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1 ed the precursors of M2 macrophages, DCs and regulatory T cells.
2 tates, including multiple distinct states of regulatory T cells.
3 rtant impacts on the suppressive activity of regulatory T cells.
4 such as myeloid-derived suppressor cells and regulatory T cells.
5 ages, tumor-infiltrating dendritic cells and regulatory T cells.
6 c T-cell engager activated both effector and regulatory T cells.
7 s II-dependent fashion and are suppressed by regulatory T cells.
8 n of gut T cells and an accumulation of skin regulatory T cells.
9 ing less targeted by FOXP3(+) CD4(+) CD25(+) regulatory T cells.
10 ls and loss of intestinal forkhead box p3(+) regulatory T cells.
11 and suppression, which is mediated mainly by regulatory T cells.
12 nt and expansion of ChgA-specific peripheral regulatory T cells.
13  cells and autoimmune-protective human blood regulatory T cells.
14 he induction of therapeutic antigen-specific regulatory T cells.
15 ing of the immune response and inhibition of regulatory T cells.
16 cose metabolism because of maintenance of AT regulatory T cells.
17 ter differentiation of T helper 17 cells and regulatory T cells.
18 t was not sufficient to restore the level of regulatory T cells.
19  engraftment, including an increase in human regulatory T cells.
20  CCL2 induced formation of highly functional regulatory T cells.
21  a transcription factor which is a marker of regulatory T cells.
22  immunity, countering negative regulation by regulatory T cells.
23 ation of atheroprotective immunity involving regulatory T cells.
24 licular helper, T follicular regulatory, and regulatory T cells.
25  cytotoxic T cells and more CD25(+)/FOXP3(+) regulatory T cells.
26 ls (MDSCs) and further decreased circulating regulatory T cells.
27 sitization and anaphylaxis, while sustaining regulatory T cells.
28  necessary for the development of T(H)17 and regulatory T cells.
29 , and a decreased presence of CD4(+)Foxp3(+) regulatory T cells.
30 e deviated toward naive cells with increased regulatory T cells.
31 id cells, which then cross-regulated Th2 and regulatory T cells.
32  and CD8(+) effector T cells and in Foxp3(+) regulatory T cells.
33 , upregulated (median [interquartile range]) regulatory T cells (5.3% [3.1%-18.2%]; P < .05), reduced
34 ns suggest that immune modulators that block regulatory T cell activity may increase responses to vir
35 te counter-regulation by T-helper 1 cell and regulatory T cell activity.
36 oportions of intestinal Foxp3(+)RORgammat(+) regulatory T cells and BALB/c mice developed increased R
37 bers correlated with increased occurrence of regulatory T cells and effector T cells and decreased na
38  immune response by inducing infiltration of regulatory T cells and expression of immune-suppressive
39  that depletes CD25-positive cells including regulatory T cells and has been approved for the treatme
40 n; numerous forkhead box protein P3 (FoxP3)+ regulatory T cells and IgG4 plasma cells; and abundant a
41                                The levels of regulatory T cells and IL-10 were elevated at the end of
42 ngside PNA-specific forkhead box P3-positive regulatory T cells and IL-10(+) and TGF-beta1(+) skin-re
43 evels, but the latter preferentially induced regulatory T cells and in general downregulated T cell c
44 expression of CD3(+)/CD4(+)/CD25(+)/FoxP3(+) regulatory T cells and increased the Teff/Treg ratio.
45 cleotidases on various immune cells, such as regulatory T cells and macrophages, as well as component
46 hich may account for the expansion of CD4(+) regulatory T cells and memory-type CD8(+) T cells.
47 possess immunomodulatory potential, in which regulatory T cells and monocytes play a key role.
48 ltration of immune-inhibitory cells, such as regulatory T cells and myeloid-derived suppressor cells,
49 tumour immune microenvironment by decreasing regulatory T cells and myeloid-derived suppressor cells.
50  The TME already shows a reduction in type 1 regulatory T cells and PD-L1+ tumor-associated macrophag
51 scribed correlations between the presence of regulatory T cells and recovery from Lyme arthritis.
52                            A small number of regulatory T cells and T helper 1 cells is also identifi
53                                     However, regulatory T cells and T(H)17 cells were found in higher
54                        It acts by attracting regulatory T cells and Th2 cells via their receptor CCR
55 oneal (i.p.) administration of MDP triggered regulatory T cells and the accumulation of a population
56 ited the induction of antigen-specific FoxP3 regulatory T cells and the prevention of food allergy by
57 nction of pro-repair immune cells, including regulatory T cells and tissue macrophages.
58 e role of mTOR in follicular helper T cells, regulatory T cells, and other T cell subsets.
59 otential by regulating macrophage functions, regulatory T cells, and secretion of proresolving mediat
60 ut neither IL-12Rbeta2 nor IL-23R, protected regulatory T cells, and suppressed Th1 and Th17 biasness
61 r function of IL-2 is to maintain functional regulatory T cells, and thus its essential function is i
62 Th2 cells, group 2 innate lymphoid cells and regulatory T cells, and to a lesser extent, on NK cells
63 tment of low HLA-DR expressing monocytes and regulatory T-cells; and transcription of immune checkpoi
64 (regs) treatment and its bystander effect on regulatory T cells are associated with CNS repair as ref
65          These data suggest that pDC-induced regulatory T cells are dependent on downstream signaling
66 ell-DC interaction preferences, and discover regulatory T cells as a major T cell subtype interacting
67 responsive RORgammat-positive FOXp3-positive regulatory T cells as critical for the maintenance of to
68 sion block that can be released by impairing regulatory T cell associated signaling pathways.
69 nce that an elevated proportion of activated regulatory T cells at birth was specific to the sensitiz
70 ed include decreased suppressive activity of regulatory T cells at steady state, which correlates wit
71                                 Depletion of regulatory T cells at various times after infection also
72                                      Adipose regulatory T cells (aTregs) have emerged as critical cel
73 latively divergent and CD8+ T cells and CD4+ regulatory T cells being relatively similar.
74 the transcriptional adaptation trajectory of regulatory T cells between lymphoid tissue and colon.
75 as independent of antioxidant mechanisms and regulatory T cells but dependent on residential alveolar
76 TLA-4(+)) and treatment-induced depletion of regulatory T-cells (CD4(+) Foxp3(+)/CTLA-4(+)) was seen
77 eanut-specific clones in the effector versus regulatory T-cell compartment, which distinguished the c
78 l receptor signaling and dysregulated T-cell/regulatory T-cell compartment.
79 trol of alloreactivity and would represent a regulatory T-cell-compatible immunosuppression.
80 pulations of tumor-associated macrophage and regulatory T cell declined.
81 atients received one of six CBMPs containing regulatory T cells, dendritic cells, or macrophages; pat
82 in II-infused mice, suggesting the effect is regulatory T cell-dependent.
83 tive effects of propionate were abrogated in regulatory T cell-depleted angiotensin II-infused mice,
84  vitro stimulation of splenocytes from these regulatory T cell-depleted mice resulted in increases in
85  onset of autoimmune T cell activation after regulatory T cell depletion in an established model of s
86                                              Regulatory T cell depletion using PC61 antibody was used
87  and proliferative states and no evidence of regulatory T cell depletion.
88                    Total T-cell or selective regulatory T-cell depletion abrogates the atheroprotecti
89 TGF-beta activation blocks immunosuppressive regulatory T cell differentiation, which is a potential
90 aralogs during Langerhans cell and inducible regulatory T cell differentiation.
91 -mannose inhibit tumour growth and stimulate regulatory T cell differentiation.
92 n of a transcriptional program that promotes regulatory T-cell differentiation.
93 ell-extrinsic factors, such as nutrients and regulatory T cells, directly and indirectly balance quie
94 nopportune induction of peripherally induced regulatory T cells during immune responses against forei
95 ts who failed withdrawal without evidence of regulatory T cell dysfunction.
96                                 Depletion of regulatory T cells enhanced response.
97 tively, these findings provide evidence that regulatory T cells existing at the time of, and possibly
98 spring exposed to UFPs, suggesting increased regulatory T cell expression and suppressed Th2/Th17 res
99 y two populations of tTreg, one in which the regulatory T cell fate is associated with unique propert
100 ating T cells mainly displayed exhausted and regulatory T-cell features.
101 ls (p = 0.006), whereas lower proportions of regulatory T cell fractions (p = 0.009), which induce an
102 ividuals, plasma TL1A levels correlated with regulatory T cell frequencies, whereas soluble DR3 was s
103 ther reduced in ALR-H-KO mice; HF/HC reduced regulatory T-cell frequency only in WT mice.
104 e (CD25(+) ) forkhead box P3-positive CD4(+) regulatory T-cell frequency was lower in ALR-H-HET than
105 doleamine-2,3-dioxygenase-1 enzyme activity, regulatory T-cell frequency, activated CD38+Human Leukoc
106 ts of AIT are attributed mainly to increased regulatory T-cell function and increased allergen-specif
107 le of regulatory lymphocyte subsets, such as regulatory T cells, gammadelta regulatory T cells, NKT10
108 ubset strictly equivalent to IL-10-secreting regulatory T cells has only recently been proposed.
109 hymal stromal cells and, to a lesser extent, regulatory T cells have demonstrated efficacy in humans.
110 tion, and the induction of allergen-specific regulatory T cells, highlighting its potential use in th
111 rom the lymphoid compartment promotes a Th17/regulatory T cell imbalance and exacerbates the developm
112  aim was to investigate the role of CCR4 and regulatory T cells in cutaneous wound healing in diabeti
113 ector lymphocytes and a higher activation of regulatory T cells in grafts with subTCMR compared to cT
114 -3 plays an important role in development of regulatory T cells in mice.
115 ease in autoreactive T cells and decrease in regulatory T cells in peripheral tissues.
116 ) PD1(+) CD73(+) ICOS(+) IL-10(+) peripheral regulatory T cells in prediabetic mice, and liposome adm
117 he serum and an increase in the frequency of regulatory T cells in the blood and spleen.
118  growth, increased the ratio of cytotoxic to regulatory T cells in tumors, and prolonged survival.
119  (all CD11c-expressing cells) expands aortic regulatory T cells in vivo, limits the accumulation of T
120 y led to a reduction in AT immunosuppressive regulatory T cells, increased AT CD8(+) T cells, and no
121  Early-relapse tumors have reduced levels of regulatory T cells, increased dendritic cells (DCs), and
122  of IL-10(+) and/or TGF-beta(+)CD4(+)CD25(+) regulatory T cells (induced Treg cells).
123       This was associated with promoting the regulatory T cell-inducing molecule retinoic acid, inhib
124  NFkappaB inhibitors were used to assess the regulatory T-cell induction pathways.
125        Manipulating autoimmunity by inducing regulatory T cells is potentially a more specific and sa
126 king antibody studies suggested that loss of regulatory T cells is the most important cause of aggrav
127 as well as an increase in the frequency of a regulatory T cell, known as Tr1, by week 12 of follow-up
128  were reduced natural killer cells, elevated regulatory T cells, M2-type macrophages, and high PD-L1
129                            Expression of the regulatory T cell markers FOXP3 and CD25, together with
130                  Assessment of cells bearing regulatory T cell markers from these mice revealed defec
131 -specific immunoglobulins, T-cell cytokines, regulatory T cells, mast cells, and basophils were quant
132 kin was characterized by the accumulation of regulatory T cells, mast cells, M2 macrophages, and mark
133               Moreover, in vitro blockade of regulatory T cell mechanisms of action with biologic mod
134 expression by cDC is not required for CD4(+) regulatory T cell-mediated control of colitis.
135              Here, we demonstrate a role for regulatory T cell-mediated restraint of T(VM) at least i
136 e T-cell responses to interleukin (IL)-2 and regulatory T cell-mediated suppression, the composition
137          Here, we used C57BL/6 "depletion of regulatory T cell" mice to assess the effects these cell
138                 However, in mice depleted of regulatory T cells, MPO-Sp administration did not protec
139  Our data suggest that the receptor CCR4 and regulatory T cells negatively affect wound healing in di
140 sets, such as regulatory T cells, gammadelta regulatory T cells, NKT10 cells, and perifollicular mast
141                        Furthermore, reducing regulatory T cells or eliminating B and T cells in Rag1
142 tudy consortium after injection of recipient regulatory T cells, or injection of donor regulatory mon
143 sLeX(+)CD4(+)T cells exclusively contain the regulatory T cell population (CD127(low)CD25(high) and F
144 he severity of psoriasis linked to a reduced regulatory T cell population and increased IL-17A expres
145                                   Cord blood regulatory T-cell populations were measured at birth.
146 sion was identified within effector, but not regulatory T-cell populations.
147 g in the balance between proinflammatory and regulatory T cells potentially favoring a proinflammator
148 e LAMP3 + DC subgroup may be able to recruit regulatory T cells, potentially taking part in the forma
149 s (Th17s) predict improved survival, whereas regulatory T cells predict poorer survival.
150                                              Regulatory T cells, previously shown to associate with p
151                  We showed that depletion of regulatory T cells prior to infection with B. burgdorfer
152                           Peripheral induced regulatory T cells (pTreg cells) play a central role in
153 anistically, GM-CSF had no adverse effect on regulatory T-cell reconstitution, but linked adaptive to
154                                              Regulatory T-cell-related mediators (IL-10 and FOXP3) we
155 er central memory T cell response, and lower regulatory T cell response, compared with controls.
156                                  A sustained regulatory T-cell response, after IT cessation, occurs i
157 nked to the acquisition and maintenance of a regulatory T-cell response.
158                    Although the frequency of regulatory T cells seemed normal in the ELGAN/ELBW prete
159    Furthermore, SLIT enhanced proportions of regulatory T cells specific to RGP.
160            However, the expression of Foxp3, regulatory T cell-specific transcription factor, was enh
161 per cell 17, CD8+ effector, CD4+ memory, and regulatory T-cell-specific signatures.
162    Follicular regulatory T (Tfr) cells are a regulatory T cell subset that controls antibody producti
163 ies of a majority of CD4(+) conventional and regulatory T cell subsets; in general, Ag-naive subsets
164 gulatory over effector T cells, expansion of regulatory T-cell subsets expressing CXCR3 or retinoic a
165 with the presence of functional effector and regulatory T-cell subsets with diverse T-cell receptor c
166 L-17 is tumorigenic in many cancer types and regulatory T cells suppress antitumor T cells.
167 tor expression patterns that interfered with regulatory T-cell suppression assays and were associated
168 ction of both conventional CD4(+) T cell and regulatory T cell (T(reg)) compartments.
169 fic CD4(+) T cells with an atheroprotective, regulatory T cell (T(reg)) phenotype in healthy individu
170  T cell (T(eff)) functions(14,15) and hamper regulatory T cell (T(reg)) suppression(16-20).
171 ng distinct T cell compartments, and several regulatory T cell (T(reg))-biasing anti-IL-2 antibodies
172                          Adipose tissue (AT) regulatory T cells (T regs) control inflammation and met
173 42 directly targets Tgfbr1 for repression in regulatory T cells (T(REG) ).
174 programs that generate a broad repertoire of regulatory T cells (T(reg) cells) able to respond to bot
175                                     Foxp3(+) regulatory T cells (T(reg) cells) are crucial for the ma
176                                              Regulatory T cells (T(reg) cells) can activate multiple
177                                              Regulatory T cells (T(reg) cells) play a pivotal role in
178                                              Regulatory T cells (T(reg) cells) restrict immune system
179                                    Depleting regulatory T cells (T(reg) cells) to counteract immunosu
180                                              Regulatory T cells (T(reg) cells), a distinct subset of
181                                              Regulatory T cells (T(reg) cells), whose differentiation
182 ne receptor 4 (CCR4) is broadly expressed on regulatory T cells (T(reg)) as well as other circulating
183   Recruitment of suppressive CD4(+) FOXP3(+) regulatory T cells (T(reg)) to the tumor microenvironmen
184 nd reduced proportion of SARS-CoV-2-reactive regulatory T cells (T(REG)).
185 lated to improved prognosis is a decrease in regulatory T cells (T(reg)).
186                               CD4(+)Foxp3(+) regulatory T cells (T(regs)) are a key cell population c
187                                              Regulatory T cells (T(regs)) maintain peripheral self-to
188 neonatal Tgammadelta17 cells are innate skin regulatory T cells that are critical for skin homeostasi
189 ast in part by generation of IL-10-secreting regulatory T cells that mediate bystander immunosuppress
190 ably the retinoic orphan receptor gamma T(+) regulatory T cells, the epithelial barrier, and healthy
191 is, where it controls immunity by recruiting regulatory T cells to dendritic cells (DCs).
192 romotes the differentiation of ROR-gammat(+) regulatory T cells to suppress FA.
193       Nrp1 also helps migrate thymus-derived regulatory T cells to vascular endothelial growth factor
194 ) regulatory T cells (Treg cells) and type 1 regulatory T cells (Tr1 cells), concomitant with a reduc
195 that results from mutations in the canonical regulatory T cell transcription factor FOXP3.
196 eing evaluated for T1D is the restoration of regulatory T cell (Treg) activity, specifically directed
197 eta-specific signaling in TGF-beta-dependent regulatory T cell (Treg) differentiation.
198             IL-2R signaling is essential for regulatory T cell (Treg) function.
199 ription factor Foxp3 specifies and maintains regulatory T cell (Treg) identity.
200  interleukin (IL)-6 and IL-2, but peripheral regulatory T cell (Treg) numbers did not increase.
201 he CD4(pos) CD25(high) CD127(neg) FOXP3(pos) regulatory T cell (Treg) pool.
202 cross-sectional study aimed to determine the Regulatory T cell (Treg) properties, together with 14 pl
203  well as a decrease in intratumoral MDSC and regulatory T cell (Treg).
204                       Increasing donor graft regulatory T cell (Treg):effector T cell (Teff) ratios c
205 he expansion of MOG(35-55)-specific FoxP3(+) regulatory T cells (Treg cells) and type 1 regulatory T
206                                              Regulatory T cells (Treg cells) are important for preven
207 required for the development and function of regulatory T cells (Treg cells).
208 ed with expansion of peripheral Foxp3CD4CD25 regulatory T cells (Treg) and increased forkhead box P3
209 athway reduced the intratumoral abundance of regulatory T cells (Treg) and the expression of the Treg
210                              CD4(+) Foxp3(+) regulatory T cells (Treg) are essential to maintain immu
211                              Whether and how regulatory T cells (Treg) are involved in this postsepti
212 d detect whether the helper T cells (Th) and regulatory T cells (Treg) could affect osteoclasts and f
213 ofiles, and differing capacities to increase regulatory T cells (Treg) during coculture assays, featu
214                                              Regulatory T cells (Treg) have proven to be a powerful i
215  the latter is due to selective depletion of regulatory T cells (Treg) in a tumor microenvironment.
216 nged with Ova in vitro, though the number of regulatory T cells (Treg) in the LN remained comparable.
217 ntained by the differentiation and action of regulatory T cells (Treg) specific for tissue Ags.
218 ts, with significantly reduced donor-derived regulatory T cells (Treg), increased cytotoxic effector
219 l transplantation, but they induce long-term regulatory T cells (Treg)-dependent graft acceptance onl
220 myeloid-derived suppressor cells (MDSC), and regulatory T cells (Treg).
221             In previous studies, deficits in regulatory T-cell (Treg) number and function at birth ha
222                             Antibody levels, regulatory T-cell (Treg) numbers, and cytokine levels we
223            Further, highly immunosuppressive regulatory T cells (Tregs) (ie, ICOShigh/PD-1-) and myel
224 une cells including activated CD4(+)Foxp3(+) regulatory T cells (Tregs) and CD4(+)Foxp3(-) convention
225 ated with expansion of CD4(+)CD25(+)FOXP3(+) regulatory T cells (Tregs) and enhanced transforming gro
226                  Surprisingly, the levels of regulatory T cells (Tregs) and interleukin-10 (IL-10) we
227 observed between relative counts of FOXP3(+) regulatory T cells (Tregs) and lung cancer risk, and sig
228 eatment also increases the percentage of the regulatory T cells (Tregs) and maintains immune toleranc
229 regulatory myeloid cells (Mregs) that expand regulatory T cells (Tregs) and suppress effector T cells
230                                              Regulatory T cells (Tregs) are a lymphocyte subset with
231                                   FOXP3+CD4+ regulatory T cells (Tregs) are critical for immune homeo
232                                              Regulatory T cells (Tregs) are crucial mediators of immu
233                                              Regulatory T cells (Tregs) are crucial mediators of immu
234                                              Regulatory T cells (Tregs) are essential for establishin
235                                              Regulatory T cells (Tregs) are non-redundant mediators o
236                                       Foxp3+ regulatory T cells (Tregs) are potent immunoregulatory c
237                                       Foxp3+ regulatory T cells (Tregs) are potent suppressor cells,
238       In this study, we report that Foxp3(+) regulatory T cells (Tregs) are the main target cells of
239 ivo and are able to increase the function of regulatory T cells (Tregs) at well-tolerated concentrati
240           mIL-2/CD25 acts not only to expand regulatory T cells (Tregs) but also to increase their ac
241  for HIV cure cannot be discarded.IMPORTANCE Regulatory T cells (Tregs) can decisively contribute to
242     To test if Ag-independent stimulation of regulatory T cells (Tregs) can prevent T1D onset, groups
243                                 Depletion of regulatory T cells (Tregs) delayed spontaneous pain reco
244                                        Human regulatory T cells (Tregs) have been implicated in cance
245                                              Regulatory T cells (Tregs) have been shown to be atherop
246                                     Although regulatory T cells (Tregs) have been studied for their r
247                                          CD8 regulatory T cells (Tregs) have enhanced apoptosis in ea
248                                              Regulatory T cells (Tregs) have unique immunosuppressive
249 , we found that the upregulation of Foxp3(+) regulatory T cells (Tregs) in the mesothelioma tumor mic
250 for uterine tropism, we explored the role of regulatory T cells (Tregs) in the pathogenesis of Brucel
251 shown to selectively expand CD4(+)Foxp3(+) T regulatory T cells (Tregs) in vivo.
252 be-induced population of RORgamma-expressing regulatory T cells (Tregs) is essential in controlling g
253    The equilibrium of T-helper 17 (Th17) and regulatory T cells (Tregs) is often found altered in pat
254 ciated toxicity and expansion of suppressive regulatory T cells (Tregs) limit its use in patients wit
255                                              Regulatory T cells (Tregs) may be key contributors to th
256 eous and systemic accumulation of Foxp3(+) T regulatory T cells (Tregs) partially at the expense of m
257                                              Regulatory T cells (Tregs) play a central role in the in
258                                              Regulatory T cells (Tregs) play essential roles in obesi
259                                              Regulatory T cells (Tregs) play important roles in limit
260 , functional imbalance of Th17 effectors and regulatory T cells (Tregs) promote inflammatory diseases
261 ntional T cells (Tcon) and immunosuppressive regulatory T cells (Tregs) remains elusive.
262 t cells, substantially increase the ratio of regulatory T cells (Tregs) to allo-activated Tconvs, and
263                            The potential for regulatory T cells (Tregs) to limit disease progression
264 mma(+) effector Th1 cells and CD4(+)Foxp3(+) regulatory T cells (Tregs) were evaluated by flow cytome
265 ition of B cell depletion to the transfer of regulatory T cells (Tregs) with indirect alloresponse fu
266                    Exhausted T and NK cells, regulatory T cells (Tregs), alternatively activated macr
267 genic potential, lower frequency of Foxp3(+) regulatory T cells (Tregs), and increased number of CD8(
268 +)) T cells, CD8(+) T cells, CD4(+) T cells, regulatory T cells (Tregs), gammadelta T cells, B cells,
269               For instance, FOXP3-expressing regulatory T cells (Tregs), have shown remarkable effect
270                                              Regulatory T cells (Tregs), in contrast, demonstrated an
271 articular effector/memory subsets, including regulatory T cells (Tregs), which also displayed hardwir
272                 However, the effect of SP on regulatory T cells (Tregs), which are functionally impai
273                       A unique population of regulatory T cells (Tregs), with a distinct transcriptio
274  the checkpoint inhibitory protein CTLA-4 in regulatory T cells (Tregs).
275 n neonates via induction of antigen-specific regulatory T cells (Tregs).
276 ecipients expanded their endogenous Foxp3(+) regulatory T cells (Tregs).
277 hown to regulate immune responses and induce regulatory T cells (Tregs).
278 H to increase the number of bone marrow (BM) regulatory T cells (Tregs).
279 gen controls the selection of autoimmune and regulatory T cells (Tregs).
280 inst hematopoietic stem/progenitor cells and regulatory T-cells (Tregs) deficiency.
281 lios(+), GITR(+), LAG3(+) CD4 T cells (i.e., regulatory T cells [Tregs]).
282                                       Thymic regulatory T cells (tTreg) are critical in the maintenan
283 cytometry for presence of ovalbumin-specific regulatory T cells, using activation markers, FoxP3, and
284 bset, it accounted for more than 25% of CD4+ regulatory T-cell variation and over 50% of CD8+ central
285   At birth, a higher proportion of activated regulatory T cells was associated with ST (OR = 2.89, 95
286           The frequency of circulating FoxP3 regulatory T cells was not altered.
287         A decrease in the population of CD4+ regulatory T cells was observed during UB-421 monotherap
288 ge-dependent expansion of ovalbumin-specific regulatory T cells was only observed in infants who (a)
289                            The proportion of regulatory T cells was reduced in mice born by CS, where
290 ed by markers associated with exhaustion and regulatory T cells, was explored by flow cytometry.
291                     To generate MPO-specific regulatory T cells, we used a modified protein-conjugati
292                        In contrast, FoxP3(+) regulatory T cells were equally upregulated by transfer
293 n of suppressive molecules, and induction of regulatory T cells were monitored.
294 ers of FoxP3(+) and FoxP3(-) IL-10-secreting regulatory T cells were reduced.
295  were significantly lower, and pro-cancerous regulatory T cells were significantly higher in Late tum
296 ing RORgammat(+) T(reg) cells and follicular regulatory T cells, were c-Maf dependent.
297 genic phenotype and promote the expansion of regulatory T cells, whereas no such effects are seen wit
298 ycosylated antigens also expanded functional regulatory T cells, which are necessary for the durable
299  peripheral homeostasis of Foxp3(+)Helios(+) regulatory T cells, which likely accounts for our findin
300 ant reductions in numbers of CD4(+) Foxp3(+) regulatory T cells within tumors.

 
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