ライフサイエンスコーパス: respiratory burst

1 on activation of the NADPH oxidase-dependent respiratory burst.

2 ice that are unable to generate a phagocytic respiratory burst.

3 is required for the TSP1-induced macrophage respiratory burst.

4 ntegrins, also prevented beta-glucan-induced respiratory burst.

5 ivate the proton conductance or for a normal respiratory burst.

6 omote neutrophil adhesion, degranulation and respiratory burst.

7 an and released an efficient plasma membrane respiratory burst.

8 some-lysosome fusion but not by generating a respiratory burst.

9 in-dependent activation of degranulation and respiratory burst.

10 yl-methionyl-leucyl-phenylalanine-stimulated respiratory burst.

11 t N. gonorrhoeae stimulated PMN to produce a respiratory burst.

12 jor role in the activation of the neutrophil respiratory burst.

13 ion of genes that contribute to an effective respiratory burst.

14 al, this bacterium suppresses the neutrophil respiratory burst.

15 utrophils), and the AGE-augmented neutrophil respiratory burst.

16 ducts in conjunction with a minimal residual respiratory burst.

17 (phox-/-)) in which PMN are incapable of the respiratory burst.

18 NADPH oxidase resulting in the absence of a respiratory burst.

19 und site, release reactive oxygen species by respiratory burst.

20 stream intermediates, thereby amplifying the respiratory burst.

21 y neutrophil activation and its accompanying respiratory burst.

22 in eosinophils that is modulated during the respiratory burst.

23 he increase of proton conductance during the respiratory burst.

24 ed in vitro for its effect on the neutrophil respiratory burst.

25 LDL blocked T/HS priming of respiratory burst.

26 al to the activation of H(+) flux during the respiratory burst.

27 ity, stayed the same or decreased during the respiratory burst.

28 cytosis of the particles and generation of a respiratory burst.

29 for the enhanced GSH uptake seen during the respiratory burst.

30 gmented size, granularity, phagocytosis, and respiratory burst.

31 ucyl-phenylalanine (fMLP)-induced neutrophil respiratory burst.

32 s with sepsis enhanced bacterial killing and respiratory burst.

33 product of lipid peroxidation induced by the respiratory burst.

34 l transmigration to IL-8, but did not affect respiratory burst.

35 sis, Ab-dependent cellular cytotoxicity, and respiratory burst.

36 and eliminated any haplotypic impact on the respiratory burst.

37 in the absence of an NADPH oxidase-mediated respiratory burst.

38 lation at the phagosome, leading to impaired respiratory burst.

39 proinflammatory cytokine production, and the respiratory burst.

40 tes to microbe elimination during macrophage respiratory burst.

41 osomal environment and inhibit the host cell respiratory burst.

42 obust, highly periodic cycles in the form of respiratory bursts.

43 ed the percentage of phagocytes undergoing a respiratory burst (66.0% +/- 6.3% versus 41.0% +/- 8.3%

44 tem's battle against pathogens includes the "respiratory burst," a rapid release of ROS from leukocyt

45 eral blood leukocytes (PBL) and enhanced the respiratory burst, acid phosphatase activity, chemotacti

46 several mechanisms, such as NO synthase, the respiratory burst, acidification, and autophagy, how hum

47 bulin (Ig)G-coated erythrocyte phagocytosis, respiratory burst, actin cup formation, and activation o

48 rradiation was also associated with enhanced respiratory burst activities and an unexpected neutrophi

49 ionyl-leucyl-phenylalanine (fMLP)-stimulated respiratory burst activity and (3)H-DG uptake are tempor

50 TNFalpha and LPS primed respiratory burst activity and increased membrane expres

51 Respiratory burst activity and phosphorylation of an NAD

52 ocytosis contributes to phagocytosis-induced respiratory burst activity and plays a critical role in

53 ed the ability of Akt to regulate neutrophil respiratory burst activity and to interact with and phos

54 a reduced capacity to phagocytose or exhibit respiratory burst activity following mycobacterial-Ag or

55 (phox) phosphorylation, which contributes to respiratory burst activity in human neutrophils.

56 class II beta-chain, and STAT1, and enhanced respiratory burst activity in macrophages.

57 m by which TNFalpha and LPS prime neutrophil respiratory burst activity is by increasing membrane exp

58 TNFalpha failed to prime respiratory burst activity or to increase membrane CD35

59 uptake compared with fMLP without affecting respiratory burst activity, and that fMLP stimulation of

60 ucts (AGEs) enhance NADPH oxidase, and hence respiratory burst activity, of stimulated neutrophils.

61 gp91(phox) are rate-limiting components for respiratory burst activity, our studies may identify rat

62 -induced chemotaxis but has little effect on respiratory burst activity.

63 phage lineages and exhibits phagocytosis and respiratory burst activity.

64 ial for life span extension, chemotaxis, and respiratory burst activity.

65 ssive protection model and induce neutrophil respiratory burst activity.

66 assessment was made of neutrophil function (respiratory burst, adhesion molecule expression, and che

67 Here we optimize the antibody-dependent respiratory burst (ADRB) assay, which assesses the abili

68 ymorphonuclear neutrophil (neutrophil [PMN]) respiratory burst after trauma and hemorrhagic shock (T/

69 ssessed no ability to augment the neutrophil respiratory burst alone.

70 ANCAs activate neutrophils inducing their respiratory burst and a peculiar form of cell death, nam

71 n RAW264.7 macrophages during the phagocytic respiratory burst and A431 cells in response to EGF stim

72 , which regulates activity of the neutrophil respiratory burst and actin assembly.

73 esulted in an impaired ability to activate a respiratory burst and also inhibited chemotaxis.

74 uppression in vitro through the induction of respiratory burst and apoptosis.

75 syk(-/-) neutrophils showed normal respiratory burst and degranulation in response to the b

76 Thus, separate signals control the respiratory burst and degranulation, and a normal rate o

77 with bacterial coinfection showed decreased respiratory burst and killing activity against H. influe

78 The respiratory burst and NO radical (NO.) made distinct con

79 including complement, mononuclear phagocyte respiratory burst and phagocytosis through retargeting o

80 ClC-3 was specifically involved in the respiratory burst and phagocytosis.

81 TF expression in neutrophils contributes to respiratory burst and subsequent trophoblast injury and

82 triggered by the release of H2O2 during the respiratory burst and that induces the uptake of GSH int

83 . phagocytophilum does not suppress a global respiratory burst and that, under identical conditions i

84 al macrophages to E. coli and induced both a respiratory burst and the release of lysozomal enzyme fr

85 0 and PD098059, revealed that priming of the respiratory burst and up-regulation of flavocytochrome b

86 ith impaired neutrophil adhesion, migration, respiratory burst, and degranulation in vitro.

87 an neutrophils, IL-8 induces chemotaxis, the respiratory burst, and granule release, and enhances cel

88 hibited compromised phagocytosis, attenuated respiratory burst, and impaired fungicidal activity in v

89 ted responses such as in vitro phagocytosis, respiratory burst, and in vivo thrombocytopenia, we inve

90 CR3 (CD11b/CD18), enhanced the intracellular respiratory burst, and increased levels of Rac2 activati

91 cisella phagosomal escape, inhibition of the respiratory burst, and intracellular survival.

92 ogates the AGE-enhanced activated neutrophil respiratory burst, and it is demonstrably stimulated in

93 included CD11b activation and up-regulation, respiratory burst, and shape changes.

94 such as tight adhesion, spreading, sustained respiratory burst, and specific granule release in vitro

95 Adenosine inhibited the respiratory burst, and, in cocultures, adenosine deamina

96 gamma), and IL-1 alpha; increased neutrophil respiratory burst; and, ultimately, increased clearance

97 eems to mediate the AGE-augmented neutrophil respiratory burst (ascertained by chemiluminescence).

98 lity in the luminol, but not the isoluminol, respiratory burst assays following stimulation with phor

99 r full-length forms was also demonstrated in respiratory burst assays, CD11b Ag expression, and intra

100 The classic respiratory burst at fertilization is the result of prod

101 Reactive oxygen species generated by respiratory burst attack iron-sulfur cluster-containing

102 nt PMN cannot generate an adhesion-dependent respiratory burst, because of markedly diminished integr

103 nyleneiodonium not only blocked a productive respiratory burst but also abrogated the survival advant

104 ation and escape response and the neutrophil respiratory burst but with little increase in the solubl

105 hagocytophilum did not produce a significant respiratory burst, but A. phagocytophilum did not inhibi

106 timulates neutrophil chemotaxis and a robust respiratory burst, but other aspects of this interaction

107 Therefore, HGE bacteria repress the respiratory burst by down-regulating gp91phox, the first

108 and plays a critical role in priming of the respiratory burst by increasing expression of membrane c

109 persists within neutrophils and prevents the respiratory burst by inhibiting gp91(phox).

110 that PMA activates the H+ efflux during the respiratory burst by modulating the properties of H+ cha

111 opsonized Burkholderia induced a significant respiratory burst by neutrophils compared to unopsonized

112 gp91phox levels and a consequent decline in respiratory burst capability.

113 se in apoptotic frequency and an increase in respiratory burst capacity, consistent with in vivo "pri

114 plained and unexpected defects in neutrophil respiratory burst, chemotaxis and calcium flux, in respo

115 Both have impairments in their neutrophil respiratory burst, chemotaxis response, and calcium flux

116 in modulating neutrophil function, including respiratory burst, chemotaxis, and apoptosis.

117 patients also have defects in the neutrophil respiratory burst, chemotaxis, and calcium flux.

118 ta led to neutropenia; defects in neutrophil respiratory burst, chemotaxis, and calcium flux; and inc

119 mpairment is reflected in reduced neutrophil respiratory burst, chemotaxis, and calcium mobilization.

120 form conventional neutrophil functions, like respiratory burst, chemotaxis, and phagocytosis.

121 -/-) macrophages having impairments in their respiratory burst, chemotaxis, calcium flux, and phagocy

122 ocytes from infected mice had an ineffective respiratory burst compared with 1%+/-1% (mean+/-SD) of t

123 (p <.05) phorbol myristate acetate elicited respiratory burst compared with buffer or T/SS.

124 n inability to kill bacteria and a defective respiratory burst compared with children without bacteri

125 d significant elevations in MIP/PAF-elicited respiratory burst compared with T/HS lymph or buffer onl

126 With stimulation of the respiratory burst, cytosolic oxidase components, p47(pho

127 ctivated by IFN-gamma or by macrophages from respiratory burst-deficient mice.

128 sion, adhesion and migratory responsiveness, respiratory burst, degranulation, and calcium mobilizati

129 syk(-/-) neutrophils failed to undergo respiratory burst, degranulation, or spreading in respon

130 CGD mice, lacking a respiratory burst, developed accentuated colitis compare

131 he NBT and Fc-Oxyburst assays could detect a respiratory burst during A. phagocytophila infection.

132 GO:0010200 (response to chitin), GO:0002679 (respiratory burst during defence response) and GO:003555

133 We describe the fungal respiratory burst during host infection, paralleled by s

134 gonococcal susceptibility to the phagocytic respiratory burst during infection and that gonococcal c

135 peritoneal-resident macrophages to maintain respiratory burst during phagocytosis via enhancing mito

136 There was no difference in respiratory burst early after injury.

137 The magnitude of respiratory burst found here paralleled the [Ca2+]i resp

138 Also, DNE led to an increase in respiratory burst frequency after AMPA injection into th

139 8-OH-DPAT transiently increased respiratory burst frequency in Lmx1b(f/f/p) preparations

140 ced leukocyte recruitment, phagocytosis, and respiratory burst functions of innate leukocytes.

141 Leukocyte phagocytosis and respiratory burst functions were enhanced after treatmen

142 RESPIRATORY BURST OXIDASE HOMOLOG F-mediated respiratory burst had a major impact and was a convergin

143 Blocking crystal-activated respiratory burst has, however, no effect on NETs.

144 ive oxygen species (ROS), which requires the respiratory burst homolog RbohB.

145 tis challenge induced a robust intracellular respiratory burst; however, this response did not contri

146 rol appear physiologic because they regulate respiratory burst in a proportional biphasic fashion.

147 ally, vitronectin reduced the silica-induced respiratory burst in AM as determined with chemiluminesc

148 Conversely, phagocytosis did not trigger a respiratory burst in blood monocytes or monocyte-derived

149 cells, cytotoxicity against hepatocytes, and respiratory burst in hepatic leukocytes.

150 ressing cells induced a significantly larger respiratory burst in human neutrophils compared with con

151 nerated during apoptosis inhibited the basal respiratory burst in human neutrophils, and those genera

152 The respiratory burst in interferon-gamma and zymosan-stimul

153 he classic PKC alpha mediates IgG-stimulated respiratory burst in macrophages, whereas the novel PKCs

154 We report here that the respiratory burst in monocytes is accompanied by an incr

155 P. aeruginosa induces a robust respiratory burst in neutrophils that is required for ex

156 f the uracil auxotrophic mutants triggered a respiratory burst in neutrophils, and ingested bacteria

157 those responsible for the pathogen-activated respiratory burst in phagocytes.

158 ivation of the proton conductance during the respiratory burst in phagocytes.

159 ropathogenic Yersinia spp. also inhibits the respiratory burst in PMNs and macrophages, and we show h

160 d association with and induction of a weaker respiratory burst in PMNs from estradiol-treated mice.

161 dose-dependent enhancement on the neutrophil respiratory burst in response to a secondary mechanical

162 OS) from Neisseria meningitidis enhances the respiratory burst in response to formyl-Met-Leu-Phe, an

163 endotoxin, prime neutrophils for an enhanced respiratory burst in response to subsequent stimulation.

164 KC isoforms in IgG-mediated phagocytosis and respiratory burst in the mouse macrophage-like cell line

165 The neutrophil respiratory burst in the presence of A. phagocytophilum

166 Respiratory burst in these cells was Ca2+ dependent and

167 n (Hv1) channels play important roles in the respiratory burst, in pH regulation, in spermatozoa, in

168 uent killing of microbes is initiated by the respiratory burst, in which nicotinamide adenine dinucle

169 We further show that respiratory burst induces antibiotic tolerance in the sp

170 AcpA is a respiratory burst-inhibiting acid phosphatase from the C

171 AcpA of Francisella spp. is a respiratory-burst-inhibiting acid phosphatase that also

172 These data demonstrate respiratory burst inhibition by A. phagocytophila in viv

173 te acetate, which was fully abrogated by the respiratory burst inhibitor diphenyleneiodonium chloride

174 The phagocyte respiratory burst is crucial for innate immunity.

175 Defects in the phagocytic cells' respiratory burst lead to life-threatening infections, i

176 e gp91(phox) gene encodes a component of the respiratory burst NADPH oxidase complex and is highly ex

177 7(phox), an essential component of phagocyte respiratory burst NADPH oxidase.

178 NOS2 (NOS2(-/-)), gp91(Phox) subunit of the respiratory burst NADPH-oxidase complex (Phox(-/-)), or

179 Neither activation of the respiratory burst nor phagocytosis of either latex parti

180 PER activation in vitro slightly reduced the respiratory burst of acidophilic granulocytes and drasti

181 regions of Pyk2, specifically inhibited the respiratory burst of cells responding to tumor necrosis

182 HNA-3a antibodies primed the fMLP-activated respiratory burst of HNA-3a+, but not HNA-3a-, PMNs and

183 that SodC protects B. abortus 2308 from the respiratory burst of host macrophages.

184 of their aerobic metabolism and through the respiratory burst of host phagocytes.

185 The respiratory burst of human neutrophils is primed by a nu

186 the addition of 2 mm glutamine increased the respiratory burst of human PMN stimulated with both phor

187 c disruption of IL-27 signaling enhanced the respiratory burst of macrophages.

188 olarization in the presence of Ac-PGP or the respiratory burst of neutrophils in the presence of a me

189 CBD treatment also attenuated the respiratory burst of neutrophils isolated from chronic p

190 Porphyromonas gingivalis (Pg), stimulate the respiratory burst of neutrophils.

191 ts have revealed a C5a-induced defect in the respiratory burst of neutrophils.

192 ly to be due to increased sensitivity to the respiratory burst of phagocytes but is, instead, due to

193 ze interactions of both molecules during the respiratory burst of phagocytes provided an excellent op

194 f L. monocytogenes by PMN, and inhibited the respiratory burst of PMN compared with vehicle-treated c

195 Granulocytes generate a "respiratory burst" of NADPH oxidase-dependent superoxide

196 cted DC in the presence of inhibitors of the respiratory burst or inhibitors of NO synthase had littl

197 ons, but not for the rapid initiation of the respiratory burst or phagocytosis.

198 It does not correlate with respiratory burst or secretory activity but may reflect

199 ction of superoxide anion, inhibitors of the respiratory burst (or NO production) did not inhibit kil

200 Mutation of Arabidopsis thaliana respiratory burst oxidase (Atrboh) genes eliminated path

201 The level of BRs was closely related to the respiratory burst oxidase 1 (RBOH1)-encoded NADPH oxides

202 l closure was dependent on the production of RESPIRATORY BURST OXIDASE 1 (RBOH1)-mediated hydrogen pe

203 The CYBB gene, which encodes the respiratory burst oxidase component gp91(phox), is expre

204 s are blocked by inhibitors of ROS-producing respiratory burst oxidase enzymes.

205 naling molecules produced by tissue-specific respiratory burst oxidase homolog (RBOH) enzymes to driv

206 be involved in the ROS burst from the plant respiratory burst oxidase homolog (Rboh) of the human ne

207 gical and genetic approaches showed that the RESPIRATORY BURST OXIDASE HOMOLOG (RBOH) pathway and an

208 BZR1 could directly bind to the promoter of RESPIRATORY BURST OXIDASE HOMOLOG 1 (RBOH1), and that RB

209 ECEPTOR-LIKE 3.5 or the mutants deficient in RESPIRATORY BURST OXIDASE HOMOLOG 1 abolished the RKN-in

210 f Ca(2+) wave propagation in the Arabidopsis respiratory burst oxidase homolog D (AtrbohD) knockout b

211 also induced increased levels of Arabidopsis respiratory burst oxidase homolog D (AtrbohD) mRNA, but

212 These studies further identified the RESPIRATORY BURST OXIDASE HOMOLOG D (RBOHD) protein as a

213 g MS, we identified the plant NADPH oxidase, respiratory burst oxidase homolog D (RBOHD), as an in vi

214 as a result of the lower expression level of RESPIRATORY BURST OXIDASE HOMOLOG D (RbohD).

215 This early response is dependent on the respiratory burst oxidase homolog D protein, and the fun

216 re1]) or reactive oxygen species production (respiratory burst oxidase homolog DF [rbohDF]).

217 re associated with increased accumulation of respiratory burst oxidase homolog F (RBOHF)-dependent re

218 Transcripts encoding the Respiratory Burst Oxidase Homolog F, signaling component

219 The RESPIRATORY BURST OXIDASE HOMOLOG F-mediated respiratory

220 nine dinucleotide phosphate, reduced oxidase RESPIRATORY BURST OXIDASE HOMOLOG PROTEIN D (RBOHD) by s

221 n species generation (due to the function of Respiratory Burst Oxidase Homolog proteins D and F) are

222 silencing of a gene encoding NADPH oxidase (Respiratory burst oxidase homolog) in the gox mutants di

223 oxidase homologs of plants termed RBOH (for respiratory burst oxidase homolog).

224 n domains of extracellular ROS donors of the RESPIRATORY BURST OXIDASE HOMOLOGS (RBOH).

225 AtPeps is absent in the double mutant of the respiratory burst oxidase homologs D and F (rbohD rbohF)

226 response to abiotic stress, the key role of respiratory burst oxidase homologs in the integration of

227 hrough the transcriptional induction of four Respiratory Burst Oxidase Homologs TUNEL-positive nuclei

228 ADPH oxidase genes, MtRbohA and MtRbohC (for respiratory burst oxidase homologs), is increased in lat

229 d with reduced expression of the Arabidopsis respiratory burst oxidase homologue AtrbohD and the SA b

230 Arabidopsis respiratory burst oxidase homologues (rboh genes) have b

231 We show that lack of A. thaliana respiratory burst oxidase protein F (AtrbohF; an NADPH o

232 The CYBB and NCF2 genes encode the phagocyte respiratory burst oxidase proteins, gp91PHOX and p67PHOX

233 ox) are components of the phagocyte-specific respiratory burst oxidase that are encoded by the NCF2 a

234 gp91phox is the catalytic subunit of the respiratory burst oxidase, an NADPH-dependent, superoxid

235 6 extends into the neighboring gene encoding respiratory burst oxidase, and (2) a commonly used STA6

236 the essential role of Rac in assembly of the respiratory burst oxidase, invasion through this nonopso

237 talytic subunit of the superoxide-generating respiratory burst oxidase, is regulated by subunits p47(

238 talytic subunit of the superoxide-generating respiratory burst oxidase, is stimulated by the regulato

239 actors, the membrane-bound components of the respiratory burst oxidase, membrane-bound adhesion molec

240 codes gp91Phox; a component of the phagocyte respiratory burst oxidase.

241 During the phagocytic respiratory burst, oxygen is converted to potent cytotox

242 in T/HS plasma increased MIP-2/PAF-elicited respiratory burst (p <.05) compared with UC or T/SS plas

243 actericidal phase, which is dependent on the respiratory burst phagocyte oxidase (phox) is succeeded

244 . pestis uses the T3SS to inhibit neutrophil respiratory burst, phagocytosis, and release of inflamma

245 e.g. debris clearance) and detrimental (e.g. respiratory burst, phagoptosis) consequences.

246 te (NADPH) 2 (NADPH oxidase 2; NOX2), termed respiratory burst (RB).

247 icient neutrophils had an enhanced phagocyte respiratory burst relative to Nbeal2-expressing neutroph

248 The microbial adaptations to the respiratory burst remain poorly understood, and establis

249 crophages, although the phagocytosis-induced respiratory burst remained intact.

250 AGE augmentation of the activated neutrophil respiratory burst requires AA generation, through which

251 cftr gene (Cftr morphants) exhibited reduced respiratory burst response and directed neutrophil migra

252 We show human PMNs generate a respiratory burst response to unopsonized hyphae.

253 also potentiated the IAV-induced neutrophil respiratory burst response.

254 reduced in the presence of inhibitors of the respiratory burst response.

255 HNPs did not increase neutrophil respiratory burst responses to IAV.

256 eater increases in neutrophil uptake of, and respiratory burst responses to, IAV than MBL.

257 arly (CD38 and CD11b) and late (neutrophilic respiratory burst) responses.

258 PKC inhibition during the respiratory burst reversed the activation of both molecu

259 The pir-b-/- neutrophils displayed enhanced respiratory burst, secondary granule release, and a hype

260 Lyn(-/-) neutrophils displayed enhanced respiratory burst, secondary granule release, and a hype

261 cytotoxic effector mechanisms, including the respiratory burst, secretion of inflammatory mediators a

262 obial and cytotoxic functions, including the respiratory burst, secretion, and apoptosis.

263 ological functions, including the phagocytic respiratory burst, sperm motility, apoptosis, and metast

264 el role for RhoG in signaling the neutrophil respiratory burst stimulated by G protein-coupled recept

265 ta indicate that the NADPH oxidase-dependent respiratory burst stimulated by Pseudomonas infection co

266 olesterol availability, Ca(2+) signaling and respiratory burst suggest that Ca(2+) influx and PMN act

267 by genetic abrogation of the host phagocyte respiratory burst, suggesting that the sigmaE regulon pl

268 gested by PMNs and induce a rapid and strong respiratory burst that is comparable to PMA.

269 ation with fresh serum, Hp triggers a modest respiratory burst that is confined to the phagosome, and

270 he macrophage, the bacteria must survive the respiratory burst that produces superoxide.

271 During the "respiratory burst," the NADPH oxidase complex of phagocy

272 ated pro-survival phenotype with an enhanced respiratory burst thought to contribute to ARDS pathophy

273 ed the relative contribution of PMNs and the respiratory burst to "inflammatory hypoxia" in vivo.

274 beta(1) integrins to include modulating PMN respiratory burst to a pathogen-associated molecular pat

275 not protein kinase C zeta in generating the respiratory burst to beta-glucan.

276 beta(1) integrin ligands did not affect respiratory burst to ingestible beta-glucan-containing p

277 Priming neutrophils for an enhanced respiratory burst together with promoting granule conten

278 TF) expression in neutrophils contributes to respiratory burst, trophoblast injury, and pregnancy los

279 urium did not cause an increase in the early respiratory burst under unprimed or primed conditions, a

280 ole in mediating p38-dependent activation of respiratory burst upon stimulation of Fc gamma RIIIb in

281 tively activates K-ras during induction of a respiratory burst via pathways involving multiple upstre

282 PMN respiratory burst was assessed using the nitro-blue tetr

283 This respiratory burst was associated with increased expressi

284 production also was rescued, and a deficient respiratory burst was corrected.

285 Finally, respiratory burst was dependent on Lyn and membrane raft

286 e role of exocytosis in the human neutrophil respiratory burst was determined using a fusion protein

287 haride on in vivo leukocyte phagocytosis and respiratory burst was examined.

288 PMN respiratory burst was initiated by using macrophage infl

289 Plasma membrane-associated respiratory burst was measured by reduction of ferricyto

290 The enhanced respiratory burst was phosphoinositide 3-kinase-dependen

291 ffer significantly between the 2 groups, but respiratory burst was significantly less (by 28%) in iro

292 ) antiport regulates pH during the phagocyte respiratory burst, we show here that voltage-gated proto

293 uding cytokine production, phagocytosis, and respiratory burst were globally impaired in macFoxp1tg c

294 (killing, phagocytosis, transmigration, and respiratory burst) were used to assess the effects of pr

295 unctional, as demonstrated by stimulation of respiratory burst when neutrophils adhered to surfaces c

296 agocytophilum did not inhibit the neutrophil respiratory burst when phorbol myristate acetate was add

297 (-/-) double-knockout mice failed to undergo respiratory burst when plated on mVCAM-1.

298 db/db neutrophils demonstrated a diminished respiratory burst when stimulated with S. aureus.

299 ction cooperatively in phagocytes during the respiratory burst, when reactive oxygen species are prod

300 Endotoxic stimulation induced a respiratory burst with the production of superoxide and