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1 rotid body (CB) connected with the brainstem respiratory center.
2 of nerve fibers terminating in the brainstem respiratory center.
3 ct as acute hypoxia sensors in the medullary respiratory center.
4 he VMS and they innervate key pontomedullary respiratory centers.
5 , initiates apnea upon invasion of brainstem respiratory centers.
6 f an A1 adenosine receptor antagonist to the respiratory centers.
8 tive to neuromodulatory inputs from multiple respiratory centers, and thus they serve as a key nexus
9 lly dyspneic and have an increase in overall respiratory center drive, as a result of increased lung
12 cally when disconnected from the rest of the respiratory centers in vivo (KYN treatment) and in vitro
13 ndicating that invasion of SD into medullary respiratory centers initiated apnea and hypoxia rather t
14 ed carotid sinus nerve discharge rate to the respiratory centers of the brain, (2) intrinsic hypoxic
15 euroinflammatory response to COVID-19 in the respiratory centers of the brain, and (2) to assess the
16 y environment, warranting exploration of the respiratory centers of the brain, namely the pons and me
17 lutamatergic neurons that innervate the main respiratory centers of the medulla oblongata and pons.
18 e mechanisms include the way dyspnea and the respiratory centers respond to low levels of oxygen, the
19 dback system, defined as the response of the respiratory center's output to changes in arterial blood
21 lief that air hunger results from heightened respiratory center stimulation combined with the incapac
22 rinsic respiratory rhythm from the brainstem respiratory centers to periodic mechanical insufflations