ライフサイエンスコーパス: resistance to antibiotics

1 A. baumannii had high level of resistance to antibiotics.

2 m biofilms, it can evade and rapidly develop resistance to antibiotics.

3 Efflux is a common mechanism of resistance to antibiotics.

4 r interactions mediated by production of and resistance to antibiotics.

5 ring RNA stability, and conferring bacterial resistance to antibiotics.

6 la enterica that are all validated to confer resistance to antibiotics.

7 solve the challenges arising from increasing resistance to antibiotics.

8 pumps significantly contribute for bacteria resistance to antibiotics.

9 f enzymes that plays a key role in bacterial resistance to antibiotics.

10 prevalent pathogen that can rapidly acquire resistance to antibiotics.

11 garnering notoriety in an era of increasing resistance to antibiotics.

12 problematic outbreaks and acquire high-level resistance to antibiotics.

13 racellular residency, biofilm formation, and resistance to antibiotics.

14 ences in phenotypes, including virulence and resistance to antibiotics.

15 ble by vaccination and is rapidly developing resistance to antibiotics.

16 ficult to eradicate because of its intrinsic resistance to antibiotics.

17 vivo fitness, cell envelope homeostasis and resistance to antibiotics.

18 ion, and potential contribution to bacterial resistance to antibiotics.

19 d with mutations, can contribute to adaptive resistance to antibiotics.

20 it has led to a rapid increase in bacterial resistance to antibiotics.

21 "intrinsic resistome," which provides innate resistance to antibiotics.

22 Gram-negative bacteria and provide intrinsic resistance to antibiotics.

23 a major health threat because of increasing resistance to antibiotics.

24 elements responsible for spreading bacterial resistance to antibiotics.

25 nificant mechanism by which bacteria develop resistance to antibiotics.

26 fication as well as their ability to develop resistance to antibiotics.

27 ons continues to be complicated by expanding resistance to antibiotics.

28 pid spread of genes such as those conferring resistance to antibiotics.

29 gly difficult to treat due to high levels of resistance to antibiotics.

30 notypic delay for the evolution of bacterial resistance to antibiotics.

31 ructural basis of catalysis and mechanism of resistance to antibiotics.

32 ulated genes points to mechanisms of biofilm resistance to antibiotics.

33 hages in the age of widespread antimicrobial resistance to antibiotics.

34 There is evidence of rapidly growing resistance to antibiotics across Africa.

35 ic outcomes and containing further spread of resistance to antibiotics among other bacteria.

36 tems are necessary for metabolic regulation, resistance to antibiotics and antimicrobials, pathogenes

37 inhibitors and often contribute to multidrug resistance to antibiotics and biocides.

38 Efflux pumps have been shown to mediate resistance to antibiotics and cationic peptides in other

39 ellular superstructure can display increased resistance to antibiotics and cause serious, persistent

40 omplex multicellular assemblies that exhibit resistance to antibiotics and contribute to the pathogen

41 ts prominence as a pathogen is its intrinsic resistance to antibiotics and disinfectants.

42 Its predilection to develop resistance to antibiotics and expression of multiple vir

43 Results showed the safety of probiotics, resistance to antibiotics and gastric acid, and potentia

44 states during 1978-2019 to examine bacterial resistance to antibiotics and heavy metals.

45 ich includes imparting significantly greater resistance to antibiotics and host immune effectors.

46 In developing countries, increased resistance to antibiotics and its cost make eradication

47 ty testing platform to recognize patterns of resistance to antibiotics and make predictions about the

48 the same genetic determinant responsible for resistance to antibiotics and metals).

49 Bacillus subtilis are broadly implicated in resistance to antibiotics and other cell envelope stress

50 e selecting drugs, chromosomal mutations for resistance to antibiotics and other chemotheraputic agen

51 sion of multiple chromosomal genes affecting resistance to antibiotics and other environmental hazard

52 ial MDRs had previously been associated with resistance to antibiotics and other toxic compounds.

53 With a high intrinsic resistance to antibiotics and the ability to overcome ch

54 to play an important role in the bacteria's resistance to antibiotics and the host immune response.

55 The emerging and sustained resistance to antibiotics and the poor pipeline of new a

56 The emergence of superbugs developing resistance to antibiotics and the resurgence of microbia

57 ted functions of these genes are: conferring resistance to antibiotics and toxic compounds, and enabl

58 an isolate, testing its properties, such as resistance to antibiotics and virulence, and monitoring

59 ucture reveals the molecular basis for broad resistance to antibiotics and will inform the design of

60 ), and sigma(X)) are induced by, and provide resistance to, antibiotics and other agents eliciting ce

61 ere pneumonia in young children), increasing resistance to antibiotics, and changes in HIV prevalence

62 ay the dominant role in conferring intrinsic resistance to antibiotics, and provide initial insights

63 fferent biofilms compositions, increased the resistance to antibiotics, and some changed the cell wal

64 outcomes of increased infectivity, intrinsic resistance to antibiotics, and subversion of the host im

65 bidity and mortality and, because of biofilm resistance to antibiotics, are difficult to treat.

66 only display distinct phenotypes, including resistance to antibiotics, but also, serve as building b

67 entical bacteria display differing levels of resistance to antibiotics, clonal yeast populations demo

68 he typical ARG profiles suggest a prevailing resistance to antibiotics commonly used in human health

69 Increasing resistance to antibiotics creates the need for prudent a

70 The rise in bacterial resistance to antibiotics demonstrates the medical need

71 However, the development of antimicrobial resistance to antibiotics, demonstrates a need to find a

72 populations exhibit non-genetic or adaptive resistance to antibiotics, despite sustaining considerab

73 eradicate because of their unusually robust resistance to antibiotics, disinfectants, and desiccatio

74 Resistance to antibiotics has become a major threat to m

75 The emergence and spread of bacterial resistance to antibiotics has developed into one of the

76 own genetic components underlying phenotypic resistance to antibiotics has increased.

77 The continued evolution of resistance to antibiotics has led to wide ranging consul

78 Bacterial resistance to antibiotics has reached critical levels, s

79 genetic material in bacteria that encode for resistance to antibiotics) have been found in the aquati

80 ly related to these shifts include those for resistance to antibiotics, heavy metals, and phage.

81 Mutations that cause resistance to antibiotics in bacteria often reduce growt

82 Bacterial resistance to antibiotics in this clinical setting furth

83 a challenge for treatment due to its evolved resistance to antibiotics, including carbapenems.

84 icult to treat due to intrinsic and acquired resistance to antibiotics, including vancomycin.

85 The emergence of bacterial resistance to antibiotics is a major health problem and,

86 The emergence of resistance to antibiotics is a serious problem often rel

87 ncing for phenotype prediction as the actual resistance to antibiotics is almost exclusively mediated

88 Resistance to antibiotics is approaching crisis levels f

89 Increased Gram-negative bacteria resistance to antibiotics is becoming a global problem,

90 The incidence of resistance to antibiotics is increasing; therefore, it i

91 jejuni is a major zoonotic pathogen, and its resistance to antibiotics is of great concern for public

92 of P. aeruginosa and its ability to develop resistance to antibiotics, it continues to be problemati

93 With the rapid increase of infection resistance to antibiotics, it is urgent to find novel in

94 he survival of bacteria and the evolution of resistance to antibiotics make it an attractive target f

95 Bacterial resistance to antibiotics makes previously manageable in

96 animal-adapted S. aureus lineages exhibiting resistance to antibiotics must be considered a major thr

97 Conversely, mutations conferring resistance to antibiotics not administered diminish and

98 deration, such as public good production and resistance to antibiotics or predation, are often assume

99 Gram-negative bacteria develop and exhibit resistance to antibiotics, owing to their highly asymmet

100 Bacterial resistance to antibiotics, particularly plasmid-encoded

101 Bacterial resistance to antibiotics, particularly to multiple drug

102 We postulate that phenotypic resistance to antibiotics, persistence, is not an evolve

103 Resistance to antibiotics poses a major global threat ac

104 The spread of bacterial resistance to antibiotics poses the need for antimicrobi

105 With increase in the incidence of resistance to antibiotics, probiotics are emerging as a

106 sion as measured by mtrCDE transcription and resistance to antibiotics, progesterone and antimicrobia

107 It has long been assumed that resistance to antibiotics reduces the fitness of disease

108 The increasing threat of pathogen resistance to antibiotics requires the development of no

109 Genes for resistance to antibiotics such as acriflavin, bacitracin

110 With the rise in resistance to antibiotics such as methicillin, there is

111 NA) modulates ribosomal function and confers resistance to antibiotics targeted to the ribosome.

112 in mixed cultures was associated with higher resistance to antibiotics than in either monoculture.

113 ncluding cell wall remodeling (and therefore resistance to antibiotics that target bacterial cell wal

114 lly characterized VgaA, LsaA and MsrE confer resistance to antibiotics that target the peptidyl trans

115 aminoglycoside O-phosphotransferases, confer resistance to antibiotics that were not administered in

116 The emergence of bacterial multidrug resistance to antibiotics threatens to cause regression

117 omised by the ability of bacteria to develop resistance to antibiotics through mutations or through t

118 Due to their resistance to antibiotics, treatment is often very chall

119 inistered drug, as well as genes that confer resistance to antibiotics unrelated to the administered

120 across multiple high-mortality settings, and resistance to antibiotics used for sepsis treatment was

121 Strains with rRNA mutations conferring resistance to antibiotics used in acne treatment were do

122 Bacterial resistance to antibiotics usually incurs a fitness cost

123 Bacteria can rapidly evolve resistance to antibiotics via the SOS response, a state

124 No genomic microevolution and no Legionella resistance to antibiotics were detected.

125 estimating the degree of E. coli JM109 cells resistance to antibiotics within 2-5h using disposable s

126 Increasing resistance to antibiotics worldwide has adverse effects