ライフサイエンスコーパス: repressive

1 xp3 expression, whereas the PBAF complex was repressive.

2 encoded by CaGPI2 and CaGPI19, are mutually repressive.

3 new regulator of iron homeostasis limits the repressive action of Fur.

4 eep the brain free of anti-proliferative and repressive action of Hox homeotic genes.

5 onal in vitro analyses demonstrated a strong repressive action of miR-30b on Mkrn3 3' UTR.

6 Repressive action of REVERBalpha in the liver therefore

7 The repressive action of the PRRs on both growth and DTG exp

8 tructure that drives both the activating and repressive actions of glucocorticoids.

9 We show that the repressive activities of exon5-coded variants are likely

10 We show that the repressive activities of the Pumilio RDs depend on speci

11 on assays indicate that the PRRs exert their repressive activity by binding directly to the PIF prote

12 However, ARID1A elicits repressive activity in an enhancer-specific, but forkhea

13 This effect correlates with the repressive activity of CDK4/6 inhibitors on homologous r

14 mutation or domain deletion impairs AML1-ETO-repressive activity.

15 Thus, a competition between Mi-2beta-repressive and activating AP1 complexes may permit rapid

16 m and reveal that alternating recruitment of repressive and activating transcription factors to share

17 local density of H1 controls the balance of repressive and active chromatin domains by promoting gen

18 n of H3K27 acetylation, respectively shaping repressive and active chromatin landscapes.

19 N-auxin interactions allow switching between repressive and de-repressive chromatin states in an inst

20 nction was originally viewed as being solely repressive and irreversible, as observed at the homeotic

21 ion of Lys(537) within the G-region enhances repressive BMAL1-TAD-CRY1 interactions.

22 elements within the IRF8 locus that mediate repressive chromatin conformation resulting in the silen

23 ed to support siRNA biogenesis in a TE-rich, repressive chromatin context is not well understood.

24 n factors activate the hTERT promoter in its repressive chromatin environment is unknown.

25 temporally by synMuv B proteins to establish repressive chromatin in a tissue-specific manner to ensu

26 ageing in male flies, and a general loss of repressive chromatin in aged males away from pericentrom

27 and differentiating into retinal neurons: 1) repressive chromatin in the promoter regions of non-phot

28 (ChIP) assays showed that TBX2 establishes a repressive chromatin mark, specifically H3K9me3, around

29 n cancer associated with broad enrichment of repressive chromatin marks and aberrant DNA hypermethyla

30 are required for piRNA-guided deposition of repressive chromatin marks and transcriptional silencing

31 atin of the host genome and were enriched in repressive chromatin marks.

32 is widely recognized as a transcriptionally repressive chromatin modification but the mechanism of r

33 ctors, or located in broad domains marked by repressive chromatin modifications.

34 latory programs and infer key activating and repressive chromatin regulators of these cell states.

35 nd rescued transcallosal dysconnectivity via repressive chromatin remodeling by the SETDB1 repressor

36 ent transposable element transcript recruits repressive chromatin remodelling activities and the de n

37 Mechanistically, lnc-IL7R mediated a repressive chromatin state of the proinflammatory gene p

38 n that HELLS activity generally results in a repressive chromatin state.

39 ns allow switching between repressive and de-repressive chromatin states in an instantly-reversible m

40 uclease mapping showed that ZL0580 induces a repressive chromatin structure at the HIV LTR.

41 hen quickly inactivated by the assembly of a repressive chromatin structure composed of deacetylated

42 propose that unintegrated HIV-1 DNA adopts a repressive chromatin structure that competes with the tr

43 uripotency-associated transcription factors, repressive chromatin-modifiers such as PRC2 and DNA meth

44 Thus, 'repressive' chromatin marks could be important specifica

45 expression and low endothelium expression of repressive cldn5-related transcription factor foxo1 are

46 -methylated regions are enriched in polycomb repressive complex (EZH2/SUZ12) recognizing regions.

47 e two interacting components of the polycomb repressive complex (L3MBTL2 [L(3)Mbt-Like 2] and MGA [MA

48 Deeper analysis of central Polycomb repressive complex (PRC) 1 and 2 components indicates su

49 Upon HS, various Polycomb Repressive Complex (PRC)1 and PRC2 subunits, including C

50 Polycomb-repressive complex 1 (PRC1) and PRC2 are critical chroma

51 Here we focus on Polycomb Repressive Complex 1 (PRC1) and trace the evolution of c

52 The Polycomb-repressive complex 1 (PRC1) family complexes are central

53 Here we examine the role of Polycomb-repressive complex 1 (PRC1) in shaping 3D genome organiz

54 te that epigenetic reprogramming by Polycomb Repressive Complex 1 (PRC1) promotes an inflammatory tum

55 -cell imaging, we observed that the Polycomb repressive complex 1 (PRC1) protein chromobox 2 (CBX2),

56 ed Sterile Alpha Motif (SAM) in the Polycomb Repressive Complex 1 (PRC1) subunit Polyhomeotic (Ph) ha

57 n of BMI1, an integral component of polycomb-repressive complex 1 (PRC1) that catalyzes H2Aub on chro

58 (BCOR) is a component of a variant Polycomb repressive complex 1 (PRC1) that is essential for normal

59 Chromobox 6 (CBX6) is a subunit of Polycomb Repressive Complex 1 (PRC1) that mediates epigenetic gen

60 atin Protein 1b (SlLHP1b), a tomato Polycomb Repressive Complex 1 (PRC1)-like protein with a ripening

61 e divided into two major complexes: Polycomb repressive complex 1 and 2 (PRC1 and PRC2).

62 rthermore, ChIP-seq analysis of the polycomb repressive complex 1 component RING1B indicated that thi

63 we find that two components of the Polycomb repressive complex 1.1 (PRC1.1), BCL6 corepressor (BCOR)

64 transcriptionally regulated by the polycomb repressive complex 2 (PRC2) and others previously implic

65 Polycomb repressive complex 2 (PRC2) catalyzes methylation of his

66 Polycomb Repressive Complex 2 (PRC2) catalyzes mono-, di-, and tr

67 In addition, LINC00313 bound to polycomb repressive complex 2 (PRC2) complex components, and this

68 Polycomb repressive complex 2 (PRC2) installs and spreads repress

69 The Polycomb repressive complex 2 (PRC2) is a crucial chromatin modif

70 Polycomb repressive complex 2 (PRC2) is a histone methyltransfera

71 Polycomb-repressive complex 2 (PRC2) is a histone methyltransfera

72 The polycomb repressive complex 2 (PRC2) is composed of three core su

73 EZHIP are competitive inhibitors of Polycomb Repressive Complex 2 (PRC2) lysine methyltransferase act

74 Polycomb repressive complex 2 (PRC2) places H3K27me3 at developme

75 Dysregulation of polycomb repressive complex 2 (PRC2) promotes oncogenesis partly

76 Coherently, binding of the Polycomb Repressive Complex 2 (PRC2) protein SUZ12 and deposition

77 Polycomb repressive complex 2 (PRC2) silences expression of devel

78 istent with this view, we show that Polycomb Repressive Complex 2 (PRC2) silencing is genetically req

79 C (FLC) involves distinct phases of Polycomb repressive complex 2 (PRC2) silencing.

80 enzymatic catalytic subunit of the polycomb-repressive complex 2 (PRC2) that can alter gene expressi

81 volutionarily conserved function of polycomb repressive complex 2 (PRC2) that mediates coordinated tr

82 ZH2, the catalytic component of the Polycomb Repressive complex 2 (PRC2) that methylates H3K27, switc

83 we show that lamin B1 recruits the polycomb repressive complex 2 (PRC2) to alter the H3K27me3 landsc

84 are involved in the recruitment of polycomb repressive complex 2 (PRC2) to CpG island (CGI) chromati

85 he stage is set for the activity of Polycomb-repressive complex 2 (PRC2) to maintain these repressed

86 ID2 is a noncatalytic member of the polycomb repressive complex 2 (PRC2) which methylates of histone

87 ent loss-of-function alterations in polycomb-repressive complex 2 (PRC2), a histone-modifying complex

88 d chromatin interaction analyses of Polycomb repressive complex 2 (PRC2), a key inducer of transcript

89 Inhibition of the Polycomb Repressive Complex 2 (PRC2), an H3K27 tri-methyltransfer

90 One of these, Polycomb repressive complex 2 (PRC2), deposits the H3K27me3 mark

91 angiosperm-specific subunit of the polycomb repressive complex 2 (PRC2), is an oxygen (O(2) )-regula

92 ethylation on lysine 27, induced by polycomb repressive complex 2 (PRC2), is responsible for downregu

93 EZH2), the catalytic subunit of the Polycomb Repressive Complex 2 (PRC2), of unknown function.

94 n of EZH2, the enzymatic subunit of polycomb repressive complex 2 (PRC2), often occurs in cancer.

95 (EZH2) is the catalytic subunit of Polycomb Repressive Complex 2 (PRC2), which minimally requires tw

96 heral nerve sheath tumors (MPNSTs), Polycomb repressive complex 2 (PRC2), which plays a crucial role

97 (EZH2) is the catalytic subunit of polycomb repressive complex 2 (PRC2), which silences transcriptio

98 2 (EZH2), the catalytic subunit of polycomb repressive complex 2 (PRC2), with a focus on EZH2 inhibi

99 vely regulated CBX6 expression in a Polycomb Repressive Complex 2 (PRC2)-dependent manner.

100 ntally controlled expression of two Polycomb repressive complex 2 (PRC2)-interacting proteins, Pcl an

101 he genome and in recruitment of the polycomb repressive complex 2 (PRC2).

102 which are the catalytic subunits of Polycomb Repressive Complex 2 (PRC2).

103 rabidopsis INCURVATA11 (ICU11) as a Polycomb Repressive Complex 2 accessory protein.

104 romatin compaction the roles of the Polycomb repressive complex 2 histone methyltransferases EZH1 and

105 tial roles of the immune system and polycomb repressive complex 2 in pathological AD.

106 (MPNST) harboring loss-of-function polycomb-repressive complex 2 mutations.

107 Enhancer of zeste 2 polycomb-repressive complex 2 subunit (Ezh2) also bound to this r

108 calization with enhancer of zeste 2 polycomb repressive complex 2 subunit (EZH2).

109 biosynthesis and downregulation of polycomb repressive complex 2 targets.

110 stically, WT HNF-1beta recruits the polycomb-repressive complex 2 that catalyzes H3K27 trimethylation

111 , a PHD protein that functions with Polycomb repressive complex 2 to epigenetically silence FLOWERING

112 Pax5-binding site by recruiting the polycomb repressive complex 2 to induce bivalent chromatin at the

113 s nitrogen-dependent recruitment of polycomb repressive complex 2 to repress branching-inhibitory gen

114 The polycomb repressive complex 2, with core components EZH2, SUZ12,

115 Mechanistically, SRC-2-bound AR formed a repressive complex by recruiting histone deacetylase 2 t

116 dentify a circZNF827-nucleated transcription-repressive complex containing hnRNP-K/L proteins and sho

117 Polycomb repressive complex I exerted a further layer of control

118 ription factor Yin Yang 1 (YY1) and Polycomb repressive complex member enhancer of zeste homologue 2

119 ers, and gene bodies, as well as in polycomb repressive complex occupancy and CTCF binding sites are

120 ich encodes a core component of the Polycomb repressive complex-2 (PRC2).

121 nteracts with SUZ12, a component of polycomb-repressive-complex 2 (PRC2), to repress the transcriptio

122 Polycomb Repressive Complexes (PRC1 and PRC2) regulate developmen

123 essed chromatin and the function of polycomb repressive complexes (PRC1/2).

124 Long noncoding RNAs (lncRNAs) cause Polycomb repressive complexes (PRCs) to spread over broad regions

125 We also extensively discuss polycomb-group repressive complexes (PRCs), which frequently play a vit

126 for spreading Xist and maintaining Polycomb repressive complexes 1 and 2 (PRC1/PRC2) along the inact

127 Involvement of Polycomb repressive complexes 1 and 2 in XCI has been intensively

128 of the Trithorax-COMPASS complexes, Polycomb repressive complexes and Clr4/Suv39 histone-modifying ma

129 ion studies confirm the involvement of these repressive complexes in regulating A3B expression.

130 The Polycomb Repressive Complexes PRC1 and PRC2 catalyse distinct chr

131 ist locus resistant to silencing by the same repressive complexes that the lncRNA recruits to other g

132 elinogenic programs and recruiting chromatin-repressive complexes to block Schwann cell differentiati

133 een Firre RNA and components of the Polycomb repressive complexes.

134 agonist/corepressor-bound transcriptionally repressive conformation.

135 changes between transcriptionally active and repressive conformations-supporting a fundamental hypoth

136 changes between transcriptionally active and repressive conformations.

137 This co-repressive consortium links gene expression to ratio of

138 Our results indicate that PKA maintains repressive control over MAPK signaling as well as a nega

139 We found that basal, repressive CPEB3 is localized to membraneless cytoplasmi

140 inase and ASXL1, a component of the Polycomb repressive deubiquitinase (PR-DUB) complex, both of whic

141 The mammalian Polycomb repressive deubiquitinase (PR-DUB) complexes catalyze re

142 at form multiprotein complexes to counteract repressive developmental gene expression programmes esta

143 set of mechanisms by which transcriptionally repressive DNA and histone methylation are targeted to v

144 Termination removes the repressive DNA structure, whereupon long K48-linked ubiq

145 in de novo deposition of H3K27me3-containing repressive domains.

146 expression is suppressed in normal cells by repressive E2F complexes and that viral or mutational di

147 Mechanistically, monomerisation relieves a repressive effect allosterically propagated from the dim

148 eIF4A2 exerts its repressive effect by binding purine-rich motifs which ar

149 The repressive effect of ANG II on Npr1 transcription and re

150 resses PHO gene expression and erases the de-repressive effect of CTD-S7A.

151 Experiments showed that the repressive effect of TGFbeta on chondrocytes treated wit

152 Promoter truncation analyses pinpointed the repressive effect of WT p53 to the -209 and -14 region o

153 We also demonstrate that ZL0580 induces repressive effect on HIV in human primary monocyte-deriv

154 reveals that PRDM16 imparts a predominantly repressive effect on the RCC transcriptome including sup

155 mid-based constructs do not demonstrate such repressive effect.

156 ic regions bound by bZIPs with promotive and repressive effects on flowering.

157 led investigation revealed that SIRT6 exerts repressive effects on multiple promoters of KSHV.

158 -4a (HSF-4a) facilitated the ANG II-mediated repressive effects on Npr1 transcription.

159 ffects on flowering, but does potentiate its repressive effects on root growth.

160 wer transcript levels, consistent with local repressive effects.

161 ysis across Danio species indicated that the repressive element is conserved in closely related speci

162 a short (22 bp) DNA element containing a key repressive element.

163 ation of these loops by interactions between repressive elements in the loci, forming a genomic subco

164 f repetitive DNA forming a transcriptionally repressive environment.

165 coincides with the establishment of a global repressive epigenetic landscape, followed by the emergen

166 27 of histone 3 (H3K27me3), which acts as a repressive epigenetic mark.

167 mming efficiency, mainly focused on removing repressive epigenetic marks such as histone methylation

168 Repressive epigenetic marks, such as DNA and histone met

169 tin context of increased levels of these two repressive epigenetic marks.

170 targeted mRNA destruction and deposition of repressive epigenetic marks.

171 etrotransposon silencing and cooperates with repressive epigenetic pathways in male germ cells.

172 g their contributions, our data suggest that repressive epigenetic systems play minor roles in mESC s

173 Prior binding of the repressive Ets transcription factor Etv6 predicts cohesi

174 De novo H3K9me3 is initially non-repressive for gene expression, but instead bookmarks pr

175 LE3, HDAC7, and NFIC, thereby blocking FOXA1-repressive function and activating the invasion genes.

176 However, when DREAM repressive function is compromised during terminal diffe

177 fined the exact timing of the switch and the repressive function of CPE elements, and identified a no

178 remodeler, as a candidate for mediating the repressive function of let-7 in HC differentiation and i

179 Our work describes a critical repressive function of REX1 in maintenance of HCC cells

180 e SBE on the DMT1 promoter did not alter the repressive function of SMAD3 or SMAD4.

181 NOTCH signaling with intrinsic transcription repressive function, causes conversion of dermal fibrobl

182 on in cancer is mediated in part through YAP repressive function.

183 d hypermethylation of THOR counteracted this repressive function.

184 tion of how Su(H) in other protostomes gains repressive function.

185 In agreement with its established repressive functions, Etv6 directly inhibits expression

186 or neural crest cells, and may be blocked by repressive guidance signals elevated in the male pathway

187 ting IRF3-mediated transcription and erasing repressive H2A ubiquitination.

188 ation at lysine 36 (H3K36me2) and/or loss of repressive H3 trimethylation at lysine 27 (H3K27me3).

189 , most notably H3K27Ac and H3K36me2, whereas repressive H3K27 di- and trimethylation (H3K27me2/3) mar

190 mice confirmed that the distribution of the repressive H3K27me3 (histone H3 trimethylated at K27) ma

191 r GSK-J4 increases genome-wide levels of the repressive H3K27me3 chromatin mark and leads to suppress

192 lated nematodes, piRNAs are clustered within repressive H3K27me3 chromatin, while in other species, t

193 e found a small subset of genes that contain repressive H3K27me3 marks near the gene body in SKBR3 ce

194 und KDOBA67 led to a genome-wide increase in repressive H3K27me3 marks with concomitant reduction in

195 3K4me3, H3K4me2, H3K4me1, H3K27ac, H3K36me3; repressive: H3K27me3, H3K9me3), CTCF binding, and gene e

196 ures in affected cell populations, including repressive H3K9me3 and H3K27me3 histone marks on extrace

197 lineage differentiation is characterized by repressive H3K9me3 and H3K27me3 marks on typical dentino

198 els but induced a site-specific reduction of repressive H3K9me3 marks at promoters of mesenchymal gen

199 l crosslinking mass spectrometry confirm the repressive helix 12 conformation.

200 Constitutive domains of repressive heterochromatin are maintained within the fis

201 enome integrity in part through establishing repressive heterochromatin at transposon loci.

202 tection of euchromatin from invasion by gene-repressive heterochromatin is critical for cellular heal

203 ina-associated domains (LADs) are regions of repressive heterochromatin positioned at the nuclear per

204 the pathways involved in maintenance of the repressive heterochromatin state are reasonably well und

205 dence that, by maintaining transcriptionally repressive heterochromatin, LSH may be essential to prev

206 as a boundary to restrict the spread of the repressive HIR/Asf1/Rtt106 complex from histone gene pro

207 arginine methyltransferase, to regulate the repressive histone arginine modifications (H4R3me2s and

208 ilial Alzheimer's disease (FAD) mouse model, repressive histone H3 dimethylation at lysine 9 (H3K9me2

209 axis in which LSD1 induces demethylation of repressive histone H3 lysine 9 dimethylation (H3K9Me2) a

210 re, we uncover a link between the removal of repressive histone H3K9 methylation and DNA methylation

211 Igf2, likely through the PRC1.1 complex; the repressive histone mark H2AK119Ub is decreased at the Ig

212 IP-seq-based genome-wide localization of the repressive histone mark H3K27me3 (mediated via the polyc

213 2 (PRC2) protein SUZ12 and deposition of the repressive histone mark H3K27me3 are increased at SCN5A.

214 ion in muscle showed increased levels of the repressive histone mark H3K9me3 in mdx mice compared to

215 hed with B-cell factors EBF1 and RBP-jK, the repressive histone mark H3K9me3, and AT-rich flanking se

216 H3K27me2 is mutually exclusive with another repressive histone mark, H3K9me2, that mainly silences t

217 he 5'UTR and stimulating local deposition of repressive histone marks at these transposons.

218 these findings suggest that rapid erasure of repressive histone marks by KDM7A and UTX is essential f

219 DNA methylation, repressive histone marks, and PIWI-interacting RNA (piRN

220 bining the in situ Hi-C data with active and repressive histone marks, CTCF binding sites, nucleosome

221 moter activity and accumulation of active or repressive histone marks.

222 ting the histone demethylase Kdm4a to remove repressive histone marks.

223 egion, recruiting KAP1 and HP1, and imposing repressive histone marks.

224 n the two chromatin segments with active and repressive histone marks.

225 sive histone acetylation and lower levels of repressive histone methylation as well as alterations to

226 ion of gene expression, but the functions of repressive histone methylation dynamics during inflammat

227 essive complex 2 (PRC2) installs and spreads repressive histone methylation marks on eukaryotic chrom

228 ional reprogramming to direct CREB or G9a (a repressive histone methyltransferase) to the Zfp189 prom

229 arate-dependent dioxygenases that remove the repressive histone modification H3K27me3 and activate di

230 GRD is a widespread enrichment domain of the repressive histone modification H3K27me3 and is further

231 ontrol the level and the localization of the repressive histone modification H3K27me3 and the histone

232 that sperm chromosomes inherited lacking the repressive histone modification H3K27me3 are maintained

233 enriched for and can distinguish between the repressive histone modification H3K9me3, H3K9me2 and H3K

234 However, it is unclear how the repressive histone modification H4K20me3 or the histone

235 istone 3 lysine 9 dimethylation (H3K9me2), a repressive histone modification mark that was increased

236 epigenetic machinery to deposit the H3K9me3 repressive histone modification, can activate expression

237 abstinence was coupled with depletion of the repressive histone modification, H3K27me3, and enrichmen

238 occurred in association with HDAC2-dependent repressive histone modifications at the mGlu2 promoter.

239 In mammals, repressive histone modifications such as trimethylation

240 ge decisions in vivo By analyzing active and repressive histone modifications, we show that stem cell

241 y loss of their activating marks and not via repressive histone modifications.

242 genomic regions marked with both active and repressive histone modifications.

243 correlation with previously curated LADs and repressive histone modifications.

244 are heavily modified by DNA methylation and repressive histone modifications.

245 the light-induced degradation of D1 relieves repressive interactions between D1 and translational act

246 HSF1 is tightly regulated via intramolecular repressive interactions, post-translational modification

247 the presence of transcriptionally active or repressive intervals flanked by 6mA-enriched regions, an

248 y, our results illustrate that an angiogenic repressive lncRNA, LINC00313, which is upregulated durin

249 is responsible for catalyzing the epigenetic repressive mark H3K27me3 and is essential for animal dev

250 s exhibits higher DNA methylation levels and repressive mark H3K9Me2 enrichment when compared to DP-M

251 dent silencing and genome-wide deposition of repressive mark H3K9me3.

252 e H3K4me3 active methyl mark to the H3K27me3 repressive mark.

253 noubiquitin from histone H2A (H2A-K119Ub), a repressive mark.

254 of HNF-1beta decreases H3K27 trimethylation repressive marks and increases beta-catenin occupancy at

255 sequencing, we found that by targeting 5hmC repressive marks in the promoter regions, C35-mediated T

256 and is further enriched with multiple other repressive marks including H3K9me3, H3K9me2, and H3K27me

257 by K27M, which mostly impairs spread of the repressive marks it catalyzes, especially H3K27me3.

258 e strongest interactions are associated with repressive marks made by the Polycomb complex and are di

259 de binding of polycomb and polycomb H3K27me3 repressive marks to pluripotency genes, thereby exerting

260 gh NKX2-1 and SOX2 are hypothesized to be co-repressive master regulators of tracheoesophageal fates,

261 on mediated by H1 as an additional, dominant repressive mechanism for silencing of repetitive major s

262 f HIF2alpha activity, suggesting an indirect repressive mechanism through DNA binding.

263 urrent findings suggest that ANG II-mediated repressive mechanisms of Npr1 transcription and receptor

264 ists of antiphasic rhythms in activating and repressive modifications generating chromatin states tha

265 signal, and is accompanied by release of the repressive nucleolar chromatin remodeling complex (NoRC)

266 ta), a scaffolding protein without intrinsic repressive or DNA-binding properties.

267 Here, we review the role that repressive pathways - and their alleviation - play in es

268 important to limit self-toxicity, endogenous repressive pathways represent a vulnerability for parasi

269 the role of PER in the dynamic nature of the repressive phase of the TTFL.

270 is peripheral heterochromatin functions as a repressive phase, mechanisms that relocate genes to the

271 er that induces dorsal mesoderm by releasing repressive polycomb group proteins from chromatin, bound

272 of wildtype but not mutant E2F promoters by repressive PRC1.6/E2F6 and DREAM/E2F4 complexes.

273 Functional studies demonstrate that PRDM16's repressive properties, mediated by physical interaction

274 ly lost without a compensatory gain in other repressive PTMs.

275 revealed neonatal alcohol decreased several repressive regulators of transcription in hypothalamic m

276 Consistent with a repressive regulatory role, DDX3 localized to cytoplasmi

277 initiating the formation of an activating or repressive regulatory sequence.

278 Consistent with a repressive role at stress response genes, genetic ablati

279 terochromatin during reprogramming and a non-repressive role for H3K9me3.

280 work shows that endothelial ERRalpha plays a repressive role in angiogenesis and potentially fine-tun

281 oiesis and globin gene switching through its repressive role in the LDB1 complex, affecting the trans

282 Further, we also show the repressive role of Delta-Notch signaling on lin28a expre

283 In supporting its repressive role, the mcrA deletion mutant conidia contai

284 tissues, indicating plausible site-specific repressive roles of HDAC1 and Sp1 in GM2-synthase mRNA e

285 mbda/iota-Erk dependent Etv5/Satb2 chromatin repressive signaling complex.

286 sary and sufficient to activate night length repressive signaling.

287 signaling, for example enabling 'negative' (repressive) signaling.

288 controls the balance between activating and repressive signals that determine the extent of osteocla

289 ese autophagy regulators, including at known repressive sites, is dependent on CDK1 but independent o

290 shifts the chromatin landscape at EIN2 to a repressive state causing a dramatic reduction of EIN2 ex

291 were regulated by proximal chromatin states; repressive states were associated with a significant red

292 Es in Drosophila embryos, and correlate with repressive states.

293 hat ATRX/DAXX does not serve as an immediate repressive switch for ALT.

294 This involves a co-repressive system in which each strain produces a signal

295 The Polycomb repressive system is an essential chromatin-based regula

296 factors, that explain U1-CPAFs' switch from repressive to activated states.

297 Many large cardiac genes transition from a repressive to an active compartment during differentiati

298 It recruits the repressive TRIM28 complex to RMER19B retrotransposons to

299 While several fatty acids are known to be repressive, we show here that cis-2-unsaturated fatty ac

300 ciated genes such as CCNA and CDK6 Moreover, repressive YAP-TEAD4 complexes containing the histone me