ライフサイエンスコーパス: respiratory mucosa

1 and activation of inflammatory cells in the respiratory mucosa.

2 s a key player that regulates the process in respiratory mucosa.

3 Ly6C(hi) monocytes and prime T cells at the respiratory mucosa.

4 nfection (p.i.) in the oral cavity and upper respiratory mucosa.

5 lass switching in B cells of the human upper respiratory mucosa.

6 ulin A (IgA) antibodies in secretions at the respiratory mucosa.

7 ulin A (IgA) antibodies in secretions at the respiratory mucosa.

8 memory CD4+ T cells from the intestinal and respiratory mucosa.

9 nds, although not in the nasal olfactory and respiratory mucosa.

10 e that this highly lethal cancer arises from respiratory mucosa.

11 which begins with colonization of the upper respiratory mucosa.

12 initiates infection by colonizing the upper respiratory mucosa.

13 . mandrillaris can cause lesions in skin and respiratory mucosa.

14 id N-specific T cell recall responses in the respiratory mucosa.

15 ting with minimal viral replication in upper respiratory mucosa.

16 8 T cell memory both systemically and in the respiratory mucosa.

17 tissue-resident memory T cells (TRM) in the respiratory mucosa.

18 t systemic immunity but poor immunity at the respiratory mucosa.

19 host IFN-alpha after viral infection in the respiratory mucosa.

20 ular precursors for IgE-secreting PCs in the respiratory mucosa.

21 portance of VA in regulating immunity in the respiratory mucosa.

22 ganism begins with colonization of the upper respiratory mucosa, a process facilitated by adhesive fi

23 isease begins with colonization of the upper respiratory mucosa, a process that involves evasion of l

24 ccination site and in distal LN draining the respiratory mucosa, although in lower numbers.

25 obligate parasite that lives in human upper respiratory mucosa and can be cultivated only on rich me

26 ant to induce robust mucosal immunity at the respiratory mucosa and can be used to design safe and ef

27 The neutralizing antibody titers in the respiratory mucosa and in the circulation, as well as th

28 coplasma gallisepticum colonizes the chicken respiratory mucosa and mediates a severe inflammatory re

29 ergens prevents inflammatory symptoms in the respiratory mucosa and provides protection against infla

30 and IgA titers in the serum, upper and lower respiratory mucosa, and distal genitourinary mucosae of

31 ocal immunity induced by FMD vaccines at the respiratory mucosa, and local responses induced in vacci

32 Finally, with respiratory mucosa as the initial coronavirus infection

33 We found that a subset of upper respiratory mucosa B cells expressed TLR3 and responded

34 tive TLR3 signaling and ongoing CSR in upper respiratory mucosa B cells from patients with CD40 signa

35 l responses, induces immunoglobulin A at the respiratory mucosa, boosts systemic immunity, and comple

36 occus pneumoniae normally inhabits the upper respiratory mucosa but can also invade and replicate in

37 ses and in activating innate immunity in the respiratory mucosa, but there is no reliable and conveni

38 trate that T cells secreting TGF-beta in the respiratory mucosa can indeed regulate Th2-induced airwa

39 occus pneumoniae, an inhabitant of the upper respiratory mucosa, causes respiratory and invasive infe

40 ubsets of BCs were found in human and murine respiratory mucosa distinguished by the expression of ba

41 jor cause of pneumonia, wherein infection of respiratory mucosa drives a robust influx of neutrophils

42 rect delivery of aerosolized vaccines to the respiratory mucosa elicits both systemic and mucosal res

43 Their positioning in the respiratory mucosa ensures their engagement in the respo

44 The respiratory mucosa formed by the airway epithelium is th

45 Immunization directly to the respiratory mucosa has been shown to promote greater pro

46 umococcal serotypes to diseases of the upper respiratory mucosa in a site-specific manner.

47 Adherence to the respiratory mucosa is a crucial event in its pathogenesi

48 Furthermore, the respiratory mucosa is enriched with extracellular matrix

49 nteractions on disease risk across the upper respiratory mucosa is not known.

50 ophilus influenzae, a commensal of the human respiratory mucosa, is an important cause of localized a

51 us influenzae (Hi), a commensal of the human respiratory mucosa, is an important cause of localized a

52 organelle) directs colonization of the human respiratory mucosa, leading to bronchitis and atypical p

53 dies in rodents suggest that exposure of the respiratory mucosa may be an efficient pathway.

54 duction in its ability to colonize the upper respiratory mucosa of mice.

55 sion was not enhanced in either digestive or respiratory mucosa of pigs following a 2-day infection w

56 infect (or become reactivated in) the skin, respiratory mucosa or other barrier tissues.

57 T cells within the human respiratory mucosa produce IL-10, which is capable of in

58 f immunobiotic Lactobacillus directly to the respiratory mucosa protects mice from the lethal sequela

59 The respiratory mucosa provides a key microbial interface wh

60 How pollens interact with the respiratory mucosa remains largely unknown due to a lack

61 ogens, especially those entering through the respiratory mucosa, such as Mycobacterium tuberculosis.

62 airway smooth muscle, and immunocytes in the respiratory mucosa, suggesting potential advantages of a

63 ctor would elicit protective immunity at the respiratory mucosa, the portal of entry and the primary

64 sal microbiota in regulating immunity in the respiratory mucosa through the proper activation of infl

65 ng from IgM to IgD occurs in the human upper respiratory mucosa to generate IgD-secreting B cells tha

66 mpted a rapid local antibody response in the respiratory mucosa, triggered upon oronasal challenge an

67 ue to their limited antiviral effects in the respiratory mucosa, whereas interventions targeting the

68 molytica with airway epithelial cells of the respiratory mucosa which might explain the different abi

69 sed in response to protective priming of the respiratory mucosa with Lactobacillus plantarum; transcr

70 We have shown previously that priming of respiratory mucosa with live Lactobacillus species promo

71 nalysis revealed restricted infection to the respiratory mucosa with stepwise changes in immune and e

72 Viruses that replicate in the human respiratory mucosa without infecting systemically, inclu