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2 nosuppressive type D retroviruses, the avian reticuloendotheliosis group including spleen necrosis vi
3 nally defined in terms of its histology as a reticuloendotheliosis, is now subdivided on the basis of
5 DNA binding activity of the NF-kappaB avian reticuloendotheliosis viral (v-rel) oncogene related B (
6 kappaB (NF-kappaB) family member v-rel avian reticuloendotheliosis viral oncogene homolog A (RELA) re
7 -kappaB transactivating subunit, v-rel avian reticuloendotheliosis viral oncogene homolog A (RELA), t
9 ving a pro-proliferative role of v-rel avian reticuloendotheliosis viral oncogene homolog B (RELB) ac
10 ) private loss-of-function variants of V-Rel Reticuloendotheliosis Viral Oncogene Homolog B (RELB).
11 a stabilization decreased the level of v-rel reticuloendotheliosis viral oncogene homolog B protein,
12 lear factor kappaB (NFkappaB) subunit c-REL (reticuloendotheliosis viral oncogene homolog) in a pilot
13 ted States were screened for the presence of reticuloendotheliosis virus (REV) sequences in their gen
14 at a virus currently present in poultry, the reticuloendotheliosis virus (REV), is actually of ancien
15 d the 5' UTR of spleen necrosis virus (SNV), reticuloendotheliosis virus A (REV-A) and human T-cell l
18 ant clone RM1 due to a solo insertion of the reticuloendotheliosis virus long terminal repeat, which
21 We identified another oncogenic retrovirus, reticuloendotheliosis virus strain T (REV-T), that upreg