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1 n through CpG islands does not inhibit their remethylation.
2  plasma homocysteine levels by impairing its remethylation.
3 ong global demethylation and drastic focused remethylation.
4 romotes transposon silencing via de novo DNA remethylation.
5 oplasm, a required cofactor for homocysteine remethylation.
6 ylate biosynthesis and impaired homocysteine remethylation.
7 crM and chromosomal DNA, thus preventing DNA remethylation.
8 uration and transmethylation or homocysteine remethylation.
9 is pathway and folate-dependent homocysteine remethylation.
10 ie, as p53 expression continues unchecked by remethylation.
11 f the 5' region for over 40 days, preventing remethylation.
12 ed by waves of genome-wide demethylation and remethylation.
13 methionine were labeled through homocysteine remethylation.
14 ined by differences in rates of homocysteine remethylation.
15 for methyl group production and homocysteine remethylation.
16 ng these cells as substrates, we defined DNA remethylation after overexpressing wild-type (WT) DNMT3A
17 th inhibition of CD8 gene expression through remethylation and concludes with up-regulation of surfac
18 tures inhibits folate-dependent homocysteine remethylation and enhances thymidylate biosynthesis.
19 but its overall contribution to homocysteine remethylation and sulfur amino acid homeostasis is not k
20 B-6 restriction on the rates of homocysteine remethylation and synthesis in healthy humans.
21  of origins that are methylated, the rate of remethylation and the effect of hypomethylation were det
22 nine coordinately regulates the flux through remethylation and transsulfuration, and glycine N-methyl
23  to first undergo demethylation, followed by remethylation as development proceeds.
24                                              Remethylation at ori-beta did not begin until approximat
25 ed IFN-gamma/IL-4 and IL-10/IL-4 ratios, DNA remethylation at the IL-4 promoter and demethylation at
26 ssues and evidence for impaired homocysteine remethylation but surprisingly exhibited depressed level
27 te deficiency at the expense of homocysteine remethylation, but the mechanisms are unknown.
28  their cognate sequences and thereby prevent remethylation by DNA methyltransferase.
29 ration of high levels of expression requires remethylation by DNMT3B.
30  cob(I)alamin to the folate (Fol) domain for remethylation by methyltetrahydrofolate (CH(3)-H(4)folat
31 ings demonstrate that defective homocysteine remethylation caused by deficiency of either MS or folat
32 o and that serine synthesis and homocysteine remethylation compete for one-carbon units in the cytopl
33 DH impairs the folate-dependent homocysteine remethylation cycle.
34 s, cancer-testis gene promoters also undergo remethylation during CTCF/BORIS switching in germ cells.
35  biosynthesis at the expense of homocysteine remethylation during folate deficiency.
36 ivity of the folate-independent homocysteine remethylation enzyme betaine-homocysteine S-methyltransf
37                                         Both remethylation events are attributable to CheR using S-ad
38 tween thymidylate synthesis and homocysteine remethylation for THF cofactors.
39                     Here, we analysed global remethylation from the mouse preimplantation embryo into
40                    Although the homocysteine remethylation gene MTHFR has emerged as a risk factor in
41 eductase, an enzyme involved in homocysteine remethylation, has been reported to render the enzyme th
42 n and its transsulfuration, and homocysteine remethylation in children with SCU.
43 onstrated that both global demethylation and remethylation in early development correlate with chroma
44 ine-derived one-carbon units in homocysteine remethylation in hepatic and whole-body metabolism.
45 coincides with genome-wide demethylation and remethylation in mice which most imprinted genes resist.
46 hese regions also exhibited higher levels of remethylation in single-cell clones and subclones derive
47  negative in vivo and because zygotic genome remethylation is a key biological event during cleavage,
48                                       Genome remethylation is essential for mammalian development but
49                             This lack of DNA remethylation leaves the Pdcd1 locus poised for rapid ex
50 0 microg/d) of vitamin B12, the cofactor for remethylation; low intake (<2.1 mg/d) of vitamin B6, the
51 riants of two genes involved in homocysteine remethylation/methionine biosynthesis--methionine syntha
52 ents in the absence of cell division, yet no remethylation occurred at CpG islands under these condit
53   On DAC withdrawal, gradual resilencing and remethylation occurred in both GFP-positive and GFP-nega
54                                              Remethylation occurred most rapidly in the p16, PAX-6, a
55  occurs in the nucleus, whereas homocysteine remethylation occurs in the cytosol.
56 urthermore, AID or Mbd4 knockdown caused the remethylation of a set of common genes.
57 brogated the protective effect of AC whereas remethylation of AC DNA reversed the effects of activati
58                Furthermore, we show that the remethylation of asparagine-bound McpB requires the resp
59                                 The rates of remethylation of both CpG islands were associated with t
60 e also examined the relationship between the remethylation of coding sequence CpG islands and gene tr
61                Genome-wide demethylation and remethylation of DNA during early embryogenesis is essen
62                  Counter-regulation included remethylation of Hcy to methionine concurrent with decre
63 bon metabolism, during which it promotes the remethylation of homocysteine -- a cytotoxic sulfur-cont
64 xes of total homocysteine remethylation, the remethylation of homocysteine from serine, and rates of
65 tilized for de novo purine synthesis and the remethylation of homocysteine in liver.
66                                              Remethylation of homocysteine is dependent on the produc
67                         Folate B12-dependent remethylation of homocysteine is important, but less is
68 antifolate drug, aminopterin (which prevents remethylation of homocysteine to methionine by methionin
69 In the cytosol, vitamin B12 functions in the remethylation of homocysteine to methionine, which regen
70                    This enzyme catalyzes the remethylation of homocysteine to methionine, with betain
71 hesis of dTMP and purine nucleotides and for remethylation of homocysteine to methionine.
72 f folate, which serves as a methyl donor for remethylation of homocysteine to methionine.
73 ltetrahydrofolate, the major carbon donor in remethylation of homocysteine to methionine.
74 hesis of three of the four DNA bases and the remethylation of homocysteine to methionine.
75 nthesis of purines, thymidylate, and for the remethylation of homocysteine to methionine.
76 synthase (MS) catalyzes the folate-dependent remethylation of homocysteine to methionine.
77 hesis of purines and thymidylate and for the remethylation of homocysteine to methionine.
78 rahydrofolate is a major methyl donor in the remethylation of homocysteine to methionine.
79 s in humans, betaine, a methyl donor for the remethylation of homocysteine, may be a therapeutic agen
80 sm is the vitamin B-12- and folate-dependent remethylation of homocysteine, which depends on methioni
81 ction of N(5)-methyltetrahydrofolate and the remethylation of l-homocysteine to form l-methionine sho
82 utilizes an unusual mechanism for control of remethylation of newly replicated DNA.
83                                              Remethylation of tHcy may be more dependent on the betai
84                 This pattern is transient as remethylation of the ALF promoter in haploid germ cell D
85                            Demethylation and remethylation of the CpG island within the LIFR promoter
86 on from myeloma to plasma cell leukemia with remethylation of the genome, particularly of genes invol
87 tivating histone marks, and is important for remethylation of the locus.
88 cytidine followed by zebularine hindered the remethylation of the p16 5' region and gene resilencing,
89  but not with the rate of cell division, and remethylation of the p16 exon 2 CpG island occurred at a
90                              The kinetics of remethylation of the p16 exon 2, PAX-6 exon 5, c-ABL exo
91 and then compared the timing and kinetics of remethylation of the p16 gene locus under conditions of
92 netics of mRNA induction, demethylation, and remethylation of the p16 promoter and second-exon CpG is
93    Furthermore, we provide evidence that the remethylation of the p16 promoter CpG island in T24 cell
94 is is susceptible to TDCPP-induced delays in remethylation of the zygotic genome, a mechanism that ma
95                                              Remethylation of these sequences was also compromised in
96 e no significant differences in homocysteine remethylation or methionine transsulfuration and transme
97 ignificantly alter the rates of homocysteine remethylation or synthesis in healthy young adults in th
98 did not affect in vivo rates of homocysteine remethylation or the appearance of homocysteine and cyst
99 ion or activity of the target enzymes of the remethylation pathway are involved in NTD risk.
100 tivity of MS in liver point out the impaired remethylation pathway as hallmarks associated with NTD r
101 t the thymidylate synthesis and homocysteine remethylation pathways compete for a limiting pool of me
102 ated disturbances in the transsulfuration or remethylation pathways for homocysteine metabolism.
103 ate between the thymidylate and homocysteine remethylation pathways.
104 functional memory cells coincided with Pdcd1 remethylation, providing an adapted program for regulati
105                We find that post-replication remethylation rate constants span approximately two orde
106                        Whereas adjustment of remethylation rates for fat-free mass tended to attenuat
107      However, women had significantly higher remethylation rates than did men (P < 0.005) and a tende
108  was detected within 1 wk, but near-complete remethylation required 6 mo.
109  primary spermatocytes followed by selective remethylation, resulting in a spermatids/sperm-specific
110                                              Remethylation studies after drug-induced hypomethylation
111         In vivo fluxes of total homocysteine remethylation, the remethylation of homocysteine from se
112  used as a source of methyl for homocysteine remethylation to methionine, but choline synthesis requi
113  stands at the intersection of two pathways: remethylation to methionine, which requires folate and v
114 yltetrahydrofolate, utilized in homocysteine remethylation to methionine.
115 ine; the rates of homocysteine synthesis and remethylation (total and vitamin B-6-dependent); and the
116                                              Remethylation was accurate, dynamic, and highly ordered,
117                                      Partial remethylation was detected within 1 wk, but near-complet
118 ulates the early embryonic demethylation and remethylation waves.
119                           Variable levels of remethylation were detected in CpG poor regions of DNA,
120 ression is associated with partial Foxp3 DNA remethylation, which may be due to an increased activity
121 e propose that [(2)H(2)]methionine occurs by remethylation with [(2)H(2)]methyl groups (as 5-methylte

 
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