ライフサイエンスコーパス: reproductive tract

1 ent in which sperm compete (e.g., the female reproductive tract).

2 effective treatments for diseases within the reproductive tract.

3 responses to Chlamydia muridarum within the reproductive tract.

4 varian and endometrial cancers in the female reproductive tract.

5 ze fertilization inside the mammalian female reproductive tract.

6 ation of patients with cancers of the female reproductive tract.

7 ely in a sensitive mucosal site, such as the reproductive tract.

8 (2+)-concentration environment of the female reproductive tract.

9 uding the gastrointestinal tract, heart, and reproductive tract.

10 olonizes healthy human skin, mucosa, and the reproductive tract.

11 ated in inflammatory syndromes of the female reproductive tract.

12 abnormalities in the development of the male reproductive tract.

13 ghters, indicating accelerated ageing in the reproductive tract.

14 eds progress of the sperm through the female reproductive tract.

15 e with oocytes despite direct contact in the reproductive tract.

16 contribute to E. faecalis persistence in the reproductive tract.

17 osal epithelium in the rhesus macaque female reproductive tract.

18 genes that are selectively expressed in the reproductive tract.

19 ne disruptors on the development of the male reproductive tract.

20 ment is dependent on factors secreted by the reproductive tract.

21 rus (HPV)-driven cancer including the female reproductive tract.

22 n for successful fertilization in the female reproductive tract.

23 n epithelial barrier protection of the lower reproductive tract.

24 an oocyte as they travel through the female reproductive tract.

25 trols human sperm function within the female reproductive tract.

26 over long distances in the mammalian female reproductive tract.

27 how that MISO and 20E interact in the female reproductive tract.

28 input of these sensory neurons to the female reproductive tract.

29 gation is driven by elongation of the female reproductive tract.

30 t both IgG and SIgA are present in the human reproductive tract.

31 SP is mediated by SP retention in the female reproductive tract.

32 hus avoiding ejection by the female from her reproductive tract.

33 this capacity during migration in the female reproductive tract.

34 nology for drug delivery to the lower female reproductive tract.

35 natural selective environment of the female reproductive tract.

36 ated in an AF-2-dependent manner in the male reproductive tract.

37 ons of the sex accessory tissues in the male reproductive tract.

38 in metabolism, transport, or activity in the reproductive tract.

39 tween the sexes that occur within the female reproductive tract.

40 stitial cells in other organs, including the reproductive tract.

41 S colonization and persistence in the female reproductive tract.

42 in the Caenorhabditis elegans hermaphrodite reproductive tract.

43 o activation once introduced into the female reproductive tract.

44 will differentiate into the internal female reproductive tract.

45 ntains SP's ability to act within the female reproductive tract.

46 e source of mucosal antibodies in the female reproductive tract.

47 axis, spinal cord, forelimbs, heart, eye and reproductive tract.

48 iding fertilization to protecting the female reproductive tract.

49 ion for improved drug delivery to the female reproductive tract.

50 face with the luminal contents of the female reproductive tract.

51 ent, however little is known about the lower reproductive tract.

52 ma cells in the lamina propria of the female reproductive tract.

53 the respiratory, gastrointestinal and female reproductive tract.

54 crine interactions with the hosting maternal reproductive tract.

55 ion shaped variant diversity in the stallion reproductive tract.

56 means to reduce ZIKV persistence in the male reproductive tract.

57 determine its pathogenic role in the female reproductive tract.

58 l to study RNA virus persistence in the male reproductive tract.

59 licle cells of ovary and epithelial cells of reproductive tracts.

60 ent and homeostasis of the airway, brain and reproductive tracts.

61 iated with diseases of the brain, airway and reproductive tracts.

62 es in the development of the male and female reproductive tracts.

63 often in specialized storage organs in their reproductive tracts.

64 ame intersex-possessing both female and male reproductive tracts.

65 hts into the Y chromosome's roles beyond the reproductive tract--a theme that promises to broaden the

66 ed constellation of increasingly common male reproductive tract abnormalities (including hypospadias,

67 Sperm storage in the female reproductive tract after mating and before ovulation is

68 ured the amount of SP retained in the female reproductive tract after mating and female egg laying af

69 y before mating but also within the female's reproductive tract after mating.

70 gg laying, or the amount of SP in the female reproductive tract after mating.

71 istic effects of raloxifene persisted in the reproductive tract after treatment had ceased.

72 by regulating sperm maturation in the female reproductive tract and by triggering key sperm physiolog

73 d inflammation and although expressed in the reproductive tract and fetal tissues, its role in the pa

74 nd to cues originating from along the female reproductive tract and from the layers of the egg in ord

75 rom the paternal compartment to the maternal reproductive tract and future embryo; 2) a molecular sig

76 on (PMCA)-competent prions within the female reproductive tract and in fetal tissues harvested from C

77 ry efficient at navigating the hermaphrodite reproductive tract and locating oocytes.

78 The tissues and organs of the female reproductive tract and pelvic floor undergo significant

79 hormone profile, which controls human female reproductive tract and peripheral tissue dynamics in sin

80 are the most common neoplasms of the female reproductive tract and primary cause for hysterectomy, l

81 conducted a thorough analysis of the female reproductive tract and report that the primary cause of

82 ary tissues, affects the virilization of the reproductive tract and results in genitourinary birth de

83 n mixtures are separately stored in the male reproductive tract and sequentially transferred to the f

84 , including persistent infection in the male reproductive tract and sexual transmission, an ability t

85 nti-Neu5Gc antibodies could enter the female reproductive tract and target Neu5Gc-positive sperm or f

86 These systems simulate the in vivo female reproductive tract and the endocrine loops between organ

87 ry site of viral persistence in the stallion reproductive tract and the persistence is associated wit

88 gastrointestinal tract, nasopharynx, female reproductive tract and the surface of the eye.

89 inning with the insemination into the female reproductive tract and, finally, leading to embryogenesi

90 ike structures that line the respiratory and reproductive tracts and the middle ear and generate flui

91 istology and cytokine expression from murine reproductive tracts and vaginal lavages collected 24 and

92 distinct but overlapping patterns within the reproductive tract, and in haf bee1 bee3 triple mutants

93 E. faecalis also colonizes the female reproductive tract, and reports suggest vaginal coloniza

94 ese stallions, EAV is detectable only in the reproductive tract, and viral persistence occurs despite

95 Congenital reproductive tract anomalies could impair fertility.

96 data of human sperm bound to excised female reproductive tract are also presented and are found to b

97 ility and reproduction, organs of the female reproductive tract are also regulated by the hypothalami

98 ns in highly folded environment of mammalian reproductive tract are important for spermatozoa swimmin

99 rse over which plugs survive in the female's reproductive tract are lacking.

100 infections that ascend from the lower female reproductive tract are the most common route of uterine

101 Female and male reproductive tracts are developed from Mullerian ducts a

102 s in the breast, prostate, cervix, and upper reproductive tract-are shared by embryo and adult.

103 mpanied by epididymitis, suggesting the male reproductive tract as 1 site of antigen persistence.

104 blish persistent infection in the stallion's reproductive tract as a mechanism to ensure its maintena

105 he growth of pollen tubes through the female reproductive tract as they seek out unfertilized ovules.

106 ll bowel, select lymphoid areas and the male reproductive tract, as confirmed by quantitative reverse

107 might contribute to dysfunction of the male reproductive tract associated with these mutations.

108 RNA and antigens can be detected in the male reproductive tract at 14 DPI and RNA can also be detecte

109 In Arabidopsis, the reproductive tract begins with the stigma, where pollen

110 eases in plasma cells not only in the female reproductive tract but also at other mucosal sites, and

111 It is plausible that MHM can affect the reproductive tract but the specific infections, the stre

112 mmensal fungus of human gastrointestinal and reproductive tracts, but also causes life-threatening sy

113 xually transmitted infections, treatment for reproductive tract cancer, and treatment of fungal and b

114 t recurrence of neoplastic disease and lower reproductive tract cancers.

115 RA and PGRB are often observed in women with reproductive tract cancers.

116 port on a study of the alterations of murine reproductive tract collagen resulting from pregnancy and

117 We hypothesized that each part of the reproductive tract contains a characteristic combination

118 sion to test whether the evolution of female reproductive tract design might have driven the evolutio

119 to survive as allogeneic cells in the female reproductive tract despite female immunity.

120 Normal reproductive tract development and anogenital distance (

121 Genetic basis solely for distal reproductive tract development is largely unknown.

122 ce of an additional merging phase for proper reproductive tract development.

123 duct epithelial progenitor cells for female reproductive tract development.

124 factor that is essential for male and female reproductive tract development.

125 LHFPL2 and a novel genetic basis for distal reproductive tract development; they also emphasize the

126 exually transmitted pathogen associated with reproductive tract disease in men and women, and it can

127 en associated with several acute and chronic reproductive tract disease syndromes in men and women.

128 ential component to managing lower and upper reproductive tract disease syndromes in women.

129 talium is an underappreciated cause of human reproductive tract disease, characterized by persistent,

130 e Notch1 intracellular domain (N1ICD) in the reproductive tract driven by a progesterone receptor (Pg

131 equirement for FGF8 in establishing the male reproductive tract ducts and implicate Lhx1 signaling in

132 to limit pathogen infiltration of the lower reproductive tract during pregnancy and thereby is prote

133 t and sequentially transferred to the female reproductive tract during spermatophore assembly.

134 CD8(+) T cells residing in the female mouse reproductive tract encountered cognate antigen, they exp

135 exosomes and sperm) and interact with female reproductive tract epithelia.

136 ction requires the virus to penetrate female reproductive tract epithelial barriers to infect underly

137 verexpressing cells and TLR2-competent human reproductive tract epithelial cell lines.

138 ential evidence for a key role of the female reproductive tract epithelium in facilitating Ab product

139 everely impaired in the cystic fibrosis F508 reproductive tract, even though stimulated fluid secreti

140 Here, we report reproductive tract EVs transmit information regarding st

141 ists in spermatozoa that navigate the female reproductive tract following insemination and are stored

142 We find infectious foci throughout the reproductive tract, from labia to ovary.

143 sible for pathogen clearance from the female reproductive tract (FRT) are incompletely defined; in pa

144 cts, immune control mechanisms in the female reproductive tract (FRT) are not well characterized.

145 Tenofovir (TFV) treatment of female reproductive tract (FRT) cells results in differential a

146 virological events that occur in the female reproductive tract (FRT) during ART that result in such

147 rt, 2011), the earliest events in the female reproductive tract (FRT) during heterosexual HIV-1 trans

148 es, which are known to infiltrate the female reproductive tract (FRT) following semen exposure.

149 hicle for virion dissemination in the female reproductive tract (FRT) in male-to-female HIV transmiss

150 ial vaginosis (BV), a disorder of the female reproductive tract (FRT) in which a healthy Lactobacillu

151 nic bacteria ascending from the lower female reproductive tract (FRT) is associated with many gynecol

152 Disruption of the epithelium in the female reproductive tract (FRT) is hypothesized to increase HIV

153 The female reproductive tract (FRT) is one of the major mucosal inv

154 The female reproductive tract (FRT) is the most common site of infe

155 mydia inoculation and that unlike the female reproductive tract (FRT) mucosa, it halts systemic Chlam

156 We show that the gut and female reproductive tract (FRT) of humanized DRAG mice have a h

157 2 T cells from blood, rectum, and the female reproductive tract (FRT) of rhesus macaques to determine

158 To establish infection in the female reproductive tract (FRT), HIV-1 in male ejaculate must o

159 The female reproductive tract (FRT), similar to other mucosal sites

160 s the vaginal epithelium in the mouse female reproductive tract (FRT).

161 re to SIVmac251 in the rhesus macaque female reproductive tract (FRT).

162 ns at mucosal epithelia including the female reproductive tract (FRT).

163 (GR) is also an integral part of maintaining reproductive tract function; disruption of GR signaling

164 Seminal fluid, which affects female reproductive tract gene expression as well as sperm surv

165 ncing on dissected tissues revealed that the reproductive tracts harbor a complex microbiome characte

166 The microbiome of the female reproductive tract has implications for women's reproduc

167 persistence of Zika virus (ZIKV) in the male reproductive tract has raised concerned for potential da

168 ging sperm as they travel through the female reproductive tract has revealed new details about fertil

169 The results indicate that female reproductive tracts have undergone extensive diversifica

170 nfections could help us to better understand reproductive tract health and improve current prevention

171 sed IFN has suggested a function for IFNs in reproductive tract homeostasis and protection from infec

172 y and space radiation on the male and female reproductive tracts, hypothalamic-pituitary regulation o

173 and precancerous lesions in the female lower reproductive tracts (ie, cervix and vagina) in the human

174 and may regulate muscle contractions in the reproductive tract, if it is expressed there.

175 ance of persistent infection in the stallion reproductive tract.IMPORTANCE EAV can persist in the rep

176 epithelial cells and to colonize the female reproductive tract in a mouse model of infection.

177 lting in complete and incomplete blockage of reproductive tract in infertile and fertile males, respe

178 differentially regulated in the female lower reproductive tract in response to conspecific mating.

179 ment of many conditions affecting the female reproductive tract, including sexually transmitted disea

180 m) in sensory neurons innervating the female reproductive tract indicate that some of these candidate

181 vaginoplasty might play an important role in reproductive tract infection prevention and neovagina he

182 Additionally, acute but not chronic male reproductive tract infection with ZIKV results in infect

183 ng studies of intrauterine contraception and reproductive tract infection.

184 d which looked at health outcomes, primarily reproductive tract infections (RTI).

185 For decades reproductive tract infections (RTIs) have been hypothesi

186 ths, screened and treated (as indicated) for reproductive tract infections (RTIs) within 1 month of e

187 Malaria and sexually transmitted infections/reproductive tract infections (STIs/RTIs) in pregnancy a

188 laria infection and sexually transmitted and reproductive tract infections (STIs/RTIs).

189 , cervicovaginal and systemic HIV-1 RNA, and reproductive tract infections every 3-6 months over 8 ye

190 of biological specimens that were tested for reproductive tract infections.

191 ght (<2,500 g) in women without malarial and reproductive tract infections.

192 ed novel GAS genes that contribute to female reproductive tract interaction that warrant translationa

193 Varying pH of luminal fluid along the female reproductive tract is a physiological cue that modulates

194 The female reproductive tract is particularly sensitive to early-li

195 e outcome of infectious diseases outside the reproductive tract is rarely considered.

196 The persistence of SP in the mated female's reproductive tract is thought to be a consequence of its

197 estrogen exposure and defects in the female reproductive tract is well established.

198 irus (ZIKV) are dampened in the lower female reproductive tract (LFRT) compared to other tissues, but

199 but are not limited to genes influencing the reproductive tract, mammary glands, bone, brain, fat dif

200 Upon mating, regions of the female reproductive tract mature and alter their function [1-3]

201 innate immune activation in the lower female reproductive tract may also affect adaptive immunity, we

202 a man other than her long-term partner, her reproductive tract may contain the sperm of both men, in

203 Secretions within the adult female reproductive tract mediate sperm survival, storage, acti

204 Female reproductive tract microbiota may affect human reproduct

205 sialyl T-antigen varied in bovine and ovine reproductive tract mucins, and terminal N-acetylgalactos

206 as) are the most common tumors of the female reproductive tract, occurring in up to 77% of reproducti

207 rine proteases specifically expressed in the reproductive tract of An. gambiae females play an import

208 ted molecules ("signaling molecules") in the reproductive tract of Drosophila melanogaster females be

209 mating transcriptional response of the lower reproductive tract of Drosophila mojavensis females foll

210 nitored cancer incidence in the female lower reproductive tract of FA-deficient mice expressing HPV16

211 s most renowned as venereal pathogens of the reproductive tract of humans and cattle.

212 tive tract.IMPORTANCE EAV can persist in the reproductive tract of infected stallions, and consequent

213 nges involved in observing events within the reproductive tract of internally fertilizing species whi

214 ly and silently colonizes the intestinal and reproductive tract of laying hens, resulting in contamin

215 cern about persistence of Ebola virus in the reproductive tract of men who have survived EVD.

216 ve immune responses to ZIKV infection in the reproductive tract of mice and that pregnancy-associated

217 tein expression were increased by 25% in the reproductive tract of mice exposed in utero.

218 ed the consequences of infection in the male reproductive tract of mice.

219 tella species are commonly isolated from the reproductive tract of women with obstetric/gynecologic h

220 expanded blastocyst were collected from the reproductive tract of zinc deficient animals on day 3.5

221 eumovirus (AMPV) infects the respiratory and reproductive tracts of domestic poultry, resulting in su

222 The reproductive tracts of female fireflies showed increased

223 indicate that E5 acts as an oncogene in the reproductive tracts of female mice.

224 is indicated by the presence of sperm in the reproductive tracts of queens.

225 The reproductive tracts of the classified heifers were obtai

226 The reproductive tracts of the mice were examined histopatho

227 dua and placenta by ascending from the lower reproductive tract or via hematogenous transmission.

228 The female reproductive tract organs of mammals, including the ovid

229 Mullerian ducts, the primordia of the female reproductive tract organs.

230 issues including intestine, stomach, kidney, reproductive tract, pancreas, brain, heart, and salivary

231 ma gallisepticum is an avian respiratory and reproductive tract pathogen that has a significant econo

232 n provide evidence that they are involved in reproductive tract pathologies.

233 Reproductive tract pathology caused by Chlamydia trachom

234 lowed the discovery of Hoxd9,10,11 redundant reproductive tract patterning function.

235 out human pregnancy, suggesting that reduced reproductive-tract progesterone receptor (PR) initiates

236 ent evidence of adaptive evolution in female reproductive tract proteins suggests this pattern may re

237 jaculates and their interactions with female reproductive tracts remain poorly understood.

238 represented by the lumen of the lower female reproductive tract remains unclear.

239 women, as the organism ascends to the upper reproductive tract, resulting in pelvic inflammatory dis

240 and changes in muscle contraction within the reproductive tract (RT).

241 However, it is difficult to reconcile the reproductive tract's many changing but coordinated event

242 on (STI) that is potentially associated with reproductive tract sequelae in women.

243 nd TNF-alpha production to Chlamydia-induced reproductive tract sequelae.

244 ulating memory B cells that enter the female reproductive tract serve as the source of rapid and robu

245 ce this competition occurs inside the female reproductive tract, she often influences the outcome thr

246 Our results suggest that the reproductive tract signals to the ovary using glandular

247 competition should not be limited to female reproductive tract-specific genes, but should focus also

248 e found that glands of the Drosophila female reproductive tract, spermathecae and/or parovaria, are r

249 No obvious morphological differences in reproductive tract structures and histology of the uteru

250 selection, where readily quantifiable female reproductive tract structures are capable of biasing pat

251 Each reproductive tract subregion displays a characteristic c

252 n Mycoplasma genitalium infection and female reproductive tract syndromes through meta-analysis, Engl

253 vived multiple checkpoints within the female reproductive tract, termed pre-fertilization events.

254 function for the MVB-exosome pathway in the reproductive tract that appears to be conserved across e

255 r coregulator in normal tissues of the human reproductive tract that is expressed at higher levels in

256 igration through and localization within the reproductive tract, thereby promoting their access to oo

257 of P. gingivalis from the oral cavity to the reproductive tract thus represents a potential mechanism

258 e expression changes, and appropriate female reproductive tract tissue architecture.

259 odeling of tenofovir (TFV) in plasma, female reproductive tract tissue, cervicovaginal lavage fluid a

260 entiating estrogen-target tissues, including reproductive tract tissues and adipocytes.

261 gene expressed at low levels in normal human reproductive tract tissues and at higher levels in castr

262 exosomes into E15 mice localized in maternal reproductive tract tissues and in intrauterine fetal com

263 The reproductive tract tissues and peripheral organs integra

264 and SEVI to intact human and rhesus macaque reproductive tract tissues to determine how it influence

265 Ascension to upper reproductive tract tissues was not detected, even among

266 rs of oxidative stress and cellular aging in reproductive tract tissues were assessed at 3 and 6 mo o

267 was limited to hemolymphatic tissues, female reproductive tract tissues, kidney, and liver, potential

268 tion factors that act redundantly to specify reproductive tract tissues.

269 and cellular effects have been identified in reproductive tract tissues.

270 e zygote but also to tissues of the maternal reproductive tract to influence fetal development.

271 pregnancy reduces the capacity of the female reproductive tract to prevent bacterial infection of the

272 d (upstream) secreted factors enter the male reproductive tract to regulate epididymal (downstream) p

273 epithelial-immune system axis in the female reproductive tract to respond to exposure to mucosal pat

274 ts that guide spermatozoa through the female reproductive tract to the mature oocyte.

275 fication of interacting ejaculate and female reproductive tract traits that mediate sperm competition

276 We conclude that reproductive tract tTregs as well as pTregs are sensitiv

277 ereas few PGRA-overexpressing mice developed reproductive tract tumors, all PGRB-overexpressing mice

278 In swine the upper reproductive tract undergoes early postnatal development

279 ng-term persistent infection in the stallion reproductive tract using next-generation sequencing and

280 the shape of female and male harbor porpoise reproductive tracts using 2D geometric morphometrics and

281 l plasticity is characteristic of the female reproductive tract: vaginal sensory innervation density

282 In the reproductive tract, virK was required in early colonizat

283 varian cancer risk among women with a patent reproductive tract was 1.13 (95% CI, 1.01 to 1.26), the

284 ction comparing women with vs without patent reproductive tracts was .15.

285 PGR isoform ratio in the maintenance of the reproductive tract, we generated mice that overexpress P

286 male's sperm progressing through the female reproductive tract were also explained by natural variat

287 mentally infected with MmuPV1 in their lower reproductive tract were housed with unmanipulated male m

288 Animals were euthanized at week 16 and the reproductive tracts were examined histologically.

289 tissues such as skin, gut, lung, tongue and reproductive tract where they provide a first line of de

290 tivated by mechanical pressure in the female reproductive tract, whereas in most other species, eggs

291 activated motility as they ascend the female reproductive tract, which enables them to overcome barri

292 anscriptional changes in cells of the female reproductive tract, which may facilitate HIV transmissio

293 minal vesicles are paired organs of the male reproductive tract, which produce and secrete seminal fl

294 ullerian ducts are the anlagen of the female reproductive tract, which regress in the male fetus in r

295 molecules that endow different parts of the reproductive tract with unique temporal and spatial iden

296 l opening, persistent diestrus, and atrophic reproductive tracts with absent corpora lutea.

297 The prostate plays a vital role in the male reproductive tract, with acute and chronic prostatitis l

298 synergistic homeotic transformations of the reproductive tracts, with the uterus anteriorized toward

299 m is established by default because the male reproductive tracts (Wolffian ducts) in the female degen

300 cuss how the sperm interacts with the female reproductive tract, zona pellucida, and the oolemma.