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1 erpin mutational selection in normal tissues remain poorly understood.
2 neural correlates of this remarkable ability remain poorly understood.
3 anisms whereby the microbiota modulates mood remain poorly understood.
4 studied, the underlying cellular mechanisms remain poorly understood.
5 ixing on the membrane biophysical properties remain poorly understood.
6 s underlying increased antiapoptosis signals remain poorly understood.
7 the mechanisms underlying metastatic spread remain poorly understood.
8 on of OHCs and their synaptic specialization remain poorly understood.
9 ent influences of ligands in these reactions remain poorly understood.
10 tterns and their impact on neutral diversity remain poorly understood.
11 al substrates engaged by muscarinic agonists remain poorly understood.
12 c acid interactions in a crowded environment remain poorly understood.
13 riving leukemic transformation at this level remain poorly understood.
14 the mechanisms linking these two phenomena, remain poorly understood.
15 ers and genomic basis of seed mass variation remain poorly understood.
16 logically restrictive tumor microenvironment remain poorly understood.
17 ion of Tim-3, but the molecular determinants remain poorly understood.
18 heir underlying drivers and societal impacts remain poorly understood.
19 s N, its turnover, and stabilization in soil remain poorly understood.
20 mechanism of action, antagonism and targets remain poorly understood.
21 but its function and mechanism of regulation remain poorly understood.
22 However, the mechanisms causing itch remain poorly understood.
23 and shape life histories among species, but remain poorly understood.
24 the corresponding bone remodeling mechanisms remain poorly understood.
25 sms contributing to the pathogenesis of DFUs remain poorly understood.
26 The interiors of giant planets remain poorly understood.
27 nal silencers have been identified, and they remain poorly understood.
28 tin establishment during early embryogenesis remain poorly understood.
29 lationship between cell number and body size remain poorly understood.
30 rigins and evolution of the llama and alpaca remain poorly understood.
31 les detect and align with the long cell axis remain poorly understood.
32 ious donor-specific antibody (DSA) responses remain poorly understood.
33 underlying cellular and molecular mechanisms remain poorly understood.
34 lved in terminating PG glycan polymerization remain poorly understood.
35 ical origins of their different morphologies remain poorly understood.
36 ress pathway in promoting lipid accumulation remain poorly understood.
37 ar mechanisms through which MIA imparts risk remain poorly understood.
38 ia to humans, but their mechanisms of action remain poorly understood.
39 ation is achieved in microsporidia, however, remain poorly understood.
40 ptional networks underlying their expression remain poorly understood.
41 the mechanisms that control myotube guidance remain poorly understood.
42 ant of Hp colonization and chronic infection remain poorly understood.
43 s metastatic process in ovarian cancer cells remain poorly understood.
44 tion of such continuously adapting pathogens remain poorly understood.
45 ced neurotoxicity, the underlying mechanisms remain poorly understood.
46 pathway neuron disconnectivity in stereotypy remain poorly understood.
47 microplastic ingestion in marine zooplankton remain poorly understood.
48 sms that drive pathogenesis and genetic risk remain poorly understood.
49 its epidemiologic and pathogenetic features remain poorly understood.
50 viduals, and the reasons for such variations remain poorly understood.
51 regulating their development within tissues remain poorly understood.
52 or some medications but, in other instances, remain poorly understood.
53 ttern formation during embryonic development remain poorly understood.
54 of these cis-isomers from trans-violaxanthin remain poorly understood.
55 r and clinical level have been reported that remain poorly understood.
56 establishment and maintenance of these sizes remain poorly understood.
57 lls and other stromal cells in breast cancer remain poorly understood.
58 ut the mechanisms of tropical tree mortality remain poorly understood.
59 ecture and neurogenetics of animal migration remain poorly understood.
60 initiation and progression of these subtypes remain poorly understood.
61 partially rescue DeltaF508-CFTR function yet remain poorly understood.
62 e preferential growth in width versus length remain poorly understood.
63 ution of damaged cells to developing embryos remain poorly understood.
64 actions with hematopoietic microenvironments remain poorly understood.
65 anisms of seizure initiation and propagation remain poorly understood.
66 lements in exerting their protective actions remain poorly understood.
67 rn alpha-syn fibrillization and LB formation remain poorly understood.
68 llopolyploidy and the underlying mechanisms, remain poorly understood.
69 peratures; however, the mechanisms behind TC remain poorly understood.
70 minal differentiation of segment populations remain poorly understood.
71 owever, the underlying biological mechanisms remain poorly understood.
72 ny aspects of their photosynthetic machinery remain poorly understood.
73 isms by which they modulate immune responses remain poorly understood.
74 d cellular mechanisms mediating this process remain poorly understood.
75 onment that elicit niche-driven inflammation remain poorly understood.
76 s species, the mechanisms behind this choice remain poorly understood.
77 uence the retention of persisting duplicates remain poorly understood.
78 ral mechanisms contributing to these effects remain poorly understood.
79 n, and interactions with cellular chaperones remain poorly understood.
80 the molecular mechanisms behind transduction remain poorly understood.
81 he mechanisms by which pathogens modulate AS remain poorly understood.
82 ts of nanodomain maintenance and persistence remain poorly understood.
83 sponding clinical relevance of prolactinomas remain poorly understood.
84 au pathology, and their effects on cognition remain poorly understood.
85 , and choking, yet relevant sensory pathways remain poorly understood.
86 tionary processes that shape the OROV genome remain poorly understood.
87 million years before photosynthesis evolved, remain poorly understood.
88 rmation; however, their mechanisms of action remain poorly understood.
89 ) O production across terrestrial ecosystems remain poorly understood.
90 nimal kingdom, but its underlying mechanisms remain poorly understood.
91 sory correlates of changes in ICMS frequency remain poorly understood.
92 he exocyst functions and how it is regulated remain poorly understood.
93 an cerebral cortex folds into its final form remain poorly understood.
94 onged eosinophil deficiency for human health remain poorly understood.
95 so that its dynamical and collective aspects remain poorly understood.
96 h pathogens overcome colonization resistance remain poorly understood.
97 the mechanisms of PROX1/VEGF-C feedback loop remain poorly understood.
98 tion with other metabolic processes in seeds remain poorly understood.
99 as well as centriole architectural integrity remain poorly understood.
100 el oxygen dynamics and fermentative pathways remain poorly understood.
101 izing bacteria, but their interrelationships remain poorly understood.
102 vation of oncogenic KRAS in epithelial cells remain poorly understood.
103 n whose evolutionary and biological function remain poorly understood.
104 ture of halite-entombed archaeal communities remain poorly understood.
105 with the breaching behaviors of large whales remain poorly understood.
106 orldwide, but the associated CO(2) emissions remain poorly understood.
107 e interneurons are produced from progenitors remain poorly understood.
108 host interactions involved in nuclear import remain poorly understood.
109 lucocorticoids regulate human erythropoiesis remain poorly understood.
110 tcomes among patients with tuberculosis (TB) remain poorly understood.
111 ents driving proteostasis failure in disease remain poorly understood.
112 late these oxygen and 2-OG sensitive enzymes remain poorly understood.
113 echanisms underlying sex differences in pain remain poorly understood.
114 aherpesvirus-driven germinal center response remain poorly understood.
115 echanisms that link these pathways, however, remain poorly understood.
116 evolutionary origins and ancestral function remain poorly understood.
117 hether it recapitulates embryonic mechanisms remain poorly understood.
118 or in T-ALL, the mechanisms of GC resistance remain poorly understood.
119 ophysiological processes modified by nesting remain poorly understood.
120 insertions to recent regulatory adaptations remain poorly understood.
121 ntibody-mediated immunity (AMI) to Y. pestis remain poorly understood.
122 at regulate fascin dynamics within filopodia remain poorly understood.
123 ammatory cells in neurodegenerative diseases remain poorly understood.
124 the mechanisms controlling digit morphology remain poorly understood.
125 nction behavior and subsequent reinstatement remain poorly understood.
126 and temperature signaling pathways in plants remain poorly understood.
127 isms and the influence of exercise intensity remain poorly understood.
128 hemical and behavioral consequences, if any, remain poorly understood.
129 questration mechanisms in estuarine wetlands remain poorly understood.
130 ble and adapt to a particular internal state remain poorly understood.
131 disease onset but the underlying mechanisms remain poorly understood.
132 onsible for severe inflammatory side effects remain poorly understood.
133 lying the persistent accumulation of Mo/MPhi remain poorly understood.
134 underlying this so-called foraging response remain poorly understood.
135 ssible health risks associated with ENDS use remain poorly understood.
136 cular mechanisms that initiate this activity remain poorly understood.
137 ctions during development by mechanisms that remain poorly understood.
138 The mechanisms mediating AMC remain poorly understood.
139 he complex mechanisms driving immune evasion remain poorly understood.
140 nisms that underlie their ionic conductivity remain poorly understood.
141 r processes underpinning flowering in nature remain poorly understood.
142 s droughts, are overlooked and their impacts remain poorly understood.
143 yofibroblasts to persist in fibrotic tissues remain poorly understood.
144 c features of this mixed valent intermediate remain poorly understood.
145 esses, but the underlying circuit mechanisms remain poorly understood.
146 ular mechanisms regulating mtDNA copy number remain poorly understood.
147 f the PD-1 pathway ligands, PD-L1 and PD-L2, remain poorly understood.
148 re remodelled upon developmental transitions remain poorly understood.
149 ses to novel classes of antagonistic stimuli remain poorly understood.
150 nisms that retain neutrophils within tissues remain poorly understood.
151 rane interactions to protein oligomerization remain poorly understood.
152 cells; however, their active site structures remain poorly understood.
153 nderlying schizophrenia and bipolar disorder remain poorly understood.
154 olecular identities and functional potential remain poorly understood.
155 egulatory elements, yet how this is achieved remains poorly understood.
156 ocessing higher up in the cortical hierarchy remains poorly understood.
157 regulation of AM development and maintenance remains poorly understood.
158 broad measures of health, such as mortality, remains poorly understood.
159 ontributes to the pathogenesis of neuropathy remains poorly understood.
160 on and their relationship with transcription remains poorly understood.
161 ontact investigations and preventive therapy remains poorly understood.
162 their trafficking across the Golgi apparatus remains poorly understood.
163 suberin biosynthesis in this group of plants remains poorly understood.
164 crucial in development and regeneration but remains poorly understood.
165 vity of these circuits is altered in obesity remains poorly understood.
166 emanding, and highly dynamic valvular system remains poorly understood.
167 DD); however, its neural mechanism of action remains poorly understood.
168 ced by Rab7, though the underlying mechanism remains poorly understood.
169 euronal subtypes to seizure pathophysiology, remains poorly understood.
170 sis and secretion of SGs, but how SGs mature remains poorly understood.
171 ory elements such as enhancers and promoters remains poorly understood.
172 ad movements affect visual cortical circuits remains poorly understood.
173 been demonstrated, and the function of Ddi1 remains poorly understood.
174 dysregulation, particularly in human cells, remains poorly understood.
175 ole biosynthesis and/or protein biosynthesis remains poorly understood.
176 lock as young adults, the mechanism of which remains poorly understood.
177 ncy virus type 1 (HIV-1), but their assembly remains poorly understood.
178 ccus pneumoniae carriage prevalence and load remains poorly understood.
179 iated cancer cell containment can be induced remains poorly understood.
180 h TLR signaling to self-ligands is regulated remains poorly understood.
181 pecies interactions in setting range margins remains poorly understood.
182 To date, the pathogenic mechanism involved remains poorly understood.
183 on of 3D chromatin structure in this process remains poorly understood.
184 ic network communities during human seizures remains poorly understood.
185 ailure, which affects a large population but remains poorly understood.
186 that the mechanism of KRAS-driven malignancy remains poorly understood.
187 but the molecular basis of thermoregulation remains poorly understood.
188 ction stage in patients with aortic stenosis remains poorly understood.
189 ed, their metabolism including glycosylation remains poorly understood.
190 function of HSs as a redox battery, however, remains poorly understood.
191 athway is organized and regulated in neurons remains poorly understood.
192 s activation states in response to cytokines remains poorly understood.
193 ifferent Arfs function(s) during replication remains poorly understood.
194 estrial plants; however, its functional role remains poorly understood.
195 lemented in the central nervous system (CNS) remains poorly understood.
196 lar localizations during meiotic progression remains poorly understood.
197 Ns), a well-recognized characteristic of AD, remains poorly understood.
198 the cell biology of endocrine tissue ageing remains poorly understood.
199 w mRNA granules interact with motor proteins remains poorly understood.
200 ficiency leads to increased immune responses remains poorly understood.
201 y investigated, the genetic etiology of NTDs remains poorly understood.
202 izational logic of the anterolateral pathway remains poorly understood.
203 ss on gene transcription in uterine fibroids remains poorly understood.
204 eral tissues in health and disease, however, remains poorly understood.
205 How effector sensing triggers NLR activation remains poorly understood.
206 cell into a malignant, heterogeneous tissue remains poorly understood.
207 he regulation of cardiomyocyte contractility remains poorly understood.
208 tree seedlings depend on different stressors remains poorly understood.
209 strates and functions besides degrading KRPs remains poorly understood.
210 ations reported in FA lead to HSC exhaustion remains poorly understood.
211 cts on gene expression in human immune cells remains poorly understood.
212 t, the role of host immunity in this process remains poorly understood.
213 are resistant to incursions of alien species remains poorly understood.
214 ole of sleep in responses to neural injuries remains poorly understood.
215 ing behaviour, infection and immune function remains poorly understood.
216 pact of hydro-dynamic coupling on ice motion remains poorly understood.
217 h which tau contributes to neurodegeneration remains poorly understood.
218 many ecological processes, but how it arises remains poorly understood.
219 ations in nutrients suppress mTORC1 activity remains poorly understood.
220 ion priorities and institutional constraints remains poorly understood.
221 m by which these oncometabolites inhibit HDR remains poorly understood.
222 tion factors are transcriptionally regulated remains poorly understood.
223 itive decline, although studied extensively, remains poorly understood.
224 to which these mechanisms respond to climate remains poorly understood.
225 ributed to many populations that live today, remains poorly understood.
226 e virus engages and exploits these filaments remains poorly understood.
227 Yet, how it remodels under force remains poorly understood.
228 al development in early-life affects fitness remains poorly understood.
229 sitive membrane remodeling by these proteins remains poorly understood.
230 efficiency for downstream immune activation remains poorly understood.
231 How nutrient signals control IME1 expression remains poorly understood.
232 e genes, but its function during development remains poorly understood.
233 which keratin filament reorganization occurs remains poorly understood.
234 challenges using a molecular mechanism that remains poorly understood.
235 The pathobiologic basis for skin aging remains poorly understood.
236 tinct types of genome-composing nucleic acid remains poorly understood.
237 n, the influence of the physical environment remains poorly understood.
238 ow this relates to behavioral abnormalities, remains poorly understood.
239 ole of systemic immunity in cancer incidence remains poorly understood.
240 iversification and adaptation in angiosperms remains poorly understood.
241 some occupancy influence the cold-induced AS remains poorly understood.
242 echanism of sickle cell hepatobiliary injury remains poorly understood.
243 ousness (LOC) and affects sensory processing remains poorly understood.
244 ntly discovered, but its mechanism of action remains poorly understood.
245 ulation of TRIM21 during bacterial infection remains poorly understood.
246 ied, precisely how gamma-TuRC nucleates a MT remains poorly understood.
247 ic regulation of leptin in polygenic obesity remains poorly understood.
248 chical levels, but its neural implementation remains poorly understood.
249 rolled to dictate the fates of cellular RNAs remains poorly understood.
250 ional-anatomical basis of this heterogeneity remains poorly understood.
251 ns receive information about reward outcomes remains poorly understood.
252 (CNS) through the blood-brain barrier (BBB) remains poorly understood.
253 but how they affect substrate conformations remains poorly understood.
254 ents are habitable for terrestrial organisms remains poorly understood.
255 he presence of variable environmental forces remains poorly understood.
256 use, and yet how the brain represents shape remains poorly understood.
257 lammasome activation and IL-1beta secretion, remains poorly understood.
258 nity against hemibiotrophic fungal infection remains poorly understood.
259 echanism that regulates Parkin protein level remains poorly understood.
260 n dissected, regulation of membrane dynamics remains poorly understood.
261 ntracellular force regulates these processes remains poorly understood.
262 nce, but their effect on the soil microbiome remains poorly understood.
263 essing extend and/or progress into adulthood remains poorly understood.
264 tructural basis of Wnt signaling through Fzd remains poorly understood.
265 role in modulating cellular immune responses remains poorly understood.
266 gram the immunological state of target cells remains poorly understood.
267 over to large-scale hydroclimate variability remains poorly understood.
268 modulate CLR binding to transducer proteins remains poorly understood.
269 tant ecological processes, but how it arises remains poorly understood.
270 napses but its role in synaptic transmission remains poorly understood.
271 cs to drug adaptation and therapy resistance remains poorly understood.
272 established in the developing epidermis has remained poorly understood.
274 t Nat machinery and its biological relevance remain poorly understood, although NTA has gained recogn
275 ms underlying these heterogeneous phenotypes remain poorly understood, although post-translational mo
276 uring infection and its role in pathogenesis remain poorly understood and are fruitful areas for futu
277 ses; however, the causes of this association remain poorly understood and they cover a range of immun
278 of individual PSMs during biofilm formation remains poorly understood and the molecular pathways of
279 traumatic spectrum disorders, their etiology remains poorly understood and their phenomenology incomp
280 crobial adaptations to the respiratory burst remain poorly understood, and establishing how the NADPH
281 anisms driving sarcomatoid dedifferentiation remain poorly understood, and information and treatment
282 g men, but the underlying molecular features remain poorly understood, and therapeutic options are li
286 egrator subunits beyond the RNA endonuclease remain poorly understood, but some can act independently
287 smission of drug-resistant tuberculosis (TB) remain poorly understood, despite over half a million in
288 nfection with Schistosoma haematobium worms, remains poorly understood due to a historical lack of an
289 ad of mosquito-borne diseases, blood feeding remains poorly understood due to technological limitatio
290 of NAc FSIs in the context of drug addiction remains poorly understood, emerging evidence suggests th
291 carriers and their transfer mechanisms still remain poorly understood, especially for the cases of li
292 mic changes of the bacterial wine microbiota remain poorly understood, especially in the context of r
293 bon out of the surface ocean and its drivers remains poorly understood, especially in the Southern Oc
294 race to silence transposable elements yet it remains poorly understood how this domain is able to evo
295 ion for variation in molar proportions, what remains poorly understood is if molar shape, or specific
296 s tidally induced biogeochemical oscillation remains poorly understood, limiting the effectiveness of
297 However, the mechanism by which they do so remains poorly understood owing to a lack of structural
298 scence competence, induction and maintenance remain poorly understood, precluding the deliberate ther