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1 (i.e., tidal volume, minute ventilation, and respiratory rate).
2 duction in dynamic hyperinflation at a lower respiratory rate).
3 adaptation of stimulation to alterations in respiratory rate.
4 ient of variation and autocorrelation of the respiratory rate.
5 gnals, skin temperature, skin hydration, and respiratory rate.
6 ef changes in blood pressure, heart rate, or respiratory rate.
7 HB renal and total clearance, also improving respiratory rate.
8 no effect on blood pressure, heart rate, or respiratory rate.
9 rterial blood pressure, body temperature and respiratory rate.
10 te the trigeminal nerve and evoke changes in respiratory rate.
11 ed by increasing tidal volume and decreasing respiratory rate.
12 oximetry/Fi(O(2)) had a greater weight than respiratory rate.
13 ts of not only volume and pressures but also respiratory rate.
14 e and age-matched WT littermates had similar respiratory rates.
15 eases in arousal, temperature, and heart and respiratory rates.
16 inergic synaptosomes from striatum had lower respiratory rates.
17 lved in the control of maximal mitochondrial respiratory rates.
18 ptoms and panic attacks, as well as elevated respiratory rates.
19 14 points [95% CI, -0.23 to 0.50]; P = .46); respiratory rate (0.17 breaths/min [95% CI, -1.32 to 1.6
20 23; 95% CI, -0.46 to -0.01; P=0.04), resting respiratory rate (-0.7 breaths/min; 95% CI, -1.2 to -0.2
21 ilated with 12 mL/kg tidal volume, 28% FIO2, respiratory rate = 12 breaths/min) were hemorrhaged to <
22 -2.2 L/min vs. 10.1+/-2.9 L/min, p<0.01) and respiratory rate (16+/-5 bpm vs. 19+/-6 bpm, p<0.01) tha
23 y pressure [PEEP], 5 cm H(2)O; Vt, 10 ml/kg; respiratory rate, 20 bpm), 2) conventional-protective (P
24 -protective (PEEP, 10 cm H(2)O; Vt, 6 ml/kg; respiratory rate, 20 bpm), and 3) near-apneic (PEEP, 10
25 s. 16%, p = .003) and had a higher intrinsic respiratory rate (22 breaths/min vs. 18, p = .03), but t
28 d) initial values for pH were 7.08 +/- 0.18; respiratory rate, 35.1 +/- 9.1 breaths/min; PetCO2, 18.6
29 P production rates (29% higher), and maximal respiratory rates (37% higher) compared with WT cells.
30 was independently associated with increased respiratory rate (4%; 95% confidence interval [CI], 1.01
31 sure-controlled mode (tidal volume, 6 mL/kg; respiratory rate, 40; FIO2, 0.6; inspiratory:expiratory,
33 eak airway pressure was similar at the three respiratory rates (66.8 +/- 8.7 vs. 66.4 +/- 9.5 vs. 67.
34 in measuring temperature, 85% for measuring respiratory rates, 98% for diagnosis, 98% for classifica
35 hours preceding ICU admission, with a higher respiratory rate, a more frequent acute kidney injury, a
36 athing 15 L/min of oxygen, plus either [1] a respiratory rate above 30/min or [2] clinical signs sugg
37 respiration was a dose-dependent decrease in respiratory rate, accompanied by an increase in tidal vo
38 matically increased and decreased changes in respiratory rate according to a set protocol: +2, -4, +6
39 lly ventilated (tidal volume V(T) = 8 mL/kg, respiratory rate adjusted to normocapnia) at low (n = 2,
41 ant peripheral oximetry, blood pressure, and respiratory rate alerts from artifacts in an online moni
42 hted average heart rate, blood pressure, and respiratory rate, along with changes-over-time for each.
45 ts have pulmonary hypertension and increased respiratory rates, although the pathophysiological basis
46 und a linear relationship between changes in respiratory rate and big up tri, openPetCO2 as well as b
49 afil, subjects had a significantly decreased respiratory rate and decreased minute ventilation at pea
51 lso is inhibited by NO, thereby reducing the respiratory rate and enhancing local oxygen concentratio
52 i, openPetCO2) as well as between changes in respiratory rate and equilibration time (teq) for mechan
53 ication included higher body temperature and respiratory rate and higher percentage of immature neutr
54 al and hippocampal activation, increased the respiratory rate and hypoglossal nerve activity, induced
55 ome measures were evaluated as a function of respiratory rate and included tidal volume, maximal alve
56 guinea pigs while having no effect on basal respiratory rate and little or no effect on reflexes att
59 eved by reducing tidal volume and increasing respiratory rate and positive end-expiratory pressure.
60 ondrial Ca(2+) handling, membrane potential, respiratory rate and production of reactive oxygen speci
61 eptor-responsive PPTn neurons also increased respiratory rate and respiratory-related genioglossus ac
62 lationship has been found between changes in respiratory rate and resulting changes in end-tidal cO2
71 reased in parabolic fashion as a function of respiratory rate and was maximal at rates of 4.3-6.8 bre
73 ted from the brains of R6/2 mice had similar respiratory rates and Ca(2+) uptake capacity compared wi
75 in lung lavage fluids of infected mice with respiratory rates and measures of outflow obstruction an
76 hat combines the low tidal volume with lower respiratory rates and minimally invasive CO2 removal.
78 ain Assessment Card) and 60-second heart and respiratory rates and sustained change in quality of lif
79 trienes were correlated with both increasing respiratory rates and the degree of prolongation of expi
80 data using Lifetouch (ECG-derived heart and respiratory rate) and WristOx2 (pulse-oximetry and deriv
81 with monitoring of jugular venous pressure, respiratory rate, and arterial oxygen saturation and tre
83 an arterial pressure, rate-pressure product, respiratory rate, and catecholamine levels were all sign
84 presents a short shelf life due to the high respiratory rate, and consequent ripening, which limits
85 splayed markedly reduced locomotor activity, respiratory rate, and energy expenditure, which were not
87 anxiolysis: reduced risk-avoidance, reduced respiratory rate, and increased positive valence, respec
88 ncluding mean arterial pressure, heart rate, respiratory rate, and oxygen saturation) were collected
89 tates, including blood pressure, heart rate, respiratory rate, and oxygen saturation, showed no signi
90 Assessing gas exchange, diaphragm function, respiratory rate, and patient comfort during high-flow o
92 astance as a function of time, tidal volume, respiratory rate, and positive end-expiratory pressure c
93 impaired mitochondrial ATP generation, basal respiratory rate, and spare capacity in microglial cells
94 used unadjusted and adjusted (for age, sex, respiratory rate, and, in analyses of HC data only, Weig
96 nd theta rhythms, instantaneous heart rates, respiratory rates, and sweat pH, uric acid, and glucose,
97 creased to 0.87 (95% CI, 0.71-0.98), whereas respiratory rate area under the curve started at 0.85 (9
98 n each case, indicators of pulmonary damage (respiratory rates, arterial oxygen partial pressures, an
99 ch intestinal ROI was sent for mitochondrial respiratory rate assessment and for metabolites quantifi
100 We show that RSV G glycoprotein reduces respiratory rates associated with the induction of subst
103 ry time (beta = -0.004; P = 0.01), increased respiratory rate (beta = 0.28; P = 0.01), and increased
104 to expiratory time: beta = -0.003, P = 0.05; respiratory rate: beta = 0.36, P = 0.01; minute ventilat
105 sed using clinical parameters such as pulse, respiratory rate, blood pressure (BP), and biochemical p
106 s, clinical events, and the Confusion, Urea, Respiratory rate, Blood pressure and age >= 65 (CURB-65)
107 modified SOFA (mSOFA), the Confusion, Urea, Respiratory Rate, Blood Pressure and Age (CURB-65) score
108 nuous single-channel monitoring (heart rate, respiratory rate, blood pressure, and peripheral oxygen
109 ninvasive monitoring parameters (heart rate, respiratory rate, blood pressure, and peripheral oxygen
110 rome (SIRS), includes changes in heart rate, respiratory rate, body temperature, and circulating whit
111 glucose stimulation of insulin secretion and respiratory rate but demonstrated two different patterns
112 low-glucose medium and alterations in their respiratory rate, citrate synthase activity, and AMP/ATP
113 rocardiography, beat-to-beat blood pressure, respiratory rate, CO-Modelflow algorithm, and central bl
114 mode of mechanical ventilation and by actual respiratory rate compared with set respiratory rate duri
115 /kg SCH50911 plus l-lactate further improved respiratory rate compared with the same dose of either a
118 ecord oxygen requirement, oxygen saturation, respiratory rate, consciousness level, and other evidenc
121 morphology, as evidenced by measurements of respiratory rates, cytochrome contents, and also clearly
123 Deletion of MORs from KF neurons attenuated respiratory rate depression at all doses of morphine.
125 by actual respiratory rate compared with set respiratory rate during the first 48 hours of mechanical
126 easing age and presentation with an elevated respiratory rate; elevated levels of venous lactate, cre
128 tidal volume (V(T) ) but otherwise increased respiratory rate (f(R) ) and net respiratory output as i
129 02-0.66 +/- 0.03 ml; p < 0.05) and increased respiratory rate (f;91 +/- 3.7-132 +/- 5.7 breaths/min;
130 d conditions, obesity, respiratory symptoms, respiratory rate, fever, absolute lymphocyte count, hypo
131 on as measured by pulse oximetry/Fi(O(2)) to respiratory rate) for determining HFNC outcome (need or
137 tions to control heart rate, blood pressure, respiratory rate, gastrointestinal motility, hormone rel
138 30.7% of patients were febrile, 17.3% had a respiratory rate greater than 24 breaths/min, and 27.8%
139 e less than 20 mEq/L, lactate concentration, respiratory rate greater than or equal to 24 breaths/min
140 essment criteria (Glasgow Coma Scale </= 14, respiratory rate >/= 22 breaths/min, or systolic blood p
141 </= 7.35, and at least one of the following: respiratory rate >/= 25/min, PaO2 </= 50 mm Hg, and oxyg
142 r 2 points for > 6 L/min; 1 point each for a respiratory rate >/= 30 and immune suppression) accurate
143 30 with arterial PCO2 > 20% of baseline, and respiratory rate >/= 30 breaths/min or use of accessory
144 ial hypertension (OR, 1.5; 95% CI, 1.1-2.1), respiratory rate >/=30 breaths per minute (OR, 1.6; 95%
145 Age >/= 80 years, heart rate >90/minute, respiratory rate >20/minute, white cell count <4 x 10(9)
146 not receiving either drug to have diabetes, respiratory rate >22/min, abnormal chest imaging finding
147 eta-blockers during hospitalization included respiratory rate >24 breaths/min (30.8% vs. 16.9%; p = 0
148 her hospital (ORadj 2.08, 95% CI 1.33-3.25), respiratory rate >33 breaths/min (ORadj 2.39, 95% CI 1.5
149 0%-92%; specificity, 47%-54%) and tachypnea (respiratory rate >40 breaths/min; LR, 1.5 [95% CI, 1.3-1
151 ed as >/=2 of 1) heart rate>90 beats/min, 2) respiratory rate>20 breaths/min, 3) body temperature>38
152 ow PiMax or high coefficient of variation of respiratory rate had a nearly three-fold higher risk of
156 dified early warning score was assessed from respiratory rate, heart rate, systolic blood pressure, b
157 eft ventricular ejection fraction, increased respiratory rate, high GRACE score, or presence of diabe
158 ventilation circuit dead space, increases in respiratory rate, higher positive end-expiratory pressur
159 demonstrated that the combination of higher respiratory rate, higher systolic blood pressure, lower
160 expiratory time will depend on the baseline respiratory rate (i.e., less reduction in dynamic hyperi
161 rus (RSV) infection in the neonate can alter respiratory rates, i.e., lead to episodes of apnea.
167 cin-sensitive oxygen consumption and maximal respiratory rates in cells derived from wild type, but n
169 Among the 488 treated neonates, the mean respiratory rates in the first 24 h were 51 (SD 8) breat
170 t of the diaphragm, and Vt decreased and the respiratory rate increased significantly from the beginn
171 ide range of trotting speeds on a treadmill, respiratory rate increases to a fixed and stable value i
172 CH50911 completely prevented the decrease in respiratory rate, indicating agonism at GABA(B) receptor
173 ) nerve fibers, causing an alteration of the respiratory rate indicative of trigeminal activation.
174 f the spatial distribution of heart rate and respiratory rate information were developed from the coe
175 icians to the potential injurious effects of respiratory rate insensitivity to chemical feedback duri
176 vity, the faster enhancement of the cellular respiratory rate is due to intrinsic factors within the
178 t rate, body temperature and blood pressure, respiratory rate is the vital sign that has been often o
180 ial blood pressure, an increase in pulse and respiratory rate, lactic acidosis, and renal failure.
181 During ripening, the fruit experience high respiratory rates leading to ascorbate depletion and a q
182 y responding minimally to chemical feedback, respiratory rate leaves the control of V. e almost exclu
184 age, lower systolic blood pressure, abnormal respiratory rate, lower Glasgow Coma Scale score, lower
186 r=90 mm Hg, Glasgow Coma Scale score <or=12, respiratory rate <10 or >29 per minute, advanced airway
187 nclusion in the AMPT score included GCS <14, respiratory rate <10 or >29, flail chest, hemo/pneumotho
189 ly presents the multiple mechanisms by which respiratory rate may induce injury during mechanical ven
190 hich concerns only assisted ventilation, the respiratory rate may induce injury in a less apparent wa
191 Changes in PaO2/Fio2 ratio, tidal volume, respiratory rate, mean airway pressure, plateau pressure
192 n between PetCO2 equilibria after changes in respiratory rate might not be dependent on moderate lung
193 Secondary outcomes included tidal volume, respiratory rate, minute volume, dynamic lung compliance
195 espiratory acidosis, encephalopathy, and the respiratory rate more quickly than air/O2 but does not p
196 .8 g/dL (odds ratio, 3.6; 95% CI, 1.1-11.9), respiratory rate more than or equal to 32 cycles/min (od
197 dal volume and its coefficient of variation, respiratory rate, neural timing components, and calculat
198 age of >65 yrs, b) altered mental status, c) respiratory rate of >30 breaths/min, d) low oxygen satur
199 rmalisation of patients' respiratory status (respiratory rate of </=20 breaths per min for adults or
200 iggers (heart rate of <40 or >140 beats/min, respiratory rate of <8 or >36 breaths/min, systolic bloo
201 ide clinical score termed quickSOFA (qSOFA): respiratory rate of 22/min or greater, altered mentation
202 vious 24 hours with persisting dyspnea and a respiratory rate of 24/min or greater were eligible prov
203 revealed a temperature of 38.1 degrees C, a respiratory rate of 48 breaths per minute, a heart rate
204 fill volume, 17.5-20 mL.kg(-1) tidal volume, respiratory rate of 5 breaths/min, inspiratory/expirator
205 an inspiratory/expiratory ratio of 1:2.5 and respiratory rate of 6.8 breaths/min appeared to provide
206 peak inspiratory pressure of 50 cm H2O, at a respiratory rate of 8 breaths.min, with an inspiratory t
207 ists for studies that reported heart rate or respiratory rate of healthy children between birth and 1
208 children aged from 1 month to 5 years with a respiratory rate of more than 50 breaths per min in chil
211 the activity, surface body temperature, and respiratory rate of the meadow jumping mouse during hibe
212 .6, 38.6 +/- 4.5, and 23.1 +/- 5.8 mL/sec at respiratory rates of 18, 12, and 6 breaths/min, respecti
215 aths 1-5 [mean+/-SD], 49+/-41% baseline) and respiratory rate often sufficient to sustain minute vent
216 therefore also be used for the titration of respiratory rate on the PetCO2 for a wider range of path
219 44, 95% CI .17-1.07, p = .076), and baseline respiratory rate (OR 2.03 per sd, 95% CI 1.38-3.08, p <
220 t rate (OR, 1.01 [95% CI 1.00-1.01]), higher respiratory rate (OR, 1.05 [95% CI 1.03-1.07]), lower ox
221 % CI: 1.17, 1.40, p < 0.001), or a very high respiratory rate (OR: 1.27, 95% CI: 1.13, 1.43, p < 0.00
223 al volume, positive end-expiratory pressure, respiratory rate, oxygen administration, and head-of-bed
224 condary end points including height, weight, respiratory rate, oxygen saturation, cough, or respirato
226 Secondary outcomes included PRAM score; respiratory rate; oxygen saturation at 60, 120, 180, and
227 001), lower PaCO2 (p < .05), and a lower set respiratory rate (p < .0001) as compared with the SIMV-t
228 sed body mass index (P = .03), and increased respiratory rate (P < .01) were associated with signific
230 improved oxygenation (P < 0.001) and lowered respiratory rate (P < 0.01), DeltaPes (P < 0.01), and pr
231 Low body mass index (P = .002) and elevated respiratory rate (P = .01) at tuberculosis diagnosis ind
232 r operating characteristic curve [AUC] 0.7), respiratory rate (p<.001, AUC 0.71), and oxygen saturati
233 diagnosis, both coefficient of variation of respiratory rate(p < 0.001) and low PiMax (p = 0.002) re
235 oth controlled and assisted ventilation, the respiratory rate per se may promote ventilator-induced l
236 sive vital sign monitoring data (heart rate, respiratory rate, peripheral oximetry) recorded on all a
238 istress Syndrome Network protocol plus lower respiratory rate plus minimally invasive Co2 removal.
240 riables: pH, Glasgow Coma Scale, spontaneous respiratory rate, positive end-expiratory pressure, and
241 spiratory rates, whereas increasingly higher respiratory rates progressively and significantly impair
243 was in direct proportion to the increase in respiratory rate reflecting the level of conditioned fea
248 s and management are based on thresholds for respiratory rate (RR) and oxyhaemoglobin saturation (SpO
249 ublished reference ranges for heart rate and respiratory rate show striking disagreement, with limits
250 gic states, but also that "normal" or higher respiratory rates significantly compromise hemodynamics,
251 has predominantly considered the coupling at respiratory rates slower than the heart rate and shown t
252 erity predictors in the final model included respiratory rate, systolic blood pressure, oxygenation,
253 r higher mass per cell results in an overall respiratory rate that is comparable with wild-type cells
254 ntilation for patients with COPD such as low respiratory rates that maximize expiratory time and care
255 tion coinciding with the increase in overall respiratory rate; this acquired capability was accompani
256 ion (OIRD), a life-threatening depression in respiratory rate thought to be caused by stimulation of
259 iratory chain activity by calcium allows the respiratory rate to change severalfold with only small o
260 to 91 (range, 62-114; P <.001) and the mean respiratory rate to increase an average of 12 breaths pe
262 es , 30 degrees , 40 degrees ) elevation and respiratory rate (to achieve a DeltaETco2 = +/-3-4 mm Hg
263 0% of eupneic VT) with and without increased respiratory rate, using controlled and assist control me
265 arly, sedation interruption led to increased respiratory rate variability for low multiple organ dysf
266 sedation reduces heart rate variability and respiratory rate variability in critically ill patients
267 nuously monitored heart rate variability and respiratory rate variability in critically ill patients.
268 er restoration of heart rate variability and respiratory rate variability in patients with low organ
269 suggest that both heart rate variability and respiratory rate variability increased during sedation i
271 and quantified by an apnoea-hypopnoea index, respiratory rate variability index and the coefficient o
273 se to injury were explored by heart rate and respiratory rate variability measured with non-invasive
274 but in contrast, a further deterioration in respiratory rate variability occurred in the high multip
277 ure was 37 degrees C, heart rate was 78/min, respiratory rate was 17/min, and blood pressure was 158/
281 /- 2.6 vs. 21.3 +/- 2.9 cm H2O, p <.01) when respiratory rate was decreased from 12 to 6 breaths/min
282 /- 2.8 vs. 23.3 +/- 2.6 cm H2O, p <.01) when respiratory rate was decreased from 18 to 12 breaths/min
283 During the lower respiratory rate condition, respiratory rate was reduced from 30.5 +/- 3.8 to 14.2 +
284 the individual vital signs and labs, maximum respiratory rate was the most predictive (AUC 0.67) and
288 onary pressure (DeltaPl), expiratory Vt, and respiratory rate were recorded on admission and 2-4 to 1
292 tochondria from Gq versus wild-type mice and respiratory rates were lower; these changes in mitochond
293 n contrast to WT hearts, complex I-dependent respiratory rates were protected against ischemic damage
297 on and acid-base status with lower-frequency respiratory rates, whereas increasingly higher respirato
298 dictive of stage 2 or 3 AKI included initial respiratory rate, white blood cell count, neutrophil/lym
299 ding pressure, small tidal volumes and rapid respiratory rates with the intent to recruit atelectatic