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1  dystrophies, which are often accompanied by respiratory dysfunction.
2 tribute to subsequent swallowing, voice, and respiratory dysfunction.
3 ion manifested later than cardiovascular and respiratory dysfunction.
4 oteomic changes that accompany mitochondrial respiratory dysfunction.
5 store normocapnia regardless of the cause of respiratory dysfunction.
6 n be fatal, including premature death due to respiratory dysfunction.
7 s and showed a higher level of mitochondrial respiratory dysfunction.
8  targets of alcohol-induced toxicity causing respiratory dysfunction.
9 g injury (ALI) results in severe, persistent respiratory dysfunction.
10 resents a reliable tool for the diagnosis of respiratory dysfunction.
11 f activating 5-HT1A receptors on post-C5 SCI respiratory dysfunction.
12 ation regulatory proteins, and mitochondrial respiratory dysfunction.
13 nd young children with NMD may contribute to respiratory dysfunction.
14 c diaphragm remodeling in obesity-associated respiratory dysfunction.
15 atory musculature, which culminate in severe respiratory dysfunction.
16  plays a major role in lung inflammation and respiratory dysfunction.
17 , including delayed communication skills and respiratory dysfunctions.
18 ld asthmatic children and adults with severe respiratory dysfunctions (7, 5, and 3 mug.h(-1), respect
19                                              Respiratory dysfunction after cervical spinal cord injur
20 al injury and the magnitude of mitochondrial respiratory dysfunction after lipopolysaccharide treatme
21 translational implications for patients with respiratory dysfunction after SCI.
22 ore sensitive to MPTP-mediated mitochondrial respiratory dysfunction and complex I inhibition than ce
23 CYP2D6 were more sensitive to toxin-mediated respiratory dysfunction and complex I inhibition than ce
24 tation would alter hemorrhagic shock-induced respiratory dysfunction and correlate with nuclear facto
25 n of mild hypothermia attenuates cardiac and respiratory dysfunction and counteracts sympathetic acti
26 Phosphocreatine in brainstem correlated with respiratory dysfunction and disability; in muscle, energ
27  in Clara cells of SP-B -/- mice resulted in respiratory dysfunction and invariable neonatal death, r
28 g mutations have a higher incidence of awake respiratory dysfunction and lower levels of cerebrospina
29  weakness starting in childhood, restrictive respiratory dysfunction and prominent contractures, alth
30 mbulatory difficulties, vocal fold weakness, respiratory dysfunction and skeletal involvement.
31 eased production of reactive oxygen species, respiratory dysfunction, and loss of cytochrome c oxidas
32 agmentation, cardiomyocyte and mitochondrial respiratory dysfunction, and rapidly progressive and let
33  mitochondria showed increasing irreversible respiratory dysfunction as well as diminished calcium bu
34  a relevant mechanism that may contribute to respiratory dysfunctions associated with obesity.
35 educed fetal growth, cardiovascular disease, respiratory dysfunction, asthma, sensitization to common
36                  Consistent with the lack of respiratory dysfunction, ATP content of cultured striata
37 death in epilepsy (SUDEP) has been linked to respiratory dysfunction, but the mechanisms underlying t
38  postnatal mortality, infertility and strong respiratory dysfunction caused by defective mucociliary
39                                              Respiratory dysfunctions caused by obesity were original
40  is increasing evidence that seizure-induced respiratory dysfunction contributes to the pathophysiolo
41 mplications of pain, nausea and vomiting and respiratory dysfunction, differences between anaesthetic
42 ses occur in the cervical region, leading to respiratory dysfunction due to damaged neural circuitry
43  mild hypothermia impacts on circulatory and respiratory dysfunction during experimental endotoxemia.
44      The mechanisms coupling hypertension to respiratory dysfunction during sleep remain, however, la
45                           Centrally mediated respiratory dysfunction has been observed to lead to dea
46 l material in stimulating IgE production and respiratory dysfunction in a C57BL/6 murine model of AHR
47          Despite this, mechanisms underlying respiratory dysfunction in DS are unknown.
48          Data revealed two general stages of respiratory dysfunction in Mecp2(-/y) mice.
49 d functionally abnormal mitochondria induced respiratory dysfunction in Mfn2-deficient mouse embryoni
50 tory muscle weakness is the primary cause of respiratory dysfunction in neuromuscular disease (NMD),
51  there was evidence of significant vocal and respiratory dysfunction in the RLN transection group, bu
52 ible for a distinct program of mitochondrial respiratory dysfunction, in addition to the activation o
53                                              Respiratory dysfunction is a common complication of obes
54                                              Respiratory dysfunction is a common endpoint for disabil
55                                              Respiratory dysfunction is a hallmark of the disease, mu
56                                              Respiratory dysfunction is a key hallmark of NPD, yet th
57                                              Respiratory dysfunction is a notorious cause of perinata
58                               Opioid-induced respiratory dysfunction is a significant public health b
59 nd, consequently, suggest that mitochondrial respiratory dysfunction is not essential for HD pathogen
60                                              Respiratory dysfunction is one of the most common causes
61 opioid receptors as a possible source of the respiratory dysfunction manifested in panic attacks occu
62 enfluramine, suggesting that seizure-induced respiratory dysfunction may be due to impairment of sero
63 r both, and 4 = severe generalized weakness, respiratory dysfunction, or both.
64 enotypes but, surprisingly, not weight loss, respiratory dysfunction, or premature lethality.
65 ubiquitination and death, forelimb motor and respiratory dysfunction, reactive astrocytosis, and redu
66 iated with a higher probability of worsening respiratory dysfunction scores the following day.
67 ve facial appearance, as well as cardiac and respiratory dysfunction that can be life-threatening.
68 ing the severe eosinophilic inflammation and respiratory dysfunction that is typical of AERD.
69  SCI for preventing PhMN loss and consequent respiratory dysfunction that occurs during secondary deg
70 on Assessment Method for ICU), for renal and respiratory dysfunction (using the ordinal renal and res
71                           In three patients, respiratory dysfunction was part of an early-onset multi
72 certain clinical features, like dysphonia or respiratory dysfunction, were exclusively detected in th
73                                              Respiratory dysfunction, while not an initial symptom in
74 ld result in survival of infants with severe respiratory dysfunction who would otherwise have died.