ライフサイエンスコーパス: renal cortex

1 (tubulo-interstitial) renal lesions in total renal cortex.

2 led DOTA-biotin localized selectively in the renal cortex.

3 are identified in the outer medulla and the renal cortex.

4 mouse renal inner medulla but much lower in renal cortex.

5 d only NADH-dependent O(2)(.-) generation by renal cortex.

6 epithelial cells cultured from normal human renal cortex.

7 higher values in regions composed largely of renal cortex.

8 lack of terminal branches in the midventral renal cortex.

9 ding protein from a cytosolic extract of rat renal cortex.

10 and degradation of nitric oxide (NO) in the renal cortex.

11 hione and accumulation of malondialdehyde in renal cortex.

12 ased AQP2 protein expression particularly in renal cortex.

13 tion of inducible NO synthase protein in the renal cortex.

14 is indicated that PTP-1D is expressed in the renal cortex.

15 fluorescence was used to detect gamma in rat renal cortex.

16 ted in marked homogeneous enhancement of the renal cortex.

17 ase-2 (COX-2) at restricted sites in the rat renal cortex.

18 -inducible factor-1alpha upregulation in the renal cortex.

19 be located to the tubular epithelium of the renal cortex.

20 nal outer stripe of the outer medulla and/or renal cortex.

21 d to proximal tubule epithelial cells in the renal cortex.

22 ding type IV collagen and fibronectin in the renal cortex.

23 l membrane vesicles isolated from the rabbit renal cortex.

24 n apical membrane vesicles isolated from rat renal cortex.

25 nephrons to cortical collecting ducts in the renal cortex.

26 accumulation of type III collagen within the renal cortex.

27 fusion confirmed the presence of MSCs in the renal cortex.

28 ulated AKT by 33% (P < 0.05) respectively in renal cortex.

29 in the tubulointerstitial compartment of the renal cortex.

30 days, and MSCs retention was demonstrated in renal cortex.

31 terstitial fibroblast-like cells in the deep renal cortex.

32 the PO2 (partial pressure of oxygen) in the renal cortex.

33 odialysis catheter was placed in the lateral renal cortex.

34 sured levels of HO-1 mRNA and protein in the renal cortex.

35 hment of their corresponding antigens in the renal cortex.

36 lateral membranes of proximal tubules in the renal cortex.

37 fewer CD3(+) and proliferating cells in the renal cortex.

38 m normal renal cortex and abundant in Alport renal cortex.

39 specific binding in the pancreas than in the renal cortex.

40 had evidence for increased apoptosis in the renal cortex.

41 me cytokines were increased significantly in renal cortex.

42 FC was minimally elevated in renal cortex (0 to 15%), the majority apparently localiz

43 dulla, 1702 msec +/- 205 and 60 msec +/- 21; renal cortex, 1314 msec +/- 77 and 47 msec +/- 10; splee

44 ammonium sulfate-mediated injury; and 3) in renal cortex 18 hours after induction of glycerol-induce

45 m (93%), cerebrum (183%), brain stem (177%), renal cortex (53%), ileal mucosa (69%), gastric antral m

46 f angiotensin converting enzyme (ACE) in the renal cortex (942 +/- 130 versus 464 +/- 60 arbitrary pi

47 wise, had a strong positive correlation with renal cortex ACE protein expression (170-kDa band) (r =

48 In renal cortex, ACE2 activity was increased in both models

49 icromol Pi/mg protein per h while decreasing renal cortex activity from 10.9 +/- 0.9 to 6.5 +/- 0.7.

50 analyzed, we identified 6 as enriched in the renal cortex and 11 in the renal medulla.

51 entified 58 proteins as more abundant in the renal cortex and 72 in the renal medulla.

52 sent in the vascular endothelium from normal renal cortex and abundant in Alport renal cortex.

53 educes the partial pressure of oxygen in the renal cortex and activates the renin-angiotensin-aldoste

54 ediates initial trafficking to the brain and renal cortex and contributes to fungal persistence in th

55 Renal cortex and cultured GEnC were examined by microsco

56 and NOS1 phosphorylation at Ser1417 in mouse renal cortex and cultured human kidney tissue.

57 Human renal cortex and heart cDNA libraries were screened for

58 Biopsies were obtained from renal cortex and hybridized to Affymetrix HG-U133A GeneC

59 ormed in four regions of interest within the renal cortex and in three regions within the medulla.

60 control and NFAT5 deficient mice as well as renal cortex and inner medulla from principal cell speci

61 rt the hypothesis that CAA is accumulated in renal cortex and is responsible for nephrotoxicity.

62 Both renal cortex and liver have similar developmental patter

63 m and mean counts of the lesion and adjacent renal cortex and maximum and mean lesion Hounsfield unit

64 Renal cortex and medulla AA metabolism were not signific

65 he expression of these genes was analyzed in renal cortex and medulla after ischemic-reperfusion inju

66 FcepsilonRI(+) cells were found in the human renal cortex and medulla and provide targets for HLA-spe

67 H(2) O(2) production was reduced in both the renal cortex and medulla in SS(Nox4-/-) rats fed an HS d

68 he corticomedullary osmotic gradient between renal cortex and medulla induces a specific spatial gene

69 ide wasting, we analyzed their expression in renal cortex and medulla of animals placed on KD diet.

70 The non-heme iron content both in the renal cortex and medulla of Heph/Cp KO mice was signific

71 Mean ADCs of renal cortex and medulla were significantly higher in gr

72 ver lobe, gallbladder, pancreas, spleen, and renal cortex and medulla).

73 ulmonary and renal artery flow probes in the renal cortex and medulla, combination fiber-optic probes

74 ons at 3.0 T (left liver lobe, pancreas, and renal cortex and medulla, P < .008), intervendor differe

75 cCD8alpha and in CD11cCD8alpha DC within the renal cortex and medulla.

76 eys with regions of interest assigned to the renal cortex and medulla.

77 ed a major 30-kDa band in membranes from rat renal cortex and medulla.

78 he expression of more than 3000 genes in the renal cortex and more than 5000 genes in the inner medul

79 Src, and tyrosine phosphorylation of ROMK in renal cortex and outer medulla in wild-type (WT) mice.

80 Immunoprecipitation of tissue lysates from renal cortex and outer medulla or 293T cells transfected

81 mmunoprecipitation of proteins obtained from renal cortex and outer medulla with ROMK antibody reveal

82 ylation of c-Jun, a transcription factor, in renal cortex and outer medulla.

83 the tyrosine phosphorylation of ROMK in the renal cortex and outer medulla.

84 s abundant CYP2J5 mRNA within tubules of the renal cortex and outer medulla.

85 sociation between c-Src and RPTPalpha in the renal cortex and outer medulla.

86 hosphorylation of c-JUN N-terminus kinase in renal cortex and outer medulla.

87 On the basis of renographic findings, renal cortex and renal medulla enhancement curves and no

88 f the liver and relatively low (<10%) in the renal cortex and renal medulla.

89 Although found at high level in liver, renal cortex and small intestine, fructokinase activity

90 tes a gradient of oxygen tension between the renal cortex and the papillary tip that results in a sta

91 l profile of microRNA expression between the renal cortex and the renal medulla and greatly expands t

92 ed by Rho-kinase, were increased in both the renal cortex and the renal medulla of endotoxemic rats.

93 Time-activity curves were derived from the renal cortex and were analyzed by the Gjedde-Patlak plot

94 p47(phox), Nox2, and Nox4 mRNA levels in the renal cortex and were unchanged in diabetic PKC-beta(-/-

95 xylases was localized in renal microvessels, renal cortex, and a striated muscle microvascular bed (c

96 osis with hemorrhage of both the adrenal and renal cortex, and an intense influx of neutrophils into

97 esistive index and reduced blood flow in the renal cortex, and decreased glomerular filtration rate.

98 ersally labeled glucose.Perfusate, end-point renal cortex, and medulla samples underwent metabolomic

99 s of the liver, and in the pancreas, spleen, renal cortex, and renal medulla.

100 diac tissue, ACE2 activity was lower than in renal cortex, and there were no significant differences

101 dopamine-derived radioactivity in the heart, renal cortex, and thyroid gland but not in the liver, sp

102 alizes sympathetic innervation in the heart, renal cortex, and thyroid gland.

103 thelial cells of the liver, lung, intestine, renal cortex, and urinary tract.

104 hat constitute the basement membranes in the renal cortex are constantly renewed in an ordered fashio

105 ule cells within the nephron segments of the renal cortex are mitochondrially dense with high oxygen

106 isplaceable uptake, was calculated using the renal cortex as a reference tissue devoid of specific VM

107 BP(ND)) was estimated voxelwise by using the renal cortex as reference tissue.

108 PLA2 activity was increased in the renal cortex as well as in the renal medulla.

109 lant, which revealed multiple lesions in the renal cortex as well as liver and spleen.

110 the proximal convoluted tubules of the outer renal cortex, assessed by Western blotting and immunohis

111 toblastoma lesions, non-tumor-bearing liver, renal cortex, blood pool in the left ventricle, and gast

112 165-kDa band in membrane fractions from the renal cortex but not from the renal medulla.

113 restricted to stromal cells in the embryonic renal cortex, but it mediates its effects on the adjacen

114 teral ureteric obstruction-operated fibrotic renal cortex, characterized by abundant CLEVER-1-positiv

115 ays indicated that cytosolic extracts of rat renal cortex contain a protein that binds to the R-2 and

116 hibitor of matrix degradation, PAI-1, in the renal cortex, contributing to significant reversal of me

117 ACE2 activity in renal cortex correlated positively with ACE2 protein in

118 We have previously shown that in renal cortex, COX-2 expression is localized to macula de

119 We generated similar datasets from renal cortex cultures, to compare with those of the glom

120 We have previously shown that in rat renal cortex, cyclooxygenase-2 (COX-2) expression is loc

121 Both renal neoplasms and normal renal cortex demonstrated significantly greater enhancem

122 njury and formation of giant vacuoles in the renal cortex, die from renal failure, a phenotype that r

123 hat are rapidly filtered in the kidneys, the renal cortex dose is approximately one-half of that pred

124 ults demonstrate a desensitization of sGC in renal cortex during endotoxemia, which may contribute to

125 hase subtypes increased significantly in the renal cortex during septic acute kidney injury but tende

126 dative stress promotes NO degradation in the renal cortex during the early stage of diabetes mellitus

127 itionally, FC/CE levels were measured in rat renal cortex either 10 days after CSA or tacrolimus ther

128 Using Young's modulus, the renal cortex elasticity of CKD patients was shown to be

129 inal vesicles, pituitary, thyroid, pancreas, renal cortex, enteric epithelium, muscles, myocardium an

130 For the adult, the absorbed dose to the renal cortex for (90)Y-labeled compounds retained within

131 nterstitial monocytes that accumulate in the renal cortex from Alport mice are immunopositive for int

132 NADPH-dependent superoxide production in the renal cortex from Asm(+/+) mice compared with that from

133 ACE2 mRNA levels in renal cortex from db/db and STZ-induced diabetic mice, b

134 beta1 subunits, is increased in glomeruli or renal cortex from diabetic animals or in mesangial cells

135 We conclude that in renal cortex from diabetic mice, ACE2 expression is incr

136 levels or their distribution were evident in renal cortex from diabetic rats.

137 parable early neutrophil infiltration in the renal cortex from P2X(7)(+/+) and P2X(7)(-/-) mice.

138 y revealed an approximately 70-kD protein in renal cortex from sham rats, the nitrotyrosine content o

139 erns for the gluconeogenic enzymes, although renal cortex generally shows greater activity than liver

140 0.5 +/- 3% of the introduced capsules in the renal cortex glomeruli.

141 of 20.5 3% of the introduced capsules in the renal cortex glomeruli.

142 a global downregulation of microRNAs in the renal cortex; however, these animals were remarkably res

143 Renal cortex immunoblot analysis using anti-peptide anti

144 l moving blood volume estimates of the right renal cortex in a volunteer when simulating different bo

145 pressed in the differentiation domain of the renal cortex in an overlapping manner with the vasopress

146 inant fibroblast chemoattractant produced by renal cortex in anti-GBM disease.

147 situ hybridization localized the mRNA to the renal cortex in both mice and confirmed equal message le

148 bular glomeruli by electrocoagulation of the renal cortex in mice.

149 eeks of age and vacuolization throughout the renal cortex in older mice.

150 endothelin-1 (ET-1) and ET receptors in the renal cortex in short-term diabetes.

151 eceptor, and signaling levels throughout the renal cortex in this animal model of type 2 diabetes.

152 srupts mitochondrial metabolic fluxes in the renal cortex in vivo.

153 onstrated in tubular epithelial cells in the renal cortex, in spermatogenic cells in the testis, and

154 fibronectin protein was markedly reduced in renal cortex including glomeruli of AS-treated diabetic

155 reases CD3(+) cells and proliferation in the renal cortex independent of changes in BP, but changes i

156 tored interleukin-6 to control levels in the renal cortex, indicating the protective effects on the p

157 FC and CE levels in renal cortex, isolated glomeruli, and proximal tubule se

158 xpression and phosphorylation of both hsp in renal cortex, isolated glomeruli, outer medulla, and inn

159 ADC and eADC values in the renal cortex measured at b1000 present a relationship wi

160 he region of interest (ROI) including normal renal cortex measurements.

161 d laser Doppler oxygen-sensing probes in the renal cortex, medulla, and within a bladder catheter in

162 ium, lungs, hepatic parenchyma, jejunum, and renal cortex/medulla) and potentially adverse (hepatobil

163 he aorta, inferior vena cava, liver, spleen, renal cortex, muscle, and fat.

164 n and diuresis associated with inhibition of renal cortex Na,K-ATPase activity and redistribution of

165 ogenous lithium clearance, 33% inhibition of renal cortex Na,K-ATPase activity, and redistribution of

166 A/I total NO production, with no decrease in renal cortex NOS activity despite a decrease in remnant

167 Core-needle biopsy of the renal cortex obtained during surgical implantation of th

168 AL-DNA adducts were present in the renal cortex of 83% of patients with A:T to T:A mutation

169 t-off point, 30,000 D) were implanted in the renal cortex of anesthetized rats and were perfused at 2

170 respectively, than those of NOS1alpha in the renal cortex of C57BL/6 mice.

171 xcept for the tumor of case 1 (5.9%) and the renal cortex of case 2 (5.6%).

172 s in vivo decreases expression of p53 in the renal cortex of control and streptozotocin-injected diab

173 ence for chronic NF-kappaB activation in the renal cortex of db/db mice and suggest a novel, diabetes

174 Status of GSK3beta was examined in vivo in renal cortex of db/db mice with type 2 diabetes at 2 wee

175 microRNA-192 (miR-192) are increased in the renal cortex of diabetic mice, and this is associated wi

176 and p38/JNK activation in the podocytes and renal cortex of diabetic mice.

177 P-1 activation in brain pericytes and in the renal cortex of diabetic mice.

178 and noncanonical activation pathways in the renal cortex of diabetic mice.

179 Finally, we show in the renal cortex of diabetic rats that increased TGFbeta was

180 In the renal cortex of diabetic rats, the increase in Akt phosp

181 cubated in high K(+) media ex vivo or in the renal cortex of mice fed a high K(+) diet for 4 days, th

182 WNK1 but increased expression of WNK4 in the renal cortex of mice.

183 ug resistance gene 1 was up-regulated in the renal cortex of mutant mice.

184 a low basal expression of RANTES mRNA in the renal cortex of nephrotic rats that did not differ from

185 DeltaR1 was increased in the renal cortex of NTN mice and in both the cortex and the

186 Analysis of DUSP4 expression in the renal cortex of patients with diabetes revealed that dec

187 istolactam (AL) and dG-AL DNA adducts in the renal cortex of patients with EN but not in patients wit

188 te mRNA expression of IP-10 and MCP-1 in the renal cortex of rats 6 to 8 days after the administratio

189 ed a large increase in TSC expression in the renal cortex of rats on a low-NaCl diet (207 +/- 21% of

190 Extracts prepared from the renal cortex of rats that were made acutely acidotic als

191 of brush border and lipid vacuolation in the renal cortex of Slc7a7Lbu/Lbu mice, which combined with

192 as malondialdehyde content was lower, in the renal cortex of SOD-Tg-db/db compared with NTg-db/db mic

193 erone, BiP, was observed in the cells of the renal cortex of the male transgenic mice, suggesting ER

194 1 protein was significantly increased in the renal cortex of these animals.

195 nd C5b-9 deposition were demonstrated in the renal cortex of two TTP patients, by immunofluorescence

196 synthase protein levels were upregulated in renal cortex of uni-x sheep (P<0.05).

197 the partial pressure of oxygen (PO2) in the renal cortex of unrestrained rats, which might in turn c

198 Diabetes increased PKC activity in renal cortex of wild-type mice and was significantly red

199 paralleled by a decrease in its abundance in renal cortex on immunoblot.

200 y, SNR measurements in the liver, aorta, and renal cortex on pre- and postcontrast images were signif

201 mal ablation zone, perirenal fat, and normal renal cortex on the MR images.

202 in levels were not increased in either mouse renal cortex or medulla after either 2 or 7 days of oral

203 radioactivity in the heart (P < 0.0001) and renal cortex (P = 0.02 and P = 0.005, respectively).

204 Increased neutrophils were observed in the renal cortex, particularly within the glomeruli where a

205 1.7% was observed between the sphere and the renal cortex phantoms.

206 d cholesteryl ester (CE) accumulation within renal cortex/proximal tubules.

207 Renal cortex R2* values correlated with gadolinium conce

208 following tissues of Brown Norway rats: the renal cortex, renal outer medulla, liver, cardiac left v

209 the site of erythropoietin production in the renal cortex, showing the greatest accumulation of renox

210 T1 and T2 SI ratio (ratio of tumor to renal cortex SI on T1- and T2-weighted images, respectiv

211 bserved transgene expression confined to the renal cortex (specifically proximal and distal tubules)

212 hat transfected shRNAs were expressed in the renal cortex starting on day 3 and continued for 24 days

213 In participants with CKD, renal cortex SWS values showed a positive association wi

214 Age-adjusted renal cortex SWS was lower in participants with glomerul

215 ociated with liver, pancreas, myocardial and renal cortex T1 time, respectively.

216 Gsalpha expression in the renal cortex (the site of PTH action) is markedly reduce

217 reas interior structures are defined for the renal cortex, the medullary pyramids with papillae (2 ve

218 of COMT to nuclei of epithelial cells in the renal cortex, the site of CE biosynthesis and where the

219 ygen tension and that when injected into the renal cortex, they incorporated into the renal parenchym

220 ameters offer quality ADC measurement in the renal cortex using b1000.

221 ELV], smart region of interest [ROI] volume, renal cortex volume [RCV]) were performed in 101 living

222 Overall renal cortex volumetry seems to be the most accurate tec

223 average difference between haematoma and the renal cortex was 5 dB.

224 Renal cortex was analyzed after sorbitol density gradien

225 days with 50 mg/kg, the level of CAA in the renal cortex was approximately 15 micromol/L.

226 2, Tie2, and VEGF expression in normal human renal cortex was examined with immunofluorescence and im

227 Accumulated activity measured in the renal cortex was significantly lower for the small-pepti

228 , and alpha1-(IV) collagen observed in OVE26 renal cortex was significantly reduced in OVE26 RKO kidn

229 RSOR expression, normally confined to the renal cortex, was markedly increased and extended into t

230 A, VEGFR2, and nephrin protein expression in renal cortex were determined by Western immunoblotting;

231 Consequently, apoptotic cells in the renal cortex were significantly decreased by 47%, wherea

232 est measurements within tumor and uninvolved renal cortex were used to calculate percentage signal in

233 These lesions concentrate in the renal cortex, where they serve as a sensitive and specif

234 wn to induce net vasoconstriction within the renal cortex while increasing medullary blood flow and o

235 , we identified over 4,000 proteins from the renal cortex with a majority of them contained only (15)

236 ate distinct binding of 11C-L-159,884 in the renal cortex with a specific binding component suitable

237 of HB-EGF on renal function, we infused the renal cortex with active rHB-EGF, prepared from transfec

238 ghest mean absorbed dose was received by the renal cortex, with 1.9 mGy per MBq of (86)Y- 6: CONCLUSI