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1 s associated with a risk of gastroesophageal reflux disease.
2 in pediatric patients with gastroesophageal reflux disease.
3 r the surgical treatment of gastroesophageal reflux disease.
4 formed surgical therapy for gastroesophageal reflux disease.
5 vel potential treatment for gastroesophageal reflux disease.
6 ently overlap with those of gastroesophageal reflux disease.
7 cribed for the treatment of gastroesophageal reflux disease.
8 ormation, 11 (41%) had objective evidence of reflux disease.
9 the presence of concomitant gastroesophageal reflux disease.
10 vely distinguishing it from gastroesophageal reflux disease.
11 an effective treatment for gastroesophageal reflux disease.
12 ognized disease that mimics gastroesophageal reflux disease.
13 rritable bowel syndrome and gastroesophageal reflux disease.
14 squamous mucosa damaged by gastroesophageal reflux disease.
15 developing complications of gastroesophageal reflux disease.
16 ease commonly confused with gastroesophageal reflux disease.
17 disorders and treatment of gastroesophageal reflux disease.
18 between BMI and symptoms of gastroesophageal reflux disease.
19 iologic system is perturbed in subjects with reflux disease.
20 ccurrence and management of gastroesophageal reflux disease.
21 s are at increased risk for gastroesophageal reflux disease.
22 dicative of severe types of gastroesophageal reflux disease.
23 ts with symptomatic GERD do not have erosive reflux disease.
24 and a reliable indicator of gastroesophageal reflux disease.
25 e research and treatment of gastroesophageal reflux disease.
26 l ulcer, gastric ulcer, and gastroesophageal reflux disease.
27 esophagus or other types of gastroesophageal reflux disease.
28 commonly associated with gastro-oesophageal reflux disease.
29 evant to the association between obesity and reflux disease.
30 to those of patients with gastro-oesophageal reflux disease.
31 heartburn in patients with gastro-esophageal reflux disease.
32 ure by waist belt on reflux in patients with reflux disease.
33 o clear and buffer the refluxate, leading to reflux disease.
34 lications or recurrence of gastro-esophageal reflux disease.
35 long-lasting treatment for gastroesophageal reflux disease.
41 pnea may be associated with gastroesophageal reflux disease, a strong risk factor for Barrett's esoph
42 , chronic rhinitis, asthma, gastroesophageal reflux disease, adenotonsillitis, sleep apnea, anxiety,
43 factors evaluated included gastroesophageal reflux disease, alcohol consumption, smoking, chronic op
44 a complication of chronic gastro-oesophageal reflux disease, although asymptomatic individuals might
45 ased abdominal pressure and gastroesophageal reflux disease, although this pathogenic mechanism has n
46 and duration of symptoms of gastroesophageal reflux disease among randomly selected participants in t
48 quality of life related to gastroesophageal reflux disease and a 50% or greater reduction in the use
49 of peptic ulcer disease and gastrosophageal reflux disease and acts by irreversibly blocking ATP4A,
50 ecreased prevalence of both gastroesophageal reflux disease and adenocarcinoma of the esophagus and c
52 nd a higher prevalence of gastro-oesophageal reflux disease and blistering/desquamating skin disorder
53 niques in the management of gastroesophageal reflux disease and constipation also may have an impact
55 ted with conditions such as gastroesophageal reflux disease and diabetes mellitus, as well as emergen
56 ties is similar except for gastro-esophageal reflux disease and dyslipidemia that appear to be more s
57 both procedures except for gastro-esophageal reflux disease and dyslipidemia, which were more success
59 evolving definition and its relationship to reflux disease and functional gastrointestinal disorders
60 osis, including the role of gastroesophageal reflux disease and proton pump inhibitor-responsive esop
61 e to design new surgical strategies to treat reflux disease and reduce complications of fundoplicatio
62 The potential roles of undiagnosed venous reflux disease and the military physical training enviro
63 Approximately 20% have gastro-oesophageal reflux disease and this can be effectively treated with
64 n with a medical history of gastroesophageal reflux disease and type II diabetes presented to the hos
67 ma, mechanical ventilation, gastroesophageal reflux disease, and aspiration or other types of pneumon
68 ological manifestation of gastro-oesophageal reflux disease, and is a major risk factor for the devel
71 isk factor for oesophageal adenocarcinoma is reflux disease, and the rising incidence of this coincid
72 is usually due to asthma, gastro-oesophageal reflux disease, and upper airway conditions, and that it
77 f gastric disorders such as gastroesophageal reflux disease, autoimmune gastritis, gastric cancer, an
78 have been identified-mainly gastroesophageal reflux disease, Barrett's esophagus, obesity, and tobacc
79 assessed 100 patients with gastroesophageal reflux disease before and after sphincter augmentation.
80 e and effective therapy for gastroesophageal reflux disease, but its effect on the LES has not been e
81 s frequently mimic those of gastroesophageal reflux disease, but the diseases are distinct in their h
82 of a confirmed diagnosis of gastroesophageal reflux disease by an abnormal esophageal pH study (body
83 ar-old men with symptoms of gastroesophageal reflux disease by Cytosponge is cost effective and would
84 that is distinguished from gastroesophageal reflux disease by the expression of a unique esophageal
91 se of these technologies in gastroesophageal reflux disease continues to accelerate, and the last 2 y
92 nterval [CI]: 1.04-2.67) or gastroesophageal reflux disease controls (OR = 1.61; 95% CI: 1.33-1.96).
93 oesophageal reflux disease (gastroesophageal reflux disease controls, n = 1332), and population-based
94 chest pain associated with gastroesophageal reflux disease, correlates abnormal ambulatory pH monito
95 , regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, laryngitis, GERD, GOR
96 , regurgitation, dysphagia, gastroesophageal reflux disease, cough, aspiration, laryngitis, GERD, GOR
97 ssive surgical treatment of gastroesophageal reflux disease decreases the rate of bronchiolitis and i
98 rugs (lipid-lowering drugs, gastroesophageal reflux disease drugs, antihypertensive drugs, sleep aids
99 sses (lipid-lowering drugs, gastroesophageal reflux disease drugs, diabetes drugs, antihypertensive d
100 as functional dyspepsia and gastroesophageal reflux disease (e.g. vomiting, disordered lower esophage
101 n the last 2 years for many gastroesophageal reflux disease endotherapies, providing some insight int
103 onse rates compared to patients with erosive reflux disease (ERD); pH metry contributes to GERD diagn
104 l complications of obesity: gastroesophageal reflux disease, erosive esophagitis, Barrett's esophagus
107 ides a clinical overview of gastroesophageal reflux disease, focusing on diagnosis, treatment, and pr
108 th those from subjects with gastroesophageal reflux disease (gastroesophageal reflux disease controls
109 e interval [CI], 2.9-12.9), gastroesophageal reflux disease (GERD) (RR, 1.9; 95% CI, 1.4-2.6), dyspep
110 thophysiological factor in gastro-esophageal reflux disease (GERD) and as a target for GERD treatment
112 incidence and predictors of gastroesophageal reflux disease (GERD) and dyspepsia and their associatio
113 hageal disorders, including gastroesophageal reflux disease (GERD) and eosinophilic esophagitis (EoE)
115 an alternative treatment of gastroesophageal reflux disease (GERD) and may provide durable reflux con
116 ential for the treatment of gastroesophageal reflux disease (GERD) and other esophagogastric diseases
117 the Montreal definition of gastroesophageal reflux disease (GERD) and the Rome III definition of fun
118 sophageal manifestations of gastroesophageal reflux disease (GERD) and to compare the most recent tec
119 recipients with documented gastroesophageal reflux disease (GERD) are at increased risk for graft dy
120 urrent diagnostic tests for gastroesophageal reflux disease (GERD) are suboptimal and do not accurate
121 ia who were thought to have gastroesophageal reflux disease (GERD) but who did not respond to medical
127 iding physicians diagnose gastro-oesophageal reflux disease (GERD) have not been evaluated in terms o
128 anges associated with acute gastroesophageal reflux disease (GERD) have not been studied prospectivel
131 as to compare recurrence of gastroesophageal reflux disease (GERD) in children randomized to laparosc
132 ly used in the treatment of gastroesophageal reflux disease (GERD) in children; however, their effica
133 cation for the treatment of gastroesophageal reflux disease (GERD) in comparison with a sham procedur
136 se or eliminate features of gastroesophageal reflux disease (GERD) in some patients whose symptoms pe
145 It has been suggested that gastroesophageal reflux disease (GERD) is a risk factor for developing rh
152 he Montreal classification, gastroesophageal reflux disease (GERD) is much more than heartburn and pa
159 iteria included symptoms of gastroesophageal reflux disease (GERD) more than once a month, use of med
161 cid peptic disorder such as gastroesophageal reflux disease (GERD) nor should it preclude a diagnosis
162 y used for the treatment of gastroesophageal reflux disease (GERD) or completing Heller's myotomy and
164 Treatment-refractory gastro-oesophageal reflux disease (GERD) remains a significant problem in t
167 ed for use in patients with gastroesophageal reflux disease (GERD) symptoms despite proton pump inhib
168 ects a higher prevalence of gastroesophageal reflux disease (GERD) symptoms or a higher degree of eso
172 re matched to subjects with gastroesophageal reflux disease (GERD) without Barrett's esophagus and to
173 f objective measurements of gastroesophageal reflux disease (GERD) would improve management of patien
176 ease (CrD), celiac disease, gastroesophageal reflux disease (GERD), and eosinophilic esophagitis (EoE
177 table bowel syndrome (IBS), gastroesophageal reflux disease (GERD), and overactive bladder syndrome (
178 ysfunction syndrome (RUDS), gastroesophageal reflux disease (GERD), and rare cases of inflammatory pu
179 r three common indications: gastroesophageal reflux disease (GERD), Barrett's esophagus (BE), and non
180 proposed for patients with gastroesophageal reflux disease (GERD), but there is little evidence of i
181 pylori may protect against gastrointestinal reflux disease (GERD), but these results could be due to
182 appraise the prevalence of gastroesophageal reflux disease (GERD), esophagitis, and Barrett's esopha
184 ysiological states, such as gastroesophageal reflux disease (GERD), functional dyspepsia and, possibl
185 tion of body mass index and gastroesophageal reflux disease (GERD), including its complications (esop
186 sophagus, a complication of gastroesophageal reflux disease (GERD), predisposes patients to esophagea
187 previous fundoplication for gastroesophageal reflux disease (GERD), underwent reoperative surgery.
188 f frequency and duration of gastroesophageal reflux disease (GERD), using data from a randomly select
191 ave a similar prevalence of gastroesophageal reflux disease (GERD), yet esophageal adenocarcinoma (EA
192 rease of 50% or more in the Gastroesophageal Reflux Disease (GERD)-Health Related Quality of Life sco
215 o included 10 patients with gastroesophageal reflux disease (GERD; age, 32-60 y; 7 women) without tro
216 ere matched to persons with gastroesophageal reflux disease (GERD; n = 316) and to population control
217 apy in patients with proven gastroesophageal reflux disease [GERD]), to document physiologic levels o
218 ntified domains (dysphagia, gastroesophageal reflux disease [GERD], nausea/vomiting, and pain) align
222 lastic progression in the gastro-oesophageal reflux disease (GORD)-Barrett's metaplasia (BM)-oesophag
224 in pediatric patients with gastroesophageal reflux disease have shown good to excellent results; how
225 in patients with normal GE (Gastroesophageal Reflux Disease Health-Related Quality of Life score 18.2
226 mptom that can be caused by gastroesophageal reflux disease; however, treatment outcome has been diff
228 associated with symptoms of gastroesophageal reflux disease in both normal-weight and overweight wome
229 correlations of BE/EA with gastroesophageal reflux disease in male individuals and obesity in female
230 in meters - and symptoms of gastroesophageal reflux disease in persons of normal weight has not been
231 cidic environment caused by gastroesophageal reflux disease in the gastroesophageal junction and asso
232 ic approach to patients with extraesophageal reflux disease involved the use of insensitive tools, wh
239 the pathogenesis of heartburn in nonerosive reflux disease is a reaffirmation of the definition of r
241 Antireflux surgery for gastro-esophageal reflux disease is followed by varying rates of re-interv
242 hagus epithelium related to gastroesophageal reflux disease, is the strongest known risk factor for t
243 ough heartburn is the most common symptom of reflux disease, it is unclear whether the severity of he
244 on "Endoscopic Therapy for Gastroesophageal Reflux Disease." It was approved by the Clinical Practic
246 s (asthma, sinusitis, and gastro-oesophageal reflux disease), mental health disorders (depression, po
247 for an esophageal etiology-gastroesophageal reflux disease, motility abnormalities, or esophageal hy
248 rcinogen treatment leads to gastroesophageal reflux disease, multilayered epithelium (MLE, a presumed
249 lication for PPI-refractory gastroesophageal reflux disease (n = 14 270 degrees LPF vs. n = 28 360 de
250 ere matched to persons with gastroesophageal reflux disease (n = 308) without Barrett's esophagus and
251 indeterminate EoE (n = 15), gastroesophageal reflux disease (n = 7), or normal esophagus (n = 21).
253 t symptom burden in patients with nonerosive reflux disease (NERD) compared with patients with erosiv
256 h improved understanding of gastroesophageal reflux disease, newer developments in diagnostic techniq
257 , including rhinosinusitis, gastroesophageal reflux disease, obesity and dysfunctional breathing.
258 cter (LES) in patients with gastroesophageal reflux disease often has a low resting pressure and a sh
259 orically distinguished from gastroesophageal reflux disease on the basis of histology and lack of res
261 indeterminate EoE) but not gastroesophageal reflux disease or normal esophagus and was correlated to
263 ry of peptic ulcer disease, gastroesophageal reflux disease, or gastrointestinal bleeding, and prior
264 This article is a review of gastroesophageal reflux disease, other types of esophagitis, benign and m
266 We randomly assigned 64 gastroesophageal reflux disease patients to radiofrequency energy deliver
267 on for selected symptomatic gastroesophageal reflux disease patients who are intolerant of, or desire
268 Too much acid can lead to gastroesophageal reflux disease, peptic ulcer disease, and stress-related
269 eosinophilic bronchitis, gastro-oesophageal reflux disease, postnasal drip syndrome or rhinosinusiti
270 a >50% improvement in their gastroesophageal reflux disease quality of life score (n = 19 [61%] vs. n
271 of GERD was by means of the gastroesophageal reflux disease questionnaire (GERDQ) while the diagnosis
272 objective is to evaluate the ability of the Reflux Disease Questionnaire (RDQ) to identify GERD acco
273 ary outcome was symptom control evaluated by Reflux Disease Questionnaire and Reflux Symptom Index.
275 Endoscopic therapies for gastroesophageal reflux disease represent a minimally invasive alternativ
276 vely high rate of recurrent gastroesophageal reflux disease requiring treatment, diminishing some of
277 f non-drug treatments for gastro-oesophageal reflux disease, safety of long-term drug treatment, and
278 igated whether patients with supraesophageal reflux disease (SERD) have impaired UES and esophageal b
279 ence of anxiety, headaches, gastroesophageal reflux disease, sleep apnea, and infections of the respi
280 fied version of a validated gastroesophageal reflux disease-specific QOL tool to patients before and
281 ty of life, measured by the gastroesophageal reflux disease-specific QOL tool, and recurrence, define
283 proved in group A, with the Gastroesophageal Reflux Disease Symptom Assessment Scale score decreasing
284 very significantly improved gastroesophageal reflux disease symptoms and quality of life compared wit
285 ue in patients with chronic gastroesophageal reflux disease symptoms is of unproven value, and recomm
286 y for patients with chronic gastroesophageal reflux disease symptoms to assess for Barrett's esophagu
287 d Kingdom with histories of gastroesophageal reflux disease symptoms, assuming the prevalence of Barr
289 pertension, hyperlipidemia, gastroesophageal reflux disease, thyroid disease, diabetes, osteoporosis)
290 tly cleared new endoluminal gastroesophageal reflux disease treatments; however, no controlled trials
294 r patients with symptomatic gastroesophageal reflux disease were older (58 for men and 64 for women)
295 tis, allergic rhinitis, and gastroesophageal reflux disease were only associated with increased exace
296 ts a potential biomarker for extraesophageal reflux disease when detected in airways, however a direc
300 (BEAC) is a complication of gastroesophageal reflux disease, with no effective chemotherapy and poor