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1 almeterol significantly reduced death of the retinal Muller cells.
2  levels induce GAPDH nuclear accumulation in retinal Muller cells.
3 r translocation and subsequent cell death in retinal Muller cells.
4 principal mediator of glutamine transport in retinal Muller cells.
5          Ang II and Ang-(1-7) colocalized in retinal Muller cells.
6  the retinal pigment epithelium (RPE) and in retinal Muller cells.
7 nding activity, and regulation of sigmaR1 in retinal Muller cells.
8 increases of VEGF and TNF-alpha secretion in retinal Muller cells.
9  and the aquaporin 4 (AQP4) water channel in retinal Muller cells.
10 e (rMC-1) that exhibits the phenotype of rat retinal Muller cells.
11 nergic agonists selectively activate ERKs in retinal Muller cells.
12  expression in response to exogenous bFGF in retinal Muller cells.
13 ured rat retinal ganglion cells and cultured retinal Muller cells.
14            Pathogenesis involves toxicity to retinal Muller cells.
15 ose-induced nuclear accumulation of GAPDH in retinal Muller cells, an event considered crucial for th
16            FRalpha and PCFT are expressed in retinal Muller cells and colocalize in the endosomal com
17 nd lipid peroxidation were determined in rat retinal Muller cells and compared with the effects of NG
18 d -beta immunoreactivity was observed in rat retinal Muller cells and in the RGC layer and blood vess
19 (4) receptor in angiogenic cell behaviors of retinal Muller cells and retinal microvascular endotheli
20  GAPDH in hyperglycemia-induced apoptosis in retinal Muller cells and the ability of R-(-)-deprenyl t
21       Taken together, these results identify retinal Muller cells as an intraretinal source of TNF al
22 green fluorescent protein expression only in retinal Muller cells at P17.
23 nregulation of insulin receptor signaling in retinal Muller cells cultured under hyperglycemic condit
24                                     In human retinal Muller cell cultures, hyperglycemic conditions i
25 d receptor alpha (PPARalpha) is expressed in retinal Muller cells, endothelial cells, and in retinal
26 wever, in contrast to GFAP-lacZ transgenics, retinal Muller cells expressed the GFP transgene in resp
27                                 Furthermore, retinal Muller cells in culture are resistant to anoxia
28 th human retinal endothelial cells (REC) and retinal Muller cells in either 5 mM (normal) or 25 mM (h
29 ment, prevents diabetes-induced apoptosis of retinal Muller cells in the rat eye, but the mechanism i
30 ied the anatomical remodeling and gliosis of retinal Muller cells in the rd/rd mouse model of photore
31                       RDH10 was expressed in retinal Muller cells, in addition to the RPE.
32                      Here, we report that in retinal Muller cells, increased glycolytic flux causes i
33                               Cultured human retinal Muller cells (MIO-M1) were treated with highly o
34                            Bergmann glia and retinal Muller cells, nonforebrain astrocytes that have
35                    Chromophore supply by the retinal Muller cells (retina visual cycle) supports the
36                      Also, incubation of rat retinal Muller cells (rMC-1) in normal glucose (5 mm) or
37                                              Retinal Muller cells span the retina and secrete several
38  Studies were performed to determine whether retinal Muller cells transcribe genes for the proinflamm
39             A recent study demonstrated that retinal Muller cells undergo hyperglycemia-induced apopt
40                                              Retinal Muller cells were cultured in normal (5 mM) or h