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1 latile anesthetic, fatigue, active time, and respiratory function.
2 e amount of contractile tissue available for respiratory function.
3 mpiled using simple measures of physical and respiratory function.
4 nal medulla, and for their control of cardio-respiratory function.
5 igand binding are what ultimately drives the respiratory function.
6 armacologic activation of TrkB would improve respiratory function.
7 thin the lung interstitium leads to impaired respiratory function.
8 e expertise in caring for patients with poor respiratory function.
9  many nuclear genes specifying mitochondrial respiratory function.
10 it alters isolated muscle kinetics, limiting respiratory function.
11 of apoE4 domain interaction on mitochondrial respiratory function.
12 d hippocampus were isolated and assessed for respiratory function.
13 abile zinc and interferes with mitochondrial respiratory function.
14 itochondrial RNA stability, translation, and respiratory function.
15 ns in respiratory supercomplex structure and respiratory function.
16 of scapular winging, contractures and normal respiratory function.
17 he effects of their toxic exoproducts impede respiratory function.
18 n, this measurement is limited as a guide to respiratory function.
19 f pulmonary integrity and edema compromising respiratory function.
20 igh-amplitude swelling and exhibited altered respiratory function.
21  obtained by spirometry, were used to assess respiratory function.
22 cores for AEX, an alternative measurement of respiratory function.
23  mutations caused little or no effect on the respiratory function.
24        All presented with a deterioration in respiratory function.
25 ruction would not lead to a deterioration in respiratory function.
26 DNA and protein content, gene expression, or respiratory function.
27 consequent barotrauma, resulting in improved respiratory function.
28 SH, but fully restored mitochondrial GSH and respiratory function.
29 ling extubation demonstrate abnormalities of respiratory function.
30 and chest wall are important determinants of respiratory function.
31 ial DNA maintenance, and it is essential for respiratory function.
32 rsible inhibition of cardiac contractile and respiratory function.
33 ving only a marginal effect on mitochondrial respiratory function.
34 ial protein involved in iron homeostasis and respiratory function.
35 -related SNPs were linked to lung cancer and respiratory function.
36 lations is a powerful strategy for restoring respiratory function.
37  both the reticular shadows in the lungs and respiratory function.
38 and improved force generation, myotonia, and respiratory function.
39 ent to restore ATP content without restoring respiratory function.
40 ival may be mediated by the drug's effect on respiratory function.
41  (NMB) expressing neurons in the RTN altered respiratory function.
42 t reduction in gas exchange surface area and respiratory function.
43             Physical therapy did not improve respiratory function.
44 ndria from these cells appear to have normal respiratory function.
45 essive measures of skeletal muscle power and respiratory function.
46 y barrier cells that hinders regeneration of respiratory function.
47 t (LS) is an essential system supporting the respiratory function.
48  severe respiratory diseases and compromised respiratory function.
49 s, and a progressive decline in mobility and respiratory function.
50 re non-viable, emphasizing the importance of respiratory function.
51 unctional Rating Scale-Revised score and the respiratory function.
52 cal role for Oma1 protease in fine-tuning of respiratory function.
53 coactivator-1alpha (PGC-1alpha) and enhanced respiratory function.
54 ormal structure and displayed a 35-43% lower respiratory function.
55  were clinically satisfactory, with adequate respiratory function.
56 ed to immediate and sustained improvement of respiratory function.
57 sociations of exposure to air pollution with respiratory function.
58 in lungs, participating in the regulation of respiratory functions.
59 (mtErbB2) negatively regulates mitochondrial respiratory functions.
60 tioning in the case of immune, hormonal, and respiratory functions.
61 e, temperature, humidity, body movement, and respiratory functions.
62 o the deterioration of lung architecture and respiratory functions.
63 ucial roles in modulating cardiovascular and respiratory functions.
64  of exogenous cytochrome c markedly restored respiratory functions.
65 the expression of numerous genes involved in respiratory functions.
66 ucing glucose availability and mitochondrial respiratory functions.
67 ons, which support other respiratory and non-respiratory functions.
68  it robustly measures the brain, cardiac and respiratory functions.
69 r discharge to induce appropriate changes in respiratory function (1).
70 latation, he is alive and has a satisfactory respiratory function 15 mo after transplantation.
71  at presentation and 20% routinely monitored respiratory function; 32% relied on symptoms as the only
72 to common allergens, and to undergo tests of respiratory function, 659 women and 500 men agreed to an
73 ty (4.2%; P<0.05), tolerance to calcium, and respiratory function (72% increase in state 3 and 23% in
74 hore crabs in maintaining osmoregulatory and respiratory function after acute exposure to both anthro
75 reported significant abnormalities in infant respiratory function after clinical recovery from bronch
76 ogram or higher had negligible impairment of respiratory function after infection, whereas control an
77 epresent an important strategy for improving respiratory function after spinal cord injury.
78 IBG pretreatment also improved mitochondrial respiratory functions after cold preservation.
79 in neural centers critical for autonomic and respiratory function alleviates the lethality associated
80 ng was defined as good cognitive, motor, and respiratory functioning, along with absence of disabilit
81 osition may play a role in the impairment of respiratory function among the abdominally obese.
82 ial permeability transition, decline in both respiratory function and activity of cytochrome c oxidas
83 ding the mechanisms regulating mitochondrial respiratory function and adaptations to metabolic challe
84 rectly affects acid-base and ion regulation, respiratory function and aerobic performance in aquatic
85 eased glycolysis, but impaired mitochondrial respiratory function and biogenesis.
86 egration of pathways directing mitochondrial respiratory function and cell growth.
87    Consequently, inhibition of HsPDF reduces respiratory function and cellular ATP levels, causing de
88 hyperaccumulation, and restore mitochondrial respiratory function and cytochrome c levels.
89 in (0.2%) in 2 month old mdx mice to improve respiratory function and end-point functional and histol
90 tion, improved motor performance, normalized respiratory function and extended lifespan up to an eigh
91 hmogenic networks, their respective roles in respiratory function and how they interact to constitute
92 rapy might provide benefits, particularly in respiratory function and in patients who have been recei
93 sphate (ATP); they had reduced mitochondrial respiratory function and increased oxidative stress, com
94                                    Decreased respiratory function and increased reactive oxygen speci
95 (P < .0001) and a faster improvement in both respiratory function and inflammatory markers.
96                            Breeding for good respiratory function and maintaining a healthy bodyweigh
97                                Mitochondrial respiratory function and metabolism were assessed in sap
98 owever, the impact of single LT on long-term respiratory function and nutritional status has not been
99                                         Poor respiratory function and obesity are associated with all
100 sts that loss of Lon1 significantly modifies respiratory function and plant performance by small but
101 es mitophagy and improves both mitochondrial respiratory function and proteostasis in aged flies.
102 of treatments have been developed to improve respiratory function and quality of life in patients wit
103                 Aberrations in mitochondrial respiratory function and reactive oxygen species (ROS) l
104 al cells to C5a led to altered mitochondrial respiratory function and reactive oxygen species generat
105     Late diagnosis significantly worsens the respiratory function and reduces the chance for normal d
106                Idebenone reduced the loss of respiratory function and represents a new treatment opti
107 process, which is necessary to regain normal respiratory function and restore the lungs to homeostasi
108  strong correlation between DM1 genotype and respiratory function and skeletal muscle power, as part
109 increases survival, rescues abnormalities in respiratory function and social recognition, and improve
110 ay enable frataxin to simultaneously promote respiratory function and stress tolerance.
111 h failure is associated with worse long-term respiratory function and survival(2,3), the determinants
112                                     Improved respiratory function and symptoms have lasted up to 144
113              Effect estimates for changes in respiratory function and symptoms were pooled by using f
114 raction between the disruption of this novel respiratory function and the loss of wild-type mtDNA.
115 hysiological basis of these vocalizations in respiratory function and to additional developmental var
116 hile sprint-interval training (SIT) improves respiratory function and upregulates pathways involved i
117 t time a physiological improvement of cardio-respiratory functions and a correction of behavioral fea
118         We examined the correlations between respiratory function, and ACT and AHQ-JAPAN.
119 ities of Fe-S cluster enzymes, a decrease in respiratory function, and an increase in oxidative stres
120 chondrial protein degradation, mitochondrial respiratory function, and cell viability are compromised
121 l muscle pathophysiology, improves motor and respiratory function, and normalizes systemic metabolism
122 ochrome b, several mutations that impair the respiratory function, and reversions that correct the de
123             Motor and cognitive development, respiratory function, and safety were evaluated, as well
124  have a role in maintenance of mitochondrial respiratory function, and this function is analogous to
125 ipid metabolism, impair muscle mitochondrial respiratory function, and uncouple oxygen consumption fr
126 piratory components for different aspects of respiratory functions, and collectively for the integrit
127 tigated whether defects in the mitochondrial respiratory function are consequences of the expression
128        Strategies to preserve and/or restore respiratory function are critical for successful treatme
129 ams that govern mitochondrial biogenesis and respiratory function are well known, posttranscriptional
130 ntilation, reduced lung injury with improved respiratory function, as compared with protective contro
131  the nos mutant likely resulted from altered respiratory function, as inhibition of NADH dehydrogenas
132  of nuclear genes required for mitochondrial respiratory function, as well as for other fundamental c
133  status, Do-Not-Resuscitate status, impaired respiratory function, ascites, hypoalbuminema, elevated
134                       Our aim was to compare respiratory function at 1 yr of age in infants assigned
135  treated with neonatal caffeine had improved respiratory function at 11 years of age compared with ch
136 t improved diaphragm muscle regeneration and respiratory function at 14 wk but not at 6 mo.
137 vidually and in groups, and ALS function and respiratory function at diagnosis.
138      38% of responding neurologists assessed respiratory function at presentation and 20% routinely m
139  fusion and fission events that can maintain respiratory function at steady-state levels amid the exi
140 eption of feeling secure, and improvement of respiratory function at the end of an SBT (most performe
141 etwork area but elicited modest effects upon respiratory function at the same time as influencing gly
142                    Metformin inhibited basal respiratory function at the same time as significantly e
143 ia in the critically ill can help to improve respiratory function, bowel function, mental status and
144 ic administration of methylxantines improves respiratory function but also leads to the development o
145  mitochondrial ROS and rescues mitochondrial respiratory function but also robustly suppresses tau ol
146 B. cepacia can lead not only to a decline in respiratory function but also to an acute systemic infec
147 t was not due to deficiency of mitochondrial respiratory function but was rather caused directly by t
148 as well as with aspirin-induced symptoms and respiratory function, but not with nasal mediators.
149 bstance P modulates the reflex regulation of respiratory function by its actions both peripherally an
150 group, patients underwent daily screening of respiratory function by physicians, respiratory therapis
151       Cpat is essential to the mitochondrial respiratory function by targeting key metabolic enzymes
152                          Bedside measures of respiratory function can predict extubation success and
153                      Bedside measurements of respiratory function can predict extubation success and
154                                    Decreased respiratory function, cardiac involvement, and intranucl
155 ransport measurement and clinical condition (respiratory function, chest radiograph score, or Shwachm
156 less secure and reported less improvement of respiratory function compared with nurses' and physician
157 hr108Met showed decreased motor, bulbar, and respiratory function, compared to a dramatic improvement
158  were associated with impaired mitochondrial respiratory function concurrent with reduced iron regula
159 s that TMZ-mediated alterations in mtDNA and respiratory function contribute to TMZ-dependent acquire
160 atures, including multiorgan dysfunction and respiratory function, could be predicted using combined
161 ter neonatal bronchopulmonary dysplasia, and respiratory function differences were similar at 11 and
162 al discharge, motor function improves, while respiratory function does not, being accompanied by evid
163 hortened spine and trunk can severely affect respiratory function during early childhood.
164  responsive to treatments aimed at restoring respiratory function, eliciting the mitochondria as a dr
165 MM, including anticancer, anti-inflammatory, respiratory function enhancement, antioxidant, anti-agin
166 t age-dependent decline in the mitochondrial respiratory function, especially COX activity, may parti
167 nt effects on blood pressure, pulse rate, or respiratory function (FEV1).
168       To determine the effects of sleep upon respiratory function, five adult patients with cystic fi
169     Oral quercetin supplementation protected respiratory function for 4-6 months during a 12 month do
170                 Aged animals showed impaired respiratory function for 45 days, mounted a weaker antiv
171 ded into those above and those below average respiratory function for their age.
172                 There was a wide spectrum of respiratory function, from normal to markedly abnormal.
173                                              Respiratory function, growth, and cognitive and motor fu
174 s examining the effect of sternal closure on respiratory function have not been published, and curren
175 erosolized brevetoxins in sea spray (reduced respiratory function); however, the reason for brevetoxi
176 g Cox1 synthesis in coa2Delta cells restores respiratory function if Cox10 protein levels are elevate
177                                      Loss of respiratory functions impairs Candida albicans colonizat
178                                          The respiratory function improvement was found to be associa
179 yridine and salbutamol resulted in motor and respiratory function improvement.
180 een identified a gene, CCC1, that maintained respiratory function in a Deltayfh1 yeast strain regardl
181 mmatory protein-2 significantly improves the respiratory function in addition to decreasing the infil
182 ed for those interested in cardiovascular or respiratory function in addition to motor function and c
183   This modified CHCHD4 protein reestablished respiratory function in AIF-deficient cells and enabled
184                    Given the high demands of respiratory function in athletes, insight into triggerin
185 ficant role in mitochondrial positioning and respiratory function in cardiac and skeletal muscle.
186  contribution of S. aureus to the decline in respiratory function in children with CF is not elucidat
187 poorer birth outcomes, neurodevelopment, and respiratory function in children.
188                                              Respiratory function in hem15Delta cells can also be res
189 heme proteins, doming is associated with the respiratory function in hemoglobin and myoglobins.
190 onchopulmonary dysplasia, but its effects on respiratory function in later childhood are unknown.
191 erved significantly lower static and dynamic respiratory function in LOPD.
192  of nuclear genes required for mitochondrial respiratory function in mammalian cells.
193 yperexpanded lung, has been shown to improve respiratory function in many patients with end-stage emp
194 replication, transcription processivity, and respiratory function in normal cells.
195 with alveolar recruitment maneuvers improves respiratory function in obese patients undergoing surger
196 d magnesium sulphate (MgSO(4)) might improve respiratory function in patients with acute asthma.
197 ruitment, is a major cause of the decline in respiratory function in patients with CF and is a leadin
198 ajor differences in objective assessments of respiratory function in patients with moderate or severe
199  to the last weeks of gestation by comparing respiratory function in preterm infants whose mothers ha
200 at NF1 is required to maintain mitochondrial respiratory function in Schwann cells by stabilizing NAD
201 ion between abdominal pattern of obesity and respiratory function in the European Prospective Investi
202 excessive sedation, and maintaining adequate respiratory function in the face of compromise resulting
203  Nurr1 has a key function in sustaining high respiratory function in these cells, and that Nurr1 abla
204          It allows monitoring of a patient's respiratory function in ways which are not possible thro
205 tified target genes, and those that regulate respiratory functions in melanocytes are among them.
206 hese results suggest that BCL-2 can regulate respiratory functions in response to mitochondrial distr
207 infected cells were adjacent to neurons with respiratory functions in the medulla.
208 ity of single neurons related to cardiac and respiratory functions in three subcortical regions: vent
209  resD mutant phenotypes, directly related to respiratory function, include streptomycin resistance, l
210    With increasing mutant ND5 mtDNA content, respiratory function including oxygen consumption and AT
211 n favor of HFOV in several other measures of respiratory function, including forced expiratory volume
212  mitochondria tolerance to calcium, enhances respiratory function (increases of 90% state 4, 220% sta
213  anti-leukemia agent, inhibits mitochondrial respiratory function, increases free radical generation,
214 hondrial complexes I and IV that limit their respiratory function independent of an upregulation of c
215 tegrates cardiovascular, musculoskeletal and respiratory function into a single index that is largely
216                                 In contrast, respiratory function is comparatively dispensable for me
217                               Thus, impaired respiratory function is evident in infants born on avera
218 ngth is only mildly affected and cardiac and respiratory function is normal.
219                   We show that as the cells' respiratory function is reduced or eliminated, the expre
220                       However, monitoring of respiratory function is suboptimal and uncontrolled oxyg
221                                              Respiratory function is the main cause of mortality in p
222  variation in metabolic, cardiovascular, and respiratory function is unknown.
223                  However, based on assays of respiratory function, it has been reported that the prim
224  with its limited effects on hemodynamic and respiratory function, it is widely used in pediatric int
225 .02), with all exhibiting a rapid decline in respiratory function leading to death.
226 ed ubiquitin and mitophagy markers, and lost respiratory function, leading to neurodegeneration.
227 lar disease, smoking, body mass index grade, respiratory function levels (forced expiratory volume in
228                                   Indices of respiratory function (maximum inspiratory pressure gener
229      This study supports the hypothesis that respiratory function may be compromised after delayed st
230 c attenuation of TGF-beta signaling improves respiratory function, mdx mice were treated from 2 weeks
231 ctional Rating Scale-Revised (ALSFRS-R), and respiratory function, measured using percentage of predi
232  illness who had complete anthropometric and respiratory function measures obtained at a health visit
233                                              Respiratory function, mitochondrial membrane potential,
234 tracted, and effect estimates for changes in respiratory function, MPDA and asthma morbidity were poo
235 e was, however, no significant impairment of respiratory function, no alteration to the structure or
236       These data indicate that mitochondrial respiratory function, not abnormal redox homeostasis, di
237                       Daily screening of the respiratory function of adults receiving mechanical vent
238                                 Although the respiratory function of blood has been studied intensive
239                                              Respiratory function of coa4Delta cells was restored by
240 the essential role of the ND6 subunit in the respiratory function of Complex I and give some insights
241 hed the complex I assembly and disrupted the respiratory function of complex I.
242  on the informatics results, we analyzed the respiratory function of mitochondria isolated from wild-
243 verexpression of Mrs1 partially restores the respiratory function of mne1Delta cells.
244  consumption of Ptpn11(E76K/+) cells and the respiratory function of Ptpn11(E76K/+) mitochondria were
245  cristae architecture is crucial for optimal respiratory function of the organelle.
246       Although the individual structures and respiratory functions of cytochromes are well studied, t
247  fitness variance is not directly related to respiratory functions of Hb.
248 ystemic administration of drugs that improve respiratory function often cause deleterious side effect
249              No differences in mitochondrial respiratory function or content were observed between le
250 er skeletal muscle mitochondrial content and respiratory function or the size of ischemic lesion afte
251 chicine did not improve functional capacity, respiratory function, or inflammatory markers.
252 es in surfactant surface activity, postnatal respiratory function, or survival.
253                   The effect of idebenone on respiratory function outcomes was similar between patien
254  changes in cardiovascular, neurological, or respiratory function, oxygen carrying capacity, and bloo
255 022), associated with improved mitochondrial respiratory function (postischemic percent respiratory c
256 iration in darkness is maximal, beyond which respiratory function rapidly declines) in upper canopy l
257  in the aged rats; (iii) brain mitochondrial respiratory function related to coupled oxidation was de
258 otion, swallow function, vocal function, and respiratory function, respectively.
259 ce mutations greatly accelerated the loss of respiratory function, resulting from enhanced oxidative
260 enal chromaffin cells, which regulate cardio-respiratory function, resulting in irregular breathing w
261 e-radical generation and exhibit declines in respiratory function(s).
262 g, reduced overall lung volume, and improved respiratory function safely and consistently.
263 th in the dark, with both photosynthetic and respiratory functions severely compromised; growth in th
264     Oxygen consumption, a sensitive index of respiratory function, showed that mtDNA from chimpanzee,
265                                              Respiratory function studies demonstrated a gradual dete
266 sm, such as thyroid and parathyroid; and for respiratory function, such as trachea and lung.
267 mphysematous changes and correlate them with respiratory function tests (RFTs).
268                                   ACT score, respiratory function tests and respiratory resistance we
269 an accelerometer during 9 days and undertook respiratory function tests.
270 killing of P. aeruginosa by IgG2 have poorer respiratory function than infected patients who do not p
271 espiratory neuroplasticity after declines in respiratory function that are related to neurological im
272 togenetic experiments identified deficits in respiratory function that were specific to manipulations
273 rogressive and irreversible deterioration of respiratory functions that lacks curative therapies.
274  minimal disturbance of gastrointestinal and respiratory function, thereby reducing the length of int
275 ce of deterioration in nutritional state and respiratory function to prevent further decline.
276                                  In tests of respiratory function, Trpa1(-/-) mice displayed profound
277 h data collected during exposures found that respiratory function varied considerably between rabbits
278                                Mitochondrial respiratory function was analyzed by high-resolution res
279 atory symptoms, total IgE, specific IgE, and respiratory function was assessed by logistic and multip
280                                              Respiratory function was compared within pairs of previo
281 ered relationship between protein import and respiratory function was confirmed through the investiga
282 on, including in the diaphragm muscle, where respiratory function was improved.
283                                              Respiratory function was measured continuously for 30 mi
284                                              Respiratory function was measured in 29 control and 29 a
285                                              Respiratory function was measured in nonanesthetized mic
286                                              Respiratory function was measured using partial and rais
287                                              Respiratory function was measured with a head-out plethy
288                                              Respiratory function was protected for the first 4-6 mon
289 ough intrinsic skeletal muscle mitochondrial respiratory function was unchanged, estimated skeletal m
290  Using barometric plethysmography to measure respiratory function, we found that the MD rat develops
291  understand the transient nature of improved respiratory function, we measured PGC-1alpha pathway act
292 cause cardiolipin plays an important role in respiratory function, we measured the energy transformat
293 in muscle fiber morphology and mitochondrial respiratory function were also assessed.
294 Functional Rating Scale (ALS-FRS) scores and respiratory function were analysed.
295                     Indexes of autonomic and respiratory function were assessed in CB intact and CB d
296 atment, and mitochondrial ultrastructure and respiratory function were assessed.
297 ory symptoms, and laboratory measurements of respiratory function were made for respiratory rate, tid
298                    The clinical outcomes and respiratory function were retrospectively compared betwe
299      The mutation had a severe effect on the respiratory function, with the activity of the bc(1) com
300 levels of mutant huntingtin on mitochondrial respiratory function within an appropriate cellular cont

 
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