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1 nge in phase difference between postural and rest tremor.
2 c component, whereas 57 patients (40.7%) had rest tremor.
3 reduced neuromuscular strength, dystonia and resting tremor.
4 including gait abnormalities, rigidity, and resting tremor.
5 0.574 for spiral drawing, 0.441 to 0.488 for rest tremor, 0.266-0.577 for postural tremor and 0.306 f
6 ng 188 with essential tremor, 90 (47.9%) had rest tremor; 75.6% with rest tremor did not have Lewy pa
8 symptoms of Parkinson's disease (PD) include resting tremor, akinesia, bradykinesia, and rigidity, an
9 kinson's disease cohort were associated with rest tremor amplitude (beta = -0.106, P < 0.05), rest tr
10 can achieve almost 50% average reduction in resting tremor amplitude and in so doing form the basis
11 to (1) the presence and severity of isolated rest tremor and (2) the diagnosis of essential tremor-Pa
12 fewer limb segments than Parkinson's disease rest tremor and essential tremor, and its frequency was
16 Two mutant mice were identified with marked resting tremor and further characterized using the SHIRP
17 t deficits in WT alphaS mice and the PD-like resting tremor and progressive motor decline of 3K alpha
18 gait deficits in WT aS mice and the PD-like resting tremor and progressive motor decline of 3K aS mi
20 to those of previous studies of parkinsonian rest tremor, and highlight an important difference in th
21 ve parkinsonism with bradykinesia, rigidity, resting tremor, and impaired postural reflexes was seen
22 , these animals have severe limb weakness, a resting tremor, and notable neuroanatomical malformation
26 , decreased overall activity, acquisition of resting tremors, and increased susceptibility to pentile
27 Egr3-/- mice develop gait ataxia, scoliosis, resting tremors, and ptosis, suggesting a defect in prop
28 lowing inclusion criteria: the presence of a rest tremor, bradykinesia, and rigidity; a modified Hoeh
29 disease include motor impairments involving resting tremor, bradykinesia, postural instability, gait
32 tremor amplitude (beta = -0.106, P < 0.05), rest tremor constancy (beta = -0.109, P < 0.05), and ind
33 porter availability was also associated with rest tremor constancy (beta = -0.380, P < 0.05) and inde
35 emor, 90 (47.9%) had rest tremor; 75.6% with rest tremor did not have Lewy pathology and 80% were not
39 ing of cortical oscillations responsible for rest tremor in the periphery by delivering tremor-freque
44 these results we conclude that parkinsonian rest tremor is driven by a neural network, which include
46 characterized by bradykinesia, rigidity, and resting tremor, is the most common neurodegenerative mov
48 or frequency that dictates that parkinsonian rest tremor may be significantly entrained by low freque
49 tive disorder characterized by bradykinesia, resting tremor, muscular rigidity, and postural instabil
51 Physiological evidence indicates that the resting tremor of Parkinson's disease originates in osci
53 falls than patients with Lewy body disease; resting tremor, pill-rolling tremor and hallucinations w
54 was assessed in a test cohort comprising 16 rest tremor recordings in tremor-dominant Parkinson's di
55 Specifically, the amplitude of parkinsonian rest tremor remained unchanged despite spontaneous chang
56 nical features of bradykinesia, rigidity and resting tremor resulting from the deficiency of dopamine
59 ve disease with movement disorders including resting tremor, rigidity, bradykinesia and postural inst
60 ) is the most common movement disorder, with resting tremor, rigidity, bradykinesia and postural inst
62 eurodegenerative disorders, characterized by resting tremor, rigidity, bradykinesia, and postural ins
63 minating in severe motor symptoms, including resting tremor, rigidity, bradykinesia, and postural ins
64 ied Parkinson's Disease Rating Scale (UPDRS) rest tremor score of 3 or greater for any limb, a Mini-M
68 ns predicted that without a voluntary drive (rest tremor) the neural drives would be more likely in p