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1  tissue level, and systemic clearance by the reticuloendothelial system.
2  resulting in extravascular hemolysis by the reticuloendothelial system.
3  release of iron from the macrophages of the reticuloendothelial system.
4 n of the injected NP is still cleared by the reticuloendothelial system.
5  is persistent bacterial colonization of the reticuloendothelial system.
6 cal and nonclassical pathways as well as the reticuloendothelial system.
7 that persists within phagocytic cells of the reticuloendothelial system.
8 ablishing persistent infection in the murine reticuloendothelial system.
9  from the recognition and elimination by the reticuloendothelial system.
10 s targeting platelets for destruction by the reticuloendothelial system.
11 uding dendritic cells (DCs) and cells of the reticuloendothelial system.
12 e biological particulate filter known as the reticuloendothelial system.
13 ism, such as the phagocytic functions of the reticuloendothelial system.
14 estration of iron in phagocytic cells of the reticuloendothelial system.
15  autocrine/paracrine mediator in the hepatic reticuloendothelial system.
16 infection and to clear the inoculum from the reticuloendothelial system.
17 esistance to toxic oxidative products of the reticuloendothelial system.
18 f their rapid uptake from circulation by the reticuloendothelial system.
19 bacteria directly within the confines of the reticuloendothelial system.
20 nd provides a signal for cell removal by the reticuloendothelial system.
21 reatment of storage diseases that affect the reticuloendothelial system.
22 d to a systemic inflammatory response in the reticuloendothelial system.
23  and spleen, due to greater avoidance of the reticuloendothelial system.
24 NA-induced protein expression outside of the reticuloendothelial system.
25     CoF itself had no effect on mesangial or reticuloendothelial system [125I]AHIgG uptake.
26 lity to home to tissues rich in cells of the reticuloendothelial system after intravenous injection i
27 erebroside substrate in cells throughout the reticuloendothelial system and clinical manifestations i
28 preference to platelet-free RBC) through the reticuloendothelial system and erythrophagocytes in the
29  size contributes to rapid scavenging by the reticuloendothelial system and poor penetration of key e
30 nships between various cell types within the reticuloendothelial system and suggesting possible targe
31  gene expression in hepatocytes, the splenic reticuloendothelial system and the bronchiolar epitheliu
32 ivatized phospholipids are able to evade the reticuloendothelial system and thereby remain in circula
33  lost from the circulation, sequester in the reticuloendothelial system, and do not return to circula
34 re found, principally involving the lung and reticuloendothelial system, and these were not clearly t
35              Drug molecules carried into the reticuloendothelial system are released from SWNTs and e
36 on of glucocerebroside in macrophages of the reticuloendothelial system, as a consequence of a defici
37 stinal colonization and dissemination to the reticuloendothelial system, as well as lower levels of i
38 n transfused platelets, inducing a transient reticuloendothelial system blockade by infusions of spec
39 the disease is primarily at the level of the reticuloendothelial system, but few virulence factors ha
40 crophages allowing multiplication within the reticuloendothelial system, but this does not preclude t
41     Iron oxide loading of macrophages in the reticuloendothelial system by means of intravenous ferum
42 ide initial cellular entry points within the reticuloendothelial system by which Listeria establishes
43 nd undergoes substantial phagocytosis by the reticuloendothelial system, causing a short blood circul
44 ls were highest among patients with mixed HC/reticuloendothelial system cell (RES) iron deposition.
45 helial cell and a cytoplasmic compartment of reticuloendothelial system cells.
46                                        Rapid reticuloendothelial system clearance of QD will require
47 tient benefits due to issues associated with reticuloendothelial system clearance, tumor heterogeneit
48 f histologic improvement seen throughout the reticuloendothelial system, even in animals that were en
49 y extracellular nucleases, scavenging by the reticuloendothelial system, filtration by the kidney, tr
50                                          The reticuloendothelial system has a central role in erythro
51 , short plasma half-life, early clearance by reticuloendothelial system, immunogenicity, inadequate i
52 injury through the specialized organs of the reticuloendothelial system, including the lungs, liver,
53 f the resident macrophage populations of the reticuloendothelial system is a key component of the com
54 d slowly and are largely retained within the reticuloendothelial system, making clinical translation
55 -containing CPPs were rapidly cleared by the reticuloendothelial system, namely Kupffer cells of the
56 d that f-SWNT are not retained in any of the reticuloendothelial system organs (liver or spleen) and
57 d biochemical improvements were found in the reticuloendothelial system organs (livers, spleens, and
58 dye molecules without severe accumulation in reticuloendothelial system organs, making them very prom
59                Consistent with uptake by the reticuloendothelial system, R2* value increased in the l
60 lation of 64Cu-labeled SCKs in organs of the reticuloendothelial system (RES) (56.0 +/- 7.1 %ID/g and
61  polyethylene glycol (PEG) used to evade the reticuloendothelial system (RES) and anisamide (AA) for
62 orter, is also expressed in the cells of the reticuloendothelial system (RES) and is likely to be inv
63  size would decrease the distribution in the reticuloendothelial system (RES) and selectively increas
64                                  Presence of reticuloendothelial system (RES) cell iron in the liver
65 patocellular (HC) pattern [63/849 (7.4%)], a reticuloendothelial system (RES) cell pattern [91/849 (1
66 iscrete components of posttransplant hepatic reticuloendothelial system (RES) function-phagocytosis a
67 60)[C(COOH)(2)](10) to possess the first non-reticuloendothelial system (RES) localizing behavior for
68 ly delayed clearance of nanomaterials by the reticuloendothelial system (RES) of mice, a highly desir
69 rial debris may accumulate in tissues of the reticuloendothelial system (RES) serving as an inflammat
70 the Fc-receptors of, cells of the phagocytic reticuloendothelial system (RES) using medronate liposom
71 on up to 1 day, relatively low uptake in the reticuloendothelial system (RES), and near-complete clea
72 umor penetration, and avoid clearance by the reticuloendothelial system (RES).
73 ating iron acquisition from the duodenum and reticuloendothelial system, respective sites of iron abs
74                                              Reticuloendothelial system-specific agents improve lesio
75 nspecific extracellular gadolinium chelates, reticuloendothelial system-specific iron oxide particula
76 s iron to be sequestered within cells of the reticuloendothelial system, suppressing erythropoiesis a
77 g circulation times, low accumulation in the reticuloendothelial system, sustained tumour retention,
78  cancer therapy is their rapid uptake by the reticuloendothelial system that decreases the systemic e
79 eostasis including the developing and mature reticuloendothelial system, the duodenum, and the pregna
80 hat may allow interactions with cells of the reticuloendothelial system to be minimized, yet permit s
81 , HFE enables the intestinal crypt cells and reticuloendothelial system to interpret the body's iron
82 d extrinsic factors (eg, the capacity of the reticuloendothelial system to remove defective RBCs).
83 ense systems, particularly the organs of the reticuloendothelial system, to remove phage particles fr
84  a new retargeting paradigm for NPs to avoid reticuloendothelial system uptake and achieve rapid prec
85 oped a new strategy to temporarily blunt the reticuloendothelial system uptake of nanodrugs, a major
86 ated RGD-PASP-IO nanoparticles and prominent reticuloendothelial system uptake.
87 limits bacterial growth in the organs of the reticuloendothelial system very quickly after infection,
88 nt upon action potentials transmitted to the reticuloendothelial system via the vagus and splenic ner
89 cholinergic antiinflammatory pathway and the reticuloendothelial system was unknown.
90 umans is safe, and cells accumulating in the reticuloendothelial system were detectable on clinical m
91 f body weight) to preload macrophages of the reticuloendothelial system with iron oxide nanoparticles
92  syndromes of childhood are disorders of the reticuloendothelial system with variable clinical manife
93 vors the liver resulting in clearance by the reticuloendothelial system, with less than 1 % of the in