ライフサイエンスコーパス: response element

1 the core promoter and a region within p53's response element.

2 umber of genes by binding to the antioxidant response element.

3 the transcriptional control of an NF-kappaB response element.

4 ream of the transcription start as a key p53 response element.

5 ing with a GATA4 molecule linked to a nearby response element.

6 promoter contains three functional NF-kappaB response elements.

7 conserved DNA sequence motifs, termed metal response elements.

8 was no decrease in HIF-1 binding to hypoxia response elements.

9 gene transcription via interferon stimulated response elements.

10 d binding with SMAD family member(s) at SMAD-response elements.

11 ize these methyl groups within their cognate response elements.

12 ts, suggesting that they function as EWS/FLI-response elements.

13 nscription factors (TF) with their chromatin response elements.

14 ial proximity to the intron 7 glucocorticoid response elements.

15 seen with the estrogen receptor and its DNA response elements.

16 e highly enriched in RXR transcripts and RXR-response elements.

17 stabilize its binding at its downstream DNA response elements.

18 nteraction at the composite antigen-receptor response element-2 site without affecting the binding of

19 ound to three different DNA sequences: a p21 response element, a puma response element and a nonspeci

20 contain a cis-acting sequence called the A2 response element (A2RE), hnRNP A2 proteins that bind spe

21 es under the control of optimized amino acid response elements (AAREs) had low basal expression and h

22 e 1 expression, and increased glucocorticoid response element activation.

23 Both TRIM28 and TRIM27 can regulate serum response element activity and were required for maintain

24 osterone (T) to E(2), which induced estrogen response element activity, epithelial proliferation, and

25 d recruitment of PPARalpha to this PPARalpha response element after bezafibrate treatment of human he

26 pathway fluxes through five transcriptional response elements against a control constitutive promote

27 NA sequences: a p21 response element, a puma response element and a nonspecific DNA sequence.

28 most prominent element, representing an ABA response element and a potential CAMTA-binding site.

29 people, with 1 SNP allele disrupting a PPAR response element and giving less activation by PPARgamma

30 nology in an AAV system by using an NFkappaB response element and revealed its potential to monitor s

31 ear export dynamics regulated by HIV-1's Rev response element and the viral Rev protein; transient ag

32 This region contains putative cytokinin response elements and is conserved in eight more legume

33 in dissociation of TACC1 from retinoic acid response elements and leads to transcriptional activatio

34 drugs increased AhR recruitment onto the AhR-response elements and significantly induced breast cance

35 The MtLAX2 promoter contains several auxin response elements, and treatment with indole-acetic acid

36 Its promoter includes oxidant-response elements, and we also discovered binding sites

37 vent immunosuppressive transcription of cAMP response element- and hypoxia response element-containin

38 Finally, signal response elements are differentially localized to respon

39 Because estrogen response elements are known to regulate genes over long

40 increased expression of the NRF2-antioxidant response element (ARE) pathway.

41 ty and stimulate higher antioxidative stress response element (ARE) reporter activity.

42 get a sequence found in a subset of androgen response elements (ARE).

43 e expression of genes containing antioxidant-response elements (AREs) in their promoter regions.

44 t from impaired Nrf2 activity on antioxidant response elements (AREs) localized to a microtubule-asso

45 o the induction of genes through antioxidant response elements (AREs).

46 the expression of genes bearing antioxidant-response elements (AREs).

47 tor Ets-2 that binds to the antigen receptor response element (ARRE)-2 of the proximal IL-2 promoter.

48 uitment of ERalpha and SRC-1 to the estrogen response element at the apoA-V promoter, implying the pa

49 table TF-cofactors chromatin interactions at response elements at the single-molecule level.

50 expression after learning requires the cAMP-response element binding (CREB) interaction domain of th

51 reased cyclic adenosine monophosphate (cAMP) response element binding (CREB)/CREB binding protein-med

52 ch decreased two transcription factors, cAMP response element binding and Nurr1, controlling synapsin

53 The transcription factors CREB (cAMP response element binding factor), SRF (serum response fa

54 ng protein of cyclic adenosine monophosphate response element binding protein (CBP), p300, and Cbp/p3

55 2 acts through ACLY to increase carbohydrate response element binding protein (ChREBP) activity, hist

56 Carbohydrate response element binding protein (ChREBP) is a key trans

57 pletion reduces the activity of carbohydrate response element binding protein (ChREBP), a cellular he

58 have shown that the activity of carbohydrate response element binding protein (ChREBP), a key regulat

59 ction via increasing phosphorylation of CAMP response element binding protein (CREB) and PSD95 after

60 ound that activated PERK phosphorylates CAMP response element binding protein (CREB) and PSD95 direct

61 horylation of the transcription factors cAMP-response element binding protein (CREB) and signal trans

62 ates with the transcriptional regulator cAMP response element binding protein (CREB) in both mouse an

63 cAMP response element binding protein (CREB) is a key regulat

64 The cyclic AMP response element binding protein (CREB) is a primary hub

65 tion all impair efficient TSH-dependent cAMP response element binding protein (CREB) phosphorylation.

66 acellular signal-regulated kinase (ERK)-cAMP-response element binding protein (CREB) signaling, as we

67 GMP) leading to increased levels of the cAMP response element binding protein (CREB), a transcription

68 novel intracellular signaling molecule, cAMP-response element binding protein (CREB), which serves as

69 homimetic mutant S386/396E bound to the cAMP response element binding protein (CREB)-binding protein

70 lation of the cyclic adenosine monophosphate response element binding protein (CREB)-regulated transc

71 racellular domain associated-1 protein, cAMP response element binding protein (CREB)-regulated transc

72 tion of the transcription factor Ca(2+)/cAMP response element binding protein (CREB).

73 orhabditis elegans homolog of mammalian cAMP response element binding protein 1 (CREB1)] isoforms on

74 nduced IRS2 expression was dependent on cAMP Response Element Binding Protein activity and that this

75 horylation of cyclic adenosine monophosphate response element binding protein and activating transcri

76 ling, leading to the phosphorylation of cAMP response element binding protein and, consequently, SMC

77 ption levels of specific members of the cAMP Response Element Binding protein gene family.

78 an enduring reduction in phosphorylated cAMP-response element binding protein levels in the NAcSh tha

79 with the magnitudes of hepatic carbohydrate response element binding protein signaling activation.

80 iding the first evidence that enhancing cAMP response element binding protein signaling can alleviate

81 veal a previously unappreciated role of cAMP response element binding protein signaling in RTT pathog

82 hyperactive, leading to a reduction in cAMP response element binding protein signaling, but this mol

83 ed DRG through the transcription factor cAMP response element binding protein-triggered transcription

84 nase-3, ribosomal S6 kinase, c-Jun, and cAMP response element binding protein.

85 ipitation experiments demonstrated that cAMP response elements binding protein regulates the expressi

86 in decreased cAMP signaling and reduced cAMP-response-element binding protein (CREB) activation, a cr

87 kinase-3'-phosphatase (PNKP), and cyclic AMP-response element-binding (CREB) protein (CBP).

88 0 production via the phosphorylation of cAMP response element-binding (CREB) protein on the IL-10 pro

89 ng and colleagues show that the carbohydrate response element-binding protein (ChREBP) coordinates an

90 1 (SP1)-regulated expression of carbohydrate response element-binding protein (ChREBP) in cells conta

91 The carbohydrate-response element-binding protein (ChREBP) is a glucose-r

92 lls in response to glucose in a carbohydrate-response element-binding protein (ChREBP)-dependent mann

93 sensitive transcription factor, carbohydrate response element-binding protein (ChREBP).

94 ilability in many cell types is carbohydrate response element-binding protein (ChREBP).

95 neration) in neonatal mice and reversed cAMP response element-binding protein (CREB) activation and a

96 ge-derived IL-10 resulted in epithelial cAMP response element-binding protein (CREB) activation and s

97 The transcription factors, cAMP-response element-binding protein (CREB) and heat-shock f

98 r (ATF)-adenosine 3',5'-monophosphate (cAMP) response element-binding protein (CREB) family transcrip

99 ng activity of the transcription factor cAMP response element-binding protein (CREB) in young adult r

100 Cyclic AMP-response element-binding protein (CREB) plays key transc

101 t via the PKA-mediated induction of the cAMP response element-binding protein (CREB) signaling pathwa

102 hase (Day 5) characterized by increased cAMP response element-binding protein (CREB) signalling respo

103 overed that HDAC2 is a direct target of cAMP response element-binding protein (CREB) that is activate

104 es converge on the transcription factor cAMP response element-binding protein (CREB) to enhance the e

105 t protein kinase II (CaMKII) and Ca(2+)/cAMP response element-binding protein (CREB) transcription fa

106 We found that loss of the cAMP response element-binding protein (CREB) transcription fa

107 tively, nuclear factor kappa B (NF-kB), cAMP response element-binding protein (CREB), and glucocortic

108 lular cAMP increased phosphorylation of cAMP response element-binding protein (CREB), which, in turn,

109 Here, we show that high glucose induced cAMP response element-binding protein (CREB)-binding protein

110 n kinase (MAPK) signaling, induction of cAMP response element-binding protein (CREB)-insulin receptor

111 tream cascade adenylyl cyclase-cAMP-PKA-cAMP response element-binding protein (CREB).

112 tivity through GPR160-dependent ERK and cAMP response element-binding protein (CREB).

113 The transcription factor cAMP response element-binding protein (CREB1) has been shown

114 ase 4D (PDE4D) gene expression, phospho-cAMP response element-binding protein (p-CREB), and cAMP resp

115 increasing occupancy of phosphorylated cAMP response element-binding protein (pCREB) at two sites in

116 reasing MCU's transcriptional regulator cAMP response element-binding protein 1.

117 Remarkably, ceramide-induced RIP of cAMP response element-binding protein 3-like 1 (CREB3L1) also

118 adenylate cyclase --> cAMP --> PKA --> cAMP response element-binding protein pathway mediating cell

119 Consequently, the cAMP-->PKA-->cAMP response element-binding protein signaling axis is inhib

120 gene, which is regulated by the carbohydrate-response element-binding protein through raised cellular

121 argets, such as ATF-2, c-Jun, and CREB (cAMP response element-binding protein), was disrupted.

122 d with a reduction in the activation of cAMP response element-binding protein, but not the activation

123 vation of the cyclic adenosine monophosphate response element-binding protein, leading to early-onset

124 ase activates the transcription factor, cAMP response element-binding protein, regulating miR-212, wh

125 moter recruited the epigenetic modifier cAMP-response element-binding protein-binding protein/p300 an

126 mouse islet alpha cells by GS/cAMP/PKA/cAMP-response element-binding protein-dependent activation of

127 r outputs: p38-dependent growth arrest, cAMP response element-binding protein-dependent cell survival

128 in lysosomes, thereby activating the sterol response element-binding protein-mediated cholesterol sy

129 on of a pathway involving Akt1/Akt2 and cAMP response element-binding protein.

130 phorylation of the transcription factor cAMP response element-binding protein.

131 panel of proteins including the ERK and cAMP response element-binding protein.

132 by the transcription factors CREB/CREM (cAMP response element-binding protein/modulator) is linked to

133 transcription factor 4 (Atf4) via C/ebp-Atf-Response-Element (CARE) enhancers.

134 othesized that genes with consensus androgen response elements (cAREs) drive proliferation but genes

135 ed concatenated tandem array of consensus TF response elements (catTFRE) approach to profile the acti

136 The DNA response elements, collectively known as kappaB sites or

137 eased Nrf2 binding to a putative antioxidant response element consensus sequence in the sterol regula

138 p53 response elements consist of two decameric half-sites, a

139 p53 tetramers bound response elements containing only one half site to form

140 iption of cAMP response element- and hypoxia response element-containing immunosuppressive gene produ

141 e element-binding protein (p-CREB), and cAMP response element (CRE)-Luc, or PDGF-induced cyclin D1 ex

142 ng function of DHX9, which acts through cAMP response elements (CREs), suggesting that SM may also ac

143 Particularly, analyses of p63 response elements differed substantially among the studi

144 lated-tau levels compared to transactivation response element DNA-binding protein 43 (TDP-43) protein

145 odified with DNA sequences known as estrogen response elements (DNA-ERE), where ERalpha binds specifi

146 Importantly, DNA hypomethylation of Myc response elements does not occur in islets from 1-year-o

147 r translocator), which recognizes the dioxin response element (DRE) in the promoter of downstream gen

148 NA let-7 mimics or inhibitors or an estrogen response element-driven reporter construct (ERE) constru

149 hat recognizes two versions of a 7-base pair response element, either 5- GAG CA-3 or 5- GAG CA-3 (whe

150 EL homozygotes treated with PDE3i was a cAMP response element enhancer 61 nt downstream from the INS,

151 l zebrafish, capable of quantifying Estrogen Response Element (ERE) activation throughout the body.

152 ound that enhancers containing full estrogen response element (ERE) motifs control ER binding at neig

153 ression via directly binding to the estrogen response elements (EREs) located on the promoter of MALA

154 promoter analysis helped identify a putative response element for activating transcription factor 3 (

155 rcadian entrainment pathway and direct light-response elements for clock genes in murine skin, simila

156 n the transgene sequences, including a metal response element from the metallothionein gene, contribu

157 howed highly enriched PGR binding to the PGR response element (GnACAnnnTGTnC), but PGR also interacte

158 s were defined by direct GR binding via a GC response element (GRE) and exclusively increased reporte

159 omoter sequences lack a consensus "whole" GR response element (GRE) but contain putative half-GREs th

160 through interaction with the glucocorticoid-response element (GRE) carrying rs6543115(C) variant.

161 -induced activity requiring a glucocorticoid response element (GRE) within each distal GBS.

162 The GR-ACTN4 complex enhances glucocorticoid response element (GRE)-driven reporter activity.

163 GR binding sites dominated by palindromic GC response elements (GRE), suggesting a non-redundant GRIP

164 n/rat CLDN1 promoters contain glucocorticoid response elements (GREs) and adjacent transcription repr

165 two functional glucocorticoid receptor (GR) response elements (GREs) that have the potential to stim

166 it contains two glucocorticoid receptor (GR) response elements (GREs).

167 proximately 100 glucocorticoid receptor (GR) response elements (GREs).

168 mide designed to bind the consensus androgen response element half-site has antitumor activity agains

169 e factor 1alpha (HIF-1alpha) binds a hypoxia response element (HRE) located within the promoter of Ep

170 GCs activated consensus hypoxia response element (HRE) reporters in a glucocorticoid rec

171 A functional hypoxia response element (HRE) was identified, which is activate

172 iminates it from the closely related hypoxia response element (HRE), and is globally affected by the

173 JQ1 prevented HIF binding to the hypoxia response element in CA9 promoter, but did not alter HIF

174 , also known as lipid rafts, are the primary response element in EF sensing.

175 1 mutation proteins 2 and 4) bind the stress-response element in gene promoters in the yeast Saccharo

176 a time-dependent, colour-based 4th dimension response element in pattern-based information codes.

177 he viral protein Rev, which binds to the Rev response element in stem IIB located on unspliced viral

178 lly, we identified two GC boxes as a key TNF response element in the CPI-17 promoter and demonstrated

179 receptor binds directly to a glucocorticoid response element in the CXCR4 promoter and recruits the

180 reased the binding of Nrf2 to an antioxidant response element in the enhancer region of the EDNRB gen

181 inducible factor 1alpha binding to a hypoxia-response element in the histoneH3-lysine27acetylation ma

182 cial for the salt tolerance of camta6 An ABA response element in the HKT1;1 promoter was found to be

183 ozygous mutant mice with deletion of hypoxia-response element in the vascular endothelial growth fact

184 ct binding sites of known RBPs, such as iron response elements in both known loci and previously unkn

185 ulates both WT and MT p53 by binding to five response elements in p53 promoter.

186 Mechanistic investigations identified NFATc2 response elements in the 3' enhancer region of SOX2, and

187 he nucleus that binds specific retinoic acid response elements in the absence of RA.

188 ligand-activated AHR to the putative dioxin response elements in the EBF1 promoter was demonstrated

189 cells and CARNs and discovered new androgen response elements in the Nkx3.1 3' UTR.

190 dies show that ACTL6A interacts with p53 DNA response elements in the p21(Cip1) gene promoter to supp

191 c leukemia 1 (MKL1), which targets the serum response elements in the promoter of rat Slc1a2 gene enc

192 f GGAA-microsatellites as EWS/FLI activating response elements in vivo and reveal an unexpected role

193 id-derived 2]-like 2) to 2 AREs (antioxidant response elements) in the proximal IL1B promoter.

194 Zta recognizes several methylated Zta-response elements, including meZRE1 (5- GAG C A-3) and m

195 nary conserved inverted repeated negative GC response element (IR nGRE)-mediated direct transrepressi

196 significantly reduces interferon-stimulated response element (ISRE) activity and the expression of i

197 d III IFNs have strong interferon-stimulated response element (ISRE) promoter motifs, whereas the exp

198 appaB (NF-kappaB) and interferon stimulating response element (ISRE) reporter assays, we identified a

199 FoxO1 binds to insulin response elements located in the promoters of insulin-li

200 85-5p expression via binding to the androgen response elements located on the promoter of miR-185-5p,

201 A/C)GATCNTY-3', which we have named the Loz1 response element (LRE).

202 notypic high-throughput screen using a serum response element luciferase promoter, we identified a no

203 onal function was measured using an estrogen response element-luciferase reporter gene assay and quan

204 onism using a cyclic adenosine monophosphate response element-luciferase reporter gene assay.

205 decoy oligonucleotide carrying wild-type p53-response element markedly attenuated OT-induced THTR1, P

206 rowth factor-A165 under control of a hypoxia-response element markedly promotes vascular volume and b

207 nd utilization of regional-specific estrogen response elements may be associated with differential ac

208 as increased cyclic adenosine monophosphate response element-mediated transcriptional activity and p

209 expression of the transcription factor cAMP response element modulator (CREM) alpha promotes altered

210 t the critical role of ventral striatal cAMP-response element modulator (CREM) in mediating impulsivi

211 as both enriched in the distal phenobarbital response element module (PBREM) and the proximal okadaic

212 inatorial interactions between sets of miRNA response elements (MREs), providing strong evidence for

213 in mammalian cells using engineered microRNA response elements (MREs).

214 lines driven by functional ROS (antioxidant response elements), NFkappaB, and mothers against decape

215 TGFbeta1 signaling through ROS (antioxidant response elements), NFkappaB, and SMAD3 in both cell lin

216 tetra acts as a super receptor by binding to response elements not accessible to the wild-type recept

217 suppressor P53, and inhibit the antioxidant response element Nrf-2, resulting in depleted glutathion

218 module (PBREM) and the proximal okadaic acid response element (OARE), a known HNF4alpha binding site.

219 d binding of ERbeta to the putative estrogen-response element of a key cholesterol-synthesis gene (Fd

220 PGR proteins were recruited on progesterone response element of Gpr64 gene in the uteri of wild-type

221 the increased binding to the glucocorticoid response element of histone deacetylase 2 (HDAC2) promot

222 1 (p21(Cip1)) and Nrf2 bound to antioxidant response elements of the Slc7a11 promoter and provide re

223 entified RORalpha response element (RORalpha response element on CYP2B6 promoter, -660/-649) within t

224 RORalpha directly bound to a specific ROR response element on the promoter of Sema3e and negativel

225 6a-5p expression via binding to the Androgen Response Elements on its 5' promoter region, which could

226 sis identified three peroxisome proliferator response elements (PPREs) within the 863-nt region and s

227 ferator-activated receptor gamma (PPARgamma) response elements (PPREs).

228 ent manner, a consequence of a Nanos/Pumilio response element (PRE) in its 3'UTR.

229 ivation and contains a putative progesterone response element (PRE).

230 atical model comprising a Polycomb/Trithorax response element (PRE/TRE) coupled to a promoter and inc

231 In Drosophila, DNA elements termed Polycomb Response Elements (PREs) recruit PcG proteins.

232 e short genomic fragments, known as Polycomb response elements (PREs), that direct Polycomb repressiv

233 ired for targeting PRC complexes to Polycomb Response Elements (PREs).

234 ntaining AU-rich elements (AREs) and Pumilio-response elements (PREs).

235 ts in its decreased binding to the ER stress response element present in the promoter region of Grp78

236 transcription by selectively binding to DNA response elements present within promoters or enhancers

237 nes expressed in response to the antioxidant response element promoter.

238 The frequency of Retinoic Acid Response Element (RARE) sequences was increased in Brazi

239 ctivates RA receptors (RAR), resulting in RA response element (RARE)-dependent gene expression in ren

240 130 bp that contains PRE half-sites and a RA response element (RARE).

241 that EtOH signals via RARgamma binding to RA response elements (RAREs) in differentiation-associated

242 s transcription via RA receptors bound to RA response elements (RAREs) of which there are thousands i

243 d greater enrichment of full-length estrogen response elements relative to ER sites.

244 caused by oxidative stress using antioxidant response element reporter gene assay models and big data

245 teract with Keap1 or activate an antioxidant response element reporter gene due to the absence of a K

246 creased transcription from NFAT or NF-kappaB response element reporters, respectively.

247 Canonical p53 response elements (REs) are made of two decameric half-s

248 on factors (TFs) that bind sequence-specific response elements (REs) in regulatory regions of target

249 CRISPR/Cas9-mediated deletion of the hypoxia-response element revealed co-regulation of NICI and the

250 c:polycytidylic acid or HIV trans-activation response element RNA in lysates of T cell lines or prima

251 loop limiting excessive accumulation of iron-response element RNA-binding activity, whose disruption

252 Transactivation response element RNA-binding protein (TRBP; TARBP2) is k

253 irectly bound to a newly identified RORalpha response element (RORalpha response element on CYP2B6 pr

254 REV-ERBalpha binds to ROR response elements (RORE) in Th17 cells and inhibits the

255 d Nlrp3 genes revealed multiple putative ROR response elements (ROREs) that were occupied by RORgamma

256 Insertion of Rev response element (RRE) allows intron 2 to be retained, a

257 tor of expression of virion (Rev) to the Rev response element (RRE) and subsequent oligomerization in

258 The HIV-1 Rev response element (RRE) is a 351-base element in unsplice

259 The HIV-1 Rev response element (RRE) is a cis-acting RNA element chara

260 Nuclear export complexes composed of rev response element (RRE) ribonucleic acid (RNA) and multip

261 The HIV-1 Rev response element (RRE) RNA element mediates the nuclear

262 structured element within viral RNA, the Rev response element (RRE), and escorts RRE-containing RNAs

263 nome encapsidation or, unexpectedly, the Rev response element (RRE), which regulates the nuclear expo

264 in with a cis-acting regulatory RNA, the Rev response element (RRE), whose sequence changes over time

265 , by binding to and oligomerizing on the Rev Response Element (RRE).

266 three proteins bind to the synaptic activity response element (SARE), an enhancer sequence found upst

267 d widespread chromatin remodeling, including response elements shared between stimulated B and T cell

268 chIFN-kappa regulated the IFN-stimulated response element signalling pathways and activated a pan

269 uption of both the -102 SF1 and the -84 GATA response elements significantly decreases the transactiv

270 condensed chromatin at the synaptic activity response element site and promoter of the Arc gene, faci

271 a, particularly at the Arc synaptic activity response element site, contributes to adult anxiety afte

272 ession and binding to the p28 IFN-stimulated response element site.

273 ease was lost when estrogen-related receptor response element sites were mutated.

274 omoter contains a putative conserved hypoxic response element suggesting that the transcription facto

275 th MYBS1 and MYBS2 could recognize the sugar response element, TA-box, in the promoter and induced pr

276 Transactivation response element (TAR) DNA-binding protein 43 (TDP-43) m

277 ction, which depends on HIV trans-activation response element (TAR) RNA and EGFR of oral epithelial c

278 nal equilibrium of the HIV-1 transactivation response element (TAR) RNA, such that the native GS conf

279 tem-loop constructs of HIV-1 transactivation response element (TAR) RNA, we achieved nucleotide-speci

280 va exosomes contain the HIV trans-activation response element (TAR), Tat, and Nef RNAs but do not exp

281 pable of binding to an interferon-stimulated response element than do cells expressing T387A STAT2.

282 AT1 (Y701) and its binding to IFN-stimulated response elements that control ISG expression.

283 ning in C. elegans, we uncover two metformin response elements: the nuclear pore complex (NPC) and ac

284 The bound MR interacts with response elements to induce or repress the transcription

285 human ITPR3 promoter revealed five putative response elements to NF-kappaB, and promoter activity wa

286 CODE mRNA/miRNA data with predicted microRNA response elements to prioritize tissue-specific ceRNA in

287 trameric transcription factor that binds DNA response elements to regulate transcription of target ge

288 rs (TRs) alpha and beta act by binding to TH response elements (TREs) in regulatory regions of target

289 ta have consistently shown GR to occupy AP-1 response elements (TREs), even in the absence of AP-1.

290 moter assays identified a functional hypoxia response element upstream of the TSS.

291 d expression was dependent on glucocorticoid response elements upstream of annexins and was reinforce

292 ded DNA and function in trans as a vitamin D response element (VDRE)-binding protein.

293 We identified and characterized vitamin D response elements (VDREs) located in both genes and show

294 ER stress response elements were identified in the catalase gene a

295 tivate the FOXM1 promoter through a Forkhead response element, whereas FOXM1 can activate AURKA expre

296 F composite element or interferon-stimulated response element, whereas R291Q retained BATF/JUN intera

297 receptor (AR) binding to different androgen-response-elements, which switch the EZH2 function from h

298 ity depends on its binding to the xenobiotic response element (XRE) in partnership with the AhR nucle

299 vation is mediated primarily through two XBP-response elements (XRE) on the ORF21 promoter region.

300 lysis revealed the presence of Zn-deficiency-response elements (ZDREs) in a number of the ZIPs.