suggested to be associated with

78 Gastroesophageal reflux disease (GERD) is the most prevalent gastrointest

79 Gastroesophageal reflux disease (GERD) is the strongest known risk factor

80 Refractory gastroesophageal reflux disease (GERD) reduces quality of life and create

81 Gastroesophageal reflux disease (GERD) seems to increase the risk of lary

82 f objective measurements of gastroesophageal reflux disease (GERD) would improve management of patien

83 table bowel syndrome (IBS), gastroesophageal reflux disease (GERD), and overactive bladder syndrome (

84 r three common indications: gastroesophageal reflux disease (GERD), Barrett's esophagus (BE), and non

85 appraise the prevalence of gastroesophageal reflux disease (GERD), esophagitis, and Barrett's esopha

86 Gastroesophageal reflux disease (GERD), functional dyspepsia (FD) and irr

87 f frequency and duration of gastroesophageal reflux disease (GERD), using data from a randomly select

88 Gastroesophageal reflux disease (GERD), which leads to acid reflux into t

89 ave a similar prevalence of gastroesophageal reflux disease (GERD), yet esophageal adenocarcinoma (EA

90 rease of 50% or more in the Gastroesophageal Reflux Disease (GERD)-Health Related Quality of Life sco

91 r the surgical treatment of gastroesophageal reflux disease (GERD).

92 ilic esophagitis (EoE), and gastroesophageal reflux disease (GERD).

93 s omeprazole monotherapy in gastroesophageal reflux disease (GERD).

94 (CNF) for the treatment of gastroesophageal reflux disease (GERD).

95 tory drugs) and symptoms of gastroesophageal reflux disease (GERD).

96 r patients with early-stage gastroesophageal reflux disease (GERD).

97 Gastroesophageal reflux disease (GORD) is a chronic and a common conditio

98 Although gastro-oesophageal reflux disease (GORD) is a common medical complaint, the

99 lastic progression in the gastro-oesophageal reflux disease (GORD)-Barrett's metaplasia (BM)-oesophag

100 patients with symptomatic gastro-oesophageal reflux disease (GORD).

101 t symptom burden in patients with nonerosive reflux disease (NERD) compared with patients with erosiv

102 apy in patients with proven gastroesophageal reflux disease [GERD]), to document physiologic levels o

103 The incidence of gastro-oesophageal reflux disease and Barrett's oesophagus is increasing.

104 nd a higher prevalence of gastro-oesophageal reflux disease and blistering/desquamating skin disorder

105 psies of patients with EoE, gastroesophageal reflux disease and controls.

106 ted with conditions such as gastroesophageal reflux disease and diabetes mellitus, as well as emergen

107 ties is similar except for gastro-esophageal reflux disease and dyslipidemia that appear to be more s

108 both procedures except for gastro-esophageal reflux disease and dyslipidemia, which were more success

109 of a confirmed diagnosis of gastroesophageal reflux disease by an abnormal esophageal pH study (body

110 that is distinguished from gastroesophageal reflux disease by the expression of a unique esophageal

111 rugs (lipid-lowering drugs, gastroesophageal reflux disease drugs, antihypertensive drugs, sleep aids

112 sses (lipid-lowering drugs, gastroesophageal reflux disease drugs, diabetes drugs, antihypertensive d

113 c antireflux surgery due to gastroesophageal reflux disease in adults (>18 years).

114 correlations of BE/EA with gastroesophageal reflux disease in male individuals and obesity in female

115 Antireflux surgery for gastro-esophageal reflux disease is followed by varying rates of re-interv

116 of GERD was by means of the gastroesophageal reflux disease questionnaire (GERDQ) while the diagnosis

117 vely high rate of recurrent gastroesophageal reflux disease requiring treatment, diminishing some of

118 r patients with symptomatic gastroesophageal reflux disease were older (58 for men and 64 for women)

119 tis, allergic rhinitis, and gastroesophageal reflux disease were only associated with increased exace

120 ts a potential biomarker for extraesophageal reflux disease when detected in airways, however a direc

121 ased abdominal pressure and gastroesophageal reflux disease, although this pathogenic mechanism has n

122 eep-disordered breathing, gastro-oesophageal reflux disease, and anxiety or depression.

123 isk factor for oesophageal adenocarcinoma is reflux disease, and the rising incidence of this coincid

124 have been identified-mainly gastroesophageal reflux disease, Barrett's esophagus, obesity, and tobacc

125 e and effective therapy for gastroesophageal reflux disease, but its effect on the LES has not been e

126 Risk factors include gastroesophageal reflux disease, central obesity, and smoking.

127 l complications of obesity: gastroesophageal reflux disease, erosive esophagitis, Barrett's esophagus

128 ry of peptic ulcer disease, gastroesophageal reflux disease, or gastrointestinal bleeding, and prior

129 f non-drug treatments for gastro-oesophageal reflux disease, safety of long-term drug treatment, and

130 evant to the association between obesity and reflux disease.

131 to those of patients with gastro-oesophageal reflux disease.

132 heartburn in patients with gastro-esophageal reflux disease.

133 ure by waist belt on reflux in patients with reflux disease.

134 o clear and buffer the refluxate, leading to reflux disease.

135 lications or recurrence of gastro-esophageal reflux disease.

136 squamous mucosa damaged by gastroesophageal reflux disease.

137 Chaperone-mediated ER protein reflux does not require E3 ligase activity, and proceeds

138 aromyces cerevisiae, we show that ER protein reflux during ER stress requires specific chaperones and

139 Gastroesophageal reflux/dysphagia and asthma/rhinitis represent a risk fa

140 ated with decreased frequency of symptomatic reflux episodes (P = 0.01) but increased frequency of re

141 ge of time with a pH < 4, the number of acid reflux episodes and the percentage of proximal esophagea

142 er one year, but the number of weakly acidic reflux episodes decreased significantly in the large sub

143 The prevalence rate of reflux esophagitis (RE) in Asia, including Taiwan, has i

144 ts should be considered high risk to develop reflux esophagitis and advised of the management conside

145 se) or is a significant risk factor, such as reflux esophagitis and gallstones.

146 Analyses of patients with severe GERD (reflux esophagitis or Barrett esophagus) showed similar

147 hree events (drug inefficacy, gastritis, and reflux esophagitis) in two patients were considered rela

148 oup and 20% of patients in the LHM group had reflux esophagitis, as assessed by endoscopy; at 24 mont

149 igraphy delineated bolus-induced deglutitive reflux events (29.6% vs. 62.5%, p=0.005) and post-prandi

150 (29.6% vs. 62.5%, p=0.005) and post-prandial reflux events (4(IQR2) vs. 4(IQR 3) events, p=0.356).

151 to evaluate the mechanisms associated with reflux events following sleeve gastrectomy (SG).

152 ressurization was associated with individual reflux events in most patients (90% in fasting state and

153 Reflux events measured by impedance were stratified by p

154 Following the meal, the mean number of reflux events with the belt was 4, vs 2 without (P = .00

155 nt manometry and pH for detailed analysis of reflux events.

156 pressure gradient (>10mmHg) underpinned most reflux events.

157 C-PAC also reduced levels of DNA damage in reflux-exposed rat esophagi, as observed by reduced leve

158 Background acidic esophageal reflux exposure appeared stable over time, whereas weakl

159 se (GERD) is complex and involves changes in reflux exposure, epithelial resistance, and visceral sen

160 percritical CO2 (SC-CO2) extraction and heat-reflux extraction (HRE), conducted in parallel.

161 those of the conventional SC-CO(2) and heat-reflux extractions, respectively.

162 -butanol in the presence of CH(3)COONH(4) at reflux followed by complexation with BF(3).OEt(2).

163 us, we found belt compression increased acid reflux following a meal.

164 1.1) with PCl(3) in toluene/triethylamine at reflux for 1 h.

165 dehydes in a cosolvent of toluene and THF at reflux for 10 h, and (ii) NaBH4 promoted regio- and ster

166 cosolvent of toluene and HOAc (v/v = 1/1) at reflux for 3 h.

167 pressants for symptoms of gastro-oesophageal reflux for more than 6 months, and had not undergone an

168 ation revealed that solution-based assembly (refluxing for days) results in the formation of large ol

169 istinguish extraesophageal manifestations of reflux from idiopathic chronic cough, laryngitis due to

170 Gastric BAs were attributed to abnormal refluxing from duodenal compartments and correlated with

171 onjugated BAs are associated with human bile reflux gastritis.

172 esophagogastroduodenal anastomosis model for reflux-generated esophageal damage were used to investig

173 Gastro-oesophageal reflux (GER) and microaspiration have been proposed as r

174 Gastro-esophageal reflux (GERD) post-SG is a critical issue due to symptom

175 tooth wear have significant gastroesophageal reflux (GERD), despite minor reflux symptoms.

176 n and characterization of gastro-oesophageal reflux (GOR), yet the two modalities frequently differ i

177 Outcomes studied included reflux grade, UTIs during the study on placebo or antibi

178 he further elucidation of the causal role of reflux in erosive tooth wear.

179 xcessive apoptosis leading to vesicoureteral reflux in newborns, which underscores the importance of

180 creasing abdominal pressure by waist belt on reflux in patients with reflux disease.

181 Microscopic bile reflux index (BRI) was calculated and a score more than

182 Mean bile reflux index score was 9.7 (range 1.77-34).

183 patients, significantly greater symptoms of reflux, indigestion and abdominal pain were reported.

184 non called "Minority MOMP." We asked whether reflux-induced esophageal carcinogenesis occurred via mi

185 ells incubated with cum-OOH and in rats with reflux-induced esophageal damage.

186 Acid reflux-induced oesophagitis and the multilayered epithel

187 l reflux disease (GERD), which leads to acid reflux into the esophagus, is a common gastrointestinal

188 Acute biliary pancreatitis, caused by bile reflux into the pancreas, is a serious condition charact

189 udden and severe condition initiated by bile reflux into the pancreas.

190 The main complication was blood reflux intra-operatively (66.7%), which resolved without

191 us may be more suitable when early stasis or reflux is a concern, in the setting of hepatocellular ca

192 ough, LPR, or asthma due to gastroesophageal reflux is difficult, as no criterion standard test exits

193 The management of vesicoureteral reflux is evolving, with advocacy ranging from a less in

194 ated BAs became prominent components in bile reflux juice, whereas almost equal amounts of conjugated

195 management of patients suspecting of having reflux, leading to rational selection of treatment and b

196 medications, anterior chamber tap, vitreous reflux, longer intervals between injections, and longer

197 ons of GERD include cough, laryngopharyngeal reflux (LPR), and asthma.

198 tations of reflux, specifically cough due to reflux, LPR, and asthma due to reflux.

199 c sphincter augmentation (MSA) with the LINX Reflux Management System (Torax Medical, Shoreview, MN),

200 In summary, chaperone-mediated ER protein reflux may be a conserved protein quality control proces

201 Acid reflux may contribute to the progression from Barrett's

202 (ERAD) factors are crippled, suggesting that reflux may work in parallel with ERAD.

203 in 30min compared to 16h by the conventional reflux method (P<0.05).

204 almost two times more than that of the heat-reflux method.

205 Prolonged biliary reflux might be the most important risk factor of gastri

206 Participants underwent prolonged wireless reflux monitoring (off PPIs for >=7 days) and a 3-week P

207 Upfront reflux monitoring off acid suppression can limit unneces

208 ence of severe esophagitis, acid exposure on reflux monitoring predicted the ability to discontinue P

209 e the clinical utility of prolonged wireless reflux monitoring to predict the ability to discontinue

210 ation half-life of 2,5-dimethyl-tetrazole in refluxing o-xylene from 300,000 years to 1 week.

211 is, chronic rhinosinusitis, gastroesophageal reflux, obstructive sleep apnoea, vocal cord dysfunction

212 Bolus-induced deglutitive and post-prandial reflux occurred in most patients.

213 We observed ventricular reflux of Gd-DOTA in SHR rats only, indicating abnormal

214 The reflux of noxious contents of the stomach may cause oeso

215 A cold finger was examined for refluxing of acid vapors to determine its impact on effi

216 es and the percentage of proximal esophageal reflux off-PPI did not change significantly after one ye

217 nd 184 of 1354 men [13.6%] had recurrence of reflux), older age (HR, 1.41 [95% CI, 1.10-1.81] for age

218 roved symptoms of watering and minimal or no reflux on nasolacrimal syringing.

219 y improved with a combination of patency and reflux on syringing.

220 patients were found to have gastroesaphageal reflux only and 4 patients were found to have esophageal

221 om 360 (48%) had dysphagia and 390 (52%) had reflux or other symptoms.

222 ents with IPF, in the absence of oesophageal reflux or symptoms.

223 stationary manometry (n=143) to characterize reflux patterns.

224 xture to a 500 W IR source for 5 min without refluxing, permitted accurate determination of all analy

225 comorbidities, including gastro-oesophageal reflux, pulmonary hypertension, coronary artery disease,

226 ion columns in batch distillation with fixed reflux rate are useful to obtain distillates or distilla

227 flux surgery was not associated with risk of reflux recurrence (HR, 1.09 [95% CI, 0.77-1.53] for hosp

228 and interviews, have reported high rates of reflux recurrence after antireflux surgery, which may ha

229 Reflux recurrence after laparoscopic antireflux surgery

230 To determine the risk of reflux recurrence after laparoscopic antireflux surgery

231 Risk factors for reflux recurrence included female sex (hazard ratio [HR]

232 gression was used to assess risk factors for reflux recurrence.

233 edian of 5.6 years, 470 patients (17.7%) had reflux recurrence; 393 (83.6%) received long-term antire

234 include potassium-competitive acid blockers, reflux-reducing agents, bile acid binders, injection of

235 on controlling gastroesophageal reflux with reflux-reducing medication (e.g., baclofen) or antireflu

236 tic workup revealed truly PPI-refractory and reflux-related heartburn in a minority of patients.

237 If patients were found to have reflux-related heartburn, we randomly assigned them to r

238 17.7% experienced recurrent gastroesophageal reflux requiring long-term medication use or secondary a

239 , and the risk of worsening gastroesophageal reflux requiring revision may be higher than previously

240 re hypotonia, pathological gastro-esophageal reflux, retinal disease, and sinus-node dysfunction, whe

241 d in terms of yield and purity: conventional reflux, sealed vessel heated in an oil bath, and microwa

242 eatment of extraesophageal manifestations of reflux, specifically cough due to reflux, LPR, and asthm

243 edicted FEV1, self-reported gastroesophageal reflux, St. George's Respiratory Questionnaire score, sm

244 20 with GERD (symptoms plus esophagitis or + reflux study) were analyzed.

245 ntervention for Children With Vesicoureteral Reflux Study, a 2-year randomized clinical trial that en

246 dysmorphism, strabismus, and vesicoureteric reflux, suggesting that EBF3 has a widespread developmen

247 ADVICE 7: Based on available evidence, anti-reflux surgery and endoscopic GERD treatment modalities

248 ventral incisional hernia repair, colectomy, reflux surgery, bariatric surgery, and hysterectomy).

249 Symptom burden was quantified using the Reflux Symptom Questionnaire electronic Diary (RESQ-eD).

250 sure in the distal esophagus with absence of reflux-symptom association (ie, negative symptom index a

251 monstrates physiologic acid exposure without reflux-symptom association (ie, negative symptom index a

252 but was also present in those referred with reflux symptoms (64 [19%] of 329 patients vs 32 [10%] pa

253 as considered diagnostic of gastroesophageal reflux symptoms (GERD).

254 azole alone in providing complete cupping of reflux symptoms and healing of esophagitis in patients w

255 ents in group 2 demonstrated full cupping of reflux symptoms at 8 weeks.

256 with asthma, dyspepsia symptoms by 44%, and reflux symptoms by 26%.

257 le in management of patients with persistent reflux symptoms despite adequate medical or surgical tre

258 n terms of postoperative outcomes, including reflux symptoms, anastomotic leakage and stricture, and

259 Data regarding the presence of reflux symptoms, dysphagia, general health, PPI use, and

260 Among patients with typical reflux symptoms, inadequate PPI response, and absence of

261 gue scores and yes/no questions to determine reflux symptoms, side-effects and satisfaction with surg

262 g 98 (44%) with dysphagia and 123 (56%) with reflux symptoms.

263 astroesophageal reflux (GERD), despite minor reflux symptoms.

264 up to 50% of patients with gastroesophageal reflux symptoms.

265 H2O2 was performed in a digester block with reflux system and heated at 200 degrees C for 150min.

266 digestion in digester block with cold finger reflux system, which ensured that the elements were not

267 In patients with gastro-oesophageal reflux, the offer of Cytosponge-TFF3 testing results in

268 Reflux therefore develops via alterations in the balance

269 dimerization of 2-arylacetyl-1-naphthols in refluxing THF under open-flask conditions.

270 mediate progression of chronic acid biliary reflux to Barrett's esophagus and cancer.

271 We found reflux to be strongly associated with SG and identified

272 lpyridines in high to excellent yields under refluxing toluene conditions.

273 le) and arylaldehydes (dual electrophile) in refluxing toluene for 3 h.

274 olyl) aurones and flavones under warming and refluxing toluene reaction conditions via the formation

275 The use of Rh2(Piv)4 in refluxing toluene results in the formation of 1,2-dihydr

276 tal catalyst; instead the addition occurs in refluxing toluene without additives.

277 ripyrrane, followed by oxidation with DDQ in refluxing toluene, gave carbaporphyrin or carbachlorin p

278 nes (2 equiv) and cyclic amines (1 equiv) in refluxing toluene.

279 on of beta-ketosulfones and arylaldehydes in refluxing toluene; (ii) Grignard reagent (R'MgBr) or red

280 ntervention for Children With Vesicoureteral Reflux trial and the Careful Urinary Tract Infection Eva

281 Patients with saphenous vein reflux undergoing treatment with endothermal ablation (w

282 er standard management of gastro-oesophageal reflux (usual care group), in which participants only re

283 te prevalence of GD-CNVs; and vesicoureteral reflux (VUR) had the fewest GD-CNVs but was enriched for

284 Vesicoureteric reflux (VUR) is the commonest urological anomaly in chil

285 acute pyelonephritis (APN) or vesicoureteral reflux (VUR) using the data of 288 patients.

286 had combined reflux [acidic + weakly acidic reflux (WAR)].

287 ERD after sleeve (POGAS) was 19% and de novo reflux was 23%.

288 The rate of gastroesophageal reflux was comparable with prior studies of both POEM an

289 All underwent gastroscopy and bile reflux was grouped as normal, yellowish bile lakes and p

290 Gastroesophageal reflux was more common among patients who underwent POEM

291 Objective evidence of reflux was present in 40% for all patients and 33% for p

292 Chronic sinusitis and gastroesophageal reflux were also associated with exacerbation frequency

293 eased by the belt, but those associated with reflux were increased (2 vs 3.5; P = .04).

294 roup B) had diagnosis of GERD: 23 had acidic reflux, whereas 9 had combined reflux [acidic + weakly a

295 S: Central obesity promotes gastroesophageal reflux, which may be related to increased intra-abdomina

296 demonstrate that preventing gastroesophageal reflux with antireflux surgery halts the progression of

297 cy and focus on controlling gastroesophageal reflux with reflux-reducing medication (e.g., baclofen)

298 When refluxing with sodium hydrogen carbonate in acetonitrile

299 atients on medication for gastro-oesophageal reflux would increase the detection of Barrett's oesopha

300 s performed with a 0.05 M ketone solution in refluxing xylene in the presence of 10 equiv of potassiu