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1 he cytoplasmic domain of the calcium pump to relieve its inhibition.
2 caspase-3, the nuclease activity of DFF40 is relieved of inhibition.
3 of the requisite tyrosine, was still able to relieve NifL inhibition.
4 Excess unsaturated but not saturated FAs relieve this inhibition.
5 ion by DNA-PKcs in the C-terminal domain may relieve this inhibition.
6 ansporter (NCCT); mutations that cause PHAII relieve this inhibition.
7 hat mutagenesis or replacement of CE(16) can relieve this inhibition.
8 nce of a Wg signal, and the role of Wg is to relieve this inhibition.
9 Rnr1 and that Mec1 and Rad53 are required to relieve this inhibition.
10 , which increases UL9 processivity, does not relieve this inhibition.
11 leavage by tolloid proteinases is thought to relieve this inhibition.
12 guanine-nucleotide exchange factor RCC1 can relieve this inhibition.
13 ne that relieve tonic proton inhibition also relieve Zn2+ inhibition.
14 tracellular pH disrupted the association and relieved the inhibition.
15 ther the T antigen or the TBP fusion peptide relieved the inhibition.
16 1 because neutralizing antibodies completely relieved the inhibition.
17 Disrupting either binding determinant relieved the inhibition.
18 al change that prevents this interaction and relieves the inhibition.
19 that strong step depolarization transiently relieves the inhibition.
20 ity, and phosphorylation at Ser-68 or Ser-63 relieves the inhibition.
21 hospholipase C-mediated degradation of PIP2, relieving SR inhibition.
22 molar ratio to dinitrogenase, and 2OG fully relieved this inhibition.
23 at mutation of the Alix binding site largely relieved this inhibition.
24 , a potential signal of nitrogen limitation, relieved this inhibition.
25 on of the ARE or removal of the poly(A) tail relieved this inhibition.
26 tion of active CG, but not inactive CG, also relieves Boc2 inhibition.
27 reas addition of FPR agonists, fMLF and F2L, relieves Boc2 inhibition.
28 hat RhoA binding to the N terminus partially relieves this inhibition.
29 6 kinase, and that PI 3-kinase-Akt signaling relieves this inhibition.
30 cAMP binding relieves this inhibition.
31 ker K+ channels; presumably, phosphorylation relieves this inhibition.
32 in APC1 and that its mitotic phosphorylation relieves this inhibition.
33 nhibitor of SERCA and phosphorylation of PLN relieves this inhibition.
34 mmasome assembly, and downregulation of cAMP relieves this inhibition.
35 ination and proteasomal degradation, thereby relieving its inhibition.
36 ds to oxidation of the quinone pool, thereby relieving the inhibition.
37 nuclear RRC3 was necessary and sufficient to relieve growth inhibition.
38 de biochemical evidence that they mimic RHEB relieving auto-inhibition.
39 ing CysC would facilitate Abeta clearance by relieving CatB inhibition.
40 fall in pH potentiates kainate receptors by relieving zinc inhibition.
41 odon-anticodon base-pairing in 48S complexes relieved eIF1's inhibition.
42 mology 2 domain in an intramolecular fashion relieving basal inhibition.
43 ng domain (GBD) of the beta subunit strongly relieved glucose inhibition.
44 tracellular Na(+) ions to K(+) binding sites relieved the CTS inhibition.
45 by MPK-1 disrupts the LIN-1/LIN-31 complex, relieving vulval inhibition.
46 , disrupt this protein-lipid interaction and relieve catalytic inhibition.
47 risea, and plant surface waxes were found to relieve this self-inhibition.
48 JARID2 weakens PRC2's binding to RNA and relieves catalytic inhibition.
49 molecule inhibitor blocks CPA production and relieves PPARgamma inhibition.
50 not reactivate the cholinesterase enzyme and relieve cholinergic inhibition.
51 129-548 with similar affinity and partially relieves nucleation inhibition.
52 rbonate can further increase sAC activity by relieving substrate inhibition.
53 del in which protons modulate ENaC gating by relieving Na(+) self-inhibition.
54 vates the epithelial Na(+) channel (ENaC) by relieving Na(+) self-inhibition.
55 g with C1b alone activates classical PKDs by relieving C1-mediated inhibition.
56 igma wedge domain that binds to PTPsigma and relieves CSPG-mediated inhibition.
57 ective modulatory effects by exacerbating or relieving tonic proton inhibition.
58 ts of peroxynitrite and NO2 on TH but do not relieve the enzyme from inhibition.
59 constitutively targeting Tec to the membrane relieves SHIP1-mediated inhibition.
60 tions of Mg(2+) do not inhibit RNase III nor relieve the Mn(2+)-dependent inhibition.
61 t pronounced effect of Gbetagamma is that it relieves competitive product inhibition.
62 nt of both BIR2 and BIR3 domains for Smac to relieve XIAP-mediated caspase inhibition.
63 ation of CFs, and this suppression is partly relieved by blocking synaptic inhibition.
64 -473 phosphorylation and entry into G2/M was relieved by PHLPP phosphatase inhibition.
65 ylation and cytosolic retention of p27, thus relieving CDK2 from p27-induced inhibition.
66 hus, calpain cleavage of beta(3) at Tyr(759) relieves c-Src-mediated RhoA inhibition, activating the
68 itivity, and concentrations of spermine that relieve tonic proton inhibition also relieve Zn2+ inhibi
69 is to autophosphorylate the AIR in order to relieve auto-inhibition and that ADP acts as a switch to
70 to pyrimidine antimetabolite inhibitors also relieve CTP inhibition and cause a dramatic increase in
73 ADR1(c) alleles, or the inactivation of Bmh, relieve the inhibition and Snf1 dependence of this activ
79 OMP sequences to the DegS-PDZ domain, which relieves allosteric inhibition and activates proteolysis
81 in the N-terminal conformation of Kv1.5 that relieves Src-mediated tonic channel inhibition and resul
84 of a single phosphorylation site on separase relieves the inhibition and rescues chromatid separation
85 tion of the core histone tails significantly relieves this inhibition and allows TFIIIA to exhibit hi
86 lyzed activation of the G protein transducin relieves this inhibition and enhances PDE6 catalysis.
87 -gated chloride channels; odour presentation relieves this inhibition and results in activation of AI
88 n on the Na/K pump, while phosphorylated PLM relieves this inhibition and stimulates pump activity.
89 e phosphatase and its dissociation from ERK, relieving ERK from inhibition and resulting in its activ
90 estrogen-induced endogenous opioid release, relieving estrogen inhibition and facilitating lordosis.
91 noma cells maintained under hypoxia, thereby relieving Siah2 inhibition and increasing its activity u
92 nteracting with the YAP oncoprotein, thereby relieving YAP inhibition and promoting malignant transfo
93 Gal83 (beta) and Snf4 (gamma) subunits that relieve glucose inhibition, and we have here identified
95 ng to the inhibitory subunit p85alpha, which relieves its catalytic inhibition, and increased p110alp
96 inhibits the ATPase; (ii) binding of peptide relieves this inhibition; and (iii) the E543K mutation s
97 However, addition of the soluble SNARE Vam7p relieves BAPTA inhibition as effectively as Ca2+ or Mg2+
98 which augmented the heparin acceleration by relieving Gla domain inhibition as previously shown for
99 Repeated trains of large depolarizations relieved toxin inhibition, as if toxin affinity for acti
100 r magnesium, and that modulation by PKCalpha relieves this inhibition, as the potentiating effects of
101 d the rate of cleavage of TnaC-tRNA(2)(Pro), relieving the growth inhibition associated with plasmid-
102 : (a) neutralizing thrombospondin completely relieved the inhibition; (b) the inhibitory activity of
103 proposed to promote establishment by merely relieving Wpl1p inhibition because deletion of WPL1 bypa
104 onstitutively active Akt construct partially relieved cell growth inhibition but was less effective t
105 REC2 subdomain that normally caps the R-loop relieved this inhibition but favoured stabilisation of u
106 lthough binding of cAMP has been proposed to relieve auto-inhibition by affecting the structure of th
107 ncentrations of glucose, conditions known to relieve GK inhibition by GKRP in vitro, only GK was tran
109 Although inhibition by 1-548 is partially relieved by RhoA, inhibition by 129-548 or 129-369 is Rh
110 Mgn gene expression in innervated muscle and relieved Mgn promoter inhibition by HDAC inhibitors.
113 portantly, tyrosine phosphorylation (of p27) relieves Cdk inhibition by p27, enabling cell cycle entr
114 cycle progression, we hypothesized that PIN1 relieves CDK2 inhibition by suppressing the CDK inhibito
115 ity of AKAP121 by the ubiquitin ligase Siah2 relieves Drp1 inhibition by PKA and increases its intera
116 sically interact in vitro, that this complex relieves NadE(Gln) negative feedback inhibition by NAD(+
121 verproduction of FliH and FliI substantially relieved the inhibition caused by FliH, suggesting that
122 ed to the in vitro CTL stimulation cultures, relieved the inhibition caused by the AKR.H-2(b) cells i
123 totagmin-1 activates fast release, likely by relieving the inhibition caused by Complexins and cooper
124 nued presence of norepinephrine, PDC or PDCL relieved calcium channel inhibition compared with contro
126 Our results support a model in which RSpo1 relieves the inhibition DKK1 imposes on the Wnt pathway.
129 tidine residues on Ca(v)3.2 T-channels, thus relieving tonic channel inhibition, enhancing Ca(v)3.2 c
130 Accumulation of fimbrin at actin patches relieves Myo1p from tropomyosin-mediated inhibition, ens
132 ic acid, decreases alpha-synuclein levels by relieving the inhibition exerted on alpha-synuclein prot
133 exoelectrogens (typically Geobacter species) relieves feedback inhibition for the fermentative consor
135 EPTOR overexpression suppresses S6K1 but, by relieving feedback inhibition from mTORC1 to PI3K signal
137 he JNK pathway described here is required to relieve the inhibition imposed by TRAF2-cIAP1 on caspase
138 an intracellular conformational switch which relieves allosteric inhibition imposed on the intracellu
140 ERK activity in immature granulosa cells by relieving an inhibition imposed by a 100-kDa phosphotyro
141 n the Bcl-2 family members Bax and/or Bak by relieving the inhibition imposed by antiapoptotic member
143 non-NtrC-dependent promoters, it was able to relieve NifL inhibition in the absence of uridylyltransf
144 on under aerobic conditions, and attempts to relieve NifL inhibition in vitro by reconstituting Fe-S
145 mutations in EB1 that disrupted APC binding relieved the inhibition in vitro and restored APC locali
147 t phosphorylation of PLB at Ser-16 or Thr-17 relieves this inhibition in cardiac sarcoplasmic reticul
149 action potential voltage waveforms can also relieve the inhibition, increasing calcium current throu
150 he mutations Q1208S, K1085N, and Y1162F each relieved intrasteric inhibition, increasing catalytic ef
151 bitory effects of ER stress inducers, and E2 relieved general translation inhibition induced by PERK.
152 ation of PLN at S16 in the cytoplasmic helix relieves SERCA inhibition, initiating muscle relaxation.
153 bsiella pneumoniae, in which unmodified GlnK relieves NifL inhibition instead of stimulating it.
154 ssion in other cell types, PP1 activation to relieve IRS1 inhibition may be a more general mechanism
155 RadA prior to reaction initiation with ssDNA relieves the inhibition observed when SsoSSB is added ei
156 olecularly with the N-terminal SH2 domain to relieve basal inhibition of the PTPase, whereas a phosph
158 itment, impair endosomal protein sorting and relieve dominant-negative VPS4 inhibition of HIV budding
159 ore a mechanotransducer that acts via PKC to relieve Egr1 transcriptional inhibition of Cav1 and cavi
162 ed signaling, as evidenced by its ability to relieve Galphai2-mediated inhibition of adenylyl cyclase
163 g1; moreover, hxk2 and reg1 mutations, which relieve glucose inhibition of Snf1, correspondingly affe
165 from the spindle pole bodies, which may both relieve Nak1 inhibition of the SIN and block MOR signali
166 notably, the same mutations in PRKWNK4 that relieve NCCT inhibition markedly increase inhibition of
168 RNAs (miRNAs) 1 and 21 bind PLN strongly and relieve PLN inhibition of SERCA to a greater extent than
169 TA1-1-mediated resistance to CHB in order to relieve potent product inhibition of the enzyme by intra
170 truncation, a beta-ENaC PY mutation did not relieve S1A inhibition of I(Na), suggesting that S1A doe
172 ExsD in ExsA binding explains how ExsC might relieve the ExsD-mediated inhibition of T3SS gene expres
173 gesting that repetitive stimulation does not relieve the G-protein inhibition of calcium channels by
175 GS and NR and the ability of ATP and AMP to relieve the inhibition of NR can therefore be explained
176 tions are reflected in an ability of MDM2 to relieve the inhibition of p63 by p53R175H, but enhance t
179 formation of a UBF-SL1 complex can partially relieve the inhibition of transcription, only the assemb
180 ed, which sheds light on the ability of 3 to relieve the inhibition of XIAP with not only caspase-9 b
182 conferred by MeCP2 and methylated DNA can be relieved by inhibition of histone deacetylase, facilitat
183 dingly, repression in the absence of MLL2 is relieved by inhibition of PRC2 and DNA methyltransferase
184 ssed by the histone modifier HDAC2, which is relieved by pharmacological inhibition of histone deacet
185 r protein family members pRB, p107, and p130 relieved E1A-mediated inhibition of transcription in res
186 5901 inhibited ERK phosphorylation, but also relieved feedback inhibition of RAS, resulting in induct
188 Acidification of the intracellular solution relieved glibenclamide inhibition of CFTR, suggesting th
189 the Asp-Phe-Gly motif on the activation loop relieved glucose inhibition of phosphorylation, resultin
193 itutively active Akt1/protein kinase B alpha relieved Hrt2-mediated inhibition of GATA-dependent tran
194 sp1161Ala mutant in the activation loop that relieved intrasteric inhibition of the unphosphorylated
196 Surprisingly, even though added H(2) also relieved N(2) inhibition of substrate reduction, the alp
197 opic expression of IRF-1 in epithelial cells relieved P. gingivalis-induced inhibition of IP-10 relea
205 tration of NifNE and NifB-cofactor (NifB-co) relieved the inhibition of FeMo-co synthesis by L127Delt
206 RGS function (GRK2-D110A/K220R) effectively relieved the inhibition of inositol phosphate generation
207 extension required for particle engulfment, relieved the inhibition of phagocytosis in the presence
210 reatment with RNA interference against SOCS3 relieved virus-induced inhibition of IFN-gamma-induced S
211 signaling mechanism in which drought stress relieves ABI1 inhibition of BIN2, allowing BIN2 activati
214 n agonist-promoted depalmitoylation of eNOS, relieves caveolin's tonic inhibition of enzyme activity.
215 decreases its affinity for beta subunits and relieves constitutive inhibition of Ca(V)1.2, observed a
216 ADP-ribose (cADPr) signaling that partially relieves DAMGO inhibition of I(Ca) and completely reliev
217 pport a cellular cascade in which inactivity relieves EVI1/HDAC-mediated inhibition of miR124 gene tr
218 ently inhibits Akt/mTOR signaling because it relieves feedback inhibition of IGF1R and other receptor
221 li such as brain-derived neurotrophic factor relieves miR-134 inhibition of Limk1 translation and in
222 ves DAMGO inhibition of I(Ca) and completely relieves MOR-mediated inhibition of high-K(+)-induced an
225 as hydrophila AhQnr, is soluble, stable, and relieves quinolone inhibition of Escherichia coli DNA gy
226 ion, a SpoIIAA-dependent mechanism partially relieves SpoIIAB inhibition of sigma F activity in the f
228 se findings provide a mechanism by which CBF relieves the auto-inhibition of Ets-1 and illustrates on
229 a putative transcription co-activator NDP52 relieves the auto-inhibition of MVI to enable DNA bindin
231 phatidylinositol 3-kinase/Akt pathway, which relieves the inhibition of B-Raf to allow the cAMP growt
233 Here we show that increased levels of CBP relieves the inhibition of glucocorticoid-mediated repre
234 f the AMP-dependent protein kinase partially relieves the inhibition of GLUT4 translocation by TBC1D1
238 proximately 1 microM to approximately 40 nM) relieves the inhibition of PDE3A, increasing its activit
239 nts dissociates the Hsp90-Apaf-1 complex and relieves the inhibition of procaspase-9 activation.
241 vity of IkappaB kinase-beta (IKKbeta), which relieves the inhibition of the NF-kappaB transcriptional
242 preferentially binds single-stranded DNA and relieves the inhibition of the strand assimilation and D
243 ing of Sonic Hedgehog (Shh) to Patched (Ptc) relieves the latter's tonic inhibition of Smoothened (Sm
244 tion experiments indicate that dFMRP OE also relieves the translation inhibition of futsch mRNA, a TD
245 MP binding to the regulatory subunits (PKAR) relieves their inhibition of the catalytic subunits (PKA
247 a- and IFNalpha-induced PML accumulation and relieves TNFalpha- and IFNalpha-mediated inhibition of E
248 ases enhance enzymatic biomass conversion by relieving cellobiose inhibition of endoglucanases and ce
249 lin/IGFs regulate beta cell proliferation by relieving Foxo1 inhibition of Pdx1 expression in a subse
251 nic phosphate is, however, less effective in relieving glucose 6-phosphate inhibition of the Gly862 -
252 phospholamban (PLN) at both Ser16 and Thr17, relieving its inhibition of the apparent Ca affinity of
253 xons, melatonin reduces cAMP levels, thereby relieving PKA-induced inhibition of group III mGluRs; th
255 of the C-terminal extension is critical for relieving the auto-inhibition of full-length Drs2p, wher
256 erol releases the receptor from lipid rafts, relieving the functional inhibition of the receptor.
259 ot removed by a washing process effective in relieving the inhibition of SHP-1 by the reversible inhi
266 n levels by Cdc5 promotes FEAR activation by relieving the inhibition on Clb2-Cdk1, the kinase for Ne
267 ylation of the docking protein Gab2, thereby relieving the negative inhibition on AKT and promoting t
268 an EXD-induced conformational change in LAB relieves this inhibition, promoting highly specific inte
270 uses that carry suppressors of RNA silencing relieved the phenotypic inhibition, restoring pigment pr
273 nation of inducer exclusion is sufficient to relieve glucose inhibition, suggesting that glucose acts
275 phorylation of PLB increases the R state and relieves SERCA inhibition, suggesting that R is less inh
276 nding that a reduced pH activated ASIC1a and relieved BK inhibition suggests that extracellular proto
278 e 2 (SSTR2) antagonism of alpha-cells, which relieve SSTR2 inhibition, thereby increasing glucagon se
279 ition; although the currents are temporarily relieved of tonic inhibition, they are still capable of
280 ctivity, whereas Smac (also known as DIABLO) relieves this inhibition through interaction with XIAP.
281 t to generate the heterotrimeric FLA8/10/KAP relieved this inhibition, thus providing a mechanistic r
282 n mammals and Reaper, Hid, and Grim in flies relieve IAP-mediated inhibition to induce cell death.
285 tein, not yet identified, may be required to relieve NifL inhibition under anaerobic, nitrogen-limiti
286 neral nitrogen regulator NtrC is required to relieve NifL inhibition under nitrogen (N)-limiting cond
287 ene(s) whose product(s) in turn functions to relieve NifL inhibition under nitrogen-limiting conditio
288 tion of superoxide dismutase in vivo did not relieve NifL, inhibition under aerobic conditions, and a
289 g visual excitation both catalytic sites are relieved of Pgamma inhibition upon binding of two activa
290 This mechanism is distinct from the one that relieves PLB-dependent SERCA inhibition upon the additio
291 at low [Ca(2+)], but the cytoplasmic domain relieves this inhibition upon Ser16 phosphorylation.
292 phosphorylation of Ser-16 and Thr-17, which relieves the inhibition, was increased approximately 2-
293 Surprisingly, however, in attempting to relieve the ACSL-mediated inhibition, we discovered that
295 , facilitate TnaC-tRNA(2)(Pro) cleavage, and relieve growth inhibition were addressed in the current
297 fection, which upon virus-signaled depletion relieves caspase inhibition, which subsequently executes
298 minated C(2)H(4) reduction but did not fully relieve its electron-flux inhibition with all four A. vi
300 clude that phosphorylating monomeric PLB can relieve SERCA inhibition without changes in the oligomer