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1  cerebral infarctions, and 1269 instances of renovascular hypertension.
2 cardial infarction, cerebral infarction, and renovascular hypertension.
3 rlying aberrant central neuronal function in renovascular hypertension.
4 thoexcitation and elevated blood pressure in renovascular hypertension.
5 roximal tubular cells, and it may ameliorate renovascular hypertension.
6 rtery angioplasty in patients with suspected renovascular hypertension.
7 m, have improved the level of BP control for renovascular hypertension.
8  utility of a noninvasive test for detecting renovascular hypertension.
9  and collagen deposition in the rat heart in renovascular hypertension.
10 r disease among patients suspected of having renovascular hypertension.
11 RABG were equally efficacious for control of renovascular hypertension.
12 ion, the swine model closely resembles human renovascular hypertension.
13 modynamics and segmental tubular function in renovascular hypertension.
14 e past 10 yr as a useful diagnostic test for renovascular hypertension.
15 ncerning the identification and treatment of renovascular hypertension.
16 potential therapeutic target for controlling renovascular hypertension.
17 vely to a cohort undergoing intervention for renovascular hypertension.
18 al chemoreflex sensitization is a feature of renovascular hypertension.
19 eflex may be a viable therapeutic target for renovascular hypertension.
20 cardial infarction, cerebral infarction, and renovascular hypertension.
21 n hypertrophied ventricles from animals with renovascular hypertension (1.6-fold, P < 0.05) and aorti
22 en during arteriographic study for suspected renovascular hypertension (42%).
23 esults indicate a possible role for GPR91 in renovascular hypertension, a disease closely linked to a
24 imately 5% of all hypertensive patients have renovascular hypertension, although its true incidence i
25 al angiograms obtained to evaluate suspected renovascular hypertension and (b) angiographically docum
26 lar tissue from neonatal and adult rats with renovascular hypertension and aortic banding, whereas ba
27 odel an absence of HO-1 leads to more severe renovascular hypertension and cardiac hypertrophy.
28 ury, chronic kidney disease, renal fibrosis, renovascular hypertension and kidney cancer.
29 ow-up (range, 1-44 months), two patients had renovascular hypertension and none had recurrence of hor
30 s (limb claudication, dizziness, angina, and renovascular hypertension), and is characterized patholo
31 hat not only can renal artery stenosis cause renovascular hypertension, but it can also lead to progr
32 s simplified the management of patients with renovascular hypertension, but long-term results are not
33 es of myocardial and cerebral infarction and renovascular hypertension by 9% (3% to 16%), 13% (7% to
34                  These results indicate that renovascular hypertension causes specific reductions in
35 cardial infarction, cerebral infarction, and renovascular hypertension, consistent with either shared
36 all accuracy of baseline parenchymal MTT for renovascular hypertension detection ranged from 54% to 5
37                             On the contrary, renovascular hypertension developed to the same extent (
38 es were used to examine the cost efficacy of renovascular hypertension diagnosis and treatment.
39  medicine: microalbuminuria, renal function, renovascular hypertension, dialysis (hemodialysis and co
40                          We show that CSD in renovascular hypertension halts further increases in blo
41                       The pathophysiology of renovascular hypertension has been linked to other intra
42                Most children presenting with renovascular hypertension have few if any symptoms, but
43                           We studied chronic renovascular hypertension in mice deficient in the induc
44                                Screening for renovascular hypertension is not cost-effective at a pre
45                                              Renovascular hypertension is the most common cause of se
46 week low-energy shockwave regimen attenuated renovascular hypertension, normalized stenotic kidney mi
47  mice would exhibit an early exacerbation of renovascular hypertension, NSE-AT(1a) and nontransgenic
48 cal indications of suspected obstruction and renovascular hypertension; part 2 also summarizes the st
49        Traditionally known in the context of renovascular hypertension, recent advances in knowledge
50 cardial infarction, cerebral infarction, and renovascular hypertension, respectively, as women with n
51 truction and 48 patients suspected of having renovascular hypertension (RVH) were randomly selected f
52  myocardial microvascular structure in swine renovascular hypertension (RVH) would be improved by sim
53 llular hyperplasia or remodeling, whereas in renovascular hypertension there is hypertrophy of vascul
54                                              Renovascular hypertension was produced by placing a silv
55                                   Effects of renovascular hypertension were then determined by using
56 rtery stenting is an effective treatment for renovascular hypertension, with a low angiographic reste