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1 nking water) or renin/angiotensin-dependent (renovascular 2-kidney-1-clip).
2                                     Although renovascular abnormalities were mild, their detection in
3                                              Renovascular abnormalities were noted in the dictated re
4 e exhibited a strong hypertensive phenotype, renovascular abnormalities, persistent constriction of t
5 es and their relationship to renal function, renovascular anatomy, and BP are delineated.
6                                              Renovascular and adrenal responses to infused angiotensi
7                                  The blunted renovascular and functional responses to acetylcholine i
8 tetraenoic acid (20-HETE), a metabolite with renovascular and tubular functions.
9 pendent factors protect against vascular and renovascular calcification, and vitamin K antagonists ma
10 ctable in intrinsic glomerular, tubular, and renovascular cells in all analyzed cases.
11 ctable in intrinsic glomerular, tubular, and renovascular cells in native kidneys.
12 mage as a result of warm ischemia, affecting renovascular circulating volume.
13 d dilution techniques may be used to measure renovascular circulating volumes during hypothermic mach
14  dilution is a reproducible method to assess renovascular circulating volumes in machine-perfused kid
15                                              Renovascular circulating volumes of machine-perfused por
16                                              Renovascular circulating volumes significantly increased
17 ility of ultrasound dilution measurements of renovascular circulating volumes was good (mean coeffici
18 mbolic events resulting in end-organ damage, renovascular complications, or hypertensive crisis at 1
19               One patient had CT evidence of renovascular compromise.
20 tion and realignment leading to fibrosis and renovascular constriction in diabetes.
21 ng II)-induced BP, dipsogenic responses, and renovascular contractility were significantly attenuated
22 hypertension, and H(2)S treatment attenuates renovascular damage.
23 Magnetic-resonance angiography showed severe renovascular disease (>50% renal-artery stenosis or occl
24 enal donors, the arteriographic incidence of renovascular disease (10.9%) was higher than previously
25 egarding the epidemiology of atherosclerotic renovascular disease (ARVD) in dialysis populations are
26                              Atherosclerotic renovascular disease (ARVD) is associated with heart dis
27 the other (n = 60) with a high prevalence of renovascular disease (group II).
28                              Atherosclerotic renovascular disease (RVD) amplifies damage in a stenoti
29                                              Renovascular disease (RVD) induces renal microvascular (
30                              Atherosclerotic renovascular disease (RVD) reduces renal blood flow (RBF
31                          Despite advances in renovascular disease (RVD) research, gaps remain between
32 ury and response to therapy in patients with renovascular disease (RVD).
33 ntly used to treat renal artery stenosis and renovascular disease (RVD); however, renal function is r
34 s; range, 27-62 years) and six patients with renovascular disease (three women; average age, 48 years
35           miR-378h modulated the capacity of renovascular disease adipose tissue-derived mesenchymal
36                         We hypothesized that renovascular disease alters the human MSC transcriptome
37             We investigated the frequency of renovascular disease among elderly people with heart fai
38 upport its routine use for the evaluation of renovascular disease among patients suspected of having
39 ing renal function and size in patients with renovascular disease and chronic renal insufficiency.
40 nction and size in patients with obstructive renovascular disease and chronic renal insufficiency.
41  subcutaneous abdominal fat of patients with renovascular disease and healthy volunteers (n=3 each),
42 may limit progressive CKD in atherosclerotic renovascular disease and may apply to other conditions i
43 tudies indicate that whereas atherosclerotic renovascular disease can accelerate both systemic hypert
44                                  Progressive renovascular disease during medical therapy can produce
45                                Patients with renovascular disease had worse renal function (mean crea
46  investigation and appropriate management of renovascular disease has remained a controversial topic.
47                           Many children with renovascular disease have abnormalities of other blood v
48                                              Renovascular disease impaired the reparative capacity of
49                                              Renovascular disease impairs the capacity of human adipo
50                                              Renovascular disease is a frequent cause of severe hyper
51                                              Renovascular disease is an uncommon but important cause
52                             Chronic azotemic renovascular disease is common in patients with atherosc
53                       Treating patients with renovascular disease is complex, particularly as imaging
54                    The anatomic diagnosis of renovascular disease is increasing in frequency due to t
55                                              Renovascular disease leads to renal ischemia, hypertensi
56 reflected in the proportion of patients with renovascular disease listed as cause of ESRD on the Medi
57                              Atherosclerotic renovascular disease may augment deterioration of renal
58                         Genes upregulated in renovascular disease MSCs versus healthy volunteer MSCs
59              Healthy volunteer MSCs (but not renovascular disease MSCs) decreased BP, improved serum
60            Some elderly patients with occult renovascular disease on ACE inhibitors will be at risk o
61                      The effect of detecting renovascular disease on donor selection was determined i
62 cular disease, and the effect of identifying renovascular disease on subsequent donor surgery was asc
63 ry stenosis mice administered with MSCs from renovascular disease pretreated with miR-378h mimic (n=5
64             Kidney injury in atherosclerotic renovascular disease reflects complex interactions among
65  of ESRD, rates of end-stage atherosclerotic renovascular disease seem to be on the rise in older pat
66 , and were more likely than patients without renovascular disease to have peripheral arterial disease
67 Cs from healthy volunteers and in those with renovascular disease were also characterized by mRNA/mic
68                           Most children with renovascular disease will need interventional or surgica
69 rovascular diseases, central aortic disease, renovascular disease, and peripheral artery disease in t
70 yperuricaemia itself causes hypertension and renovascular disease, and that lowering of serum urate m
71 gh technical success rate in atherosclerotic renovascular disease, but little is known about the clin
72 the other (n = 60) with a high prevalence of renovascular disease, defined with angiograms.
73 iency and global obstructive atherosclerotic renovascular disease, renal artery stenting improves or
74   We used captopril renography to screen for renovascular disease.
75 ficity of 94.3% (67.6-97.3) for detection of renovascular disease.
76 e placed in 32 patients with atherosclerotic renovascular disease.
77 rculation and alleviate renal dysfunction in renovascular disease.
78 oninvasive intervention in the management of renovascular disease.
79 R-26a might be a novel therapeutic target in renovascular disease.
80 c targets for recovery of kidney function in renovascular disease.
81 ic intervention for preserving the kidney in renovascular disease.
82 nal microcirculation in chronic experimental renovascular disease.
83 sport on tissue oxygenation in subjects with renovascular disease.
84 d help identify patients suspected of having renovascular disease.
85  pathobiology of chronic kidney diseases and renovascular disorders by expanding the understanding of
86 igger CKD in SCD through the gradual loss of renovascular EPCR.
87 acellular matrix (ECM) metabolism and reduce renovascular fibrosis.
88 n hypertrophied ventricles from animals with renovascular hypertension (1.6-fold, P < 0.05) and aorti
89 en during arteriographic study for suspected renovascular hypertension (42%).
90 truction and 48 patients suspected of having renovascular hypertension (RVH) were randomly selected f
91  myocardial microvascular structure in swine renovascular hypertension (RVH) would be improved by sim
92 al angiograms obtained to evaluate suspected renovascular hypertension and (b) angiographically docum
93 lar tissue from neonatal and adult rats with renovascular hypertension and aortic banding, whereas ba
94 odel an absence of HO-1 leads to more severe renovascular hypertension and cardiac hypertrophy.
95 ury, chronic kidney disease, renal fibrosis, renovascular hypertension and kidney cancer.
96 ow-up (range, 1-44 months), two patients had renovascular hypertension and none had recurrence of hor
97 es of myocardial and cerebral infarction and renovascular hypertension by 9% (3% to 16%), 13% (7% to
98                  These results indicate that renovascular hypertension causes specific reductions in
99 all accuracy of baseline parenchymal MTT for renovascular hypertension detection ranged from 54% to 5
100                             On the contrary, renovascular hypertension developed to the same extent (
101 es were used to examine the cost efficacy of renovascular hypertension diagnosis and treatment.
102                          We show that CSD in renovascular hypertension halts further increases in blo
103                       The pathophysiology of renovascular hypertension has been linked to other intra
104                Most children presenting with renovascular hypertension have few if any symptoms, but
105                           We studied chronic renovascular hypertension in mice deficient in the induc
106                                Screening for renovascular hypertension is not cost-effective at a pre
107                                              Renovascular hypertension is the most common cause of se
108 llular hyperplasia or remodeling, whereas in renovascular hypertension there is hypertrophy of vascul
109                                              Renovascular hypertension was produced by placing a silv
110                                   Effects of renovascular hypertension were then determined by using
111 s (limb claudication, dizziness, angina, and renovascular hypertension), and is characterized patholo
112 esults indicate a possible role for GPR91 in renovascular hypertension, a disease closely linked to a
113 imately 5% of all hypertensive patients have renovascular hypertension, although its true incidence i
114 hat not only can renal artery stenosis cause renovascular hypertension, but it can also lead to progr
115 s simplified the management of patients with renovascular hypertension, but long-term results are not
116 cardial infarction, cerebral infarction, and renovascular hypertension, consistent with either shared
117  medicine: microalbuminuria, renal function, renovascular hypertension, dialysis (hemodialysis and co
118 week low-energy shockwave regimen attenuated renovascular hypertension, normalized stenotic kidney mi
119  mice would exhibit an early exacerbation of renovascular hypertension, NSE-AT(1a) and nontransgenic
120        Traditionally known in the context of renovascular hypertension, recent advances in knowledge
121 cardial infarction, cerebral infarction, and renovascular hypertension, respectively, as women with n
122 rtery stenting is an effective treatment for renovascular hypertension, with a low angiographic reste
123 r disease among patients suspected of having renovascular hypertension.
124 RABG were equally efficacious for control of renovascular hypertension.
125 ion, the swine model closely resembles human renovascular hypertension.
126 modynamics and segmental tubular function in renovascular hypertension.
127 e past 10 yr as a useful diagnostic test for renovascular hypertension.
128 roximal tubular cells, and it may ameliorate renovascular hypertension.
129 ncerning the identification and treatment of renovascular hypertension.
130 vely to a cohort undergoing intervention for renovascular hypertension.
131 potential therapeutic target for controlling renovascular hypertension.
132 al chemoreflex sensitization is a feature of renovascular hypertension.
133 eflex may be a viable therapeutic target for renovascular hypertension.
134 cardial infarction, cerebral infarction, and renovascular hypertension.
135  cerebral infarctions, and 1269 instances of renovascular hypertension.
136 cardial infarction, cerebral infarction, and renovascular hypertension.
137 rlying aberrant central neuronal function in renovascular hypertension.
138 thoexcitation and elevated blood pressure in renovascular hypertension.
139 rtery angioplasty in patients with suspected renovascular hypertension.
140 m, have improved the level of BP control for renovascular hypertension.
141  utility of a noninvasive test for detecting renovascular hypertension.
142  and collagen deposition in the rat heart in renovascular hypertension.
143 cal indications of suspected obstruction and renovascular hypertension; part 2 also summarizes the st
144 NCs obtained from sham, but not in MNCs from renovascular hypertensive (RVH) rats.
145  similar inward currents in MNCs of sham and renovascular hypertensive (RVH) rats.
146 iology and real-time PCR in MNCs in sham and renovascular hypertensive (RVH) rats.
147 magnocellular neurosecretory cells (MNCs) in renovascular hypertensive (RVH) rats.
148 imbalanced excitatory/inhibitory function in renovascular hypertensive rats (RVH).
149  subunit genes was examined in ventricles of renovascular hypertensive rats.
150 e mice but only in the clipped kidney of the renovascular knockout mice.
151 ographic techniques have been used to detect renovascular lesions, but none has yet demonstrated cons
152 ilar changes in GluR1 density are found in a renovascular model of hypertension, the DOCA-salt rat, a
153 lnerability to ventricular fibrillation in a renovascular model of LVH.
154 ency (creatinine >1.5 mg. dL(-1)) and global renovascular obstruction (bilateral renal artery stenosi
155 e performed in selected patients for primary renovascular pathology.
156  Here, we investigated the role of increased renovascular rarefaction and the resulting substantial l
157 this study, we hypothesized that HHcy causes renovascular remodeling by DNA hypermethylation, leading
158 n selected patients slows the progression of renovascular renal failure and may delay the need for re
159 e and after stent insertion in patients with renovascular renal failure.
160                             The responses of renovascular resistance and arterial pressure to norepin
161 etween plasma prorenin concentration and the renovascular response to captopril in diabetes supports
162  correlation between plasma prorenin and the renovascular response to captopril in patients with diab
163 r events and type 2 diabetes; (4) attenuated renovascular responses to angiotensin II; (5) improved c
164 ubjects, OSA subjects demonstrated decreased renovascular sensitivity (ERPF, -153 +/- 79 vs. -283 +/-
165 hyma resulted in similar generation of human renovascular units in vivo.
166  whether this would result in generation of "renovascular units" comprising both vessels and tubules
167  Future studies should evaluate the value of renovascular volume in pretransplantation kidney viabili

 
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