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1 pattern of marrow replacement and changes in reticuloendothelial activity after enzyme replacement th
2                                    Depressed reticuloendothelial cell function may have contributed t
3  no mosaic color was seen in regions lacking reticuloendothelial cells (e.g., tumors).
4 ity of focal hepatic lesions with negligible reticuloendothelial cells (eg, metastases).
5 ng impairment in the movement of iron out of reticuloendothelial cells and hepatocytes.
6 these mice shows abundant iron stores within reticuloendothelial cells and hepatocytes.
7  germinal centers of lymphoid tissue, and in reticuloendothelial cells in the spleen.
8 ysosomal storage is present primarily within reticuloendothelial cells such as Kupffer cells and cell
9  iron absorption and cause iron retention in reticuloendothelial cells, resulting in iron-restricted
10 ein, which resides on the plasma membrane of reticuloendothelial cells.
11 heir ability to be recognized and cleared by reticuloendothelial cells.
12  primarily due to rapid clearance by hepatic reticuloendothelial cells.
13  the apoB48R may provide essential lipids to reticuloendothelial cells.
14 mately 3.8-kb mRNA is expressed primarily by reticuloendothelial cells: monocytes, macrophages, and e
15  extensive proliferation of inflammatory and reticuloendothelial cells; however, anti-HMGB1 treatment
16  of RBC labeling with the NPs also prevented reticuloendothelial detections and the activations of in
17 ravenously as a biomarker for normal hepatic reticuloendothelial function and SPECT was repeated.
18 ited increased Fpn1-mediated iron uptake and reticuloendothelial iron overload as young adult mice.
19 oportin 1 (FPN1), lead to autosomal dominant reticuloendothelial iron overload in humans.
20 ring chronic inflammatory states, leading to reticuloendothelial iron sequestration and an associated
21 osomal storage is marked within hepatocytes, reticuloendothelial Kupffer cells, and cells of the sinu
22                     An important function of reticuloendothelial macrophages is phagocytosis of senes
23         FPN1 is also abundantly expressed in reticuloendothelial macrophages of the liver, spleen, an
24     The expression of ferroportin1 (FPN1) in reticuloendothelial macrophages supports the hypothesis
25 ption in enterocytes, iron recycling through reticuloendothelial macrophages, and iron release from s
26    Together, our results suggest that murine reticuloendothelial macrophages, but not those in the bo
27 ropoiesis and profound iron sequestration in reticuloendothelial macrophages, duodenum, and other tis
28 ble via the intermediary of spleen and liver reticuloendothelial macrophages.
29  through NTBI transporters, independently of reticuloendothelial macrophages.
30 creased bacterial burden was detected within reticuloendothelial organs of iNOS(-/)- mice only beyond
31 ysis revealed that the ASB activities in the reticuloendothelial organs of this animal, as well as tw
32 poor enzymatic stability, rapid clearance by reticuloendothelial organs, immunostimulation, and coagu
33 t concentrations of MAC were observed in the reticuloendothelial organs, with a maximum of 6.9 log10
34 rse, B. neotomae replicated and persisted in reticuloendothelial organs.
35 ion and then spreads to the spleen and other reticuloendothelial organs.
36 (marrow iron absent) on the basis of visible reticuloendothelial (RE) marrow iron stores.
37                In several LSDs, cells of the reticuloendothelial (RE) system are the primary targets
38 USB was taken up primarily by tissues of the reticuloendothelial (RE) system.
39            In addition, stereotyped abnormal reticuloendothelial responses, including excessive react
40 g opsonization, macrophage phagocytosis, and reticuloendothelial sequestration.
41 ion from the intestine and iron release from reticuloendothelial stores.
42 lation of 64Cu-labeled SCKs in organs of the reticuloendothelial system (RES) (56.0 +/- 7.1 %ID/g and
43  polyethylene glycol (PEG) used to evade the reticuloendothelial system (RES) and anisamide (AA) for
44 orter, is also expressed in the cells of the reticuloendothelial system (RES) and is likely to be inv
45  size would decrease the distribution in the reticuloendothelial system (RES) and selectively increas
46                                  Presence of reticuloendothelial system (RES) cell iron in the liver
47 patocellular (HC) pattern [63/849 (7.4%)], a reticuloendothelial system (RES) cell pattern [91/849 (1
48 iscrete components of posttransplant hepatic reticuloendothelial system (RES) function-phagocytosis a
49 60)[C(COOH)(2)](10) to possess the first non-reticuloendothelial system (RES) localizing behavior for
50 ly delayed clearance of nanomaterials by the reticuloendothelial system (RES) of mice, a highly desir
51 rial debris may accumulate in tissues of the reticuloendothelial system (RES) serving as an inflammat
52 the Fc-receptors of, cells of the phagocytic reticuloendothelial system (RES) using medronate liposom
53 on up to 1 day, relatively low uptake in the reticuloendothelial system (RES), and near-complete clea
54 umor penetration, and avoid clearance by the reticuloendothelial system (RES).
55     CoF itself had no effect on mesangial or reticuloendothelial system [125I]AHIgG uptake.
56 lity to home to tissues rich in cells of the reticuloendothelial system after intravenous injection i
57 erebroside substrate in cells throughout the reticuloendothelial system and clinical manifestations i
58 preference to platelet-free RBC) through the reticuloendothelial system and erythrophagocytes in the
59  size contributes to rapid scavenging by the reticuloendothelial system and poor penetration of key e
60 nships between various cell types within the reticuloendothelial system and suggesting possible targe
61  gene expression in hepatocytes, the splenic reticuloendothelial system and the bronchiolar epitheliu
62 ivatized phospholipids are able to evade the reticuloendothelial system and thereby remain in circula
63              Drug molecules carried into the reticuloendothelial system are released from SWNTs and e
64 n transfused platelets, inducing a transient reticuloendothelial system blockade by infusions of spec
65     Iron oxide loading of macrophages in the reticuloendothelial system by means of intravenous ferum
66 ide initial cellular entry points within the reticuloendothelial system by which Listeria establishes
67 ls were highest among patients with mixed HC/reticuloendothelial system cell (RES) iron deposition.
68 helial cell and a cytoplasmic compartment of reticuloendothelial system cells.
69                                        Rapid reticuloendothelial system clearance of QD will require
70 tient benefits due to issues associated with reticuloendothelial system clearance, tumor heterogeneit
71                                          The reticuloendothelial system has a central role in erythro
72 f the resident macrophage populations of the reticuloendothelial system is a key component of the com
73 d that f-SWNT are not retained in any of the reticuloendothelial system organs (liver or spleen) and
74 d biochemical improvements were found in the reticuloendothelial system organs (livers, spleens, and
75 dye molecules without severe accumulation in reticuloendothelial system organs, making them very prom
76  cancer therapy is their rapid uptake by the reticuloendothelial system that decreases the systemic e
77 hat may allow interactions with cells of the reticuloendothelial system to be minimized, yet permit s
78 , HFE enables the intestinal crypt cells and reticuloendothelial system to interpret the body's iron
79 d extrinsic factors (eg, the capacity of the reticuloendothelial system to remove defective RBCs).
80  a new retargeting paradigm for NPs to avoid reticuloendothelial system uptake and achieve rapid prec
81 oped a new strategy to temporarily blunt the reticuloendothelial system uptake of nanodrugs, a major
82 ated RGD-PASP-IO nanoparticles and prominent reticuloendothelial system uptake.
83 limits bacterial growth in the organs of the reticuloendothelial system very quickly after infection,
84 nt upon action potentials transmitted to the reticuloendothelial system via the vagus and splenic ner
85 cholinergic antiinflammatory pathway and the reticuloendothelial system was unknown.
86 umans is safe, and cells accumulating in the reticuloendothelial system were detectable on clinical m
87 f body weight) to preload macrophages of the reticuloendothelial system with iron oxide nanoparticles
88  syndromes of childhood are disorders of the reticuloendothelial system with variable clinical manife
89  lost from the circulation, sequester in the reticuloendothelial system, and do not return to circula
90 re found, principally involving the lung and reticuloendothelial system, and these were not clearly t
91 on of glucocerebroside in macrophages of the reticuloendothelial system, as a consequence of a defici
92 stinal colonization and dissemination to the reticuloendothelial system, as well as lower levels of i
93 the disease is primarily at the level of the reticuloendothelial system, but few virulence factors ha
94 crophages allowing multiplication within the reticuloendothelial system, but this does not preclude t
95 nd undergoes substantial phagocytosis by the reticuloendothelial system, causing a short blood circul
96 f histologic improvement seen throughout the reticuloendothelial system, even in animals that were en
97 y extracellular nucleases, scavenging by the reticuloendothelial system, filtration by the kidney, tr
98 , short plasma half-life, early clearance by reticuloendothelial system, immunogenicity, inadequate i
99 injury through the specialized organs of the reticuloendothelial system, including the lungs, liver,
100 d slowly and are largely retained within the reticuloendothelial system, making clinical translation
101 -containing CPPs were rapidly cleared by the reticuloendothelial system, namely Kupffer cells of the
102                Consistent with uptake by the reticuloendothelial system, R2* value increased in the l
103 ating iron acquisition from the duodenum and reticuloendothelial system, respective sites of iron abs
104 s iron to be sequestered within cells of the reticuloendothelial system, suppressing erythropoiesis a
105 g circulation times, low accumulation in the reticuloendothelial system, sustained tumour retention,
106 eostasis including the developing and mature reticuloendothelial system, the duodenum, and the pregna
107 ense systems, particularly the organs of the reticuloendothelial system, to remove phage particles fr
108 vors the liver resulting in clearance by the reticuloendothelial system, with less than 1 % of the in
109                                              Reticuloendothelial system-specific agents improve lesio
110 nspecific extracellular gadolinium chelates, reticuloendothelial system-specific iron oxide particula
111 esistance to toxic oxidative products of the reticuloendothelial system.
112 f their rapid uptake from circulation by the reticuloendothelial system.
113 bacteria directly within the confines of the reticuloendothelial system.
114 nd provides a signal for cell removal by the reticuloendothelial system.
115 reatment of storage diseases that affect the reticuloendothelial system.
116 d to a systemic inflammatory response in the reticuloendothelial system.
117  and spleen, due to greater avoidance of the reticuloendothelial system.
118 NA-induced protein expression outside of the reticuloendothelial system.
119  tissue level, and systemic clearance by the reticuloendothelial system.
120  resulting in extravascular hemolysis by the reticuloendothelial system.
121  release of iron from the macrophages of the reticuloendothelial system.
122  is persistent bacterial colonization of the reticuloendothelial system.
123 cal and nonclassical pathways as well as the reticuloendothelial system.
124 that persists within phagocytic cells of the reticuloendothelial system.
125 n of the injected NP is still cleared by the reticuloendothelial system.
126 ablishing persistent infection in the murine reticuloendothelial system.
127 s targeting platelets for destruction by the reticuloendothelial system.
128 uding dendritic cells (DCs) and cells of the reticuloendothelial system.
129 e biological particulate filter known as the reticuloendothelial system.
130 ism, such as the phagocytic functions of the reticuloendothelial system.
131 estration of iron in phagocytic cells of the reticuloendothelial system.
132  from the recognition and elimination by the reticuloendothelial system.
133  autocrine/paracrine mediator in the hepatic reticuloendothelial system.
134 infection and to clear the inoculum from the reticuloendothelial system.
135 ductase of replicating amastigotes in hosts' reticuloendothelial tissues.
136 cation of bacteria inside macrophages within reticuloendothelial tissues.
137 events nonselective accumulation of qdots in reticuloendothelial tissues.

 
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